GI Bleed Flashcards

(43 cards)

1
Q

Upper Gastrointestinal Bleeding: Esophageal Origin

A

chronic esophagitis-GERD, irritating medications, alcohol, cigarettes, esophageal varices

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2
Q

Upper Gastrointestinal Bleeding: Stomach and Duodenal Origin

A

Bleeding peptic ulcers caused by: NSAIDS, ASA, corticosteroids (irritate and disrupt mucosal barrier

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3
Q

melena

A

Black tarry stools, indicative of an upper GI bleed

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4
Q

small amounts of blood in gastric secretions

A

occult bleeding

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5
Q

Diagnostics for a GI Bleed?

A
  • Diagnosed by Endoscopy (EGD)
  • Angiography
  • CBC-low H/H
  • BUN-elevated
  • Electrolytes-loss of electrolytes through emesis/stool
  • PT-increased risk for bleeding
  • Liver enzymes
  • ABG - monitor resp status
  • Blood type/cross match-blood type
  • Occult bleeding-assess stool/emesis
  • UA-looking for dehydration
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6
Q

Liver failure increases the risk of _________ (GI related).

A

esophageal varices

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7
Q

Why would the BUN be elevated in a GI bleed?

A

due to blood proteins broken down by GI tract

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8
Q

GI bleeds are diagnosed by?

A

Endoscopy (EGD)

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9
Q

Nursing Assessments for a GI Bleed?

A
  • Monitor VS q15-30 min
  • Emphasis on ABC’s & early identification of Shock
  • Assess LOC, BP, HR, peripheral pulses and perfusion, presence or absence of neck vein distension, abdominal assessment
  • Frequent Resp, CV, GI, I&O assessments
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10
Q

GI BLEED Treatment

A
  • Patient is NPO
  • Hydrate (Insert 2 large bore IV’s)
    • Isotonic crystalloid fluids (LR)
  • Blood transfusions
    • Watch for fluid overload
    • Monitor urine output
  • IV infusion or IV push Proton Pump Inhibitor (Protonix)
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11
Q

1st line tx for GI bleed?

A

Endoscopic hemostasis - hopefully within 24hrs of the bleed

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12
Q

Nursing Interventions: Teaching for GI Bleed

A
  • Smoking cessation
  • Avoid alcohol, stress, OTC drugs
  • Medication adherence
  • S/S of upper and lower GI bleed
  • Side effects of medications (ASA, NSAIDS, COUMADIN)
    • Potential for bleeding even with low dose aspirin taken daily
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13
Q

Inflammation and breakdown of the normal gastric mucosal barrier by HCL acid and pepsin

A

Gastritis

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14
Q

Gastritis Caused by?

A
  • Drugs-ASA, NSAIDS
  • Diet-alcohol, spicy
  • Microorganisms-H-Pylori
  • Environmental-Smoking
  • Pathophysiologic
    • hiatal hernia, stress
  • Other- NG tube, EGD
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15
Q

S/S of Acute Gastritis?

A
  • anorexia
  • N/V
  • epigastric tenderness
  • feelings of fullness
  • hemorrhage is associated with alcohol and may be only symptom
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16
Q

Gastritis is diagnosed by ?

A

endoscopy (EGD) with biopsy

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17
Q

Treatment of Gastritis?

A

NPO, IVF, Possible NG Tube, PPI

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18
Q

Nursing Interventions: Teaching for Gastritis?

A
  • diet modification (Individualize)
  • Decrease alcohol intake
  • Smoking cessation
  • Tx H-Pylori,
19
Q

Erosion of the GI mucosa from the digestive action of HCL acid and pepsin

A

Peptic Ulcer Disease (PUD)

20
Q

Peptic Ulcer Disease (PUD) is caused by?

A
  • H. Pylori
  • alcohol
  • nicotine
  • stress
  • ASA & NSAIDS
21
Q

Manifestations of Duodenal Ulcers?

A
  • Mid-epigastric pain may radiate to back
  • Burning or cramping 2-5 hours after eating –relief obtained with antacids and food
  • More common in men esp. 35 -45 age group
22
Q

Manifestations Gastric Ulcers

A

High epigastric dyspepsia occurring 1-2 hours after eating
Food aggrevated deep ulcers
More prevalent in women & older adults

Higher mortality rate

23
Q

Duodenal Ulcer Risk factors

A
  • H. pylori
  • alcohol
  • smoking
  • COPD
  • Chronic Renal Failure
24
Q

Gastric Ulcer Risk factors

A

H. Pylori, bile reflux, smoking, medications

25
Peptic Ulcer Disease is diagnosed by?
Endoscopy (EGD) with biopsy
26
Diagnostics for Peptic Ulcer Disease?
- Barium Contrast Study - Lab test - CBC - H/H - Similar to GI bleed if hemorrhage or perforation present - WBC - Liver enzymes - Amylase - Stool Studies
27
Nursing Interventions for Peptic Ulcer Disease?
- Advance diet as tolerated - start with clear liquids - Teach: - Avoid foods that cause gastric distress (spicy, acidic) - Alcohol and smoking cessation - Side effects of OTC medications - Drug therapy - Decrease stress - S/S of recurrence
28
Drug therapy for Peptic Ulcer Disease?
- PPI or H2 receptor blockers, - antacids - antibiotics if H. Pylori present
29
Complications of Peptic Ulcer Disease?
- Hemorrhage (most common) - Vomiting of blood, change in VS - Perforation (most lethal- may cause infection) - Gastric outlet obstruction
30
Signs of perforation of a Peptic Ulcer?
- Sudden severe pain, - rigid abdomen - found with knees drawn in - grunting respirations
31
Signs of a Gastric outlet obstruction with a Peptic Ucler?
N/V, abdominal distention despite having NG tube
32
Treatment of PUD Complications: hemorrhage?
- NPO - IVF - Hemorrhage (if present) - Stop Bleeding - Restore blood volume
33
Treatment of PUD Complications - Perforation (if present)?
- NPO - IVF - Stop bleeding - Surgical intervention - Restore blood volume - Antibiotics-delay OR to get antibiotics started
34
Treatment of PUD Complication - Gastric outlet obstruction (if present)?
- NPO - IVF - NG to decompress stomach - Fluid and electrolytes
35
partial removal of the lower 2/3 of the stomach
Gastrectomy
36
severing of the vagus nerve
Vagotomy
37
englargement of the pyloric sphincter
Pyloroplasty
38
Vagotomy
- severing of the vagus nerve | - Decreases gastric acid secretion
39
Gastrectomy
partial removal of the lower 2/3 of the stomach
40
Pyloroplasty
englargement of the pyloric sphincter
41
- Mid-epigastric pain may radiate to back - Burning or cramping 2-5 hours after eating –relief obtained with antacids and food - More common in men esp. 35 -45 age group
S/S of a duodenal ulcer
42
- Sudden severe pain, rigid abdomen, found with knees drawn in, grunting respirations
signs of perforation of a Peptic Ulcer
43
- High epigastric dyspepsia occurring 1-2 hours after eating - Food aggrevated deep ulcers - More prevalent in women & older adults - Higher mortality rate
Manifestations Gastric Ulcers