Exam 1 Flashcards

1
Q

what does your body need to clot effectively?

A

functioning liver, Ca, phospholipids, platelets

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2
Q

what labs measure clotting?

A

PT/INR, PTT<,aPTT, anti-Xa

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3
Q

what would you find on assessment if your patient has prolonged clotting?

A

bleeding, easy bruising, hematuria

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4
Q

what would you expect to find if pt has decreased clotting time?

A

-stroke, thrombosis, limb ischemia, renal injury, heart attack, DVT< PE

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5
Q

what are three different types of coagulation drugs?

A

antiplatelets (on arterial), anticoagulation (venous) , and thrombolitics (both )

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6
Q

when would your pt be on an antiplatelet vs an anticoagulant

A

venous clot vs arterial clot

anticoagulation– DVT, a-FIB (prevent a stroke that comes from the heart)

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7
Q

how does Coumadin/warfarin work?

A

reduces your risk of clots by reducing the time it takes to make a clot

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8
Q

what is sinus tacky caused by? how to treat?

A

external influences on the heart, NOT cardiac conditions –> treat the underlying cause

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9
Q

what are signs of a basilar skill fracture? why is this so dangerous?

A

racoon eyes and battles sign (behind the ears) - dangerous because you can get a CSF bleed -> infection (meningitis) & HA & herniation

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10
Q

SCI care: C5

A

breathing - yes
neuro - can move their face, can talk, but they are quadriplegic
cardiac - low cardiac output (we want to create constriction with ted hose and binding)
GI- incontinent, increased sphincter tone (we need stool softeners)
GU - spastic bladder
integ - risk for pressure injuries

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11
Q

SCI care: T3

A

a lot of the same as C5 applies, but there is more that they can do
-they might have some movement capabilities in their upper extremities

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12
Q

SCI care: T9

A

the big difference between T3 and T9 is that T9 has full function of their upper extremities

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13
Q

SCI care: L1

A

often independent in their wheelchairs
GI:
decreased tone –> we want to keep stools firm
GU - incontinent
the urine is flowing out but they are unable to completely empty the bladder so their is still some urinary retention

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14
Q

which patient would you educate about autonomic dysreflexia?
- incomplete spinal
-pt with T8 injury
-pt with complete T4
-all pts with spinal cord injury

A

complete at T6 or above
so the complete T4 is the right answer

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15
Q

what are the hallmarks of autonomic dysreflexia? how do we treat?

A

spike of hypertension
bradycardia
thermo disregulation
vasodilation above (flushing and hot)
vasoconstriction below (cold pale)
**
we treat by removing the stimulus

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16
Q

what are the differences between spinal and neurogenic shock

A

-spinal shock happens first, is a temporary loss of reflexes and sensations
-paralysis below the level of injury
-risk of secondary injury to the chord
—-
neurogenic shock is distributive shock, so it includes hypotension and bradycardia, loss of motor tone
warm or cold extremities

17
Q

what are somethings we can do to limit secondary injuries?

A

-stabilize the spine
-keep pt calm
-catheter and ng tube
-monitor for resolution

18
Q

TBI nursing management

A

managing ICP- positioning, medications, surgery , drain
prevent secondary injury
calm environment

19
Q

sinus bradycardia treatment

A

atropine and possible pacing

20
Q

first degree AV block treatment

A

monitor and continue to observe

21
Q

Central cord syndrome manifestations

A

-more weakness in the upper extremities
- “man in a barrel”

22
Q

anterior cord syndrome manifestations

A

-loss of pain and temperature sensation
-keep proprioception and vibration

23
Q

brown-sequard syndrome manifestations & rap

A

-crossed symptoms
loss of ipsilateral motor sensation
loss of contraletal pain and temp
“Im a hip ViP, I walk with a limp”
“Contemplate the pain I will create”

24
Q

as ICP goes up

A

CPP goes down

25
Q

CO2 is a potent

A

vasodilator