Exam 2 Week 4 Flashcards
(32 cards)
headache prevalence, migraine, tension HA and costs
93-98% have HA each day 16% migraine: 10-12% annually tension: 38% annually $13 billion (work days and impairment)
what are cervicogenic headaches attributed to, and what affects them
abnormalities of the C-spine.
cervical movement
what is the primary pain pattern of a cervicogenic HA
occipital, and radiating into the head and face. 70% HA sufferers complain of neck pain with their HA too.
what is the estimate of cervicogenic HAs
1 in 5 of the population
what seems to be the basis for the pain pattern of a cervicogenic HA
trigeminocervical nucleus.
afferents from CN 5 and Spinal nerves C1-C3.
how can we tell a migraine from a sinus from a tension from a cluster HA
migraine: one sided, lasts hours to days, and throbbing
sinus: widespread, until you treat it and dull
tension: widespread, hours, and dull
cluster: one sided, minutes to hours and sharp (like an ice cream HA)
what are some possible signs of intracranial pathology
sudden onset of severe HA, increasing intensity, persistent unilateral, HA that wake them up at night, stiff neck (sign of meningitis), systemic symptoms (weight loss, fever, malaise), neuro signs and symptoms.
what must we be mindful of (red flag) when we are seeing patients with HA’s.
2/100,000 with internal carotid dissection
1/100,000 vertebral artery dissection.
most present with head the neck pain.
BIG complaint: HA like never before
are the pain distributions for the internal carotid and vertebral arteries very helpful
no, overlap
when someone comes in with a HA, what is concerning, about their history
pain in the head and neck, unlike anything I’ve had ever before
what in a patients past medical history are you looking for when treating someone you suspect has an artery dissection
atherosclerosis risk factors, for potential damage
stroke, diabetes, smoker, MI, angina, TIA, CVA, PVD family history
HTN
when examining for cervical artery dysfunction with cervicogenic HA, what are you looking for
5 D’s and 3 N’s
dizziness, drop attacks, diplopia, dysarthria, dysphagia,
ataxia
nausea, numbness, nystagmus.
what are other red flags for cervical artery dysfunction
taste changes, face sensation, visual changes, facial numbness, tinnitus, gait changes, horsiness, vomiting, member or motor loss
what is the gold standard for diagnosis of cervicogenic HA
a greater occipital nerve block, which can abolish the pain for up to 30 days.
what are some subjective things with cervicogenic HA
pain in the area, posterior head and neck, unilateral, with shoulder and arm pain. Provoked with sustained and awkward positions, and increasing frequency of short lasting HA attacks.
what objective things are demonstrated with cervicogenic HA’s.
neck movement or sustained movements, trigger options, abnormal joint mobility testing, +CFRT, poor neck flexor endurance tests
what is the cervical flexion rotation test (CFRT)
looking for a significant difference in C1-2 ROM, have the neck flexed, and turn the head side to side. Positive if less then 32 degrees.
what is the cranial cervical flexor test (CCFT)
purpose is to assess deep cervical flexor strength. you have the patient lay supine, and flex their cervical spine, by doing a chin tuck. make sure there is no SCM activity (not too much). the bladder under the neck should be able to generate 26-30mmHg of pressure for 10 seconds without compensation.
what is the neck muscle endurance test
assess deep cervical flexion strength. tuck their chin, and hold your head up, and raise head off table 2.5 cm. see how long they can hold it. normal is more then 38 seconds.
what is the diagnostic cluster or cervicogenic HA
- decreased AROM cervical extension
- palpably painful somewhere from OA-C3-4 joint dysfunctions
- deep cervical flexor strength impairments with CCFT (cranio-cervical flexion test)
- NOT USUALLY PRESENT WITH MIGRAINE HA
whats another diagnostic cluster for cervicogenic HA
palpably painful C1-2 joint dysfunctions, and pec minor length shortened.
people with cervicogenic HA have muscle tightness in…
upper trap, levator, scalenes, SCM, pec major and minor
what was found, with cervicogenic HA, with the CCFT?
cervicogenic group had significantly greater activation of the superficial neck flexors, like the SCM.
treatment for cervicogenic HA, as found by research and evidence
neck exercises, low endurance training, and spinal manipulation, are effective for short and long term.