EXAM #2: COMPLICATIONS OF MI Flashcards

(34 cards)

1
Q

When are arrhythmias most common in the setting of MI?

A

Acute phase b/c of ischemia

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2
Q

Do PVCs require specific management in the post-MI setting?

A

No

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3
Q

What is an accelerated idioventricular rhythm?

A

Ventricular rhythm with a rate of 60-110 caused by ischemia induced automaticity of purkinje fibers

*Most often seen post-cath. and an indication of reperfusion

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4
Q

What kind of VT is associated with ischemia and how does this differ from VT associated with a post-MI scar?

A

Ischemia= Polymorphic

Scar= Monomorphic

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5
Q

What is the treatment for VT?

A

1) Immediate cardioversion

2) Amiodarone

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6
Q

Late VT/VF occurring 48 hours post-MI is associated with _____?

A

Increased risk of sudden cardiac death

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7
Q

What are the two indications for sudden cardiac death prophylaxis post-MI?

A

1) Late VT/VF

2) EF less than 35%

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8
Q

What causes sinus bradycardia in the acute phase of a MI?

A

1) Sinus node ischemia
2) High PNS tone
- Associated with inferior wall MI

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9
Q

When an inferior MI causes a heart block, what level of the conduction pathway is being most affected? Is a permanent pacemaker required?

A

AV node

Typically, a permanent pacemaker is NOT required b/c this is due to high vagal tone that is TRANSIENT

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10
Q

How does an anterior MI leading to heart block differ from inferior?

A

Anterior is:

1) Below the AV node
2) Requires permanent pacemaker

*Also much more rare

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11
Q

What is the difference between Killip class 2 and 3?

A

Class 2= mild pulmonary edema

Class 3= fulminant pulmonary edema

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12
Q

What is the difference between acute and chronic phase MI associated HF?

A

Acute= diastolic and/or systolic dysfunction

Chronic= systolic dysfunction

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13
Q

How is HF managed in the acute phase of a MI?

A

1) Vasodilator i.e. NTG
2) Judicious morphine
3) BiPap

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14
Q

How is HF managed in the post-acute phase of a MI?

A

1) Diuretic
2) ACE-inhibitors
3) Aldosterone antagonists

*In contrast to the acute phase, these patients are retaining fluid

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15
Q

What is a cardioembolism? What is this most commonly associated with?

A
  • Anterior wall dysfunction leads to emboli formation
  • Embolism is a cause of “cardioembolic stroke”

*Note that these can embolize to other locations as well e.g. bowels, legs…etc.

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16
Q

How are cardioembolisms treated?

A

Anticoagulation with warfarin

17
Q

When is percarditis typically seen post-MI?

A

Early= within the first week
- Focal inflammation of pericardium overlying the involved myocardium

Late= 1-8 weeks

18
Q

What is the treatment for early pericarditis? What treatments must be AVOIDED?

A

1) ASA
2) Colchicine as an adjunct

*Avoid NSAIDs and sterodis

19
Q

What is Dressler’s Syndrome?

A
  • Autoimmune disease
  • Autoantibodies against the percardium
  • Associated with malaise, arthralgias, pleural/ pericardial effusion
20
Q

What labs are associated with Dressler’s Syndrome?

A

High ESR and CRP

21
Q

What increases the risk of an Acute VSD post-MI?

A

1) Delayed or absent reperfusion
2) Elderly
3) Female
4) HTN
5) First MI

22
Q

List the sx. of an Acute VSD s/p MI.

A

Chest pain
Dyspnea
Hypotension
Biventricular failure

23
Q

What are the PE findings associated with an Acute VSD?

A
  • New murmur
  • Left low sternal border
  • Thrill in 1/2 of patients
24
Q

How is an Acute VSD diagnosed?

A

1) Echo*
2) Right heart catheterization

*Gold standard

25
What is an oxygen saturation "step up" on right heart cath. pathogoominic for?
Acute VSD
26
What is the most common etiology of acute mitral regurgitation?
Papillary muscle dysfunction
27
In the setting of inferior MI, what papillary muscle is damaged to cause acute mitral regurgitation?
Posteromedial papillary muscle rupture
28
What are the symptoms of acute mitral regurgitation?
1) HF sx. | 2) Hemodynamic collapse
29
How does acute mitral valve rupture differ from Acute VSD?
VSD= thrill in 1/2 Mitral Valve Regurg.= no thrill
30
How do the murmurs of VSD and MR differ?
VSD= loud at LSB MR= faint *Note that VSD is associated with both acute and inferior MI vs. MR that is associated with inferior
31
When does free wall rupture most commonly occur post MI?
1-5 days post MI
32
How does free wall rupture present?
- Sudden hemodynamic collapse - Cardiac tamponade - Feeling of impending doom
33
How is free wall rupture diagnosed?
1) Echo | 2) PA cath. with blunted Y-descent (emptying is impaired)
34
What is the difference between a LV true and pseudoaneurysm?
True= 3x walls bulge out - No rupture Pseudo= tear of endocardium, myocardium--epicardium is the only thing holding this together - Likely to rupture and require immediate surgical repair