Head Injuries Flashcards

(69 cards)

1
Q

head injuries

A
  • cause of 1.4 million ER visits are for traumatic brain injuries
  • 235,000 are hospitalized
  • 50,000 are fatal
  • TBIs (traumatic brain injuries) make up about half of all trauma victims
  • about 50-99% of patients who survive are left with some degree of permanent neurologic disability.
  • MVC are the leading cause of TBI’s of those 5-65 years of age
  • These injuries can be obvious and also extremely unnoticeable
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2
Q

scalp

A
  • strong flexible mass of skin, facia, muscular tissue
  • highly vascular
  • hair provides insulation
  • SCALP
  • Skin
  • Connective tissue
  • Aponeurotica
  • Layer of areolar tissue
  • Periosteum of skull
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3
Q

skull

A

-facial bones
-cranium
0vault for the brain
-strong, light, rigid spherical bone (flat)
-unyielding to increased intracranial pressure (ICP)
-Bones:
-Frontal
-Parietal
-Occipital
-Temporal

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4
Q

anatomy and physiology of the head

A
  • the interior of the cranial vault is not smooth -> not a lot of flexibility for trauma
  • This is problematic for any motion of the brain.
  • The only opening of the cranial vault is through the bottom called the Foramen magnum.
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5
Q

meninges

A
  • from the interior of the skull the brain is covered by 3 separate membranes
  • outer to inner
  • epidural space
  • dura mater
  • subdural space
  • arachnoid space
  • subarachnoid space
  • pia mater
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6
Q

epidural space

A

-under normal circumstance it does not exist, middle meningeal arteries follow grooves in the temporal bone here

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7
Q

dura mater

A
  • Made of rough fibrous tissue
  • Forms Tentorium:
  • Internal Support Structure that divides cerebrum and cerebellum
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8
Q

subdural space

A
  • Space that is spanned with Veins

- Low pressure

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9
Q

arachnoid space

A

Covering over the brains vasculature

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10
Q

subarachnoid space

A

Gap in which brains vasculature runs

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11
Q

pia mater

A

Thin covering Directly over the brain.

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12
Q

cerebrum

A

-Divided into left and right hemispheres
-four lobes:
-Frontal- Contains emotions, motor function, expression of
speech
-Parietal- Contains sensory function and spatial orientation
-Occipital- Contains vision
-Temporal- Reg. memory functions, are of speech
reception & integration

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13
Q

cerebellum

A

Involved in gross motor function

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14
Q

brainstem

A

Contains Midbrain, Pons, Medulla

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15
Q

medulla (oblongata)

A

-Acts as a path way for ascending and descending nerve tracts
-Controls several body functions
-Regulations of heart rate, blood vessel diameter,
breathing, swallowing, vomiting, coughing,
sneezing

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16
Q

pons

A
-Contains ascending and descending nerve
tracts
-Relays information from the cerebrum to
cerebellum
-Houses the sleep center and respiratory
center
-Like medulla helps in the regulation of
breathing
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17
Q

midbrain (mesencephalon)

A

-Involved in hearing through audio pathways
in the CNS
-Responsible for visual tracking of moving
objects, turning the eyes
-Coordinates regulation of the automatic
functions that require no conscious thought .

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18
Q

cranial nerve

A

Head Contains 12 cranial nerves that originate from the brain and brain stem.

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19
Q

mean arterial pressure

A
  • ensure circulation to brain tissue
  • diastolic pressure + (1/3 pulse pressure) = MAP
  • minimal value is 60 mmHg to profuse organs
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20
Q

cerebral perfusion pressure

A
  • amount of pressure that is needed to push blood through the cerebral circulation
  • accounts for cerebral pressure
  • CPP = MAP - ICP (intracranial pressure)
  • normal intracranial pressure = 7-15 mmHG
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21
Q

cerebral blood flow

A
  • most important factor for the brain

- brain retains this flow through autoregulation

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22
Q

traumatic brain injuries

A
  • are categories into 2 categories
  • primary- result of direct injury
  • secondary- result of on going injury process that is set in motion by the primary injury
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23
Q

cranial injury

A
  • trauma must be extreme to fracture skull
  • linear- crack
  • depressed- depressed and goes downward into brain
  • open- exposed to environment -> infection
  • impaled object- passing through the skull
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24
Q

basal skull

A
  • unprotected
  • bottom skull
  • spaces weaken structure
  • relatively easier to fracture
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25
signs of basal skull fracture: battle signs
- retroauricular ecchymosis - bruising behind ear | - associated with fracture of auditory canal and lower of skull
26
basal skull fracture signs: racoon eyes
- bilateral periorbital ecchymosis | - associated with orbital fractures
27
basilar skull fracture
- may tear dura - permit CSF to drain through an external passageway - may mediate rise of ICP - evaluate for target or halo sign - clear part of the CSF will be on the outside ring of a blood drop coming from the ear
28
brain injury
- as defined by the national head injury foundation - a traumatic insult to the brain capable of producing physical, intellectual, emotional, social and vocational changes - classification: - Primary- caused by forces of trauma - Secondary- caused by factors resulting from the primary injury
29
direct brain injury types
- coup- injury at site of impact (usually more severe) | - contrecoup- injury on opposite side from impact -> brain sloshes back
30
direct brain injury categories: focal
- occur at a specific location in brain - differentials: - cerebral contusion - intracranial hemorrhage - >epidural hematoma - >subdural hematoma - intracerebral hemorrhage
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direct brain injury: diffuse
- concussion - moderate diffuse axonal injury - severe diffuse axonal injury
32
cerebral contusion: focal injury
- blunt trauma to local brain tissue - capillary bleeding into brain tissue - common with blunt head trauma - confusion - neurologic deficit - personality changes - vision changes - speech changes - result from coup-contrecoup injury
33
intracranial hemorrhage: epidural hematoma: focal injury
- bleeding between dura mater and skull - blood is where the brain should be -> herniation - involves arteries - middle meningeal artery most common - rapid bleeding and reduction of oxygen to tissues - patients will have lucid interval - unconscious goes unconscious again
34
intracranial hemorrhage: subdural hematoma: focal injury
- bleeding within meninges - beneath dura mater - above arachnoid - slow bleeding- superior sagittal sinus - signs progress over several days - slow deterioration of mentation - blood is displacing the brain
35
intracranial hemorrhage: intracerebral hemorrhage: focal injury
- ruptured blood vessel within the brain - presentation similar to to stroke symptoms - Signs and symptoms worsen over time
36
intracranial perfusion
- cranial volume is fixed - 80% = cerebrum, cerebellum, and brainstem - 12% blood vessels and blood - 8% CSF - increase in size of one component diminishes size of another - inability to adjust = increased ICP - increase in any one of these things will displace CSF and eventually brain and eventually herniation
37
compensating for pressure building up
- compress venous blood vessels - reduction in free CSF - pushed into spinal cord - pressure builds
38
decompensation for pressure
- increase in ICP - rise in systemic BP to perfuse brain - further increase in ICP - dangerous cycle !
39
role of carbon dioxide
- increase of CO2 in CSF - cerebral vasodilation - encourage blood flow - reduce hypercarbia - reduce hypoxia - contribute to increased ICP - causes classic hyperventilation and hypertension - reduced levels of CO2 in CSF -> cerebral vasoconstriction -> cerebral anoxia
40
factors affecting ICP
- vasculature constriction - cerebral edema- swelling - systolic blood pressure - low BP- poor cerebral perfusion - high BP- increased ICP - carbon dioxide - reduced respiratory efficiency
41
cascade
-cranial insult -> tissue edema -> increased ICP -> compression of arteries -> decrease cerebral blood flow -> decrease O2 with death of brain cells -> edema around necrotic tissue -> increase ICP with compression of brain stem and respiratory center -> CO2 accumulates -> vasodilation -> increase ICP due to increased blood volume -> death
42
increased pressure
- compresses brain tissue - herniates brainstem - compromises blood supply
43
signs and symptoms of upper brainstem displace (from pressure)
- vomiting - Altered mental status - Pupillary dilation
44
signs and symptoms of medulla oblongata displace (from pressure)
- Respiratory - Cardiovascular - Blood pressure disturbances
45
diffuse brain injury
- Due to stretching forces placed on axons - Pathology distributed throughout brain - Types: - Concussion - Moderate diffuse axonal injury - Severe diffuse axonal injury
46
diffuse brain injury concussion
- Mild to moderate form of diffuse axonal injury (DAI) - Nerve dysfunction without anatomic damage - Transient episode of Confusion, disorientation, event amnesia - Suspect if patient has a momentary loss of consciousness - Frequent reassessment of mentation - ABCs
47
diffuse brain injury moderate diffuse axonal injury
- “Classic Concussion” - Same mechanism as concussion - Additional: minute bruising of brain tissue - Unconsciousness -> confusion - If cerebral cortex and RAS involved - May exist with a basilar skull fracture - Signs and Symptoms - Unconsciousness or persistent confusion - Loss of concentration, disorientation - Retrograde and antegrade amnesia - Visual and sensory disturbances - Mood or personality changes
48
diffuse brain injury severe diffuse axonal injury
- Brainstem Injury - Significant mechanical disruption of axons - Cerebral hemispheres and brainstem - High mortality rate - Signs and Symptoms: - Prolonged unconsciousness - Cushing’s reflex - Decorticate (flexion) or decerebrate (extension) posturing
49
altered mental status
- altered orientation - alteration in personality - amnesia
50
cushing's reflex (triad)
- increased BP - bradycardia - erratic respirations - these 3 symptoms are really only ever seen together for brain injury
51
vomiting
- without nausea | - projectile
52
body temperature changes
- changes in pupil reactivity (maybe dilated) | - decorticate posturing
53
blood glucose
-obtain blood glucose level on all patient with AMS if there is a altered mental state
54
brain injury: things to look for
- cushings reflex - body temperature changes - vomiting - altered mental status
55
pathophysiology of changes
- frontal lobe injury- alterations in personality - occipital lobe injury- visual disturbances - cortical disruption- reduced mental status or amnesia - > retrograde- unable to recall events before injury - > antegrade unable to recall events after trauma -> repetitive questioning - focal deficits- Hemiplegia, weakness, or seizures
56
upper brainstem compression
- Increasing blood pressure - Reflex bradycardia - Vagus nerve stimulation - Cheyne-Stokes respirations - Pupils become small and reactive - Decorticate posturing - Neural pathway disruption
57
middle brainstem compression
- Widening pulse pressure - Increasing bradycardia - Central Neurogenic Hyperventilation - Deep and rapid - Bilateral pupil sluggishness or inactivity - Decerebrate posturing
58
lower brainstem injury
- Pupils dilated and unreactive - Ataxic respirations - Erratic with no pattern - Irregular and erratic pulse rate - ECG changes - Hypotension - Loss of response to painful stimulus
59
brain injury: eye signs
- Indicate pressure on - CN-II, CN-III, CN-IV, and CN-VI - CN-III (Oculomotor nerve) - Pressure on nerve causes eyes to be sluggish, then dilated, and finally fixed. - Reduced peripheral blood flow - Pupil Size and Reactivity - Reduced pupillary responsiveness - Depressant drugs or cerebral hypoxia - Fixed and dilated - Extreme hypoxia
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recognition of herniation
- cushings reflex- increasing blood pressure, decreasing pulse rate, respirations that become erratic - lower level of consciousness - GCS < 9 and dropping - singular or bilaterally dilated and fixed pupils - Decerebrate or decorticate posturing - No movement with noxious stimuli
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glasgow coma scale
-know this
62
Trauma to the eye or Orbit
- Eyelid laceration: Laceration to eyelid, - Cover Both Eyes with lose dressing. Lac of globe is possible. - Corneal abrasion: Scratch to epithelial covering of the cornea - Cover both eyes, to limit light and sympathetic movement - Blow out fracture: Increased pressure due to outside insult causes the base of orbital socket to fracture inward
63
Trauma to Nasal
-Usually simplistic injury, Be ware of basilar skull injuries
64
midface injuries
- Le fort 1: Involves a horizontal detachment of the maxilla from the nasal floor - Le fort 2- Fracture of the right and left maxillae, medial orbital floor and nasal bones. - Le fort 3- Involves the facial bones being fractured off the skull “Craniofacial disjunction”
65
head injury management: airway
- Suctioning - Patient positioning - OPA and NPA use - Endotracheal intubation- Orotracheal; RSI - Cricothyrotomy - Oxygen - 15 LPM/NRB - Begin at 10-12 breaths/min - adjust breathing rate to maintain ETCO2 at 35–40 mmHg - Continuous waveform capnogrpahy
66
head injury management: circulation
- Hemorrhage Control - Blood pressure maintenance - Fluid resuscitation to SBP of 90 mmHg
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hypovolemia
- Reduces cerebral perfusion and hypoxia. - Consider early management with 2 large bore IVs and isotonic fluids. - Prevents slower compensatory mechanism. - Maintain SBP 90 mmHg in an adult. - Maintain SBP 80 mmHg in a child. - Maintain SBP 75 mmHg in a young child. - Maintain SBP 65 mmHg in an infant.
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definitive care: nonsurgical candidates
- Patients with small Hemorrhages Patients with minimal - Neurological deficits - Patients who have GCS less that or equal to 4 Unless emergent brain stem release
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definitive care: surgical candidates
- Patient with hemorrhages greater than 3cm - Patients that are neurologically deteriorating - intracranial Hemorrhage associated with structural lesion - Improve chance of good outcome - Young Patients with a moderate lobar hemorrhage with deterioration