final - ortho Flashcards
(34 cards)
sport hx and physical
-Goal of preparticipation physical evaluation (PPE) -> promote health and safety
-Primary objectives: Screen for life-threatening or disabling conditions that may predispose to 2ndary injury/illness
-Secondary objectives: Establish a medical home, determine general health, assess fitness for specific sports, counseling on injury prevention and health related issues
-Ideal timing for exam is 6-8 weeks prior to training
sport preparticipation hx: cardiavascular hx
-Routine use of ECG and echo screening is debated
-Pertinent medical hx: Chest pain/discomfort with exercise, syncope/pre-syncope with exercise, excessive SOB or fatigue assoc with exercise, hx of heart murmur, hx of elevated BP
-Family hx: Premature death <50yo due to heart ds, disability from heart disease (< 50yo) in a close relative, knowledge of other specific cardiac conditions
-Physical: Auscultation of heart murmur in supine/standing position, palpation of radial, femoral pulses, physical stigmata of Marfan syndrome!, brachial BP in seated position
-Sudden cardiac arrest is leading cause of sudden death in young athletes
-No outcome-based studies that demonstrate effectiveness of PPE in preventing SCD in athletes
-MCC in U.S. is HCM and congenital coronary artery anomalies
-Any athletes with CVS sx require further eval before allowing sports
-Restrictions/disqualifications in consultation with cardiologist
sports preparticipation hx: hx of htn
-Any hx of HTN requires investigation for 2ndary causes and end-organ damage
-Athlete with HTN who exercises -> increases BP even more -> increases risk for complications
-Ask about stimulant use and family hx
-Athletes with pre-HTN are eligible to participate in sports (with counseling on lifestyle modifications)
-Athletes with stage I HTN in absence of end-organ damage may also participate in sports with appropriate subspecialist referral if symptomatic, has assoc heart ds/structural abnormality, or has persistent elevated BP on 2 additional occasions despite lifestyle modifications
-Athletes with stage II HTN or end-organ damage should not be cleared to participate in competitive sports until BP is evaluated, treated, and under control
sports preparticipation hx: CNS, chronic ds, surgical, mono, MSK, nutrition
-CNS: Hx of frequent exertional HA, seizure disorders, concussions/head injuries, recurrent stingers or burners, or cervical cord neuropraxia requires further eval, rehab, or informed decision-making prior to clearance
-Hx of Chronic Ds: Reactive airway disease, exercise-induced asthma, diabetes, renal ds, liver ds, chronic infections, or hematologic ds
-Surgical Hx: Full recovery with no long-term impact on athletic performance required prior to clearance
-Infectious Mono:
-Risk for splenic rupture is highest within first 3 weeks of illness and can occur in the absence of trauma
-Avoid physical activity during first 3-4 weeks after infection starts
-May return to sports once clinical symptoms are resolved/risk for splenic rupture assessed as minimal
-MSK- Limitations/Prior Injuries: Joints with limited ROM, muscle weakness
-Menstrual History: Presence of oligo- or amenorrhea (possible Female Athlete Triad/RED-S – caloric intake does not keep up with expenditure)
-Nutritional Issues- Assess RF for disordered eating or low energy availability -> May lead to persistent or recurrent injury, including bone stress injuries
-Vitamin D deficiency common in athletes
-Medication History: Rx, OTC, supplements
-Mental Health: Eating disorders, anxiety/depression screening
sports preparticipation physical
-routine vitals (including BP), cardiovascular exam, MSK exam, and other specific areas:
-Skin: Lesions (herpes, impetigo)
-Vision: Visual problems, retinal problems, eyes intact
-Abdomen: Hepatosplenomegaly
-GU: Testicular abnormalities, hernias
-Neurologic: Problems with coordination, gait, or mental processing
-Sexual Maturity: Tanner staging
sports clearance/recommendations
-Cleared for sports w/o restrictions
-Cleared for all sports w/o restrictions with recommendations for further eval or tx
-Not cleared pending further evaluation (for any sports, or for certain sports)
-Recommendations may be body system-specific
concussion
-Direct blow to body or head translates forces to brain -> transient alteration of neurologic function
-Usually not assoc with structural changes in brain detectable by imaging
-Sx evolve over first few hours
-Confusion, HA, visual disturbance, posttraumatic amnesia, balance alterations (does not have to involve LOC)
-suspected in any athlete with somatic, cognitive, or behavioral complaints
-resolve within 1-4wks
-Any suspicion -> immediately remove from play!
-Should not return same day of injury
-CT considered if deteriorating/AMS, prolonged LOC, repeated vomiting, severe HA, signs of skull fx, focal neurologic deficit, severe mechanism of injury
-CT Rarely indicated beyond first 24 hrs after injury
concussion tx
-Acute tx: Early physical and cognitive rest (1-2 days)
-Return to school with light noncontact physical activity -> early in recovery period
-Young athletes: Modified school attendance, decreased schoolwork, reduction in tech stimulation, proper nutrition/hydration, rest/sleep
-Before full return of sports: Sx resolved at rest and with exercise w/o aid of meds
-Return to play is 6-step process (each step 24 hrs):
-1. Asymptomatic at rest
-2. light aerobic exercise
-3. sport-specific exercise
-4. noncontact drills
-5. contact practice drills
-6. release to game play
-requiring medical clearance prior to return to sports (state dependent)
-Retirement from sports: Considerations include # of concussions, increasing frequency, occurrences with serially less force, prolonged/severe/permanent sx/signs
osgood schlatter disease (tibial tubercle apophysitis)
-Caused by recurrent traction on tibial tubercle apophysis
-occurs in jumping/running sports
-Fragmentation and microfractures of tibial tubercle
-occur during time of rapid growth
-pre-teen and adolescent years, -MC in boys 12-15 and girls 11-13 years
-Pain localized @ tibial tubercle -> aggravated by activities using eccentric quadriceps muscle movement
-Imaging: Fragmentation or irregular ossification of tibial tubercle
-Tx:
-Typically resolves spontaneously
-Pain control with NSAIDs
-PT and stretching the hamstrings/application of ice after workouts helpful
growing pains
-37% of school-age kids
-MC- 3-12yrs, M > F
-Deep, crampy pain in legs (not centered in joints)
-evening/nocturnal pain that awakens child from sleep
-exacerbated by activity
-dx based on hx and PE
-Tx:
-Reassurance and regular bedtime ritual of stretching and relaxation/massage, nighttime analgesic, resolution over time
torticollis
-Injury to SCM muscle during delivery or ds affecting cervical spine in infancy
-Chin rotates to side opposite of affected muscle causing head to tilt toward side of contracture
-May follow URI (swelling in upper cervical spine > rotary subluxation) or mild trauma
-Other causes: Tumors, syringomyelia, and RA
-Tx:
-Passive stretching effective in up to 97% of cases
-Surgical release of muscle origin and insertion
Head tilts toward the affected SCM (contracted side)
Chin rotates away from the affected side
scoloiosis**
-Lateral curvature of spine assoc w/ rotation of involved vertebrae and classified by anatomic location (thoracic or lumbar spine)
-4 categories: Idiopathic, congenital, neuromuscular, syndromic
-Idiopathic (80%): F > M, 10-12yo or sooner, genetic component/multifactorial etiology
-Congenital (5-7%): Vertebral abnormalities due to failure of formation or segmentation of affected vertebrae
-Typically, no back pain
-curvature of >= 30 degrees -> deformity of rib cage and asymmetry of waistline
-Lesser curves detected with Adams Forward Bend test
-Rotation of spine measured with scoliometer
-Assoc with marked rib hump and lateral curvature increases severity
-Imaging- radiographs of entire spine- standing (PA/lateral): Primary curve evident with compensatory curvature to balance body
-Tx:
-Depends on curve magnitude, skeletal maturity, and risk of progression
-dependent on Cobb angle – measured on standing PA x-ray
- < 20 degrees: No tx
- 20-40 degrees: Bracing in skeletally immature child
- > 40 degrees: Resistant to tx with bracing
-40-60 degrees: Surgical correctio -> Spinal instrumentation (rods, screws, hooks) and fusion (bone graft)
-Thoracic curvatures > 70 degrees: Poor pulmonary function
nursemaids elbow ** how to reduce it
-Subluxation of radial head as a result of being lifted or pulled by hand
-presents w/ elbow fully pronated and painful (point tenderness over radial head)
-Reduction: Palpable/audible click
-stabilize the radial head with one hand (thumb) -> Place elbow in full supination and slowly moving arm from full extension to full flexion
-Holding elbow at 90-degree angle of flexion, then slowly hyperpronating the wrist
-Following reduction, immediate relief of pain
-May immobilize in sling for comfort x 24hr (or longer, as needed)
-refractory- posterior splint and fu with ortho
growth plate fractures **
-Fx involving growth plate -> ~ 20% of all fx in skeletally immature pt
-MC- Distal radius, distal tibia, and distal fibula
-Growth plates are most susceptible to torsional and angular forces
-Described using Salter-Harris classification!!!!!!
-Type I: Transverse fracture through the physis; growth disturbance unusual
-Type II: Fracture through portion of physis and metaphysis; MC type (75%)
-Type III: Fracture through portion of physis and epiphysis; may result in complication because of intraarticular component/disruption of growing/hypertrophic zone of physis
-Type IV: Fracture through metaphysis, physis, and epiphysis; high risk of complication
-Type V: Crush injury to the physis with poor functional prognosis
-Tx:
-Types I and II: Closed reduction
-Types III and IV: Anatomic alignment for successful treatment
-Type V: Rare, often result in premature closing of the physis
torus (buckle) fracture
-very common
-kids bones bend
-wrist and ankle
-“buckling” of cortex due to compression of bone
-MC in distal radius or ulna (FOOSH injuries)
-Simple immobilization for 3wks typically sufficient (soft bandage/cast)
🧠 Mnemonic:
“Kids FOOSH → Cortex Squish”
Torus = Latin for “bulge” or “swelling”
FOOSH = Fall On OutStretched Hand → classic MOI
Cortex buckles, doesn’t break
🧱 Mental Image Hook
A kid’s bone = bendy plastic straw
Push the ends = it buckles at the middle, not breaks = torus fracture
supracondylar fx
-kid falling from monkey bar (fall from ht)
-Supracondylar region of humerus has thinner area of cortical bone -> more susceptible to injury/fx
-3-6yo
-MC elbow fx in kids
-Proximity to brachial artery in distal arm creates a potential danger
-Absence of distal pulse is a strong indicator of 2ndary arterial injury
-Swelling may be severe (usually assoc with significant amount of trauma)
-Dx- anterior and posterior fat pad (sale sign)
-Tx:
-Closed reduction with percutaneous pinning
-Complications: Volkmann ischemic contracture of the forearm due to vascular compromise and cubitus varus (“gunstock” deformity) 2ndary to poor reduction
toddler’s fx
-trampoline, learning to walk
-Oblique fx of distal tibia w/o fibular fx
-typically, no significant trauma
-1-3yo, but can be as old as 6
-Present with limping and pain with wt-bearing versus minimal pain/swelling
-Initial x-ray may not show fx -> repeat in 7-10 days (with oblique view)
-Tx: Immobilization, usually heal within 3-4wks w/o disruption in growth
osteogenesis imperfecta
-Genetic connective tissue ds characterized by multiple and recurrent fx
-1 in 12,000-15,000
-Several forms: Each assoc with mutation of different gene, varying levels of severity, and a range of characteristic features
-Severe fetal type: Multiple intrauterine or perinatal fx
-Moderately affected children: Numerous fx and exhibit dwarfism (acquired bone deformities and decreased growth velocity)
-Physical characteristics: Reduced cortical thickness in shafts of long bones, accessory skull bones, blue sclerae, thin skin, hyperextensibility of ligaments, otosclerosis with hearing loss, hypoplastic/deformed teeth
-Tx:
-Surgery: Correction of long bone deformities
-Intramedullary rods to decrease incidence of fractures and prevent deformity from fracture malunion
Individuals with osteogenesis imperfecta can’t BITE: Bones (recurrent fractures), I (“eye” = blue sclerae), Teeth (dental abnormalities), Ears (hearing loss).
Bone fractures from osteogenesis imperfecta are easily mistaken for signs of child maltreatment.
developmental dysplasia of hip **
-abnormal relationship btwn proximal femur and acetabulum
-Dislocated hip: Femoral head is not in contact with acetabulum
-Dislocatable hip: Femoral head is within acetabulum, but can be dislocated with a provocative maneuver
-Subluxatable hip: Femoral head comes partially out of joint with provocative maneuver
-Acetabular dysplasia: Insufficient acetabular development (radiographic dx)
-4 major RF: 1st born child, female, breech, and family hx
DDH: clinical manifestations **
-Clinical dx in newborns is dependent on demonstrating instability of joint by placing infant on back and obtaining complete relaxation
-Barlow sign: Adduction of hip (hip slips posteriorly) -> palpable “clunk” as head dislocates
-Ortolani sign: Hips flexed to 90 degrees, abducted from midline, attempt made to lift greater trochanter forward > feeling of “slipping” as head relocates
-Limited abduction (60 degrees) with hip at 90 degrees flexion is most sensitive sign for detecting a dysplastic hip
-Signs of instability less evident after first month of life
-Galeazzi sign: Knees at unequal heights when hips and knees flexed -> dislocated hip on side with lower knee
-Dx after walking begins:
-Painless limp/lurch to affected side
-Trendelenburg sign: Standing on affected leg > dip of pelvis evident on opposite side (weakness of gluteus medius muscle) = swaying gait
-B/L dislocation = waddling gait
DDH: imaging and tx **
-Imaging:
-US- most useful in newborns
-Radiologic evaluation more valuable after first 6 weeks of life: Lateral displacement of femoral head
-Tx:
-Dysplasia is progressive with growth unless the instability is corrected – early diagnosis and treatment is therefore essential
-Pavlik harness: Maintains reduction by placing hip in a flexed and abducted position
-If hip cannot reduce easily on exam, Pavlik harness is CI
-Closed reduction with arthrogram, followed by hip spica cast
-Still unstable with closed reduction > open reduction
avascular necrosis of proximal femur (Legg-Calve-Perthes) **
-Vascular supply to proximal femur is interrupted > necrosis of bone, followed by replacement (creeping substitution)
-Rapid growth of secondary ossification centers in epiphyses in relation to their blood supply subject them to avascular necrosis (also with trauma/infection)
-Highest incidence between 4 and 8 years of age
-Persistent pain with possible limp and/or limitation of motion
-Tx:
-Protection of joint by minimizing impact
-Nonoperative (casting) and surgical approaches to promote containment of the femoral head within the acetabulum and abduction of the hip
-Prognosis:
-Functional result following replacement of necrotic femoral head depends on the amount of deformity that has developed
-Better outcomes for < 6 years of age
-Poorer prognosis for older aged children, more completed involvement of epiphyseal center, metaphyseal defects, more complete involvement of femoral head
avascular necrosis of proximal femur (legg-calve-perthes)- imaging **
-IMAGING STAGES ARE NOT ON TEST
-Early findings: Effusion of the joint associated with slight widening of the joint space and periarticular swelling
-Few weeks: Decreased bone density in and around the joint
-Necrotic ossification center appears denser than surrounding viable structures and femoral head is collapsed or narrowed
-Fragmentation of epiphysis as replacement of necrotic ossification center occurs
-Widening of femoral head with flattening/coxa plana
-If infarction extends across growth plate > radiolucent lesion within metaphysis
-If growth center of the femoral head has been damaged and normal growth arrested > shortening of femoral neck
-Eventually, complete replacement of epiphysis
-Final shape of head depends on the extent of the necrosis and collapse of weakened bone
Early: widened joint space + soft tissue swelling
Mid: dense, fragmented femoral head
Late: flattened, widened head (coxa plana)
Severe: growth plate damage → short femoral neck
slipped capital femoral epiphysis (SCFE): dx and tx **
-Physical: Limitation of internal rotation of the hip
-Imaging: AP/lateral pelvis radiograph
-Steel sign, widening of physis on affected side, epiphysis below Klein line
-kline should go through epiphysis
-Tx:
-NWB on crutches and immediate referral to an orthopedic surgeon
-ORIF is standard of care due to high risk of AVN of femoral head associated with closed reduction
-Prognosis is guarded:
-Most patients continue to be overweight and overstress hip joints
-High rate of premature degenerative arthritis (even without AVN)
-30% of pts have B/L involvement which may occur as late as 1 or 2 years after primary episode
rf: obese, AA; needs ORIF
\Obese adolescent with hip/knee pain + limited internal rotation
Get AP + frog-leg lateral pelvis X-rays
Look for Klein’s line missing the epiphysis
Tx = urgent ortho + in situ pinning