EXAMINATION Flashcards

(34 cards)

1
Q

WHAT SPINAL LEVELS ARE TESTED WITH THE BICEPS REFLEX

A

C5-6

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2
Q

WHAT SPINAL LEVELS ARE TESTED WITH THE BRACHIORADIALIS REFLEX

A

C6-7

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3
Q

WHAT SPINAL LEVELS ARE TESTD WITH THE TRICEPS REFLEX

A

C7-8

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4
Q

WHAT ARE THE MOVEMENT USED TO TEST MOTOR STRENGTH AT C4-T1

A

4= SHOULDER SHRUG
5=GH ABDUCTION
6=ELBOW FLEXION
7=ELBOW EXTENSION
8=FINGER FLEXION
1=FINGER ADDUCTION

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5
Q

WHAT ARE THE RATINGS 5-0 FOR MUSCLE STRENGTH

A

5=COMP ROM, GRAVITY, FULL RESISTANCE
4=COMP ROM, GRAVITY, SOME RESISTANCE
3= COMP ROM, GRAVITY, NO RESISTANCE
2=COMP ROM, SOME ASSISTANCE, GRAVITY ELIMINATED
1= SLIGHT MUSCLE CONTRACTION, JO MOTION
0=NO EVIDENCE OF MUSCLE CONTRACTION

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6
Q

WHAT NERVES ARE TESTED BY ULNDT1

A

MEDIAN NERVE, ANTERIOR INTEROSSEOUS NERVE (5,6,7)

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7
Q

WHAT NERVES ARE TESTED BY ULNDT2

A

MEDIAL, MUSCULOCUTANEOUS, AXILLARY

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8
Q

WHAT NERVE IS TESTED BY ULNDT3

A

RADIAL

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9
Q

WHAT NERVES ARE TESTED BY ULNDT4

A

ULNAR NERVE, C8-T1 NERVE ROOTS

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10
Q

WHAT IS THE GRADING FOR PULSES

A

3= BOUNDING
2= NORMAL
1= WEAK/ DIMINSHED
0- ABSENT

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11
Q

WHAT REGIONAL OBSERVATIONS WOULD YOU DO WHEN ASSESSING THE CERVICAL SPINE

A

CERVICAL SCREEN
UPPER EXTREMITY REGIONAL SCREEN

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12
Q

WHAT SPINAL LEVEL IS THE HYOID BONE IN LINE WITH

A

C2/3

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13
Q

WHAT SPINAL LEVEL IS THE THYROID CARTILAGE IN LINE WITH

A

C4/5

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14
Q

WHAT SPINAL LEVEL IS THE CRICOID RING IN LINE WITH

A

C6

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15
Q

WHAT ARE THE THREE PARTS OF SEGMENTAL DEFINITON

A

DIAGNOSTIC PALP, JOINT PLAY (TRANSLATION), SEGMENTAL ARTICULATION

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16
Q

WHAT DOES SHARP PURSER TEST FOR

A

CERVICAL LIGAMENT INSTABILITY

17
Q

WHAT DOES ALAR LIG STRESS TEST, TEST FOR

A

ALAR LIGAMENT INSTABILITY/ LAXITY

18
Q

WHAT ARE THE ATTACHMENTS OF THE ALAR LIGAMENT

A

DENS - BASE OF SKULL

19
Q

WHAT ARE THE ATTACHMENTS OF THE TRANSVERSE LIG

A

SMALL TUBERCLE ON THE MEDIAL SURFACE OF THE LATERAL MASS’ OF ATLAS

20
Q

WHAT IS SPURLINGS TESTING FOR

A

FACET ISSUES AND SPINAL NERVE COMPRESSION

21
Q

WHAT IS THE DISTRACTION TEST TESTIONG FOR

A

SPINAL NERVE COMPRESSION

22
Q

WHAT ARE THE 6 THERAPUTIC MECHANISMS OF SOFT TISSUE TECHNIQUES

A

REDUCTION IN PAIN
INCREASED FLUID DRAINAGE
STRETCHING SHORTENED TISSUES
PROMOTION OF HEALING AND COLLAGEN REMODELLING
RELEX MUSCLE RELAXATION
REDUNCE ANXIETY, PROMOTE RELAXATION

23
Q

WHAT ARE THE 5 THERAPUTIC MECHANISMS OF COUNTERSTRAIN

A

NOCICEPTOR MODEL
NEUROLOGICAL MODEL
CIRCULATORY MODEL
FIBROBLAST MODEL
IDEOMOTION

24
Q

WHAT IS TEH IDEOMOTION MODEL

A

BASED OFF THE BIOPSUCHOSOCIAL MODEL, AND HOW ONE PRECEIVES PAIN WHERE IF SOMEONE IS HELPING US MOVE, DO WE MOVE BETTER?

25
WHAT IS THE NOPCICEPTOR MODEL
THE MORE SIGNALS THAT YOU GET IN TO THE TISSUES MODERATES AND DULLS DOWN THE PAIN, DESENSITISING THE PAIN SIGNALS
26
WHAT ARE THE THERAPUTIC MECHANISMS OF MET
INHIBITION OF PAIN (MECHANORECEPTORS) INPROVED FLUID DRAINAGE IMPROVED MOTOR CONTROL AND PROPRIOCEPTION LENGTHENS MUSCLES AND TISSUES
27
WHAT ARE THE LEVERAGES OF UPSLOPE HVLA
PRIMARY ROTATION SECONDARY SB
28
WHAT ARE THE LEVERAGES OF DOWNSLOPE HVLA
PRIMARY SB SECONDARY ROTATION
29
WHAT ARE THE ABSOLUTE CONTRAINDICATIONS FOR HVLA
BONE = ANY PATHOLOIGY THAT HAS LED TO BOEN WEAKENING NEURO = ANY COPMPRESSION OF SPINAL NERVES, CORD OR CAUDA EQUINA VASCULAR = CAD LACK OF DX LACK OF CONSENT PATIENT POSITIONING CANNOT BE ACHIEVED
30
WHAT ARE SOME RELATIVE CONTRINDICATIONS OF HVLA
PREVIOUS ADVERSE REACTION REGNANCY OSTEOPOROSIS VERTIGO PSYCH DEPENDENCE ON HVLA
31
WHAT ARE THE THERAPUTIC MECHANISMS OF HVLA
INCREASED ROM DECREASED PAIN
32
HOE DOES HVLA INCREASE ROM
when two joint surfaces are pulled apar there is a change in volume and pressure withiun the joint cavity, once the pressure of c02 is reached, the dissolved co2 in the synovial fluid combines to form one bubble that then collapses aka tribonucleation. it is speculated whether the sound comes from the formation or collapse of the bubble. as joint volume is icreased there is less kg of force required to create mm of gaps between the joint surfaces, hence increasing ROM
33
HOW DOES HVLA REDUCE PAIN
pain is transported from perceived stimulus picked up via nociceptors to the spinal cord via first order neurons, which then synapse with second order neurons and carry the signals to the brain order neurons, hvla acts to inhibit this pathway through the activation of interneurons which stops the synapse between 1st and 2nd order neurons, stopping the transportation of pain signals, decreasing perception of pain. they can also resuce main through the activation of mechanoreceptors that create a 'pain gate' which also stops the transduction of the afferent signals
34
what are the risks of HVLA
TENDERNESS BRUSING DISC HERNIATION, NERVE ROOT COMPRESION, CERVICAL AND THORACIC STRAIN STROKE AND NEURO DEFICITS