Headaches: Tension, Cluster, Migraine, IHH, Temporal arteritis, Medication Overuse Flashcards

1
Q

IHH
-presentation

A

Persistant frontal, retroorbital
Bilateral, dull

Worsened by coughing, physical activity, pressing

Papilloedema => Ongoing progressive visual loss - different to migraine
Enlarged blind spot
If CNVI involved => diplopia

N+photophobia

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2
Q

IHH
-pathophysiology
-risk factors

A

High ICP

Most common - obese females in 20-30s
Pregnancy
Drugs
-COCP, CS, tetracycline, VitA, Li

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3
Q

IHH
-investigations, diagnosis
-management

A

Find any underlying causes
CT, MRI
ICP monitoring

IIH diagnosis of exclusion

Lifestyle - weight loss

Medication - diuretics, antiepileptic (topiramate)

Surgical
-repeated lumbar puncture
-optic nerve sheath decompression and fenestration

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4
Q

Migraine
-epidemiology, pathophysiology

A

Young females

Result of abnormal brain activity affecting nerve signals, chemicals, blood vessels => pain

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5
Q

Migraine
-diagnosis

A

Min 5 attacks lasting 4-72hrs

Min 2 of

unilateral
pulsation
moderate/severe
worse with activity
Min 1 of

N+V
photophobia/phonophobia

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6
Q

Clinical course of migraine

A

Prodrome - 48hrs due to hypothalamic involvement
-fatigue
-cravings

Aura - 20min per symptom, last for 1hr - hypothalamic activity spreads to other brain areas
-marching progression through visual => sensory => motor, aphasia
-LOSS OF FUNCTION

Headache - 72hrs
-photophobia, phonophobia
-N+V

Resolution
-fatigue

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7
Q

Migraine management
-acute
-preventative

A

Acute treatment
1st line - paracetamol, ibuprofen at first signs of headache
2nd line - triptan (before its at its worse+ antiemetics (metoclopramide or domperidone)

Preventative
-topiramate OD (antiepileptic)
others - propanolol/amitriptyline

Identify and avoid triggers - migraine diary
-date, time, duration
-warning signs
-symptoms
-medication

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8
Q

Common migraine triggers

A

Tired/stress
Alcohol
COCP, periods
Hungry, thirsty
Bright light
Cheese, chocolate, red wine, citrus

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9
Q

Medication overuse headache
-prevalence
-pathophysiology

A

More common in women
Pathophysiology a mystery :(

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10
Q

Medication overuse headache criteria

A

Preexisting headache disorder
15+ headache days/month
Regular overuse of 3 months+ of acute/symptomatic headache treatment

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11
Q

Medication overuse headache management

A

Definitive - withdrawal of overused drug
-IMMEDIATELY - simple + triptans
-GRADUAL - opioids

warn that symptoms may initially worsen but should improve over weeks
Keep headache diary

Reassess underlying cause

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12
Q

Cluster headache
-diagnostic criteria

A

Min 5 attacks with the same presentation

15mins-3hrs
Severe unilateral eye pain, same side everytime

Restlessness/agitation
Ipsilateral to pain
-Tears, runny nose. sweating
-Eyelid edema
-miosis, ptosis
Frequency ranging from 1 every other day - 8 a day
-attacks will cluster

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13
Q

Cluster headache management
-acute
-preventative

A

Acute - high flow O2
-2nd line - triptan (SC, IN) - can only use it 2x a day due to increased risk of side effects with prolonged use

Confirmation needed with neuroimaging

Preventative - verapamil whilst they have episodic clusters

taper off when clusters end
alts - topiramate, lithium

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14
Q

Primary vs secondary headache

A

Primary more likely if

headache type known for years
gradual onset
no neuro deficit
Secondary more likely if

new unknown headache
sudden onset (as if something fell on your head = ASSUME VASCULAR UNLESS PROVEN OTHERWISE WITH CT, LP
electric shock-like - trigeminal?
neuro deficit, altered consciousness
50+
positional changes, precipitated by something
systemically unwell

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15
Q

Thunderclap headache
-possible causes

A

SAH
ICH
Cerebeal venous thrombosis
Arterial dissection - intracranial/extracranial

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16
Q

Headache due to CSF pressure change
-differentiating between high and low pressure headaches

A

Raised pressure - SOL, bleed, abscess, IIH

worse in morning
better upright
worse with Valsalva
Low pressure - dural tear (idiopathic/trauma)

worse as day progresses
better recumbent

17
Q

Differentiating between primary headaches
-duration
-localisation
-accompanying symptoms
-intensity

A

Duration

constant - tension?
seconds - trigeminal?
mins to hours - cluster?
Localisation

same unilateral attacks - trigeminal?, cluster?
bilateral - tension?
unilateral - migraine
Accompanying symptoms
-nausea, photophobia, lacrimation, aura?

Intensity, changes with physical activity?

worsens on mv - migraine?
improves on mv - tension? cluster?

18
Q

Trigeminal neuralgia
-presentation
-pathophysiology
-management

A

Severe unilateral pain limited to divisions of CNV
-electric shock like
Evoked by light touch (washing, shaving, talking, brushing teeth)

Most idiopathic
Compression by tumour/vascular (common: superior cerebellar artery)

Carbemazepine
Failure to respond => neuro referral

19
Q

Tension
-epidemiology
-pathophysiology
-presentation
-management

A

Common in both sexes, 40-50
Pathophysiology unclear but related to increased central sensitization

Tight band-like pressure, bilateral
Lower intensity
No aura, N/V
Not worsened by physical activity

Acute
-aspirin/paracetamol/NSAID
Prophylaxis
-address triggers - stress, poor sleep, anxiety, depression
-up to 10 sessions of acupuncture over 5-8wks

20
Q

Temporal arteritis/GCA
-epidemiology and associations
-pathophysiology

A

70, white females
Associated with PMR

Genetic, environmental (seasonal, geographic clustering?), immune dysregulation => vascular inflammation and damage

21
Q

Temporal arteritis/GCA
-presentation

A

Rapid onset headache
Jaw claudication
Tender palpable temporal artery

VISION TESTING VITAL
-temporary visual loss
-sudden permanent visual loss - most feared complication

Consitutional - tired, depression, low-grade fever, anorexia, night sweats
PMR - aching, morning stiffness in proximal limbs, no weakness