Gingival and periodontal health in children Flashcards

(45 cards)

1
Q

children with teeth with the condition of amelogensis imperfecta

A

sensitive teeth
rough/little enamel
hard to brush teeth therefore calculus builds

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2
Q

consqeunes of children fed by a gastrostomy tube

A

may not have a swallow reflex
not generating as much saliva as they are not eating
therefore can develop extensive calculus

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3
Q

which side is gngitivus less prevalent in

A

LHS compared to RHS

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4
Q

periodontal screeing for children under 18 years

A
Code 0 ¬ healthy
Code 1 ¬ bleeding on gentle probing
Code 2 ¬ calculus present and/or plaque retention factors
Code 3 ¬ 4-5mm pocket
Code 4 ¬ 6mm or more pocket
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5
Q

what is Code *

A

furcation

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6
Q

what are the codes used in those 7-11 years

A

0-2

may be false pocketing as the teeth erupt

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7
Q

codes used in those 12-17 years

A

0-4

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8
Q

gingival disorders in children

A

chronic gingivitis
gingival hyperplasia
traumatic lesions
acute gingivitis

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9
Q

chronic gingivitus

A

common
plaque indced
reversible with improved OH

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10
Q

symptoms of chronic gingivitus

A
painless 
red
swelling of gingiva
no loss of function
gums bleeding due to not brushing enoug
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11
Q

what is the bacteria flora in chronic gingitus in childre

A

flora similar to those in adults
gram+ rods and cocci
gram - anaerobic rods and spirochetes

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12
Q

what can make chronic gingivitis worse

A

exfoliating teeth
malocclusion
presence of orthodontic applicances

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13
Q

challenged to toothbrushing

A

reteroclined teeth in cleft like and palate patients
abnormalities of tooth morphology
orthodontic applicances
sensitive teeth - amelogenesis imperfect
learning disabilites
physical disabilities e.g. cerebral palsy

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14
Q

what is another name for localised gingival recession

A

stilmans cleft

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15
Q

localised gingival recession

A

usually labial to lower incisors

associated with misaligned teeth, self inflicted injury, tooth brushing habits

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16
Q

what is gingival hyperplasia

A

gingival overgrowth

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17
Q

what drugs can induce gingival hyperplastia (i.e. drug induced)

A

Pheytoin
cyclosporin
nifedepine

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18
Q

pheytoin

A

anti epileptic

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19
Q

cyclosporin

A

immunosuppressant
inhibits T lymphocyte proliferation
effects of fibroblasts promoting protein synthesis and collagen formation

20
Q

nifedepine

A

calcium channel blocker

21
Q

what is cylcosporin used for

A
  • used mainly to prevent graft rejection
22
Q

gingival hyperplasia as a systemic disease

A

sarcoid

cyclic neutropenia

23
Q

sarcoid gingival hyperplasa

A

tubrercular cells laid down

interdental papilla overgrown

24
Q

cyclic neutropenai

A

can be inherited
very immunocomprised
leads to gingival overgrowht

25
types of traumatic gingival injury (self inflicted injury)
type A B C
26
what is type A traumatic gingival injury
injuries are superimposed upon a pre existing source of irritation e.g. exfoliating tooth
27
type B traumatic gingival injury
injuries are secondary to another established habit e.g. biting nails
28
Type C traumatic gingival injury
injuries are complex aetiology and are a physical manifestation of underlying emotions disturbance resistance to conventional treatment
29
acute gingival conditions and infections
1) Acute herpetic gingivostomatitis 2) Necrotising ulcerative gingivitis 3) hand foot and mouth 4) Herpengina
30
acute herpetic gingivostomatitis
HSV type I infection can present as subclinical infection i.e. under the weather 5-7 incubation period before symtoms
31
sign/symtoms of AHGS
``` high temperature (pyrexia) lymphadenopathy malaise and irritability profuse salivation refusal to eat sore throat and mouth symtoms for 7-10days ```
32
clinical features of acute herpetic gingivostomatic (AHGS)
``` small irregualr ulcers on gingiva tongue and palate erthyematous gingiva occasional extraoral lesions salivation lympathdenopathy ```
33
what can acute herpetic gingivasomatis recurre as
herpes labialise in 30%
34
management of AHGS
fluids and soft diet(don't get dehydrated) analgesics and antipyrexics isolation of drinking and eating materials OH improvement- chlorhexidine and sponges, soft toothbrish not acylovir (antivirals)unless immunocompromised
35
necrotiising ulcerative gingivitis and periodontiss
necrosis and ulceration (interdental papilla bleed profusely, distinctive hallitosis) broad anarobic infection treatment wit OH, hydrogen peroxide mouthwash, metronidazole 3 day
36
cancrum orbis
starts in the mouth | can necrosis soft tissues too
37
systemic conditions that may present with gingival changes e.g. bleeding, ulceration
``` HIV chrons disease leukaemia langerhans cell histocytosis scurvy ```
38
langerhams cell histocytosis
aggressive proliferation of histocytes multisystem condition
39
periodontal disease subheadings
aggressive periodontitis chronic periodontits with systemic disease/gentic conditions
40
clinical features of aggressive PD disease in children
tooth mobility/drifiting periodontal abcess rapid periodontal attatchnt loss healthy apart from periodontis
41
bacteria common in aggressive PD
a actinomycetescomitans | p gingivalis
42
body deformities with PD (Ie immune system issues)
phagocyte abno | hyperrepsonsive macrophage phenotype
43
management of periodontal diseas
standard mechanical periodontal therapy systemic or local drug therapy maintenance therapy periodontal surgery
44
what can be used to treat PD
metronidazole and amoxycillin 1 week
45
systemic or genetic conditions where PD is exacerbated
- insulin dependant diabetes - down syndrome - papillion lefevre syndrome - enlers danlos syndrome - Langerhans cell histiocytosis - neutropenia - hypophosphatasia (abnormal cementum)