HD2 Flashcards

1
Q
  1. Below are two CTG traces. In each case, name and briefly describe the condition the fetal heart trace is showing (1 mark for each). (4 marks in total)
A

Trace A – tachycardia (1 mark). Fetal heart rate (FHR) consistently raised above the normal range (160 bpm) (1 mark)

Trace B – Late decelerations or Type II dips (1 mark). FHR returns to baseline after the contraction has ended (1 mark).

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2
Q
  1. Name three of the five criteria that are used to evaluate the clinical status of the newborn baby using the Apgar score. (3 marks)
A

Any three from the following list:
Appearance (skin colour), Pulse, Grimace (reflex irritability), Activity (muscle tone) and Respiration

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3
Q
  1. A newborn boy with Down syndrome is vomiting all his feeds. What is the most likely cause for this? (1 mark)
A

Duodenal atresia

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4
Q
  1. A 27 year old woman who is 12 weeks pregnant attends the antenatal clinic for the results of her nuchal translucency scan and serum screening tests. The results reveal she has an increased nuchal translucency and decreased pregnancy-associated plasma protein A (PAPP-A) levels. What is the single most appropriate intervention at this point and why? (3 marks)
A

Chorionic villus sampling (1 mark)
Her results indicate a risk of her baby having Down syndrome (1 mark)
CVS would give a definitive diagnosis via karyotyping (1 mark)

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5
Q

How does placenta previa present? [1]

A
  • Bleeding without pain in third trimester
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6
Q

What is normal newborn resp rate? [1]

A
  1. Newborn respiratory rate: 30-60 breaths/min
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7
Q

State 4 acyanotic heart lesions

A
  • Atrial septal defects
  • Ventricular septal defects
  • Patent ductus arteriosus
  • Patent ductus arteriosus:
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8
Q

Describe difference in pre- and post-ductal coartation of the aorta? [2]

A

Pre-ductal: Ductus arteriosus persists to compensate for blood flow

Post-ductal: Collateral circulations needed to establish proper perfusion of body and legs (low BP after coarctation)

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9
Q

Describe 3 cyanotic heart lesions

A

Persistent truncus arteriosus:
 Single artery arises from the heart
 Large ventricular septal defect (blood mixing)
 Progressive heart failure

Transposition of the great vessels:
 No spiral septum formed (straight)
 Usually also open ductus arteriosus
 Catheterization and opening of fossa ovalis to allow blood mixing and buy time

Tetralogy of Fallot:
 Ventricular septal defect
 Overriding aorta (on top of septal defect)
 Pulmonary stenosis
 Right ventricular hypertrophy
 Boot shaped on xray

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10
Q

State 5 CV changes in mother during pregnancy [5]

A

o Fluctuation in Hb and haematocrit
o Increased WBC
o Reduction in blood volume
o Reduction in CO / HR
o Reduced progesterone -> reduced tissue fluid

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11
Q

Describe the structural changes to new born nn the onset of ventilation and clamping of cord [5]

A
  • Decreased pulmonary resistance
  • Increased systemic resistance
  • Increased oxygenation leads to closure of ductus arteriosus (aided by decreased PGs, anatomically closes within weeks) Functional: 12-15h / anatomical: 4-7d)
  • Atrial pressure changes close foramen ovale
  • Umbilical vein forms ligamentum teres (liver)
  • Umbilical arteries:
     Partially become medial umbilical ligament
     Partially open as branch of the internal iliac
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12
Q

Describe 5 consequencies of meconium aspiration [5]

A

o Airway obstruction -> increased lung resistance / hypoxia
o Infection
o Pulmonary inflammation
o Surfactant inactivation
o Persistent pulmonary hypertension

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13
Q

State 5 causes of non-reassuring fetal status [5]

A

 Hypoxia
 Maternal anaemia
 Gestational hypertension
 Intrauterine growth retardation
 Meconium stained amniotic fluid

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14
Q

Presentation of non-reassuring fetal status [3]

A

Irregular heart beat
* Drop after maternal contraction can be sign of uteroplacental insufficiency

Problems with muscle tone / movements

Oligohydramnios

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15
Q

Complications of perinatal asphyxia? [4]

A

 Hypoxia
 Hypercapnia
 Acidosis
 Bradycardia

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16
Q

Management of perinatal asphyxia? [3]

A
  • Management:
     Oxygen to mother
     C-section
     Mechanical breathing
17
Q

Complications [4] and management [3] of shoulder dystocia?

A

Complications:
 Erb’s palsy (brachial plexus)
 Fetal fracture
 Hypoxemic-ischaemic brain injury
 Maternal tearing / bleeding

Management:
 Change mother’s position
 Turn baby’s shoulders (McRoberts maneuver)
 Episiotomy

18
Q

Describe difference between type 1 & 2 of excessive bleeding after pregnancy [2]

A

 Primary: > 500ml within 24h of delivery
 Secondary: 24h-6wks after delivery

19
Q

Most common cause of PPH? [1]

A
  • Most common cause: lack of uterine tone
20
Q

Management of primary PPH?

A

 Ergotamine
 Misoprostol
 Uterine massage
 Blood transfusion
 Removal of retained placenta
 Tying off blood vessels
 Laparotomy
 Examination under anaesthetic
 Bakri balloon
 Compression sutures
 Artery embolisation
 Hysterectomy

21
Q

Signs of uterine rupture? [5]

A

 Abnormal fetal heart rate
 Abdominal pain
 Slow progress of labor
 Vaginal bleeding
 Rapid maternal heart rate

22
Q

Risk factors for uterine rupture? [3]

A
  • Most likely if previous C-section / large baby / induction
23
Q

Complications of uterine rupture? [4]

A

 Fetal hypoxia
 Aspiration of amniotic fluid / infection
 Maternal excessive bleeding
 Laceration of cervix / vagina

24
Q

How long does rapid labour occur in ?[1]

A
  • Labour lasts 3-5h instead of 6-18h
25
Q

Which is preffered: ventouse or forceps? [1]

A

ventose: fewer complications

26
Q

Name two complicatins of ventouse delivery [2]

A

 Vaginal laceration due to suction
 Bruising / laceration of the face / fetal scalp

27
Q

State what tachy- and bradycardia in fetus [2]

A

Tachycardia > 160bpm

Bradycardia < 100 bpm

28
Q

Describe why a fetus HR decelerates during stress [1]

A

 Hypoxic stress fetus reduces its heart rate to preserve myocardial oxygenation and perfusion

Unlike an adult, a fetus cannot increase its respiration depth and rate. This reduction in heart rate to reduce myocardial demand is referred to as a deceleration.

29
Q

State three causes of late deceleration [3]

A
  • Maternal hypotension
  • Pre-eclampsia
  • Uterine hyperstimulation
30
Q

What do late fetal HR deceleration indicate? [1]

A

 Show insufficient uteroplacental flow -> hypoxia / acidosis to the fetus

31
Q

Describe why variable decelerations occur [4]

A

 Umbilical cord compressed often in oligohydramnios
 Vein gets compressed -> acceleration
 Artery gets compressed -> rapid deceleration
 Pressure reduced -> baseline