equine derm 2 Flashcards

(99 cards)

1
Q

what infectious dz is this

signalment, dx, tx

A

Papilloma
may be congenital
mainly in younger horses (1 to 4-years old)
equine papilloma virus (‘everywhere’)
multiple wart like lesions, mainly head,
incidentally elsewhere
diagnosis on clinical presentation
treatment usually not warranted
prognosis is good
cryosurgery, surgery, creams

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2
Q

common areas for sarcoids

A

ears,groin, axila, face, and eyelid

it is a fibro epithelial tumor

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3
Q

clinical appearance of occult sarcoid

A

it manifest as a ring of alopecia with slight scaling or skin thinning

they are commonest in the medial thigh region

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4
Q

dicsuss lesions of verrucose saicoid

A

the lesions are cauliflower(warty) in appearance.

they can be focal or diffuse

single or multiple

the lesions usually produce large amounds of keratin and so have a flaky grey or scabby appearance

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5
Q

discuss the types of nodular sarcoids

A

type A and B

in the type A there is no epithelial component and the skin and the tumor can be moved independently of each other.

in type B there is significant epithelial component which results in the binding of the tumor mass under the skin so that independent movement is not possible.

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6
Q

this type of sarcoid has a fleshy appearance very like granulation tissue but

A

fibroblasticsarcoid

affected wounds may be extrememly difficult to tx

type 1 fibroblast has has a narrow pedunculated stalk attachment to the skin and therefore there may be obvious involvement of the subcutis in the region of the attachment while type 2 the tumor has a wide base usually wider than the underlying exuberant mass

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7
Q

what kind of sarcoid tumor is this one

A

mixed sarcoid tumor

this tumor is simply a mixture of the other types in varying proportions

in reality many of the common lesions fall under this category

therapy is difficult/

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8
Q

the most common skin tumors in horses

A

sarcoids

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9
Q

dx for sarcords

A

bx

differentiate types of sarcoids

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10
Q

tx for sarcoids

A

cryosx and bcg immunotherapy are possible

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11
Q

px for sarcoids

A

guarded

they dnt metastasis but therapy is nt always successful

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12
Q

what causes this dz

how is it transmitted and cured

A

ehv3

contagious veneral disease
caused by EHV-3
transmission by coitus (via insects,
fomites and inhalation)
incubation about 7 days
systemic corticosteroids may
reactivate the diseases
depigmentation may persist after
lesions have healed

rest the animal and dont breed till the dz has healed

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13
Q

which dz is this, clinical appearance ,pathogenesisas well as characteristics

A

Dermatophilus congolensis
bacteria invade epidermis and root shafts
in rainy weather in horses at pasture
dorsum and lateral parts of the body
painful but no pruritus
crusts under which there is a pink-red open lesion
underside of crust is moist and yellow-greenish
pus may be present
in laboratory samples mention ‘dermatophilosis’

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14
Q

tx for drmatophylosis

A

improvement of the management
removing crusts (painful!)
clip as far as possible
repeated bathing and drying
systemic antibiotics (costs!)
disease is not highly contagious but it is
sensible to isolate affected horses

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15
Q

which dz is this,etiology, common areas, characterisitics, tx

A

Folliculitis and furunculosis
several bacteria
most often on tack-areas
papels – pustules
often quite painful, rarely pruritic
pustules may open and exude pus
treatment is hygiene !
sometimes systemic antibiotics indicated

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16
Q

which bacteri cuses this

common areas

A

abscess

Corynebacterium pseudotuberculosis

mostly pectoral muscle
ventral abdomen
groin

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17
Q

cs of corynebcterium pseudotubeculosis infection

A

lame or reluctant to walk
depression
weight loss
small to very large abscesses
complications like purpura
haemorrhagica or ulcerative
lymphangitis
bacteria is difficult to culture

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18
Q

discuss tx for corynebacterium pseudotuberosis

A

external abscesses
- mature abscesses (hot pack)
- abscess drainage and flushing
- NSAID’s (bute)
internal abscesses
- long course proc-penicillin / TMPS
limb infection (ulcerative lymphangitis

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19
Q

px for corynebacterium psydotubercolosis

A

external abscesses good
internal abscesses poor
ulcerative lymphangitis guarded

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20
Q

what causes cellulitis

A

several bacteria, often Staphylococci
often primary ‘entrance’ not detected
acute onset of severe swelling and pain of
one limb, often a hind limb
often extremely lame
often tachycardia and febrile
complications: cutaneous necrosis and
sloughing, laminitis, bacteremia

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21
Q

tx for cellulitis

A

immediate systemic antibiotics
hydrotherapy
support bandages other limbs
NSAID’s
if within 24 hours no improvement
corticosteroids

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22
Q

name the dz

characterists

A

Tail pyoderma
folliculitis and furunculosis of the tail
may cause severe pruritus and
automutilation
therapy
- clipping infected area and bathing
- systemic treatment with antibiotics
prognosis is guarded

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23
Q

name this condition

etiology

lesions,tx

A

Botryomycosis = bacterial pseudomycetoma
mostly coagulase positive staphylococci
mostly solitary nonpruritic nodular growths
incidentally multiple pustules, nodules and
draining tracts over large areas of the body
affected horses are otherwise healthy
surgical excision for solitary lesions - good
TMPS systemically for multiple lesions - poor

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24
Q

which lesion is dz

A

dermatophytosis

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25
types of dermatophysis
superficial skin dz trichophyton spp and microspora spp
26
pathogenesis for dermatophytosis
they primarily invade the hair shaftsto weaken it and cause breakage some produce substances that can act as an irritatant or allergens and therefore produce pruritus
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describe the lesions of dermatophytosis
it often starts with small papules with erected hairs. then the classical ring shaped lesions with alopecia and some scalling may develop or may be obvious as some lesions may coalesce or appear as paint brush type areas.
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dx for drmatophytosis
it is done thru clinical appearance but confirmation can be done through microscopic examination of hairs and crusts for hyphae and conidia. while cx is the only definitive way.
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transmission of fungal infections
they are contracted through direct contact and theough indirect contact by saddle girth, brushes etc
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tx for dermatophytosis
it is self limiting tx is recommended to stop transmission or reduction in performance use antifungal topicals
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characteristics of fungal infections, incubation period, signalemnt, early signs
incubation period 6 days – 6 weeks young and debilitated horses predisposed early signs may be urticaria variable degree of associated crust and scale which is often ‘silvery’ hair can be lost in annular patches = ‘ring worm’ lesions of dermatophyte infection overlap with bacterial infection dermatophytosis is a zoonosis,
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tx for dermatophytosis
reduce contagion to the environment maximise horse’s ability to respond (correct predisposing factors) hasten resolution of the infection topical therapy systemic therapy? No! vaccination
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vaccination protocol for dermatophytosisi
Insol® Dermatophyton where to vaccinate? deep intra-muscular muscle is preferably ‘relaxed’ thigh is preferred pectoral muscle is also suitable preferably not in the neck alternate sides between vaccinations
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When to vaccinate with Insol®Dermatophyton
horses with clinical signs of dermatophytosis possible infected horses horses due to travel (second injection has to be administered at least 14 days prior to departure) all horses on premises with historically problems hygiene is essential !!! n.b horse has dermatophytosis, but does not show clinical signs (yet), these symptoms will show soon after vaccination
35
When not to vaccinate with Insol® D ?
reduced immunity - foals younger than 5 months - sick or stressed horses pregnant mares ?? close before important events! hygiene is essential !!!
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where do u normally find malazezia
mostly in intertrigenous areas (axilla, groin, udder, prepuce, incidentally coronitis)
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what are the lesions of malaszia
very rare in horses greasy, waxy, often malodorous dermatitis
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example of a deep fungal infection
sporothrichosis it is a chronic progressive sporadic skin and sq/ lymphatic infection caused by sporothrix schenckii infection is usually introduced thru small skin wounds
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cs of sporottricosis
firm,well dermacated, painless sq nodulesassociated with cored hardened lymphaticscommnly on limb regions (fetlock most common) it is potentially zoonotic
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tx for sporotrichosis
sodium iodide sx removal of nodules is also possible
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what infection is this
3-year old Quarter horse mare - phaeohyphomycosis
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common transmission of mange
direct or indirect through grooming materials
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this mange live in skin surface and feed on skin debris
choriotic mange it is caused by chorioptic equi no interspecies transmission and the mite doesnt affect pple survival off host 70 days
44
season for choriotic mange and area they are mostly found in the body
is often identified in winter in horses with feathered fedlocks. transmission is thru direct contact. the dz may be transmitted from season to season by horses that show no cs
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cs of chorioptic mange
cs are often limited to distal limbs and consists of scalling, fine papular eruptions but in heavy infectations the whole body can be affecte leading to general signs of pruritus, irritability and even wt loss horses may stump their feet because of irritation they may rub posts and bite at the limbs
46
contributing factors for mange and season
mostly late winter – early spring contributing factors - crowding - prolonged stabling - suboptimal nutrition - environmental temperature limited host specificity
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characteristics of sarcoptic mange
0.25-0.6 mm in diameter tunnel through epidermis feed on tissue fluids + epidermal cells life cycle on host ± 2-3 weeks off-host survival time only a few days ‘endangered species’ N.B it is a digging mite while choriotic is a non burrowing mite
48
location of sarcoptic mange, and clinical appearance
starts on the head very severe pruritus anaemia may occur secondary infections may occur mites very difficult to find often ‘diagnostic therapy’
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tx for sarcopts mange
treatment with appropriate spray or dip at least two times - 10-14 days between few or even no products licensed in the Netherlands we use foxim – Sebacil® ivermectine should be useful reportable disease in many countries
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characteristics of this mange
Psoroptes equi 0.4-0.8 mm long nonburrowing feed on tissue fluids life cycle on host ± 10 days 14-18 days off-host survival time but incidentally up to 84 days?
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clinical appearance of psoroptes mange
pruritic dermatitis of trunc and/or ear disease with head shaking mane and tail seborrhoea mites rarely found
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treatment for psoroptes mange
ivermectine is very effective in the treatment of psoroptes otoacariasis and/or dermatitis eggs are not killed – second dose after 14 days necessary efficacy of ivermectine may be explanation for ‘extinction’ often unnecessary treatment in the ears??
53
characterists of this ectoparasite
Thrombiculidiasis thrombiculid larvae ± 0.2-0.4 mm long thrombiculid adults and nymphes are freeliving larvae normally feed on tissue fluids of small rodents, but incidentally horses generally in late summer and fall entire life-cycle 50-70 days (7-10 days feeding as larvae on host)
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lesions of Thrombiculidiasis dx and tx
infestation primarily in pastured horses papules and wheals muzzle, face, distal limbs to make the diagnosis: chigger larva seen in the centre of the lesion variable pruritus often self-limiting / topical treatment
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characteristics of this ectoparasite
Demodex ± 0.2 mm long (D.caballi + D. equi ) normal residents of the skin in the hair follicles and sebaceous glands feed on normal debris life cycle takes ± 20-35 days on the host survival off host few hours to days probably not a contagious disease
56
when does demodex become clinical
only clinical after long-term glucocorticoid treatment most times asymptomatic alopecia and scaling, over the face, neck, shoulders and forelimbs papules and pustules may be seen deep skin scrapings necessary treatment not attempted
57
this parasite is contacted by horse which live close to chickens
Dermanyssus gallinae ± 0.6-1.0 mm long incidentally on horses pruritic papules and crusts in contact areas horse can be sprayed or dipped eradication of the mite from the premises (and the poultry)
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which tick is this plus cs, season
Dermacentor reticulatus same as ixodes spring n summer incidentally in spring and summer may attack any part of the body surface paples, pustules, wheals later crusts, erosions, ulcers, alopecia pain and pruritus are variable anaemia
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which ectoparasite is this discuss its cs, season and lesions
Werneckiella equi 3-6 mm in lenght (nits 1-2 mm) ‘moving dust’ more in winter months feeds on cutaneous debris prefers dorsal trunk pruritus mild to moderate varying degrees of scaling and alopecia
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which parasite caused this lesions
Werneckiella equi
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name this parasite, its favourable areas in the body
Haematopinus asini 3-6 mm in lenght (nits 1-2 mm) very rare in our area feeds on blood and tissue fluid favor main, tail and fetlocks may cause anaemia
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this parasite is active in summer and adult females glue eggs to hairs on legs and shoulders
Gastrophiliasis – bots Gasterophilus intestinalis = G. equi G. nasalis, G. haemorrhoidalis, G. pecorum, G. inermis active in summer adult females glue eggs to hairs on legs and shoulders cutaneous gastrophiliasis – 1-2 mm wide greyish-white crooked streaks
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Gastrophiliasis – bots
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which parasite is this
Hypoderma bovis and H. lineatum close contact to cattle egg – larvae – skin penetration – subcutaneous tissue H. bovis – spinal canal and epidural fat H. lineatum – submucosal connective tissue of the oesophagus second stage larvae to dorsum swellings of the back with breathing pore
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Hypoderma – therapy
confirm diagnosis !! enlarging breathing pore and extracting the larva surgical excision of the whole nodule wait and allow larvae to drop out by themselves prevention: deworming with ivermectin or moxidectin
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common places for this parasite
Hippobosca equina – louse fly adult louse suck blood tender to cluster in the perineal and inguinal regions only in import horses horses may be ‘irritated’ therapy: insecticide
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this fly adult blowflies lay clusters of light-yellow eggs in wounds
**Lucilia sericata – myiasis** adult blowflies lay clusters of light-yellow eggs in wounds larvae hatch within 8-72 hours larvae reach full size in 2-19 days treatment - cleansing and debriding - topical insecticide - symptomatic therapy
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where do u find oxyubiris
Oxyuris equi , Probstmayria vivipara lives in coecum and colon adult females lay eggs around anus variable pruritus incidentally restless and irritable acetate tape – diagnosis therapy: routine worming also knon as pin worms
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clinical dz for habronemiasis discuss season
summer sores swamp cancer granular dermatitis begins in spring and summer regresses partially or completely in winter
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discuss the lyf cycle of hebronemiasis
H. muscae, H. majus + Draschia megastoma adults inhabit the stomach eggs and larvae passed with faeces Musca domestica + Stomoxys calcitrans infectious larvae deposited on the horse in moist areas or open wounds larvae near mounth are swallowed – complete parasitic life-cycle
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treatment for habronemiasis
perhaps partly a hypersensitivity disorder - sporadic (one animal in a herd) - seasonal - recurs in same horse every summer - systemic corticosteroids may be curative no optimal therapeutic protocol (surgery, local and systemic therapy)
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which parasite did this
Parafilaria multipapillosa parafilariasis haemorrhagic filariasis summer bleeding only import horses in the Netherlands adult worms (3-7 cm) live in the connective tissues coiled within nodules nodules open at the surface and discharge a blood exsudate (eggs, larvae) therapy: time + avermectines?
73
which parasite is this where does it live in the body its intermidiate host
Onchoceriasis O. cervicalis, O. reticulata, O. gutturosa adult worms live in lichamentum nuchae or in connective tissues of tendons microfilaria numerous in ventral midline Culicoides spp. are intermediate host in the Netherlands not diagnosed for over 25 years
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name the parasite and its characteristics
Halicephalobiasis Halicephalobus gingivalis = Halicephalobus deletrix = Micronema deletrix = Rhabditis gingivalis details of life cycle not known multiple organ systems can be infected cutaneous lesions uncommon diagnosis confirmed by biopsy treatment ??
75
proper way of cooling the horse
30 s. whole body cooling with cold water 30 s. walking in a circle temperature should fall 1oC / 10 min. allow horse to drink
76
when should you stop cooling the horse
if body temperature \< 38.0-39.0 oC if skin over the quarters is cool after walking if respiratory rate \< 30 breaths/min if horse shivers
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what condition is this
Arabian fading syndromeo
78
what condition is this
melanoma
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what lesion is this
sarcoid
80
what dz is this
melanoma
81
what condition is this
aloppecia areata
82
which paraste causes this
choriotic mange
83
which dz is this
Leucosis Eosinophilic granuloma
84
what condition is this
saicoid wound that grows
85
what condition is this
habronemiasis wound that grows
86
what parasite is this one
sucking lice
87
what parasites causes this
Chorioptes equi Chorioptes equi , often lives with many!
88
how is this dz dx
diagnosis by ‘diagnostic treatment’.
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what is going on here
Thick bite – Ixodes ricinus
90
which tick spp is this
Hyalomma marginatum
91
which tick spp i this
Dermacentor reticulatus http://upload.wikimedia.org/wikipedia/commons/7/77/Dermacentor\_reticulatus\_M\_070825.jpg
92
which condition is this
Dermatophilus congolensis – specific crusts Painful and pink lesions
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