Exam 1 Flashcards
(140 cards)
risk factors for sepsis
immunocompromised, CLABSI, open wounds, malnutrition, invasive procedures, cancer, older than 80, alcoholism, diabetes mellitus, transplants, hepatitis, HIV/AIDS, GI ischemia, Hepatitis, transplant recipient, CKD, infection with resistant oraganisms, mucous membrane fissures in prolonged contact with bloody/ drainage soaked packing
stages of sepsis
SIRS, Sepsis, Severe Sepsis, Septic Shock, MODS
s/s of infection and sepsis
SIRS and confirmed infection
SIRS=2/4 criteria
body temp >100.5 or <96.8
HR>90
RR>20/PaCO2<32
WBC >12,000 or <4,000 or >10% immature bands
Infection:
fever
tachycardia
pain
swelling
warmth
redness
inc WBC
labs to assess for sepsis
sepsis labs: increased WBC, Decreased PLTs and Clotting factors, increased lactate, increased procalcitonin, BUN/creatinine, ABG, H&H, blood cultures, ALT, AST
labs to assess for infection progress
CRP(C-reactive protein), ESR (erythrocyte sedimentation rate) to track inflammation(is there a downtrend?), blood cultures, and labs listed for sepsis
nursing interventions for sepsis
hour-1 bundle
observe for organ dysfunction/worsening of s/s (decreased output, low BP, cyanosis, jaundice, etc)
qSOFA
SOFA
prioritization of nursing interventions for sepsis
hour-1 bundle
1. measure Lactate level
2. obtain blood cultures before adminstering antibiotics
3. administer broad-spectrum antibiotics
4. begin rapid adminstration of 30mL/kg crystalloid for hypotension or lactate > 4mmol/L
5. apply vasopressors if hypotensive during or after fluid resuscitation to maintain a mean arterial pressure >65 mm Hg
tx of sepsis
treat infection/fungal (broad-spectrum first, specific after blood cultures result), treat hypotension with vasopressors, start fluids 30mL/kg
prioritization of tx for sepsis
obtain blood cultures and lactate level, begin broad-spectrum abx, start 30mL/kg crystalloid fluid, adjust abx as blood culture sample gets back, monitor for worsening of s/s, monitor VS, and for rxns to abx and fluids.
adrenal function related to sepsis
decreased kidney function= toxins not released->furthering inflammation and decreasing perfusion r/t edema–> can indicate/lead to MODS
blood glucose levels related to sepsis
high glucose levels released from liver due to stress response; the more severe the response, the higher the glucose level
expected outcomes to sepsis tx
improvement in BP, return of WBC counts, increased O2 sat, decreased lactate level, increased perfusion, lack of edema, decreased procalcitonin
what has the highest risk of death over sepsis alone?
septic shock
DIC
disseminated intravascular coagulation
Disseminated intravascular coagulation; patho
microthrombi formed where not needed widespread; this consumes AVAILABLE PLTs and clotting factors and leads to hypoxia–> metabolic acidosis and organ dysfunction(inc hypoxia= inc metabolites-toxic-which amplifies inflammation and repeats poor gas exchange and perfusion)
discharge teaching for a pt at risk for sepsis
s/s of sepsis:
s/s of infection:
identify pt at risk for CV disease
male, over 40/45,smoker, alcohol, family hx, obese, sedentary lifestyle, african american, high cholesterol diet, uncontrolled HTN, uncontrolled stress
Modifiable risk factors
lifestyle, diet, exercise, smoking, drinking alcohol
non-modifiable risk factors
age, gender, ethinicity, family hx
NSR
normal sinus rhythm
describe normal sinus rhythm
impulse intiated by SA node
reg rhythm
rate 60-100bpm
normal P wave in lead
P wave before each QRS
normal PR, QRS, and QT intervals
sinus bradycardia
impulse initiated in SA node
sinus rhythm w/ rate <60
normal p wave
p wave before each QRS
causes for sinus bradycardia
vagal, drugs, ischemia, disease of nodes, ICP, hypoxemia, athletes(normal)
s/s of sinus bradycardia
can be asymptomatic
OR
confusion, SOB, CP, diaphoresis, syncope, lightheadedness, hypotension