Exam 2 Flashcards

(36 cards)

1
Q

Dysphagia

A

Difficulty in swallowing (subjective)

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2
Q

Causes of dysphagia

A

Motility dysfunction
Obstructive lesions
Hang up

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3
Q

Heart burn

A

Esophageal reflux (retrosternal pain, bitter tast)

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4
Q

What motion can instegate heart burn?

A

Intraabdominap pressure increase: bending over, laying down, heavy lifting and obesity

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5
Q

Odynophagia

A

Painful swallowing

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6
Q

What is associat d with odynophagia?

A

Acute esophageal inflammation and ulceration

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7
Q

Sliding hernia

A

90% of hernia cases

Gastroesophageal junction shifted above hiatus

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8
Q

Manifastation of sliding hernia symptoms occur when?

A
50% asymtamatic
Heart burn (regurgitation) and with increased intraabdominal pressure
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9
Q

Paraesophageal hernia

A

Rolling hernia

Section of fundus reflected up through hiatus next to esophagus

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10
Q

What are the complications of paraesophageal herniations?

A

Postprandial bloating
Paraesophageal bleeding
Ulceration

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11
Q

Esophagitis

A

Contributes to esophageal mucosal injury

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12
Q

The potential risks of esophagitis are?

A

Erosion
Ulcers/bleeding
Metaplasia
Inflammatory scarring

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13
Q

Barretts espohagus (columnar cell metaplasia), mucosal inflamation, erosion and/or ulcers are associated with chronic or acute esophagitis?

A

Chronic esophagitis

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14
Q

Whar are the complications of acute and chronic esophagitis?

A

Ulceration
Dysphagia
Increaed risk of cancer

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15
Q

Morphology of esophageal carcinoma

A

Mostly squamous cell ca (except Barretts esophagus presenting with adenocarcinoma)

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16
Q

How will carcinoma of the esophagus present?

A
Mechanical obstruction(solids more so)
Hematemsis
Poor prognosis (10% make it 5y)
17
Q

What are the effects of food and drink on Achalasia and carcinoma of the esophagus?

A

Achalasia- food and drink

Carcinoma- solids more

18
Q

What do Helicobacter pylori do?

A

Secretes enzymes that degrade mucin and produces ammonia.

The associated alkalinity stimulates HCL secretion

19
Q

Is acute gastritis reversable?

20
Q

25% of people taking aspirin daily might experiance what over time?

A

Acute gastritis

21
Q

The range of superficial g., atropic g. And gastric atrophy reffer to what kind of gastritis?

A

Chronic gastitis

22
Q

Autoantibodies against parietal cells and IF (intrensic factor) are what type of gastritis?

A

Chronic gastritis

Fundic- type A

23
Q

Fundic-type A is associated with what?

A

Loss of parietal cells and penicious anemia
Decrease iron absorp.
No ulcers
Increased risk g.ca (10%)

24
Q

What is a decrease in red blood cells when the body can’t absorb enough vitamin B-12 called?

A

Pernicious anemia

25
What is Antral gastritis-type B?
80% most common Mucosal atrophy Dyspepsia Increased risk chronic ulcers and g. ca (2-4%)
26
Whar will contribute to type B (antral gastritis)?
Age, chronic duodenum reflux, alcohol abuse, aspirin and helicobacter pylori
27
Percentages of those who are exposed to helicobacter pylory are asymptamatic or symptomatic.
Asymptomatic10-50% in US | Symptomatic 60-90% with type B
28
Multiple shallow small ulcerative lesions with rapid onset are what?
Acute, stress ulcers
29
Acute ulcerative burn lesions are called what and associated with what?
Curling’s | Circulatiry alterations leading to mucosal ischemia
30
Acute ulcerative cranial/trauma lesions are called what and associated with what?
Cushing’s | Incresed HCL
31
T/F | Are acute, stress ulcers irreversable?
F
32
What does punched out referr to?
Seen in chronic peptic ulcers where there are distinct mucosal defects having smooth margins.
33
Chronic peptic ulcer
Single lesions in regions exposed to peptic juices
34
List common regions of the chronic peptic ulcer.
``` Duodenum Stomach Esophagus Mechels diverticulum-(g. Mucosa) Jejunum-(rare) ```
35
Where are 98% of chronic peptic ulcers located?
Duodenum and stomach
36
jejunal ulcers are associated with what?
Zillinger-Ellison syndrome