Final Exam Flashcards

(145 cards)

1
Q

DSM classification broadly identifies mood disorders as either:

A

Unipolar: a single depressive mood experience
Bipolar: involves mania and depression

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2
Q

2 separate categories of mood disorders

A

Depressive disorders
Bipolar and related disorders

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3
Q

Major depressive disorder (MDD) is defined by the presence of: (8)

A
  • one or more major depressive episodes or irritable mood episodes
  • loss of pleasure (anhedonia)
  • change in weight or appetite
  • sleep problems
  • fatigue or loss of energy
  • feelings of worthlessness or guilt
  • difficulty thinking or concentrating
  • thoughts of death or suicidal thoughts and behaviour
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4
Q

DSM diagnostic criteria of MDD

A
  • must have 6 out of 8 symptoms
  • symptoms must be present for at least 3 consecutive days and must last for at least 2 weeks
  • must cause individual clinically significant stress or impairment
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5
Q

Persistent depressive disorder (dysthymia) diagnostic criteria

A
  • same symptoms of MDD but symptoms are less severe and more chronic
  • symptoms must be present for at least 1 year
  • must be clinically significant and cause distress or impaired functioning
  • along with 2 or more of the following symptoms:
    Poor appetite or overeating
    Sleep disturbances
    Low energy or fatigue
    Low self esteem
    Difficulty concentrating or making decisions
    Feelings of hopelessness
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6
Q

When can MDD appear in children

A

Depression can start as early as preschool

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7
Q

What is the most prevalent form of affective disorder among children and adolescents?

A

Major depressive disorder (MDD)

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8
Q

Epidemiology of depression (clinical vs community)

A

-Clinical sample: rates in children range from 80% MDD
-Community sample: rates in children range from 0.4-2.5%
-Lifetime prevalence rates of diagnosable depressive disorders are 20-30%
-Typically occurs more in females than in males after the age of 12

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9
Q

SES, ethnic, and cultural considerations for depression

A
  • research suggests that lower SES associated with higher rates of depression
  • possible influences on income on MDD:
    Chronic stress (on mood and physical symptoms of MDD)
    Family disruption
    Environmental adversities
    Racial and ethnic discrimination
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10
Q

Biological influences on depression (genetics- family history of depression)

A
  • highly heritable but not the only factor
  • higher rates in first degree adult relatives
  • genetic effects may influence personality and temperament
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11
Q

Biological influences of depression (neurochemistry and brain functioning)

A
  • serotonin, norepinephrine, acetylcholine
  • builds on research that finds certain classes of medication are effective as antidepressants
  • low levels of serotonin and norepinephrine are results of too much reabsorption by the neuron and the breakdown of neurotransmitter too efficiently
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12
Q

Social-psychological influences on depression (2)

A

Separation and loss
- can produce adverse circumstances including lack of care, changing in family structure, socioeconomic problems

Cognitive and interpersonal perspective
- interpersonal skills, cognitive distortions, views of self, control beliefs, self regulation, and stress
- the way a person relates to others, is viewed by others, and view themselves contributes to how depression developed and/or is maintained

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13
Q

Impact of parental depression

A

Children from homes with a depressed parent are:
- greater risk of developing MDD and other disorders
- less likely to get treatment

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14
Q

Garber and Flynn (2001): longitudinal study of children with depressed mothers - findings

A

Possible that behaviour of depressed parents may be accompanied by anger, frustration, and hostility
- alters the parent’s ability to parent effectively
- parents may be detached, withdrawn, and inattentive
- depressed behaviour is maintained by parent-child interactions

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15
Q

Assessments of depression

A

Self report measures
- children’s depression index (CDI)
- revised children’s anxiety and depression scale

Parental/teacher measures
- behaviour assessment system for children (BASC)

Observations and clinical judgment

For children, might not use the DSM; would instead use evidence based on that particular child

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16
Q

Treatment of depression (medications and CBT)

A

Medications
- past: tricyclic antidepressants
- present: SSRIs and second-generation antidepressants

CBT and interpersonal psychotherapy
- challenge maladaptive thoughts and negative attributions, teach problem solving/coping skills
- understand interpersonal issues and problem solving

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17
Q

Bipolar disorder definition

A

Involves the presence of mania as well as depressive symptoms

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18
Q

Mania and euphoria definitions

A

Mania: period of abnormally elevated euphoric mood
Euphoria: characterized by inflated self esteem, high rates of activity, speech and thinking, distractibility, exaggerated feelings of physical and mental well being

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19
Q

DSM-5 criteria for manic episode

A

Persistent elevated, and expansive or irritable mood
3 of the following:
- inflated self esteem
- decreased need for sleep
- more talkative than usual
- thoughts racing
- distractibility
- psychomotor agitation
- excessive pleasure seeking

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20
Q

Types of bipolar disorder

A

Bipolar I: involves a history of MDD and mania
Bipolar II: involves a history of MDD and hypomania (euphoric mood that is shorter in duration - about 4 days - and less severe than manic episodes)
Cyclothymic disorder: chronic but less severe fluctuations in mood. Does not meet criteria for depression or BPD

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21
Q

Comorbidity of bipolar disorder and ADHD?

A

60%-90% of children

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22
Q

FIND (frequency, intensity, number, duration) criteria for BD (Kowatch et al. 2005)

A

-Exceedingly happy or silly (no apparent reason)
- Intense outbursts or anger/hostile
-Frequent irritable moods
-Less sleep than usual (full of energy)
-State of grandiose views of their abilities and plans
-Intense concentration on activity but becomes increasingly disorganized
-Rapid/unintelligible and difficult to follow speech
-Flight of ideas
-Poor judgement

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23
Q

Epidemiology of BD - Blader and Carlson (2007)

A

National representative sample (0-19yo) of doctor visits for mental health related issues = 6.67% in 2002-03
Between 1996-2004: children = 1.4 to 7.3 per 10000; teens = 5.1 to 20.4 per 10000

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24
Q

Prevalence of BD

A

0-6% in a community sample of children and adolescents
Distribution of males and females are equal
No significant cultural/ethnic differences
Less prevalent in prepubertal youth

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25
Diagnostic challenges of BD
-Same diagnostic criteria are used for adults and children -Children tend to have very short episodes, very frequent mood shifts, mixed mood, chronic difficulty in regulating moods -Children present with co-morbid problems -Mania in adolescents - associated with antisocial behaviour
26
BD developmental course
- in some children, major depressive disorder may be an early stage of bipolar disorder —> more likely in those youngsters with an earlier onset of depression - experience relatively early onset of affective difficulties (Median duration for manic episode = 10.8 months; first affective episode = 11.75 years) - retrospective and prospective studies suggest that these children continue to show symptoms of affective disorders, social and academic impairment
27
Risk factors and stoplights of BD
Family history of bipolar disorder (genetics) - biological siblings and parents (immediate family members) - adult twins and adoption demonstrate a strong genetic component - multiple genes affecting amygdala and hormonal and neurotransmitter process, but shared with other disorders Environmental stressors - stressful life events, family relationships, parenting styles
28
Assessment of BD
Structured diagnostic interview: K-SADS Mania scales: Young Mania Rating Scales and GBI Parent/teacher/self-interviews
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Treatment of BD
Pharmacotherapy is the first and most effective treatment Family involvement in treatment is very important
30
Conduct problems listed in the DSM-5
Oppositional defiant disorder Conduct disorder Intermittent explosive disorder Antisocial personality disorder Pyromania and kleptomania
31
Antisocial personality disorder
Pervasive pattern of disregard of and violation of rights of others Met criteria for CD before the age of 15 ADHD—> ODD —> CD—> APD
32
Oppositional defiant disorder - DSM-5 criteria
A pattern of negativistic, hostile, and defiant behaviour Under age 5: symptoms have to occur on most days. Over age 5: symptoms have to occur at least once a week Has to impair functioning (ex. Academically, socially) At least 3 of these behaviours present for at least 6 months: - loses temper - touchy and easily annoyed - angry and resentful - argues with adults - actively defies or refuses to comply with adult request/rules - deliberately annoys others - blames others for own mistakes - is spiteful or vindictive
33
Conduct disorder - definitions
A pattern of repetitive and persistent acts of violence that violates the basic rights of others and goes against societal norms Mild, moderate, or severe Limited prosocial emotions (feeling guilty or not)
34
DSM-5 symptoms of conduct disorder
At least 3 of these behaviours are present during the last 12 months with at least one of them present in the last 6 months: Aggression toward people or animals - bullies, threatens, or intimidates - initiates physical fights - has used a weapon - is physically cruel to people or animals - has stolen while confronting a victim - has forced someone into sexual activity Destruction of property - engaged in fire setting with the intent to cause damage - has deliberately destroyed other’s property Deceitfulness or theft - has broken into house, building, or car - often lies to obtain goods or favours or to avoid obligations - has stolen items of nontrivial value without confronting victim Serious violation of rules - stays out at night despite parent prohibitions - has run away from home overnight at least twice - is often truant from school - beginning before age 13
35
Empirically derived externalizing behaviours associated with CD (2 types of behaviours)
Two syndromes: - aggressive behaviour: argues a lot, destroys things, purposefully disobedient, fights - rule-breaking behaviour: breaks rules, lies, steals, is truant Children can exhibit one or both of these problems Aggressive behaviour carries a higher degree of heritability than rule breaking behaviour
36
Gender differences in conduct problems
Expressed differently in boys and girls Aggression in boys: - defined as an intent to hurt or do harm to others (ex. Hitting, pushing, or threatening behaviours) Aggression in girls: - more directed to hurting another person’s feelings (relational aggression) - ex. leaving someone out of play/excluding from peer group, telling a person you won’t like them unless they do as you say, lying about someone so others won’t like her
37
Epidemiology of conduct problems
Rate 1%-15% of ODD Rate 2%-10% of CD Comparable across countries
38
Developmental paths of conduct problems
Loeber’s 3 pathways model: Authority conflict - stubborn behaviour, defiance, disobedience, truancy Covert - property damage, lying, fire setting, theft, burglary Overt - physical violence, bullying, and sever forms of violence (rape, attacks, strong arm)
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Aggression as a learned behaviour
Children can clearly learn to be aggressive by being rewarded for such behaviour Can also learn through exposure to aggressive models - vicarious learning Can build the child’s repertoire and lead to disinhibition of aggression - family violence is a source of modelled aggression - parents who punish their children physically serve as models of aggressive behaviour Children who exhibit excessive or antisocial behaviours - likely to have siblings/parents/grandparents with histories of conduct problems and records of aggressive and criminal behaviour
40
Family variables in conduct disorder
Low family SES Marital disruption Poor quality parenting Parental abuse and neglect Parenting style Handing down non-effective parenting from generation to generation Stressors that impact parenting quality Parental psychopathology: alcoholism or other parental psychopathology can impact parental effectiveness
41
Biological influences of CD
Aggressive and conduct disorder related behaviours tend to run in the family Longitudinal studies suggest a genetic component to CD Inherited characteristics (body build, temperament, sensitivity to alcohol, irritability, sensation seeking, impulsivity) make an individual prone to conduct disorders - mediated by social conditions, family variables, and social learning experiences
42
Assessment of CD
Parent completes Likert scale 36 items for CD
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Steps in the cognitive processing of social-emotional cues (CD) (5 steps)
Encoding (looking for and attending to aggression) Interpreting (misinterpreting of own and others’ emotions) Looking for alternative responses Selecting a specific response Enacting the response
44
Youth with conduct problems display these problematic social-cognitive processes: (5)
Poorer problem solving skills Attribute hostile intent to neutral actions Generate fewer responses - and those generated are aggressive Expect that aggressive responses will produce positive outcomes Label arousal in conflict situations as “anger”
45
Pharmacological interventions for ODD and CD
With co-morbid ADHD: stimulant medications Mood stabilizers: for extreme aggression and conduct disorder behaviours but works best with parent training and other interventions
46
Parent training interventions for ODD and CD
Best option! Common features of parent training programs: - how to give commands - how to reinforce behaviours - how to discipline and ignore - prepare for difficult situations Incredible Years Training Series - parenting skills for use with children diagnoses with ODD and CD - comes with standardized videos to model parenting skills Cognitive problem solving skills - targets the interpersonal and social-cognitive aspects of CD behaviour - used in children 4-8 years of age - taught to cope with stressful situations - taped vignettes and discussions - uses child sized puppets, colouring books, and cartoon stickers and prizes Parent and child programs are superior in combination
47
3 types of ADHD
ADHD predominantly inattentive subtype ADHD predominantly hyperactive/impulsive subtype ADHD combined type
48
ADHD diagnoses are provided to those who: (5)
Symptoms are present before the age of 12 Symptoms are displayed for at least 6 months Behaviours go beyond that expected developmentally Impairment identified in social/academic functioning Symptoms must occur in at least 2 different settings
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ADHD primary & secondary features
Primary features: - inattentive - hyperactive/impulsive Secondary features: - motor skills - intelligence - cognition (executive functioning skills) - adaptive functioning skills - social behaviour - sleep (lack of sleep) - accidents (very accident prone)
50
Parents and teachers often report that children who are inattentive: (5)
Jump around from one task to another Does not attend to what is being said Is easily distracted Daydreams Has difficulty concentrating
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Confirmed ADHD children pay less attention to their peers; they have a reduced capacity for: (2)
Selective attention Sustained attention
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Parents and teachers often report that hyperactive children are: (3)
Restless Fidgety Unable to sit still
53
Objective measures of hyperactivity can be measured using:
Actigraphs: little accelerometers that register movements in different planes
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What time of day does hyperactivity increase?
In the afternoon
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Parents and teachers often report that impulsivity in children appears as:
Difficulty controlling their own behaviour Ex. The child might act without thinking, interrupt others, cut in line, engage in dangerous behaviours, appear careless and irresponsible, immature and rude
56
Motor coordination problems in children with ADHD (4) + how many children diagnosed with ADHD have these problems?
Clumsiness Delay in motor milestones Poor performance in sports Difficulties with fine motor coordination and timing (particular difficulty in tasks involving complex movements and sequencing, like dancing) About 50% for children with ADHD have motor coordination problems
57
Intelligence and ADHD
Overall, children perform worse on intelligence tests Many have co-morbid learning difficulties/disorders Reduced academic achievement 56% need tutoring 30% have to repeat a grade 30-40% end up I’m at least 1 special education placement 10-35% fail to graduate high school
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Cognition and ADHD
Neuropsychological deficits cluster around executive functioning skills (ie. inhibition, working memory, sustained memory, etc.) Executive functioning skills include those involved in goal-directed behaviour and are involved in planning, organizing, and self-regulation
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Adaptive functioning skills and ADHD
Deficiencies in everyday adaptive functioning skills (ie. brushing teeth, washing their face, getting ready for school) Deficits are in doing rather than knowing Parallel with clumsiness
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Social behaviour and ADHD
-Quickly alienates themselves from peers —> worse if the child is non-compliant and aggressive -Can correct social behaviours but are disorganized, impulsive, or distracted during social activities -May not adequately process social emotional cues -Overrates peer relationships as positive
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Sleep problems associated with ADHD (3)
Problems falling asleep Needing less sleep Involuntary movements (leg restlessness, grinding teeth)
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Accidents and ADHD
Children with ADHD tend to have more accidents and injuries (broken bones, bruises, lost teeth, poisonings) Parents report that children with ADHD are inattentive in risky situations and unaware of consequences of their actions (ie. walking into traffic)
63
ADHD epidemiology
Marked increased over the last 20 years 5-9% in school aged children 2-7% in community based samples and similar rates around the world Assessments using evidence based measures estimate about 20% of children have clinically significant concerns Presentation changes over time Diagnosis usually occurs in elementary school aged children and decrease into high school and beyond Some suggestions of differences in prevalence across social class, ethnicity, and culture
64
ADHD gender ratios (general & clinical population)
2:1 or 3:1 (boys:girls) in the general population 4:1 (boys:girls) in a clinical population
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ADHD presentation in boys and girls
Boys: aggressive and antisocial behaviours, excessive running, climbing, escaping, etc. Girls: inattentive and disorganized - girls often do not meet diagnostic criteria on DSM - can go undiagnosed and miss out on early intervention - girls show many secondary features: executive functioning difficulties, academic problems, negative peer evaluations, higher rates of anxiety, mood disorders, and conduct problems
66
Developmental course of ADHD
Symptoms emerge early and children do not often “outgrow” them (but sometimes do) Community based sample of low income boys between ages 2-10 show 4 developmental pathways: - chronic - medium - moderate - low
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What what thought to be the primary cause of ADHD which we now know is not true?
Brain damage
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Which parts of the brain are smaller in children with ADHD? (3)
Right frontal corticies Caudate nucleus Globes palidus
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Neurobiological mechanisms of ADHD
ADHD involves the under-arousal of frontal cortices ADHD involves dopamine and norepinephrine deficiencies ADHD is heterogeneous- there are a number of expressions of symptomology that is reflected in the disruption of different brain regions or circuitry But the underlying neuropathology is still unclear
70
What is the genetic rate of ADHD in first-degree family?
10-35% Strong genetic component!
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Environmental factors that influence ADHD
Prenatal exposure to maternal smoking and alcohol has been linked to the risk of ADHD in the offspring Exposure to lead has been implicated in disrupting the development of brain areas associated with the control of attention/activity level and executive functioning skills
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Assessments of ADHD need to be broad based and include: (4)
Developmental/family history Perinatal period/acquisition of milestones Examination of home/school environments Examination of co-occurring disorders
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Measuring Sustained Attention using the Continuous Performance Test (visual and auditory)
Used in the diagnosis of ADHD in children and adults Involved monitoring a continuous stimulus set for the Occitan ce of a target Typical results in ADHD: make errors in omission and commission
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Pharmacotherapy treatment for ADHD
In 1937, Bradley and co observed that Benzidine improved the behaviour of children in an inpatient residential care ward Most commonly used medications are stimulants: methylphenidate, dextroamphetamines, and amphetamine Ritalin: peaks 2 hours and out of the system within 4.5 hours Concerta: peaks in 4 hours and out of the system within 8 hours Adderall: blend of dextroamphetamines and amphetampnes Strattera: norepinephrine reuptake Medications have limited time releases
75
Behavioural treatment strategies for ADHD (3)
Consequences for behaviour: - positive behaviour —> lead to desired social or play activities - negative behaviour —> lead to loss of opportunities, time out, loss of earned reinforcer Parent training programs: - teach parents child management techniques (for 4-12 year olds) Classroom management: - working with teachers and administrators to set up a code of conduct and consequences
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ADHD treatment concerns - stimulants
Don’t work on 10-20% of cases and behavioural/inattention changes reach optimal levels in only 1/2 of the children who are responders Biological side effects: sleeping problems, decreased appetite, stomach pains, headaches, irritability, jitteriness, growth suppression Increase risk of child’s addiction to medication/recreational drugs?
77
Multimodal treatment study of ADHD
Ages 6-9 600 kids total 4 treatment groups: medication only, behavioural interventions, combined treatment (medication and behavioural), community care (treatment as normal) Plus a normal comparison group (no ADHD) Measured core adhd symptoms at several time points First 14 months was most intense (weekly check-ins, medication, teacher follow ups) Findings: - medication alone and the combined treatment were both better than behavioural alone and community based treatment - medication is most effective, and the addition of behavioural treatments have additional benefits, but behavioural treatments alone are not as effective - if a child has both ADHD and anxiety, behavioural treatment was just as effective as medication and combined treatments - high income families benefited the most from combined treatment
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Receptive language
The comprehension of language used by others
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Expressive language
The production of language
80
Language development is shaped by:
The environment (ex. Immigration and bilingualism)
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Language development - 6 months (receptive & expressive)
Receptive: turns to source of sound, startles in response to sudden sounds, watches your face as you talk Expressive: makes different cries for different needs, imitates coughs or other sounds
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Language development - 9 months (receptive & expressive)
Receptive: responds to their name, understands being told “no” Expressive: gets what they want through sounds and gestures, plays social games with you, repeats babble sounds
83
Language development - 12 months (receptive & expressive)
Receptive: follow simple 1-step directions, look across the room to something you point and look at Expressive: uses three or more words, uses gestures to communicate - “waves”, gets your attention using sounds, gestures, and pointing while looking at your eyes
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Language development - 24 months (receptive & expressive)
Receptive: follows 2-step directions Expressive: uses more than 100 words, uses at least 2 pronouns “you” “‘me” “mine”, combines two or more words in short phrases
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Language development - 30 months (receptive & expressive)
Receptive: begins taking short turns with other children in reciprocal communication, shows concern when another child is hurt or sad Expressive: uses adult grammar - “two cookies” “I jumped”, uses more than 350 words, uses action words “run” “spill” “fall”, produces words with two or more syllables “com-pu-ter”
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Language and communication disorders are usually associated with one or more of these 6 specific language impairments:
Graphemes Phonemes Syntax Grammar Semantics Pragmatics
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The DSM-5 classification of language disorders has a distinction between:
Whether the child has deficits in expression and/or receptive language
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Speech sound disorder
Child fails to display developmentally appropriate and dialect appropriate speech sounds - difficulty articulating speech sounds - fail to display age-appropriate speech sounds - course of speech sound delayed - incorrect speech sounds - misarticulation - denotes
89
Receptive-expressive disorder (language)
Difficulties comprehending the communication or others and difficulties with expressing language - difficulties with comprehension and production of language - may present as silent (mistaken for selective mutism) - may not respond to speech - may respond inappropriately to others’ speech
90
Expressive disorder (language)
Standardized assessments reveal expressive deficits that fall below nonverbal intelligence and level of receptive language or comprehension - production of language is abnormal in standardized measures - limited amount of speech, small vocabulary, parts of sentences may be missing, generate unusual words, make errors is using plurals and verb tenses
91
Epidemiology of language disorders
Overall, rates of language impairment range from 3-7% of children in the general population Boys are reported as having higher rates than girls Language disorders usually appear by 3-4 years - but some impairments may only become evident with increased demand in schoolwork and language complexity
92
Types of learning disabilities (4)
Language disorders - impairments in language expression and reception Reading disabilities - deficits in the ability to discern the meaning from words in text Written expression disabilities - deficits in the ability to transcribe and generate text Mathematics disabilities - deficits in numerical understanding, learning, representations, and retrieval of basic arithmetic facts
93
Guidelines for diagnosing if a child’s language and learning skills are below expectations
Discrepancy between IQ and academic achievement level Performance on a measure of general cognitive capacity (IQ) significantly exceeds performance on a specific test of achievement The discrepancy of 2 or more standard deviations between IQ and achievement scores is required
94
How learning disorders are identified in the school system (3 levels)
Core instruction - 75-90% (level I) Supplemental - 10-25% (level II) Intense - 2-10% (level III)
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Learning disorders vs learning disabilities
Learning disorders: these disabilities are specific deficits that appear discrepant with intelligence and other abilities Learning disabilities: refer to specific developmental problems in reading, writing, and arithmetic
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Learning disorders have the highest comorbidity with ?
ADHD
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DSM-5 classification- reading disorders
Involves deficits in the ability to discern the meaning from words in running lines of text A number of defects in processes are involved: - visual-perceptual deficits - phonological processing - phonological awareness - irregular word forms - syntax, semantic components, and working memory
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Reading disorder prevalence
4-10% of school aged children 3:1 or 4:1 (boys:girls) High comorbidity with ADHD and CD (3.5% with both) Difficulties can emerge around grade 4 Difficulties can persist into adolescence and adulthood
99
DSM-5 classification- disorder or written expression
Children will have difficulty in transcription and text generation or composition Transcription involves putting ideas into written form Deficiencies in poor handwriting, spelling, punctuation, capitalization, and word placement Handwriting: fine motor skills Spelling: draws on phonetic skills, word recognition, retrieval of learned letters, and words from memory Text generation: memory for words, understanding sentence structure, higher order executive functioning, and meta cognitive skills
100
Disorder of written expression prevalence
6-10% of school aged children
101
DSM-5 classification - mathematic abilities
Involves the understanding of numbers and the learning, representation, and retrieval of basic arithmetic facts In younger children, arithmetic abilities build on skills in understanding number, numerosity, and counting
102
Mathematic disability prevalence
5-8% of school aged children
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Learning disorders- social and emotional problems (4)
Children often have difficulties with peers (less popular and face rejection) Have fewer friends, lower quality friendships, and higher levels of loneliness Have lower social competence than peers Have difficulties identifying emotions, understanding social situations, and in social problem solving
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Learning disorders- self concept and motivation
Learning disorders associated with a lowered sense of self worth Compared to same aged peers, students with learning disorders report more helplessness, lower self esteem, even when their school grades are comparable Consequences: academic failure —> give up in the face of difficulty —> experience more failure —> reinforces belief in their lack of ability and control —> academic failure…
105
Assessments for language and learning disabilities
Need to identify discrepancy between language or learning domain and IQ Uses standardized testing - Academic standardized testing - language specific testing
106
Intellectual disability definition
Intellectual disability is characterized by significant limitations both in intellectual functioning and in adaptive behaviour as expressed in conceptual, social, and practical adaptive skills. This disability originates before age 18. -AAIDD ID is linked to the social environment and level of supports that should be assessed
107
Intellectual disability levels and approximate IQ range (4)
Mild: 50-70 Moderate: 35-50 Severe: 20-35 Profound: below 20
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Changes in the educational policy of ID have made educators focus more on _____ than _____
Focus more on functional descriptions of the child’s needs than IQ
109
Intelligence definition
Intelligence involves the knowledge possessed by a person, ability to learn or think, or the capacity to adapt to new situations. General ability, g, plus other abilities (motor, verbal)
110
Generally, intelligence is measured according to____
How individuals process information
111
Measure by IQ is ____ and _____
Stable and valid
112
Adaptive functioning definition
What people do to take care of themselves/ relate to others in daily living Conceptual skills, social skills, and practical skills
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Adaptive functioning skills - younger children
Sensorimotor behaviours Communication Self help Primary socialization skills Ex. Can they: sort cups into different sizes? Organize by colour? Zip up their own zipper? Wave? Use a spoon to feed themselves? Say hi or bye? Follow directions? Ask for help?
114
Adaptive functioning skills - older children
Reasoning/ judgements about the environment Reasoning/ judgement about social relationships Ex. Can they: determine something looks dangerous? Recognize social norms? Dress themselves? Understand they need a coat because it’s cold out?
115
Developmental outlooks of the 4 ID levels
Mild: - usually develops social and communication skills in preschool - 6th level grade by late teens - may need guidance but often successful in the community Moderate: - usually develops communication skills in early childhood - unlikely to progress beyond 2nd grade - can adapt to supervised community living Severe: - may learn to talk at school age - limited ability to profit from pre-academic training - can adapt to community living Profound: - often has a neurological condition - sensorimotor impairments - requires constant supervision
116
Rates of other physiological issues in individuals with ID…
May be 2-4x that found in the general population Most common are ADHD and ODD/CD
117
Difficulties with identifying co-occurring disorders with ID
Overshadowing Cognitive/communication impairments of ID make it difficult to identify Criteria do not apply well to lower levels of disability
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Epidemiology of ID
1-3% of the population has ID, which is greater than predicted by normal distribution Prevalence of ID depends on age and severity of ID Gender and SES also influence rates of ID
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Pathological influences of ID
Biological condition accounting for ID Genetic processes, prenatal/perinatal/postnatal issues, or a combination of factors
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Multi genetic influences of ID
At least 50% of variance in intelligence due to transmission of multiple genes More likely for mild ID
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Psychosocial influences of ID
Higher prevalence of ID in lower SES - possibly due to less access to good nutrition, which is really important for development
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ID genetic syndromes - Down syndrome
Most common (1/800-1200) Trisomy 21 - error during meiosis Reduced brain size, reduced number/density of neurons, abnormal dendrites Moderate to severe disability Deficits in short-term memory/ auditory processing, delayed speech acquisition; good visual-spatial abilities
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ID genetic syndromes - fragile X syndrome
1/4000 males, 1/6000 females Moderate to severe ID Weakness in visual-spatial cognition, sequential information processing, etc. Many boys also have ASD
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ID genetic syndromes - Williams syndrome
1/777-20000 Small deletions of several genes on chromosome 7 Mild to moderate ID Abnormal brain activation during tasks involving response inhibition, visual processing, and auditory processing of music/noise Deficits in short-term visual-spatial memory; short-term verbal memory and verbal ID typically stronger Excessively outgoing/friendly; generalized anxiety/ specific phobias
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ID genetic syndromes - Prader-Willi syndrome
1/10000-15000 Micro deletion of chromosome material - genomic imprinting Abnormal functioning of hypothalamus/serotonin Hyperphagia (eating everything) is the leading cause of death IQ often >70 Relative strengths in spatial-perceptual organization and visual processing; relative weakness in short-term motor/auditory/visual memory Skin picking, concerns with exactness/order/cleanliness/sameness in environment (highly co-morbid with OCD)
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Developmental assessments for ID (2)
Bayley scale - used in children 1-42 months - sensorimotor focus - developmental index rather than ID - identifies developmental delays Mullens scale of early development - used in children 0-48 months - expressive, receptive, gross motor, fine motor, visual reception - developmental index - assess early ID - ex. Putting different sized cups into each other, matching shapes to their places, counting blocks
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Intelligence tests for ID
Wechler scales of intelligence Stanford Binet 5th edition - fluid reasoning, general knowledge, qualitative reasoning, visual spatial processing, and working memory Leiter international performance scale 3rd edition - completely nonverbal measure of intelligence for nonverbal individuals - used in cognitively delayed, non-English speaking, hearing impaired, speech impaired, or individuals with ID - tests 3-75 years Individuals with ID typically fall below 70 for IQ (well below average)
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Adaptive functioning assessment domains (Vineland adaptive behaviour scales) (5)
Communication domain - receptive, expressive, written Daily living skills domain - personal, domestic, and community Socialization domain - interpersonal relationships, play and leisure, coping skills Motor skills domain - gross and fine motor Maladaptive behaviour domain - maladaptive behaviours index
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Dual diagnosis
Refers to an individual with a mental health disorder and a co-occurring developmental disability Approximately 1-3% of Canadians have a developmental disability, but prevalence rates of a dual diagnosis are really hard to determine Individuals with a DD often struggle to articulate symptoms related to a mental health disorder
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Treatments of IDs
No direct treatments to get rid of the disorders, but programs to help build daily living skills that may allow them to live independently
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Autism core challenges and features (3)
Communication and social interaction difficulties Subscribe to specific interests Repetitive behaviours
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Autism - additional challenges that interfere with functioning (3)
Sensory difficulties (ex. Loud noises, hates the feeling of jeans on legs) Cognitive skills (those with higher IQ have fewer autistic symptoms) Motor skills
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Prevalence rates of autism
1 in 68 children Comparable rates between socioeconomic, ethnic, and racial groups 1:4 girls to boys (but this is changing)
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Reasons for increasing prevalence of autism (4)
Awareness Widening criteria for diagnosis Diagnosis substitution Environmental factors
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Historical contest of autism (Leo Kanner and Hans Asperger)
Leo kanner: Worked in a psych ward; found that some individuals (mostly boys) had very similar major characteristics which included communication difficulties, echolalia, language difficulties, high desire for repetition, very reactive to loud noises Hans Asperger: - worked in a psych ward; observed boys that had very similar behaviours, including those above, with the differences of average intelligence and average language
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DSM-5 states that ASD is characterized by:
Persistent deficits in social communication and interactions Presence of restricted, repetitive, and stereotyped behaviours or interests
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Core diagnostic symptoms of ASD
Social communication and interaction deficits (language use, nonverbal behaviours, communicative intent) Repetitive, restrictive, and stereotyped interests Inflexible and non-functional routines and rituals Insistence on sameness Repetitive motor mannerisms
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Diagnostic criteria of ASD
Impairments must be present in early developmental period Clinically significant impairments (functional, interferes with quality of life) A global developmental delay or intellectual disability can co-occur, but not always - majority of individuals on the spectrum have average or above average IQ
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Asperger’s in the DSM-4 vs now
DSM-4: No clinically significant delay in language Higher cognitive ability Fewer presentations of core symptoms Now: No longer recognized by the DSM-5
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Psychological and behaviour characteristics of ASD (not part of diagnostic criteria) (7)
Sensory and perceptual sensitivities (hyper or hypo) - auditory, visual, smell, touch, synesthesia Intellectual challenges and strengths - 40-50% have some associated ID (IQ<70), higher IQ associated with decreased impairments Emotional and behavioural challenges - fear, aggression, hyperactivity, self-injurious behaviour High co-morbidity (70%) - ADHD, anxiety and depression, OCD, substance use, etc. Adaptive functioning skills - daily living skills, gross and fine motor skills, self-help skills, communication and social skills Splinter skills - music, mathematic, spelling; 25% with splinter skills Savant skills (autism savant syndrome) - remarkable skill/talent, exceeds typical development, 5% display savant skills
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Developmental pathway of ASD
Heterogeneous - core symptoms may improve (but not disappear) - features fluctuate - mental health challenges
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ASD prevalence
1 in 59 children 4:1 boys to girls
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ASD interventions
Pharmacological: - targets associated behavioural features, but not core features Behavioural intervention: - most supported treatment - targeted (specific deficits) or comprehensive (numerous features) Discrete trial training (DTT): - most common behavioural approach - teaches complex skills by breaking them down into smaller discrete components or skills Psychosocial interventions: - focus on improving social skills and emotional functioning
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At what age are autism symptoms often recognized?
Age 2 However, age 4-5 is the average age of diagnosis
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Neurobiological impairments in ASD
Altered brain growth, most implicated in the frontal lobe, temporal lobe-limbo system, and cerebellum