exam part 2 Flashcards

(60 cards)

1
Q

what a nurse should know before a trauma patient arrives

A

What type of trauma is it
What are the haemodynaic parameters
Any other parameters
Is there anyone else who needs to be there
Do we need to notify anyone else

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2
Q

primary survey for trauma patients

A

Catastrophic haemorrahge control
Airway and cervical spine
Ensure adequate oxygenation
Face and neck injuries can cause compromised airway
Breathing and adequate ventilation
Circulation and hemorrhage contro
Disability neurology and pupils
Exposure but keep warm

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3
Q

what are causes of airway compromise

A

Hemorrhage
Swelling
Foreign bodies
Decreased loc
Displaced tongue

Signs and symptoms
Change in voice
Noisy breathing
Tachypnoea
Dyspnoa
Bleeding secretions
Agitation and or altered loc

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4
Q

breathing assessment of trauma patients

A

Management of tension pneumothorax
Immediate decompression
Needle decompression
Finger thoracostomy
Chest drain
Promote oxygenation
Insertion of chest drain
Large bore ivc
Transfusion of blood products

Management for all breathing
Promote oxygenation
Cxr
Removal of abnormal air or blood
Analgesia as required
CHip

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5
Q

Open tension pneumothorax
Management

A

Promote ventilation and oxygenation
Cover wound with 3 sided dressing
Insert chest drain
Analgesia
Iv antibiotics

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6
Q

Assessment circulation
in trauma patients

A

Skin colour
Temperature
Cap refill
Pulse
Blood pressure
External bleeding
Alerted mentation
Management
Recognise it
Find out whats causing it
Sto0p it

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7
Q

blood products considerations

A

Determined by clinical parameters and response to mx
Life threatening circulation requires activation of massive transfusion
Massive transfusion defined as >10 units PRBC in 24 hours or more than 4 units in 1 hours
Blood products =tranexamic acid
Aim 1:1:1

Uncontrolled bleeding and >30 min delay to OT 100-200 ml boluses to maintain BP 80-90 mmHG
caution in elderly
Contraindicated in unconscious pts with impalpable bp
Contraindicated in tbi
Does that patient need code crimson
Does the patient require urgent or embolization
Can we manage this patient
Do we need to transfer out
Who do we need to call

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8
Q

disablity for trauma patients

A

Head injury
Effects of alchohol or drugs
Hypoxia
Hypovoemia
Assessment
Pupils size equality reaction
AVPU scale
Glasgow coma scale
Management
Urgent ct scan
Prevent secondary brain injury
Good oxygenation
Good bp
Not too much o2

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9
Q

patient risk factors for sepsis

A

Age over 65
Surgical history
Invasive lines
IDC
Drains and open wounds
Multiple health care professional interaction
Medical history

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10
Q

Deteriorating patient early signs in sepsis

A

RR less than 10 greater than 30
Decrease in LOC less than 2 points
Alteration in mental status
Tachy or bradycardia
hypo/hypertension
Decreased urine
Desat
New uncontrolled pain chest pain
Unexpected increase in output
Family concerns

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11
Q

late symptoms in sepsis

A

Bp less than 80 and over 240
Hr less than 40
Gsc lower 9
Airway obstruction/ stridor
RR less than 5 or greater than 40
Sats above 90
Paco2 greater than 60
Seizures
Bgl 2 vs 25
Anuria

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12
Q

symptoms of sepsis

A

Abo pain
Lung cough sob
Neuro
Altered loc new onset of confusion neck stiffness
Skin
Wound, cellulitis
Urine
Dysuria, frequency, odor

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13
Q

Sepsis risk factors paeds

A

Less than 3 months
Re presented in 48 hours
Immunocompromised
Indwelling medical device
Recent surgery
High level of parental concern

Less than 3 months
Re presented in 48 hours
Immunocompromised
Indwelling medical device
Recent surgery
High level of parental concern

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14
Q

overview of the cardiovascular system

A

Fluid - blood
Adequate volume
Adequate sodium levels
Haemostatic processes

Pump - heart
Dependent on
Strength of contractions
Hr within normal limits to ensure adequate pump
Diastolic function - heart can relax and refill and start again
Venous return to ensure enough circulating volume

Delivery system
Intact vessels to prevent fluid leakeage
Vessels damage loose contractility but have that pressure

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15
Q

ATP cycle

A

Without it cannot survive long
Glucose is required for transfer of atp in a process called cellular respiration
3 main steps in this process
Glycolysis - does not require o2 byproduct of lactic acid
Krebs cycle - aerobic process requires oxygen
Electron transport chain - also aerobic

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16
Q

what is Dysoxia

A

Anaerobic metabolism ineffective atp production
Cellular death

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17
Q

what is shock

A

a failure of the circulatory system to maintain effective tissue perfusion resulting in cellular dysfunction and acute organ failure 4

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18
Q

treatment of shock

A

Recognize
Respond
Treat
Prevention is better than cure
Lactact is important measure absence of other cv symptoms

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19
Q

what is the sepsis 6

A

Oxygen
Blood culture
Lactate
Antiobitocis
Fluids
Monitor urine

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20
Q

what is the function of the skin

A

Temp reugaltion
Sensory
Interface
Immune system
Control or fluid loss
Metabolic function
Pscyh-social function

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21
Q

jacksons burn wound model

A

from deepest to highest

zone of hyperamia
zone of stasis
zone of coagulation

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22
Q

Severity of burn

A

Depth = time and temp
Severity of local injury
However depth is not the most important predictaor

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23
Q

circulatory effects of burns

A

Increase in cap permeability
Loss of fluid

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24
Q

what parts of the body do burns affect

A

Affects all major organ vessels
Heart and blood vessles
Lungs
Gut
Immune system
Neuro humeral regulation
Kidney
Bone mineralisation and growth

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25
Metabolic effects of burn
Secretion of stress hormones - tachycardia Neural response via sympathetic nervous system and hypothalamus - hyperthermia Suppression of anabolic hormones and development of massive catabolic response - protein wasting Depression of immune response - susceptibility to infection
26
first and immediate response to burns
Stop drop cover roll Cool the burn Asap Avoid hypothermia Cdontinue 20 mins If chemical irrigate copiously with water
27
assessment of depth
epidermos dermis - capillaries and nerves fat
28
burn depth assessment color
epidermal red superficial pale pink mid dermal dark pink deep dermal blotchy red full thickness white
29
epidermal burn
Skin intact red brisk capillary refill erythema not included Heal spontaneously
30
Superficial dermal burn
Blisters present Pink brisk cap refill Should heal within 7-14 days With minimal dressing requirements
31
Mild dermal burn
Heterogeneous Dark pink sluggish cap refill Should heal within 14-21 days Deeper areas over a joint may need surgical intervention and referral
32
Deep dermal burn
Heterogeneous variable depth Blotchy red white sluggish to absent cap refill Surgical intervention
33
Full thickness burn
Outer skin and some underlying tissue dead White brown red black No cap refill Surgical intervention and long term scar
34
what is the rule of 9 and palmar methods
relating to burns 9% of body parts to compromise the entire body palm and fingers of the patient useful for small and scattered burns
35
Fluid management
Increased cap permeability persist for 24 hours How to give Large canulas fluid resus give hartmans solution half in first 8 hours of injury and second halfd in next 16 hours Extra fluid is required in Inhalation injury Electrical injury Delayed resus Dehydration Intoxcitarted patients and fire fighters
36
how to monitor fluid resus
Urine output reflects perfusion Desired hourly output Assess output over several hours for adequacy
37
what is an inhalation burn
Can be caused by heat chemical compounds in the atmosphere Upper airway supra glottic Lower airway subglottic Systemic intoxication (CO, HCN, poisoning) Obtain history Give o2 Examine signs of airway burn Consider intubation early Monitor o2 saturation
38
what is circumferential burns
Brunt skin -rigid Around limbs - venous occlusion Venous occlusion -arterial occlusion around chest - restricted expansion Childs front - restrict diaphragm Elevation reduces swelling
39
how to transfer a patient with burns
Analgesia Plastic wrap Contact burn Clean dry sheet Keep warm prevent hypothermia Consult and transfer to burn unit Documentation
40
what is a catergory 1 triage
Obstructed airway Severe respiratory distress Severe haemodynamic compromise No pulse Skin pale moist mottled Uncontrolled hemorrhage HR less than 60 Ecg Gsc lower than 8
41
what is a category 2 triage
Patent airway Moderate respiratory distress Moderate haemodynamic compromise Thready pulse Cap refill 2-3 seconds Gsc 9-12 Severe pain
42
what is a category 3 moderately severe
Mild respiratory distress Mild haemodynamic compromise Altered vital signs Skin warm pale Gsc 13 Moderate pain Moderate neurovascular compromise
43
what is a category 4 or 5 in triage
Airway patent No respiratory distress no haemodynamic compromise Normal gsc Mild pain Mild neurovascular compromise
44
mental health approach to triage
Primary survey approach Consideration to appearance, behavior and conversation Based on clinical criteria acute behavior disturbances and risk of harm to self or others
45
What does the triage nurse do
Perform quick accurate assessments Prioritize Function under stress Communication Think ahead Control patient flow Diffuse conflict Initiate first aid
46
The skills required in triage
Possess good public relation skills Possess good crisis intervention skills Deal with barriers to communication Process referrals Know the operational policy of the ED Have a working knowledge of the pre hospital system
47
times when you need to alter communication for patients
An intoxicated patient Verbally abusive person Confused elderly person Prisoner in police custody Intellectually disabled person Non english speaking person Sexually abused
48
what is an endocrine emergency
Endocrinology refers to hormones Collection of glands Responsible for sending chemical signals Hormones fall into 2 major classifications Amino acids derived Lipid derived eg steroids Then you have catecholamines
49
what is the endocrine systems responsibilities include
Responsibilities include Aiding with metabolism growth sleep reproduction mood Responds to external stimulus flight or fight Interal Ay be product of an organ Thyroid is one of the most primary glands Most common endocrine disturbance is diabetes
50
most common endocrine emergencies
DKA HYPOGLYCEMIA Thyroid storm Acute adrenal crisis
51
T1 vs T2 diabetes
Autoimmune Lack of insulin Destruction of insulin producing beta cells in pancrea Cannot be prevented Developed resistance Insulin resistance Unable to transfer glucose
52
what is DKA
Lifethreanting metabolic state characterised by high bgls ketosis and acidosis If untreated with result in severe dehydration, cerebral oedema, coma and eventually death Insulin deficency - hypergylcermia prolonged hyperglycemia activates a acascade of events Cellular starvation causing release of counter -regulatory hormones These hormones produce more glucose in an attempt to supply energy to cells Bgls exceed kidneys ability to reabsorb glucose- resulting to glucose in urine and polyuria High glucose spills into urine porcess of osmotic diuresis Polyuria, polydispia, hyperkaemia Counter regulatory hormone activate lipolysiss Fats and proteins transferred to glucose called beta oxidation Weight loss Ketone production (mainly acetone) Fuel for brain during glucose stravation Large amounts of ketones result in metabolic acidosis Weight loss polyphagia keto-breath Initially - body buffers acidosis with bicarbonate buffering system but this system is quickly overwhelmed and nother mechanisms Eg hyperventilation to lower the blood co2 leveles compensatory respiratory alkolisis Kussmaul breathing
53
presentation of DKA
Severe dehydration Weight loss Hyperventilation Acetone breath Flushed cheek Abdo pain Disorientation Shock poly dipsia and polyuria Cerebral oedema Severe dehydration Prolonged hyperkaelmia Thrombosis Hyperglycemia hypermosolar state High bgl
54
management of DKA
Abcdgef Minimum 2 large bore IVC Symptomatic mangemange Treat the cause CXR FBC UA NSU Weight ECG ongoing cardiac monitoring Nursing responsibilities Assess and manage threats to life Symptomatic treatment Idc Strict fbc Insulin Check ketones Check k Fluid replacement
55
what is hypoglycemia
Bgl less than 4 mild to severe Symptoms Hunger Shakiness Anxiety Relestness Decreased loc Diaphoretic dizziness Changes in sensrioum most common
56
treatment of hypoglycemia
have 15 grams of fast acting carbohydrate 6-7 jelly beans fruit jucie recheck bgl 15 mins
57
what is the thyroid gland
Responsible for growth and metabolism Secretes 3 mian hormones T3 t3 and calcitonin T3 and t4 increase metabolic rate for growth when we are cold Relies on pituitary gland to tell it to make it more or less Could be overactive or underactive
58
what is a thyroid storm and presentation
Life threatening Excess amounts of thyroid Mutil organ dysfunction Common in young teenage and women Presentation Thermoregulatory dysfunction Altered loc seizure coma psychosis Cardiovascular af tachycardia hypertension Tachypnea Diarrha abdo pain nausea vomiting
59
precipitating factors and treatment of thyroid storm
Severe infection Undertreated hyperthyroidism Dka Surgery Trauma to thyroid Pulmonary embolism Surgical manipulation of thyroid gland Radiotherapy Treatment Early intervention A-g immediate management prn Symptoms management and supportive care
60
what is the adrenal gland
Secrete hormones to regular metabolism bp and stress response Cortisol aldersterone adrenaline and noradrenaline