GDV Flashcards

1
Q

Describe acute gastric dilatation

A
  • stomach is in the normal position but very distended

- usually associated with over-eating

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Describe chronic gastric volvulus

A
  • partial turn or misplacement of pylorus
  • causes decreased ability of eructation or increased gastric retention
  • gastropexy to fix
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Describe the pathophysiology of GDV

A
  • stomach fills with gas or fluid, which alters sphincter position
  • pylorus moves to the left as the rest of the stomach moves to the right
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What are the cardiovascular effects of GDV?

A
  • compression of veins (decreased BP)
  • secretion of catecholamines (vasoconstriction)
  • arrhythmias
  • reperfusion injury
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

What are the respiratory effects of GDV?

A
  • distension puts pressure on diaphragm

- decreased excursions results in accumulation of CO2 (acidosis)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What are the GIT effects of GDV?

A
  • reduced blood supply to mucosa of stomach results in sloughing off and necrosis
  • bacterial translocation and septicemia
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What are the metabolic effects of GDV?

A
  • poor tissue perfusion
  • cellular hypoxia
  • anaerobic metabolism
  • increased lactate
  • metabolic acidosis
  • liver/kidney buffer
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

What are the immune effects of GDV?

A
  • hypoxemia causing mucosal ischemia
  • loss of protective barrier
  • bacterial translocation
  • damage to lymphatics
  • portal hypertension
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What are the renal effects of GDV?

A
  • vasoconstriction results in decreased GFR
  • decreased urine (oliguria)
  • acute renal failure
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What is the typical history associated with GDV?

A
  • looking/biting at abdomen
  • “praying” position
  • non-productive retching
  • distended abdomen
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What are the physical findings of GDV?

A
  • distended, painful, tympanic abdomen
  • active retching
  • hypersalivation
  • tachypnea, tachycardia
  • collapse
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What are the laboratory findings associated with GDV?

A
  • increased WBC
  • increased ALT, bilirubin, BUN/Cr
  • hypokalemia
  • hypoglycemia
  • increased lactate
  • DIC
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Which radiographic view is diagnostic for GDV?

A

right lateral

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

What are the treatment options for GDV?

A
  • fluids (shock doses)
  • decompression
  • pain management
  • antimicrobials and radical scavengers
  • surgery
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What are the methods of gastric decompression?

A
  • orogastric tube
  • trocharization
  • emergency gastrostomy
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

What are the disadvantages of trocharization?

A
  • will not decompress stomach as well as tube
  • risk of lacerating stomach
  • can push needle into another organ
17
Q

Which type of drug is contraindicated in GDV patients?

A

glucocorticosteroids

18
Q

Describe gastric de-rotation

A
  • check position and palpate esophagus
  • push stomach dorsal and to the left
  • pull pylorus to the right
  • pass stomach tube
19
Q

How do you check for gastric wall viability?

A
  • presence of peristalsis
  • pink/red serosal color
  • palpate for thinning or friability
  • pulsation of vessels
  • bleeding of cut surfaces
20
Q

What does gastric necrosis normally occur in GDV?

A

along greater curvature of the stomach

21
Q

What are the techniques for a partial gastrectomy?

A
  • cut and sew
  • stapling
  • partial invagination
22
Q

Describe gastric invagination

A
  • necrotic section sloughs off and is digested
  • risk of gastric ulceration
  • can obstruct gastric outflow
23
Q

How is splenic viability evaluation?

A
  • venous congestion
  • vessel thrombosis
  • splenic torsion
24
Q

Describe incisional gastropexy

A
  • incision on right ventro-lateral wall
  • roll the wall over and make incision through peritoneal and muscular layers
  • lay pyloric antrum against this area and suture incisions together
25
Describe belt loop gastropexy
- parallel incisions through peritoneal and muscular layer to create tunnel - create belt/flap off pyloric antrum - tie suture to end of flap and feed through the loop, then reattach to stomach
26
Describe circumcostal gastropexy
- make tunnel around last rib - create belt/flap off pyloric antrum - feed flap around the rib
27
Describe tube gastropexy/gastrostomy
- stab incision into stomach and pass catheter - tighten purse string around catheter - other end comes out abdomen - place multiple mattress sutures - secure tube with finger trap
28
Describe incorporating gastropexy
- stomach wall is incorporated in linea alba incision | - not recommended
29
Describe laparoscopic-assisted gastropexy
- dorsal recumbency - pull stomach up to abdominal wall - make incision into abdominal cavity and exteriorize that section of the stomach - do an incisional gastropexy
30
Describe endoscopic-assisted gastropexy
- place scope in esophagus and stomach - use tip of scope to push antrum into right abdominal wall - drive large suture through skin to hold stomach - make incision into stomach adjacent to suture
31
What are the causes for post-op death?
- shock - gastric necrosis - reperfusion injury - arrhythmias
32
What is done for post-op care?
- NPO for 24 hours - continue fluid for 24 hours - correct hypoK, hypoCl and metabolic acidosis - H2 blockers - coating agents - analgesics
33
When is treatment of VPCs indicated?
- associated with weakness or syncope - persistent tachycardia - pulse deficits or poor pulse quality - multifocal VPCs
34
Which drug can be used to help reverse VPCs?
Lidocaine
35
What is the prognosis for GDV?
- a leading cause of death in large breeds - 10-33% mortality rate - recumbent patients have higher mortality - lacate levels > 6 mm/L indicate higher incidence of gastric necrosis