EXAM 2 Flashcards
(38 cards)
FVD
-loss of extracellular fluid exceeds intake of water
-dehydration
Hypovolemia (FVD) causes
-vomiting, diarrhea, nasogastric suctioning
-excessive skin loss w/o sodium and water replacement
-diuresis (polyuria), kidney disease, adrenal insufficiency
-third spacing burns
-anorexia, nausea, impaired swallowing, confusion, NPO
dehydration (FVD) causes
-hyperventilation
-prolonged fever
-diabetic ketoacidosis
-diabetes insipidus
-osmotic diuresis
-excessive intake of salt, salt tablets, or hypertonic fluids
-hemorrhage or plasma loss
FVD clinical manifestations
-Rapid weight loss
-Tented skin turgor
-oliguria/concentrated urine (late sign)
-postural hypotension (orthostatic hypotension)
-rapid, weak pulse
-hyperthermia
-lack of energy
-thirst
-nausea
-muscle weakness and cramps
-decreased CVP (late sign)
-oliguria
-tachypnea
-hypoxia
-seizures (rapid/severe dehydration)
FVD lab values
-normal: 10:1, FVD: 20:1, >25mg/dl due to hemoconcentration
-increased hematocrit
-sodium >145 meq/L with dehydration
-USG >1.030
-blood osmolarity >295 w/ dehydration/hypernatremia
FVD nursing management
-administer oral/iv fluids
-monitor I/O
-monitor weight every 8 hrs (1 Kg = 1 L fluid loss or gained)
-check skin turgor
-assess for gait stability
-encourage pt to stand up slowly (orthostatic hypotension)
-provide oral care
Hypovolemic shock
-occurs when a significant amount of fluid is lost (cells are no longer able to carry oxygen)
-administer oxygen and monitor oxygen
-check v/s q 15 minutes
-provide IV fluids (crystalloids: LR or 0.9% NS) (colloids: PRBCs or plasma)
-administer vasoconstrictors: norepinephrine, phenylephrine, and dopamine
-perform hemodynamic monitoring
FVE
-Fluid overload: excess of fluid causing hemodilution
-decreases hematocrit
-excess of water and electrolytes
-Risk for CHF and pulmonary EDEMA
Hypervolemia causes
-heart failure
-kidney disease
-cirrhosis
-an overdose of fluids
-fluid shifts following major burns
-corticosteroids
-severe stress
-hyperaldosteronism
Overhydration causes
-water replacement w/o electrolytes, excessive water intake
-SIADH (too much adh)
-excessive administration of IV D5W or hypotonic fluids
FVE clinical manifestations
-tachycardia
-HTN
-Tachypnea
-Edema
-DISTENDED NECK VEINS
-crackles, cough, dyspnea
-bounding pulse
-increased weight and urine output
-increased CVP
-seizures (if severe hyponatremia)
FVE nursing management
-Monitor I/O and weight
-assess lung sounds/edema
-promote adherence to fluid restrictions and sodium
-encourage REST (favors diuresis)
-discuss certain meds (some have Sodium)
-fowler’s or semi-fowler position
-turn and reposition pt.
Pulmonary Edema causes
-FVE
-clinical manifestations: anxiety, PVCs, dyspnea at rest, change in LOC, restlessness, lethargy, ascending crackles, pink tinged sputum
-put into high-fowlers pos.
-administer oxygen, positive airway pressure
-administer nitrates, morphine, diuretics if possible (BP has to be adequate)
hyponatremia
-< 136 mEq/L
-net gain of water or loss of sodium rich foods
-water moves from ECF to ICF
-caused by:
-adrenal insufficiency
-water intoxication
-SIADH
-vomiting, diarrhea, sweating,
diuretics
hyponatremia clinical manifestations
-poor skin turgor (hypovolemic)
-dry mucosa (hypovolemic)
-decreased salivation (hypovolemic)
-decreased BP and increased HR (hypovolemic)
-headache
-nausea
-abdominal cramping
-neurologic changes
hyponatremia nursing management
-encourage intake of foods and fluids high in sodium (beef broth, tomato juice)
-monitor dietary sodium and fluid intake (no more than 12 mEq in 24 hours to prevent neuro dmg demyelination)
-daily weight
-check VS and neuro status
Severe hyponatremia
-can cause comas, resp arrest, seizures
-implement seizure precautions
-give hypertonic oral and IV fluids
Hypernatremia
->145 mEq/L
-increased sodium levels = increased hypertonicity of blood
-water shifts out of cells, causing dehydration
-caused by:
- water loss > sodium loss
- excess sodium
administration
-diabetes insipidus
-heatstroke
-hypertonic IV solutions
-kidney failure
-aldosteronism
-cushing’s
syndrome/glucocorticoids
Hypernatremia clinical manifestations
-Thirst
-elevated temperature
-tachycardia
-dry, swollen tongue
-sticky mucosa
-Restlessness,
weakness, muscle
twitching to muscle
weakness, decreased
DTRs, seizures, coma
Hypernatremia nursing management
monitor LOC and ensure safety
Monitor VS and heart rhythm
Provide oral hygiene and other
comfort measures for thirst
Monitor I&Os
Assess for over-the-counter
sources of sodium
Offer and encourage fluids to
meet needs
Provide sufficient water with
tube feedings
Hypokalemia
- < 3.5 mEq/L
GI losses- vomiting, gastric suctioning, diarrhea
Medications- K-losing diuretics, corticosteroids
Alterations of acid–base balance- due to shifts of H and K ions between cells and ECF
Hyperaldosteronism- increases renal K wasting
Poor dietary intake
Alkalosis
Water intoxication
Hypokalemia clinical manifestations
Dysrhythmias
Cardiac arrest
PROMINENT U WAVE
Fatigue
Anorexia
N/V, hypoactive bowel sounds, constipation, abdominal distention, paralytic ileus
Muscle weakness and cramps
Paresthesias
Decreased muscle strength
Decreased deep tendon reflexes
Respiratory failure
Hypokalemia nursing management
Administer prescribed potassium replacement (max 10 mEq/hour; NEVER IV PUSH or IM subq anyone)
Monitoring of electrocardiogram
Monitor bowel sounds
Monitor clients receiving digoxin for toxicity
Arterial blood gases
Avocados, broccoli, dairy products, dried fruit, cantaloupe, bananas, juices, melon, salt substitutes
NO IV BOLUS (risk for arrest)
Hyperkalemia
- > 5.0 mEq/L
Increased risk of cardiac arrest
Impaired renal function
Hypoaldosteronism- deficient adrenal hormones lead to Na loss and K retention
Tissue trauma- burns, crushing injuries, severe infections
Acidosis- K moved from cell to ECF
Uncontrolled Diabetes