GI Flashcards

(49 cards)

1
Q

Pediatric differences in GI system

A

Duodenum- digestion takes place

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2
Q

Enzymes that aid in digestion

A

Amylase- saliva; digests carbs
Lipase- enhances fat absorption
Trypsin- breaks down protein into polypeptides & some amino acids

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3
Q

Signs/ symptoms of GI disorders in infants/ children

A

GI sx immature at birth
Liver function immature at birth & next few weeks
GI structures in 2nd year of life more mature

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4
Q

Review of GI system

A

Stomach- secretes HCl & digestive enzymes to break down fats (gastric lipase) & proteins (pepsin)- little absorption
Small intestine- digestion is completed here by pancreatic enzymes, bile & small intestine enzymes
Large intestine- major fxn is water absorption
Liver- bile production, detoxification, vitamin storage
Gallbladder- stores bile

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5
Q

GI assessment

A

Subjective- lifestyle/ family factors (including family hx)
Diet- gaining weight; through h/o feeding pattern, any problems w/ breastfeeding
Allergies- lactose intolerance, celiac
Elimination patterns- I’s/O’s, encopresis, constipation
Mental status- ask parents/ caregiver
Auscultation
Percussion/ palpation- rebound tenderness

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6
Q

Physiologic differences

A

Minimal saliva
Decreased stomach capacity; newborn- 10-20ml, 1 yo- 210-260ml, adolescent- 1500ml
Reverse peristalsis- leads to reflux
Increased gastric emptying time
Lg. intestine relatively short, less reabsorption of water, stools softer w/ greater water loss during diarrhea- high risk of dehydration

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7
Q

nursing implications

A

dehydration- assess I & O, fluid replacement
malnutrition/ poor growth- assess growth, feeding methods, nutritional intake, quality of stools, other s/s
inflammation/ infection- monitor for fever, pain; assess abdomen/ bowel sounds, stool

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8
Q

cleft lip/ palate

A
congenital malformation (failure of maxillary processes to fuse)- during 6-12 wks gestation
may appear by itself/ together/ varying severity
most common craniofacial deformities in US
multifactorial causes
key is to do surgery before speech begins
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9
Q

cleft lip/ palate

A

abnormal opening in lip, palate, nasal cavity
multifactorial inheritance pattern (teratogenic, viral, seizure meds during wks 9-12)
failure of foregut to close during fetal development
1st sign may be formula coming from the nose
surgical correction- done early to stimulate pleasure when sucking
lip repair- usually by 12 wks
palate- palatoplasty; usually btwn 6-24 mos to maximize speech
complications- cosmetic, speech/ hearing, feeding, orthodontic, otolaryngology

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10
Q

cleft lip

A

opening btwn nose & lip
apparent @ birth
should be documented during newborn assessment
assess child’s ability to suck & swallow
cleft lip repair is performed during 1st month of life
special feeding techniques if surgery is delayed

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11
Q

cleft lip/ palate pre-op nursing care

A

feeding- upright position to prevent aspiration; breastfeeding if possible; Haberman nipple- longer/ crosscut nipple w/ compressible bottle; frequent burping
facilitate bonding
growth & monitoring
logan bar- protects surgical repair

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12
Q

cleft lip

A

surgery- usually done early (1st few days to 1st month of life)- improve parental bonding/ feeding
closure of lip defect precedes correction of palate
z-plasty to minimize retraction of scar
protect suture line w/ Logan bow/ other methods
plastic surgery- staggered suture line (often in shape of letter Z to minimize scarring
Logan bar used post-op

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13
Q

cleft palate

A

repaired surgically btwn 12-18 months prior to talking
parents will care for child @ home until surgical repair
altered dentition/ speech dysfunction may occur
frequent episodes of otitis media before being fixed

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14
Q

cleft lip/ palate post-op nursing

A

restraint- no-no for at least 6-8 days (longer w/ palate repair); remove 1 at a time q 2 hrs for 15 min; swaddling for babies
feeding- no straws, pacifiers, spoons, fingers by mouth for 7-10 days; use shorter nipple; can feed w/ side of spoon for older children; advance diet as tolerated
suture care- lip protective device (logan bar) to prevent tension on suture site; no brushing teeth for 1-2 wks (clean suture line w/ water after each feed); don’t place on stomach; no oral temps/ tongue depressors
referrals- hearing, speech, orthodontic

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15
Q

nursing management

A

prevent injury to suture line
promote adequate nutrition
encouraging infant-parent bonding
providing emotional support

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16
Q

esophageal atresia/ tracheoesophageal fistula (TEF)

A

congenital malformations in which esophagus terminates before it reaches stomach &/ or a fistula is present that forms an unnatural connection w/ trachea
foregut fails to lengthen, separate, fuse into 2 parallel tubes (@ 4-5 wks gestation)
assoc. w/ maternal polyhydramnios (fetus can’t swallow/ absorb amniotic fluid in utero)
over half have other abnormalities
*medical emergencies

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17
Q

EA/ TEF

A

manifestations- 3 C’s- coughing, choking, cyanosis
excessive oral secretions, choking, resp. distress
NG tube can’t be passed into stomach
child needs NG tube bc unable to tolerate own oral secretions which will land in blind pouch
diagnostic eval- prenatal
treatment- emergency actions to prevent pneumonia
teaching- gastrostomy tube feedings, skin/ stoma care, nutritive sucking; oral hypersensitivity/ food aversion

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18
Q

nursing management of TEF

A
pre/ post-surgical interventions:
initiate NPO
elevate HOB
monitor hydration
assess/ maintain orogastric tube/ prevent aspiration
O2/ suction equipment available
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19
Q

pyloric stenosis

A

hypertrophy of circular muscle that surrounds pylorus causing obstruction of gastric emptying

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20
Q

hypertropic pyloric stenosis

A

unknown cause
manifestations- projectile vomiting 30-60 min after feeding @ about 2 wks of age; movable, palpable, olive-shaped mass; dehydration w/ metabolic alkalosis
treatment- pyloromyotomy- incision of pyloric muscle to release obstruction around pyloric sphincter

21
Q

pyloric stenosis

A

pre-op nursing interventions- assess for dehydration/ acid-base imbalance; examine abdomen/ listen for bowel sounds; I’s & O’s, NG tube drainage; oral care; treat dehydration/ electrolyte imbalance prior to surgery
post-op nursing interventions- feeding according to surgeon protocol; usually pedialyte after 4-6 hrs, advance to full strength breast milk/ formula
teach parents signs of problems- abdominal distention, excessive vomiting, redness/ purulent drainage @ surgical site

22
Q

abdominal wall defects- gastroschisis/ omphalocele

A

congenital defect of ventral abdominal wall- herniation of abdominal viscera outside abdominal wall
gastroschisis- occurs to side (usually right) of umbilicus
omphalocele- through umbilical cord
week 11 of gestation
multifactorial causes

23
Q

omphalocele

A

protrusion of abdominal contents through abdominal wall @ junction of umbilical cord
may include intestines, stomach, & liver
organs covered by thin transparent layer/ membrane of amnion
size of sack depends on extent of protrusion of abdominal contents through umbilical cord

24
Q

gastroschesis

A

similar to omphalocele- except defect is far from umbilicus (not on it)/ organs not contained by membrane

25
management of omphalocele & gastroschesis
bowel returned to abdomen if defect is large- staged repair done w/ silastic pouch/ "silo" TPN dependent potential for infection
26
omphalocele & gastroschesis (newborn)
both are congenital anomalies of anterior abdominal wall omphalocele- membranous sac covers exposed organs gastroschesis- organs not covered by membrane nursing management- minimize fluid loss, protect exposed abdominal contents from trauma/ infection, postop care, surgical repair of both defects occur after birth/ are done in stages
27
intussusception
telescoping/ invagination of 1 portion of intestine into another- causes obstruction decreases blood flow to that area- causes ischemia/ pain, WBC may form pus/ bleeding occasionally d/t intestinal lesions, but often cause is unknown severe pain lethargy "currant-jelly" stools, gross blood sudden onset of intermittent crampy abdominal pain most common cause of intestinal obstruction in children <2 manifestations- sudden onset of pain, draw up legs, vomit; blood in stool; sausage-shaped abd. mass may be palpated; extreme paroxysmal pain (subsides then recurs); assess for shock treatment- US-guided saline, barium, air enema to reduce if no response- surgical emergency to avoid bowel necrosis laparoscopy- post-op treatment; gradually increase feeding after NG removal high risk of recurrence- have to be hospitalized for 24 hrs after
28
malrotation & volvulus
``` twisting of intestine obstruction of feces occurs d/t intestinal malrotation manifestations- pain, vomiting, signs of obstruction surgical emergency nursing care similar to intussusception ```
29
Hirschsprung's disease
absence of ganglion cells in rectum & upward to colon- inadequate motility in part of intestine absence of ganglionic innervation leading to no persistalsis waves mechanical obstruction from inadequate motility of intestine incidence- 1 in 5000; more common in males & DS delayed passage of meconium- signs of obstruction ribbon-like stool (passing through small narrow segments) diagnosis- by rectal biopsy treatment- medical- stool softeners surgical removal of aganglionic segment w/ or w/out colostomy assess for enterocolitis (acute illness); completel bowel cleansing (enema/ GoLytely); bowel sterilization, IV abx pain management, I&O, VS, colostomy care, enterostomal therapy temporary ostomy/ "pull-through" procedure
30
anorectal malformations
imperforated anus identified in newborn period level of defect--> outcome of care staged repair for corrective surgery w/ ostomy major challenge- finding adequate nerve & muscle structures for normal evacuation associated anomalies absence/ stenosis of rectal cancal stool in urine d/t fistula to perineum stool in vagina d/t fistula DX- digital rectal exam, ultrasound, abd x-rays, CT scans treatment- low lesions w/ anal membrane- serial rectal digital dilations are effective higher lesions- 2-stage surgical repair
31
anal stenosis
thickened/ constricted anal wall | characteristic ribbon-like stool
32
anal atresia (imperforate anus)
PE reveals absent anal opening | failure to pass meconium during 1st 24 hrs also diagnostic
33
meckel's diverticulum
pouch on wall of lower part of intestine (congenital) diverticulum may contain tissue that's identical to tissue of stomach/ pancreas digested food can cause diverticulitis remant of fetal duct (fiberous band connected from sm. intestine to umbilicus) *most common congenital malformation of GI tract surgery to remove diverticulum to prevent complications- ulceration, bleeding, intussuception, intestinal obstruction
34
umbilical hernia
hernia- protrusion/ projection of organ/ part of organ through muscle wall of cavity that normally contains it imperfect closure of umbilical muscle ring danger of incarceration/ strangulation protrusion of intestines through umbilicus shouldn't be hard, should be reducable should hear bowel sounds most spontaneously resolve by 3-4 yrs as muscular ring closes surgery if s/x of strangulation, increased protrusion >2 yrs of age/ no improvement in large defect after 4 yrs
35
appendicitis
inflammation of appendix caused by obstruction | dull steady umbilical pain that localizes to RLQ at mcburney's pt
36
nursing considerations for constipation
``` administer enema hx of bowel patterns, medications, diet educate parents/ child dietary modifications *common in school-aged children ```
37
constipation/ encopresis
constipation- failure to achieve complete evacuation of lower colon encopresis- soiling of fecal contents into underwear beyond age of expected toilet training (4-5 yrs) may be 2ndary to other disorders- Hirschsprung, toilet refusal, bathroom accessibility, SCI, med side effects chronic constipation- envt/ psychosocial factors (stress) clean out w/ enema/ laxative, ensure hydration/ regular bowel regimen
38
vomiting
``` common in childhood prevention of complications from loss of fluid antiemetic meds observe & record *metabolic alkalosis ```
39
diarrhea
leading cause of illness in children <5 acute infectious diarrhea- variety of causative organisms 20% of deaths in developing countries r/t diarrhea/ dehydration types- acute, acute infectious gastroenteritis, chronic, intractable diarrhea of infancy, chronic nonspecific diarrhea metabolic acidosis etiology- rotavirus, salmonella, shigella, campylobacter organisms, giardia, cryptosporidium, clostridium difficile, abx therapy
40
dehydration
health hx- diarrhea, vomiting, decreased oral intake, sustained high fever diagnostic evaluation- wt. is most important determinant of percent of total body fluid loss in infants/ children VS, skin turgor, fontanels, mucous membranes, eyes, mental status, urinary output oral rehydration therapy nursing considerations- weight, I&O
41
oral rehydration
rehydration solution of 75-90 mEq of Na+/ L give 40-50ml/ kg over 1st 4 hrs maintain hydration w/ solution of 40-60 mEq Na+/ L daily volume of maintenance hydration <150 ml/ kg/ day
42
formula for fluid maintenance
100 ml/kg for 1st 10 kg 50 ml/kg for next 10 kg 20 ml/kg for remaining kg add together for total ml needed per 24 hr period divide by 24 for ml/ hr fluid requirement
43
oral candidiasis (thrush)
fungal infection of oral mucosa children @ risk- immune disorders (corticosteroid inhaler use, chemotherapy, abx use), transmitted btwn infant & breastfeeding mother therapeutic management w/ nystatin/ fluconazole
44
appendicitis
obstruction of fecal matter, swollen lymphoid tissue, or parasite periumbilical pain w/ nausea, RLQ pain, & vomiting w/ fever ruptured appendix N/V, diarrhea, constipation WBC- 15-20, anorexia, fever, chills appendectomy IVF/ IV antibiotics pain management *watch for s/s of perforation- fecal material spills into abdomen causing peritonitis
45
GER/ GERD
vomiting vs "spitting up" overflowing (regurgitation) vs projectile esophagus is shorter distance to stomach, stomach contents come back up much easier GER- backward flow of stomach contents up into esophagus/ mouth; happens to everyone; in babies- small amount is normal & almost always goes away by the time child is 18 mos old GERD- when complications from GER arise- failure to gain wt, bleeding, resp. problems, cough, esophagitis *eg. after feeding, baby sticks back out immaturity of cardiac sphincter (LES) causes reflux of stomach contents into esophagus manifestations- regurgitation, fussiness 30-60 min after meal, poor growth 45-50% of healthy infants <2 mos have GERD, peaks @ 4 mos, then decreases when LES matures; resolves by 1 yo
46
GERD treatment
nursing considerations- identify children, educate parents in home care, feeding, positioning, meds non-pharmacologic tx- smaller feedings, thicken formula, keep baby upright before/ after feeding, burping, if bottle feeding- find nipple that makes good seal to prevent air into mouth prokinetics- increase motility, usually w/ meds that inhibit acid antacids may be tried 1st in school-aged children 2 major therapies- H2 blockers (-ine)/ PPIs (-zole) 1st line is H2, then PPI
47
lactose intolerance
malabsorptive disorder absence/ deficiency of lactase--> inability to digest lactose more common in Asians, Native Americans, African Americans frothy diarrhea w/ presence of reducing substances, abd. pain, & distention treatment is avoidance of milk-based products, soy formula, elimination of milk-based products for breastfeeding mothers, use of Lact-Aid calcium/ vit. D supplementation
48
short bowel syndrome (SBS)
malabsorptive disorder decreased mucosal surface area, usually d/t small bowel resection prognosis has advanced w/ TPN & nutrition administer/ monitor nutritional therapy
49
celiac disease
AKA gluten-induced enteropathy & celiac sprue immunologic disorder in which gluten causes damage to small intestine 4 characteristics: steatorrhea general malnutrition abd. distention secondary vitamin deficiencies