Gallbladder Flashcards

(50 cards)

1
Q

Impacted stone in the cystic duct, cystic duct remnant or gallbladder neck

A

Mirizzi syndrome

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2
Q

Inflammation around stone may cause partial mechanical obstruction of the CHD

A

Mirizzi syndrome

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3
Q

RUQ pain
Jaundice
Recurrent cholangitis
Cholangitic cirrhosis

A

Clinical presentation of Mirizzi syndrome

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4
Q

Sonographic finding of Mirizzi Syndrome

A

Dilatation of CHD and intrahepatic ducts above impacted stone w/ normal CBD

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5
Q

GB considered contracted if measures:

A

Less than 2 cm in diameter after appropriate fasting

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6
Q

Non visualization of GB

A

In 15-25% of PTs with cholelithiasis

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7
Q

“Double arc” sign

A

WES sign

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8
Q

WES triad

A

Wall
Echo
Shadow

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9
Q

Chronic cholecystitis

A

Gallbladder wall fibrosis due to recurrent episodes of acute cholecystitis

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10
Q

Lab values w/ chronic cholecystitis

A
May be elevated: 
AST
ALT
ALP
direct bilirubin
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11
Q

Sonographic findings of Chronic Cholecystitis

A

Contracted GB w/ shadowing from stones
Hyperechoic wall 4-5mm
May see sludge
Decreased response after CCK injection

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12
Q

Cholecystomegaly AKA

A

Hydropic GB

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13
Q

Hydropic GB

A

GB distention w/o wall thickening
Prolonged total obstruction of cystic duct
GB filled with mucous secretions from wall

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14
Q

Difference between Hydropic GB and courvousier’s GB

A

Courvoisier’s due to obstruction distal to cystic duct

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15
Q

Risk factors for cholecystomegaly

A
Obstruction of cystic duct/GB neck
Kawasakis disease
Scarlet fever
Recent surgery
Prolonged biliary stasis
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16
Q

Sonographic findings of Hydropic GB

A

TRV/AP > 4cm
Thin walls
Evaluate for Mirizzi syndrome

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17
Q

Courvoisier GB

A

GB distention w/o wall thickening due to obstruction outside of GB

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18
Q

Causes of Courvoisier GB

A

Pancreatic head mass
Duodenal papilla mass
Ampulla of Vater mass
CBD mass

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19
Q

Sonographic findings of Courvoisier GB

A

Same as Hydropic GB

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20
Q

Porcelain GB

A

Complete/patchy calcification of GB wall

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21
Q

Incidence of porcelain GB

A

In 0.6-0.8% of PTs

More frequent in females (5:1)

22
Q

Risk factors for porcelain GB

A

Cholelithiasis

23
Q

Sonographic findings of Porcelain GB

A

May appear as stone filled but lacks WES sign

24
Q

Adenoma

A

Benign epithelial tumor

Localized overgrowth of epithelial lining

25
Adenoma incidence
<5% of all GB polyps
26
Risk factors for Adenoma
Cholelithiasis | Chronic cholecystitis
27
Sonographic findings of Adenoma
``` Solitary <1 cm in diameter Homogeneous, hyperechoic immobile mass DO NOT shadow or move Usually near fundus Thickening of GB wall near polyp increases malignancy suspicion ```
28
Adenomyomatosis AKA
Diverticulosis of GB
29
A form of hyperplastic cholecystosis
Adenomyomatosis
30
Adenomyomatosis occurs where in most cases?
Almost exclusively in fundus
31
Sonographic findings of Adenomyomatosis
Thickening of GB wall Intraluminal diverticula may be seen Comet tail artifacts in B mode Twinkle artifact w/ color Doppler
32
Small cholesterol stones/crystals lodged in Rokitansky-Aschoff sinuses
Adenomyomatosis
33
Degenerative or proliferation changes due to deposits of cholesterol in GB or mucosal membranes
Cholesterolosis | AKA hyperplastic cholecystosis
34
Focal form of cholesterolosis
Cholesterol polyps
35
Strawberry GB
Diffuse form of cholesterolosis
36
How big do polyps usually measure with cholesterolosis?
2-10 mm
37
Strawberry GB is diagnosed with sonography. | T/F
False
38
Primary GB carcinoma
Malignant neoplasm of GB wall
39
Represents 98% of neoplasms in GB
Adenocarcinoma
40
Incidence of primary GB carcinoma
Most common biliary malignancy Most in PTs >50 years More common in Native Americans and Hispanic Americans w/cholelithiasis More common in females (3:1)
41
Risk factors for Primary GB carcinoma
``` Obesity Smoking Female Chronic salmonella typhi infection Exposure to industrial chemicals Cholelithiasis Chronic cholecystitis Porcelain GB Polyp >2 cm Primary sclerosis cholangitis Congenital biliary anomalies IBD ```
42
Most common pattern of disease with Primary GB carcinoma
Mass from GB fossa replacing GB and invading adjacent liver
43
Patterns of GB carcinoma
Irregular GB wall thickening Polyploid intraluminal lesions w/ irregular borders “Trapped stone” Tumors in infundibular/fundal region May grow into cystic duct and porta hepatis mimicking cholangiocarcinoma
44
Direct extension Mets commonly arise from:
Cancers of stomach, pancreas, and bile ducts
45
Hematogenous and lymphatic Mets
Lung Kidney Esophagus Malignant melanoma
46
Differential of Mets from primary GB carcinoma
Cholelithiasis usually absent
47
Non-inflammatory causes of GB: Diffuse
``` Normal contracted GB Hypoalbuminemia-hypoproteinemia Ascites Acute hepatitis CHF Renal disease AIDS Pancreatitis Cirrhosis Sepsis ```
48
Non-inflammatory causes of GB wall thickening: | Focal
``` Adenomyomatosis Adenomatous polyp Cholesterol polyp Papillary Adenomas Primary GB carcinoma Mets ```
49
Pseudo-wall thickening
TGC too high Beam average artifact Tumefactive sludge
50
Thickened GB wall
>3 mm