Environmental Emergencies Flashcards
(89 cards)
Definition and classification of Hypothermia
Def: core temp = 95F
Mild: 89.6-95F (32-35C)
Mod: 86-89.6F (30-32C)
Severe: <86F(<30C)
Conduction
Transfer of heat by direct contact (water immersion)
Convection
Transfer of heat by movement of heated material (wind disrupting heat around body)
Radiation
electromagnetic transmission
evaporation
conversion of liquid to vapor - usually accounts for 10-15% heat loss (sweat, resp processes)
Etiology of hypothermia
medical illness (DM, PVD, ASVD, Neuropathy, Psych)
Ethanol (MCC)
wind chill
clothing
smoking
homeless
High risk pt for hypothermia and MC
age extremes, altered sensorsium
MC: males 30-49 y.o; extremities
Pathophys of hypothermia
Initially have increased HR, vasoconstrict + incr O2 consumption but decrHR after 32C
Hypoventilation w/ CO2 retention (=hypoxia, rest acid)
Decreased mucocillaiary clearance (incr secretions, dear gag/cough = incr aspiration)
Slowed mentation, motor, speed of reasoning
dear plt fan 2/2 sequester in portal system (= incr blood viscos + thrombi)
Decr. colga factor activity
cold diuresis
imp. insulin release
shift Oxyhemo curve to Left (harder to unload)
Hypothermia: what happens after trx started
cold, acidotic peripheral blood returns to central circ = temp decr. further and incr. risk for arrhythmia
Hypothermia: 3 main priority organs
brain, heart, kidney
hypothermia: clinical abnormalities
shivering stops @ 90 (32.2C)
incr. arrhythmias @ <86(30): Osborn J wave
w/hold card meds and defib until temp >82.4F (28C)
Hypothermia: common EKG signs
Osborn J-wave; no bunnies in v1/v2, but +in v3
Hypothermia: trx
warms O2 vent + warmed IV fluids
Active external rewarm (blankets) 1 deg C/hr
Gently circulating water (104-107.6F, 40-42C)
Active core rewarm (incr 2 deg/hr) = 2 cutes tubes each side w/ warm fluid in top, out bottom; warm NGT/urinary catheter + IVF
Frostbite Pathophys
- Cold Exposure
- Formation of extra cell ice crystals damaging cell membranes and osmotic gradient
- Intracell dehydration
- intracell ice crystal formation
- Cell death
Frost bite classifications
first deg: anesthetic central white plaque w/ peripheral erythema
2nd deg: clear or milky-filled blisters surrounded by erythema and edema
3rd deg: hemorrhagic blisters that progress to hard black eschar
4th degree: complete necrosis and tissue loss
Frostbite Treatment
- elevate and splint extremity
- wrap in dry gauze
- debride white/clear blisters
- aloe vera q 6 hr
- tetanus
- analgesics (ASA, NSAIDS, narc)
- Abx no role
- no smoke
UV Keratitis
-snowstorm/flare on slit lamp
damage to anterior chamber of eye
-develop w/in 1 hr of exposure; no symp until 6-12hrs
-severe pain, foreign body sensation, tearing, conjunctival injections
-bad far vision
UV keratitis Trx
- self-limited
- analgesics, cold compress
- cyclogel helps spasm
- polarized sunglasses
- patching not recommended
Heat Injuryies high risk pt
age extremes, confusional states, limited water access, ETOHics, Menta illness, chronic dz
Heat injury: when does radiation occur
when air temp < body temp
What is hyperthermia
a rise in body temp when heat production exceeds heat loss –fever is rise of core body temp in response to circulating cytokines
Heat Injuries Pathophys
-incr endogen heat prod
-decr. heat dispersion
-thirst is poor gauge of hydration status
excerise incr. metabolic rate 20-25x
What meds incr. heat production?
neuroleptics, hallucinogens, amphetamines, anesthetics, LSD, cocaine
What meds inhibit sweating?
Antihistamines, Neuroleptics, TCAs, Atropine, Antispasmodics