Exam 2 - Cardio Flashcards
(48 cards)
How do beta blockers work?
Beta-blockers slow down your heart rate and reduce the force at which blood is pumped round your body (cardiac afterload)
Which is the first elevated substance during MI?
Troponin
Main cardiac enzymes
SGOT
LDH (LD)
CPK (CK)
Troponin
All go up following MI (can take up to 6 hours to elevate enough to be noticed)
Blood pressure medicine that ends in “ide”
Thiazide diuretics
Remove unwanted fluid from the body, which helps lower blood pressure
Blood pressure medicine that ends in “pine”
Calcium channel blockers
Make the artery walls relax, making them wider, which lowers blood pressure
Blood pressure medicine that ends in “artan”
Angiotensin receptor blockers (ARBs)
Control hormones that affect blood pressure
Blood pressure medicine that ends in “pril”
ACE inhibitors
Help to control hormones that affect blood pressure
Coarctation of the aorta
Physical assessment findings / What happens above and below the area of coarctation?
- Higher blood pressure in upper extremities than the lower extremities
- Bounding pulses in arms
- Weak or absent femoral pulses
- Cool lower extremities
- Headache
Aortic stenosis
- Narrowing of aortic valve, causing resistance to blood flow from LV into aorta
- Characteristic murmur is present
- Increased afterload/work of LV
- Decreased cardiac output, left vent hypertrophy, pulmonary vascular congestion
Which health disorder lead to aortic regurgitation
- Result of an incompetent aortic valve that allows blood to flow back to the left ventricle during diastole
- May result from conditions that cause scarring of the valve leaflets or from enlargement of the valve orifice to the extent that the valve leaflets no longer meet
- Rheumatic fever, idiopathic dilation of the aorta
- Congenital abnormalities
- Infective endocarditis
- Marfan syndrome
Which health disorders lead to mitral regurgitation
- Most commonly from mitral valve prolapse
- Rheumatic heart disease is associated with rigid and thickened valve that does not open or close completely
- Rupture of chordae tendonae and papillary muscles, papillar muscle dysfunction, or stretching of the valve structures due to dilation of LV or valve orifice
Which health disorders lead to mitral stenosis
- Most commonly result of rheumatic fever
- Less common: congenital and manifests during infancy or early childhood
Varicose Veins – what changes occur within the veins?
- Dilated, tortuous veins
- Prolonged exposure to increased pressure causes the venous valves to become incompetent so they no longer close properly
- Reflux of blood causes further venous enlargement – pulls valve leaflet apart, causing more valvular incompetence
- Secondary – flow in the deep channels is impaired/blocked (most common cause is deep vein thrombosis)
Raynaud Phenomenon/Disease – Pathophysiology & clinical manifestations
- Affects otherwise healthy women
- Hyperreactivity of sympathetic nervous system has been suggested as contributing cause
- Vasospasm of arterioles and arteries of upper & lower extremities
- Vasospasm causes constriction of the cutaneous vessels
- Attacks are intermittent and occur with exposure to cold or stress
- Affects primarily fingers, but can affect toes, ears, cheeks
- Fingers start becoming white – then blue – then red
- (PAD disease)
Stable angina
Pain when heart’s oxygen demand increases
o Occurs with activities that involve exertion or emotional stress; relieved with rest or nitroglycerin
o Pain described as constricting, squeezing, or suffocating sensation
o Usually steady pain, increasing in intensity only at the onset and end of the attack
o Pain commonly located in the precordial or substernal area of the chest
o Similar to MI in that it may radiate to the left shoulder, jaw, arm, or other areas of the chest
Unstable angina
Does not follow a pattern
o Occurs with an unpredictable degree of exertion or emotion and increases in occurrence, duration, and severity over time
o No evidence of serum markers for myocardial damage
o Occurs at rest or with minimal exertion, severe, frank pain
Variant angina
Pain when coronary arteries spasm
o Results from coronary artery spasm – may occur at rest or with minimal exercise, and frequently occurs nocturnally
o Arrhythmias often occur when the pain is severe, and most persons are aware of their presence during an attack
o ECG changes significant – ST-segment elevation or depression, T-wave peaking, inversion of U-waves, and rhythm disturbances
Silent myocardial ischemia
Myocardial ischemia without pain
o Impaired blood flow from the effects of coronary atherosclerosis or vasospasm
o 3 populations – persons who are asymptomatic without other evidence of CAD, persons who have had an MI and continue to have episodes of silent ischemia, persons with angina who also have episodes of silent ischemia
o Ischemic episodes may be shorter and involve less myocardial tissue than those producing pain
Nursing management for MI
- Administer Aspirin to patient
- Put patient to bed to reduce the amount of oxygen requirement for the affected area of myocardium
- Pain management: Reducing as much pain as possible for the patient
- Oxygen therapy
Endocarditis – Risk factors
- IV drug users or alcohol use
- Immunodeficiency, malignancy, or therapeutic immunosuppression
- Diabetes
- Has had valve replacements or repair of valves with prosthetic materials
- Other structural cardiac defects
- Occult source into the oral cavity or gut
Left Heart Failure—Clinical manifestations (S/S)
Left ventricle cannot maintain adequate cardiac output to the body – pressure backs up into the lungs
- Tachycardia
- Dypsnea, orthopnea, nocturia
- Decreased cerebral perfusion
- Pulmonary congestion and edema (crackles may be heard on auscultation)
- Urine output less than 30 mL/hr
- Cool, clammy extremities
- Excessive fatigue
- Coughing, wheezing, tachypnea, frothy sputum
- Systolic – not enough blood pumped to body
- Diastolic – not enough blood from lungs
Right Heart Failure—Clinical manifestations (S/S)
Right ventricle cannot handle the venous return – pressure backs up into the venous system
- Peripheral edema
- Jugular vein distention
- Ascites hepatomegaly, pain in upper right quadrant
- Weight gain
- Tachycardia
- Clear lungs, paradoxical pulse (BP increases on expiration)
ECG changes during an acute myocardial infarction (NOT ischemia)
- ST elevation MI (STEMI), T wave inversion
- An abnormal Q wave may also be present
- Hours to days after the MI, ST and T wave changes will return to normal but the Q wave changes usually remain permanently
Buerger disease – Clinical manifestations
- Occlusive disease of median and small arteries and veins
- Distal upper and lower limbs are affected most commonly
- Typically affects young men who smoke
- Affects digital, tibial, plantar, ulnar, palmar arteries
- Symptoms:
o Intermittent claudication
o Affects primarily toes – tenderness and hair loss in affected area
o Ischemic pain occurring in the digits while at rest
o Aching pain that is more severe at night
o Cool, numb, or tingling sensation
o Diminished pulses in the distal extremities
o Extremities that are cool and red in the dependent position
o Development of ulcerations in the extremities
o Can lead to gangrenous lesions