Final(new material) Flashcards

(73 cards)

1
Q

Herpes zoster

A

Shingles
-Caused by chicken pox virus(rubella zoster)
-Travels on dermatome=maculopapular vesicular rash, medical emergency in eye=blindness
-Rash does not typically cross midline, usually thoracic dermatomes

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2
Q

herpes zoster teaching prevention

A

Shingles vaccine-2 shots “shingrix” for older adults 50 and above(recombinant zoster)
-varicella vaccine (VZV) for children
A/E: local rxn at injection site, HA
Contraindications: clients who are immunocompromised, pregnancy, tx with meds that alter immune system
-contact with vesicular fluid or breathing in virus particles from blisters until they dry and scab over

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3
Q

Herpes zoster teaching

A

Rash=painful itchy blisters that scab over in 7-10 days, clearing up within 2-4 weeks
-contact with vesicular fluid or breathing in virus particles from blisters until they dry and scab over

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4
Q

Herpes zoster tx

A

-most effective in 72 hours of s/s onset
-Antivirals to accelerate lesion healing and reduce lesion production and viral shedding, decreasing acute pain severity]
-acyclovir,valacyclovir,famiciclovir

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5
Q

herpes zoster complications/manifestations

A

long-term nerve pain(postherpetic neuraliga, PHN)
Serious complications: vision loss (if rash on face), pneumonia, hearing problems, encephalitis, death
S/S: fever, HA, chills, upset stomach

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6
Q

psoriasis description

A

Dry scaly skin (plaques)
-patchy,itchy, flaky(inc cell production)
-thickened skin with silvery white scales with bilateral distribution

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7
Q

Psoriasis vulgaris

A

lifelong disorder with exacerbations/remissions
-scaling disorder r/t dermal inflammation
-abnormal growth of epidermal cells in outer skin layers
-caused by inc cell division
-autoimmune rxn/genetic predisposition

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8
Q

psoriasis tx

A

-topical steroids(betamethasone or triamcinolone)=reduce secondary inflammatory response of lesions and suppresses cellular division/proliferation
-topical tar(made from coal and trees)=suppress cellular division/prliferation and reduce inflammation
-UV light

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9
Q

actinic keratosis description

A

Premalignant lesions
-Rough, scaly patch caused by years of sun exposure
-usually affects older adults and men
-commonly on face, lips, ears, back of hands, forearms, scalp, neck

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10
Q

actinic keratosis prevention teaching

A

reduce sun exposure
-spf, hats, UV shirts, limiting exposure in peak sunshine hours
-usually removed as precaution since it can become cancerous
Tx=photodynamic therapy, freezing, tissue scraping, topical anti tumor meds, chemo, NSAIDs

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11
Q

contact dermatitis description

A

-Acute or chronic
-red inflammatory vesicular rash

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12
Q

contact dermatitis causes

A

-secondary to contact with an irritant/allergen
-Cell mediated immune rxn

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13
Q

contact dermatitis tx

A

OBTAIN HX-attempt to identify causative agent
-avoidance therapy
-steroid therapy(topical, systemic-IV,PO,IM) to suppress inflammation
-cool/moist dressings over topical steroids can increase absorption
-occlusive dressings should be avoided with steroid

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14
Q

skin cancer-ABCDE

A

asymmetry, border irregularity(well defined and shaped odd), color(dark?), diameter(>6mm), evolution(does it change?) can be elevated

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15
Q

skin cancer-basal cell

A

metastasis=rare
Chronic irritation, genetic predisposition, starts at small fleshy bumps
-basal cell layer of epidermis

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16
Q

skin cancer-squamous cell

A

Cancer of epidermis
Metastasis=common
-can be r/t chronic skin damage
-most common skin CA in persons with darker skin

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17
Q

basal cell carcinoma manifestations

A

small fleshy bump, elevated
Looks like a mole or a wart
Small, waxy nodule with superficial blood vessels and well defined borders

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18
Q

squamous cell carcinoma manifestations

A

crusty, flat breaking of the skin (like a sore that wont go away), open red and flakey
-rough, scaly lesion with central ulceration and crusting

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19
Q

tx-basal cell

A

Topical chemotherapy with iniquitous
-stimulates production of interferon , which attacks cancer cells

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20
Q

tx-squamous cell

A
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21
Q

prevention teaching-basal cell

A

annual dermatology screenings

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22
Q

prevention teaching squamous cell

A
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23
Q

malignant melanoma

A

irregular shape and borders with multiple colors
-new moles or change in an existing mole
-itching, cracks, ulcerations bleeding
-common pin upper back and lower legs; palms and soles for darker skin clients -rapid invasion and metastasis with high morbidity andmortality

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24
Q

tx for malignant melanoma

A
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25
Nursing interventions following surgical removal-malignment melanoma
26
pressure ulcers-staging
27
scabies-describe and tx
8 legged mite that burrows linearly in skin, poop causes inflammation
28
caring for EOL-hospice
29
caring for EOL- palliative
30
caring for EOL- respite care
31
Non-pharmacological interventions-EOL
32
common problems at end of life
dyspnea, constipation, fatigue,anorexia, cachexia, cough, N/V, anxiety, deliriumn
33
nursing interventions EOL-dyspnea
34
nursing interventions EOL-constipation
35
nursing interventions EOL-fatigue
36
nursing interventions EOL-anorexia
37
nursing interventions EOL-cachexia
38
nursing interventions EOL cough
39
nursing interventions EOL-N/V
40
nursing interventions EOL-anxiety
41
nursing interventions EOL-delirium
42
Advanced directives
43
end of life issues
44
signs of death
45
care of body-EOL
46
chronic pain-pharmacological interventions-long acting
47
chronic pain pharmacological interventions-short acting
48
SE of long acting pain meds
49
SE of short acting pain meds
50
routes of long acting meds-chronic pain
51
routes of short acting meds-chronic pain
52
non-pharmacological interventions-chronic pain
53
management of acute vs chronic pain: how do they differ
54
best tx for each- acute vs chronic pain
55
Main focus for HF pt
FVO -oral diuretics -fluid restriction -daily weights -low sodium diet - Beta blockers(inc CO to avoid fluid retention)
56
HF exacerbation interventions
diurese-IV -echocardiogram -telemetry -smoking cessation
57
HF medications
furosemide, ACE inhibitors, ARBs, Beta blockers, CCB, Digoxin
58
furosemide-HF
monitor K+, potassium wasting
59
Lisinopril- HF
ACE inhibitor Causes dry cough Monitor BP and for hyperkalemia
60
Losarstan-HF
ARB Lowers BP, monitor for hyperkalemia
61
Metoprolol-HF
beta blocker, lowers HR and BP Hold if HR <60bpm
62
amlodipine-HF
CCB, rhythm regulator, affects HR and BP
63
digoxin-HF
inc strength of myocardial contractions, affects HR -narrow therapeutic index=monitor levels for toxicity -toxicity= yellow halos, N/V -hold if HR<60bpm
64
A-fib
irregularly irregular Both the spacing of complexes and the QRS abnormal -missing p wave -stable vs unstable
65
a-fib risk…
clotting-> atrial appendage=stroke, PE, DVT
66
stable a-fib intervention
cardiovert with chemicals-adenosine -perfuming well, no LOC changes
67
what HR is perfuseable?
60-100, above 150 not perfuming well
68
RVR
>150bpm, not perfuming Cardiovert with vagal stimulation, adenosine IVP, amiodarone drip OR electrical hard reset mechanically
69
a-fib medications
anticoagulants, digoxin, CCB, Beta blockers, adenosine,amiodarone
70
warfarin blood monitoring
PT/INR Vit K levels (antidote)
71
Xa inhibitor
anticoagulant, golden standard bc no restrictions except bleeding precautions -Rivaroxaban, xarelto, eloquence -not great for high fall risk or high risk of injury bc of bleeding risks
72
adenosine administration needs…
fast slam IVP with flush following -telemetry, defibrillator and monitor -IV, crash cart -oxygen -rapid response/code team
73
difference between a-fib and a-flutter
has P wave and QRS is regular and evenly spaced