Health Problems of Toddlers and Preschoolers Flashcards

(74 cards)

1
Q

COMMUNICABLE DISEASES

A
  • VARICELLA
  • DIPHTHERIA
  • MUMPS
  • MEASLES
  • PERTUSSIS
  • POLIOMYELITIS
  • RUBELLA
  • SCARLET FEVER
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2
Q

VARICELLA (Chickenpox)

A
  1. Causative agent: Varicella zoster virus (VZV)
  2. Source
    - Primary secretions of respiratory tract of infected
    persons
    - to a lesser degree, skin lesions (scabs are NOT infectious)
  3. Incubation period
    - 10 to 21 days
    - with common incidence at
    14–16 days following exposure
  4. Period of communicability
    - 1 day before eruption of rash or lesions (prodromal period)
  5. Mode of Transmission
    - Direct contact or indirect contact of saliva or open vesicles, droplet (airborne) spread, and contaminated objects
  6. Immunity
    - Contracting the disease offers lasting immunity to varicella
    - however because VZV is latent, it causes herpes zoster (shingles) when it is reactivated at a later time
  7. Hallmark sign
    - 2-3 mm vesicle on an erythematous base
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3
Q

CLINICAL MANIFESTATIONS
VARICELLA (Chickenpox): Prodromal stage

A
  1. rash (highly pruritic) accompanied by low grade fever and malaise occurs in the first 24 hours
  2. begins as macule, rapidly progresses to papule and then vesicle (surrounded by erythematous base
  3. becomes umbilicated and
    cloudy
  4. breaks easily and forms crusts
  5. all four stages (macule,
    papule, vesicle, crust) present in varying degrees at one time
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4
Q

CLINICAL MANIFESTATIONS
VARICELLA (Chickenpox): Distribution

A

Centripetal starting from the trunk progressing outward to arms, face, legs, and mucosal surfaces but sparse on distal limbs and less on areas not exposed to heat (i.e., from
clothing or sun)

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5
Q

VARICELLA (Chickenpox) S/Sx

A

Elevated temperature from
lymphadenopathy, irritability from pruritus

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6
Q

VARICELLA THERAPEUTIC MANAGEMENT

A
  1. Topical oatmeal-based cream and antihistamine such as
    diphenhydramine (Benadryl) for pruritus
  2. Antipyretics such as acetaminophen (paracetamol) for fever
  3. Antiviral agent acyclovir (Zovirax)
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7
Q

VARICELLA COMPLICATIONS

A

Secondary bacterial infections (abscesses, cellulitis, necrotizing fasciitis, pneumonia, sepsis)

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8
Q

VARICELLA Preventive Measures

A

Childhood immunization
1. Active artificial immunity
- Attenuated live virus vaccine

  1. Passive artificial immunity
    - varicella zoster immune globulin (VZIG) or immune globulin intravenous (IGIV) after exposure in high risk children
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9
Q

VARICELLA NURSING CARE MANAGEMENT

A
  • Maintain Standard, Airborne, and Contact Precautions if
    hospitalized
  • Keep child in home away from susceptible individuals.
  • Administer skin care: give bath and change clothes and linens daily.
  • Avoid use of aspirin.
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10
Q

DIPHTHERIA

A
  1. Causative agent
    - Corynebacterium diphtheriae
  2. Source
    - Discharges from mucous membranes of nose and
    nasopharynx, skin, and other lesions of infected person
  3. Transmission
    - Direct contact with infected person, a carrier, or contaminated articles
  4. Incubation period
    - 2–5 days, possibly longer
  5. Period of communicability
    - Variable
    - until virulent bacilli are no longer present (identified by 3 negative culture results)
    - usually 2 weeks but as long as 4 weeks
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11
Q

DIPHTHERIA CLINICAL MANIFESTATIONS

A
  1. Nasal
    - resembles common cold
    - serosanguinous mucopurulent nasal discharge without constitutional symptoms
  2. Tonsillar–pharyngeal
    - malaise
    - anorexia
    - sore throat
    - low grade fever
    - pulse increased above expected for temperature within 24 hours
  3. Laryngeal
    - fever
    - hoarseness
    - cough
    - with or without previous
    signs listed
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12
Q

DIPHTHERIA THERAPEUTIC MANAGEMENT

A
  • Equine antitoxin to neutralize the diphtheria toxin
  • Antibiotics (penicillin G, procaine or erythromycin) in addition to equine antitoxin
  • Complete bed rest
  • Tracheostomy for airway obstruction
  • Treatment of infected contacts and carriers
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13
Q

DIPHTHERIA COMPLICATIONS

A

Toxic cardiomyopathy (2nd to 3rd weeks)

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14
Q

DIPHTHERIA Preventive Measures

A

Childhood immunization - Pentavalent Vaccine
(DTP-HepB-Hib)

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15
Q

DIPHTHERIA NURSING CARE MANAGEMENT

A
  • Administer antibiotics in a timely manner.
  • Administer complete care to maintain bed rest.
  • Administer humidified oxygen as prescribed.
  • Observe respiration for signs of obstruction
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16
Q

MUMPS

A
  1. Causative agent
    - Paramyxovirus
  2. Source
    - Saliva of infected persons.
  3. Transmission
    - Direct contact with or droplet spread from an
    infected person
  4. Incubation period
    - 14 to 21 days Period of communicability
  5. Most communicable immediately before and after swelling begins
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17
Q

MUMPS CLINICAL MANIFESTATIONS

A
  1. Prodromal stage
    - Fever, headache, malaise, and anorexia
    for 24 hour followed by “earache” that is aggravated by chewing
  2. Parotitis
    - By third day, parotid gland(s) (either unilateral or bilateral) enlarges and reaches maximum size in 13 days
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18
Q

MUMPS THERAPEUTIC MANAGEMENT

A
  1. Preventive
    - Childhood immunization Measles Mumps Rubella (MMR) Vaccine
  2. Symptomatic and supportive
    - Analgesics for pain and antipyretics for fever
  3. Intravenous Fluid
    - if needed for child who refuses to drink
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19
Q

MUMPS COMPLICATIONS

A
  • Sensorineural deafness
  • Post infectious encephalitis
  • Myocarditis
  • Arthritis
  • Hepatitis
  • Epididymoorchitis: inflammation of both the epididymis and testis
  • Oophoritis; inflammation of the ovaries
  • Pancreatitis
  • Sterility (extremely rare in adult men)
  • Meningitis
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20
Q

MUMPS NURSING CARE MANAGEMENT

A
  • Maintain isolation during period of communicability
  • Encourage rest
  • Give analgesics for pain
  • Encourage Fluids and soft, bland foods.
  • Apply hot or cold compresses to neck.
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21
Q

MEASLES (Rubeola)

A
  1. Causative Agent
    - Measles virus (a paramyxovirus)
  2. Source
    - Respiratory tract secretions, blood, and urine of infected person
  3. Transmission
    - Usually by direct contact with droplets of
    infected person
  4. Incubation period
    - 10 to 20 days
  5. Period of communicability
    - From 4 days before to 5 days after rash appear
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22
Q

MEASLES CLINICAL MANIFESTATIONS

A
  1. Prodromal (catarrhal) stage
    - Fever and malaise
    - followed in 24 hours by coryza (common cold), cough, conjunctivitis,
    Koplik spots (small, irregular red spots with a minute, bluish white center first seen on buccal mucosa opposite molars 2 days before rash)
  2. Rash
    - Appears 3 to 4 days after onset of prodromal stage
  3. S/Sx
    - Anorexia
    - abdominal pain
    - malaise
    - generalized lymphadenopathy
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23
Q

MEASLES (Rubeola) THERAPEUTIC MANAGEMENT

A
  1. Preventive
    - Childhood immunization ( Measles Mumps Rubella (MMR) Vaccine)
    - Vitamin A supplementation
  2. Supportive
    - Bed rest during febrile period
    - Antibiotics to prevent
    secondary bacterial infection
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24
Q

MEASLES (Rubeola) Complications

A

Otitis media, Pneumonia

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25
MEASLES (Rubeola) Management NURSING CARE MANAGEMENT
1. Fever - instruct parents to administer antipyretics 2. Eye care - clean eyelids with warm saline solution to remove secretions or crusts. 3. Coryza, cough - use cool mist vaporizer 4. Skin care - keep skin clean - use tepid baths as necessary
26
PERTUSSIS (Whooping Cough)
1. Causative agent - Bordetella pertussis 2. Source - Discharge from respiratory tract of infected persons 3. Transmission - Direct contact or droplet spread from infected person. 4. Incubation period - 6 to 20 days - usually 7 to 10 days 5. Period of communicability - Greatest during catarrhal stage before onset of paroxysms
27
PERTUSSIS (Whooping Cough) CLINICAL MANIFESTATIONS
1. Catarrhal stage - begins with symptoms of upper respiratory tract infection, such as coryza, sneezing, lacrimation, cough, and low grade fever 2. Paroxysmal stage - Cough most common at night; consists of short, rapid coughs followed by sudden inspiration associated with a high pitched crowing sound or “whoop” 3. Additional symptoms in adolescents - difficulty breathing - post tussive vomiting
28
PERTUSSIS THERAPEUTIC MANAGEMENT
1. Childhood Immunization - Pentavalent Vaccine (DTP HepaB Hib)Hib) - Antimicrobial therapy 2. Supportive - Increased oxygen intake and humidity - Adequate fluids.
29
PERTUSSIS COMPLICATIONS
- Pneumonia - Apnea (infants <1 year) - Atelectasis - Otitis media - Seizures - Hemorrhage
30
PERTUSSIS (Whooping Cough) NURSING CARE MANAGEMENT
- Maintain isolation during catarrhal stage - Encourage oral fluids; offer small amount of fluids frequently. - Provide humidified oxygen - Encourage compliance with antibiotic therapy for household contacts.
31
POLIOMYELITIS
1. Causative agent: Enteroviruses - type 1, most frequent cause of paralysis, both epidemic and endemic - type 2, least frequently associated with paralysis - type 3, second most frequently associated with paralysis 2. Source - feces and oropharyngeal secretions of infected persons, especially young children 3. Transmission - Direct contact with persons with apparent or inapparent active infection 4. Incubation period - Usually 7 to 14 days, with range of 5 to 35 days 5. Period of communicability - Not exactly known
32
POLIOMYELITIS CLINICAL MANIFESTATIONS
May be manifested in three different forms 1. Abortive or inapparent - Fever - uneasiness - sore throat - headache - anorexia - vomiting - abdominal pain 2. Nonparalytic - Same manifestations as abortive but more severe, with pain and stiffness in neck, back, and legs 3. Paralytic - Initial course similar to nonparalytic type followed by recovery and then signs of central nervous system paralysis
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POLIOMYELITIS THERAPEUTIC MANAGEMENT
1. Childhood immunization - Oral Polio Vaccine (OPV) - Inactivated Polio Vaccine (IPV) 2. Supportive - Complete bed rest during acute phase - Physical therapy for muscles after acute stage
34
POLIOMYELITIS COMPLICATIONS
- Permanent paralysis - Respiratory arrest - Hypertension - Kidney stones
35
RUBELLA (German measles)
1. Causative agent - Rubella virus 2. Source - Primarily nasopharyngeal secretions of person with apparent or inapparent infection - virus also present in blood, stool, and urine 3. Incubation period: - 14 to 21 days 4. Period of communicability - 7 days before to about 5 days after appearance of rash 5. S/Sx - Occasionally low grade fever - headache - malaise - lymphadenopathy
36
RUBELLA (German measles) CLINICAL MANIFESTATIONS
1. Prodromal stage - Absent in children - present in adults and adolescents - low grade fever, headache, malaise, anorexia, mild conjunctivitis, coryza, sore throat, cough, and lymphadenopathy 2. Rash First appears on face and rapidly spreads downward to neck, arms, trunk, and legs
37
RUBELLA (German measles) THERAPEUTIC MANAGEMENT
Childhood immunization - Measles Mumps Rubella (MMR) Vaccine
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RUBELLA (German measles) Complications
- Rare (arthritis, encephalitis, or purpura) - greatest danger is teratogenic effect on fetuses
39
RUBELLA (German measles) NURSING CARE MANAGEMENT
- Institute Droplet Precautions. - Reassure parents of benign nature of illness in affected child. - Avoid contact with pregnant women - Monitor rubella titer in pregnant adolescents.
40
SCARLET FEVER
1. Agent - Group A β-hemolytic streptococci 2. Source - Usually from nasopharyngeal secretions of infected persons and carriers. 3. Transmission - Direct contact with infected person or droplet spread - indirectly by contact with contaminated article. 4. Incubation period - 2 to 5 days, with range of 1 to 7 days 5. Period of communicability - Incubation period and clinical illness is approximately 10 days - first 2 weeks of carrier phase, although may persist for months
41
SCARLET FEVER CLINICAL MANIFESTATIONS
1. Prodromal stage - Abrupt high fever - pulse increased out of proportion to fever, vomiting, headache, chills, malaise, abdominal pain, halitosis 2. Enanthema - Tonsils enlarged, edematous, reddened, and covered with patches of exudates 3. Exanthema - Rash appears within 12 hours after prodromal signs
42
How to spot scarlet fever
- Fine red rash, feels like sandpaper - White coating on tongue that peels after few days leaving it swollen and red (AKA "strawberry tongue") - Fever over 38.3 C (101F) - Flushed red face, but pale around mouth - Swollen glands on neck
43
SCARLET FEVER THERAPEUTIC MANAGEMENT
- Full course of penicillin - Antibiotic therapy for newly diagnosed carriers - Rest during febrile phase - analgesics for sore throat
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SCARLET FEVER COMPLICATIONS
- Peritonsillar and retropharyngeal abscess - Sinusitis - Otitis media - Acute glomerulonephritis - Acute rheumatic fever - Polyarthritis (uncommon)
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SCARLET FEVER NURSING CARE MANAGEMENT
- Institute Standard and Droplet - Ensure compliance with oral antibiotic therapy - Encourage rest during febrile phase - Relieve discomfort of sore throat with analgesics, gargles, lozenges, antiseptic throat sprays, and inhalation of cool mist. - Encourage fluids during febrile phase
46
CONJUNCTIVITIS
(inflammation of the conjunctiva) infection of the thin conjunctiva covering the eye globe.
47
Causes of CONJUNCTIVITIS in Children
- viral, bacterial, fungal organisms - allergic or related to a foreign body.
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CONJUNCTIVITIS S/Sx
- eyes watery with reddened conjunctiva - sensitivity to light - sticking of eyelids with pustular drainage
49
CONJUNCTIVITIS TRANSMISSION
- Direct contact with infected eye secretions - then touching your own eyes - indirectly by contact with contaminated article or surfaces - air borne droplet spread - allergens and chemical or irritants (not contagious)
50
Clinical Manifestations of different types of Conjunctivitis
1. Bacterial Conjunctivitis ("Pink Eye") - Purulent drainage - Crusting of eyelids, especially on awakening - Inflamed conjunctiva - Swollen eyelids 2. Viral Conjunctivitis - Usually occurs with upper respiratory tract infection - Serous (watery) drainage - Inflamed conjunctiva - Swollen eyelids 3. Allergic Conjunctivitis - Itching - Watery to thick, stringy discharge - Inflamed conjunctiva - Swollen eyelids 4. Conjunctivitis Caused by Foreign Body - Tearing - Pain - Inflamed conjunctiva - Usually only one eye affected
51
CONJUNCTIVITIS Therapeutic Management
1. Viral conjunctivitis is self limiting - treatment is limited to removal of the accumulated secretions. 2. Bacterial conjunctivitis - traditionally been treated with topical antibacterial agents such as polymyxin and bacitracin Poly sporin ), sulfacetamide Sulamyd ), or trimethoprim and poly myxin B ( Polytrim ).
52
CONJUNCTIVITIS Nursing Care Management
- Nursing care includes keeping the eye clean and properly administering ophthalmic medication. - Remove accumulated secretions by wiping from the inner canthus downward and outward away from the opposite eye. - Keep the child’s washcloth and towel separate from those used by others.
53
INTESTINAL PARASITIC DISEASE
- diseases, including helminths (worms) and protozoa - constitute the most frequent infections in the world. - Young children are especially at risk because of Young children are especially at risk because of typical hand mouth activity and uncontrolled fecal activity.
54
CLINICAL MANIFESTATION Intestinal Parasitic Diseases: ASCARIASIS (Roundworm)
1. Facts - common roundworms - largest of the intestinal helminths - helminths; affects 1 to 4 years old children - prevalent in warm climates 2. Transmission - transferred to mouth by way of contaminated food, fingers or toys 3. Signs and symptoms - anorexia - irritability - nervousness - enlarged abdomen - weight loss - fever - intestinal colic 4. Treatment - Albendazole - Mebendazole
55
CLINICAL MANIFESTATION Intestinal Parasitic Diseases: HOOKWORM DISEASE (Hookworm)
1. Transmission - transmitted by discharging eggs on the soil, which are picked up, causing infection from direct skin contact with contaminated soil - recommended to wear shoes 2. Signs and symptoms - mild to severe anemia, malnutrition - itching and burning followed by erythema and papular eruption in areas to which the organism migrates 3. Treatment - Albendazole - Mebendazole
56
CLINICAL MANIFESTATION Intestinal Parasitic Diseases: STRONGYLOIDIASIS (Threadworm)
1. Transmission - transmitted by discharging eggs on the soil, which are picked up, causing infection from direct skin contact with contaminated soil - autoinfection common in older children and adult 2. Signs and symptoms - respiratory s/sx (cough, wheezing, DOB) - abdominal pain - distention - nausea and vomiting - diarrhea (large, pale stools, often with mucus), may lead to severe nutritional deficiency 3.Treatment: - Ivermectin (preferred) - Albendazole (alternative)
57
CLINICAL MANIFESTATION Intestinal Parasitic Diseases: TRICHURIASIS (Whipworm)
1. Transmission - transmitted from contaminated soil, vegetables, toys and other objects; - prevalent in warm, moist climates - occurs in undernourished children living in unsanitary conditions 2. Signs and symptoms - abdominal pain and distention, diarrhea 3. Treatment - Albendazole - Mebendazole
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GIARDIASIS
1. Causative agent - flagellated protozoan Giardia lamblia 2. Source - the child ingests the cyst then it develops in the intestines into the mature form of the organism - it is contagious as long an the infected person still has excreted cysts 3. Mode of Transmission - fecal oral route and from fecal contaminated food and stools - from person to person or person to animals
59
GIARDIASIS CLINICAL MANIFESTATIONS
1. Infants and young children - diarrhea - weight loss - vomiting - anorexia - growth failure (failure to thrive) if chronic exposure 2. Children older than 5 years of age - abdominal cramps - intermittent loose stools - constipation
60
GIARDIASIS Therapeutic Management
1. Treatment - metronidazole (Flagyl) the drugs of choice - tinidazole (Tindamax), and nitazoxanide (Alinia). Tinidazole is said to have an 80% to 100% cure rate after a single dose. 2. The most important nursing consideration is prevention of giardiasis and education of parents, child care center staff, and others who assume the daily care of small children. 3. Prevention - proper handwashing and improve sanitation
61
ENTEROBIASIS (Pinworms)
1. Fact - most common helminthic infection in the United States. 2. Causative agent - Enterobius vermicularis 3. Transmission - Infection begins when the eggs are ingested or inhaled - The eggs hatch in the upper intestine and then mature and migrate through the intestine. - eggs persist in the indoor environment for 2 to 3 weeks, contaminating anything they contact 4. Treatment - Albendazole - Mebendazole - Pyrantel pamoate
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ENTEROBIASIS (Pinworms) Clinical Manifestations
Intense perianal itching (principal symptom); evidence of itching includes - General irritability - Restlessness - Poor sleep - Bedwetting - Distractibility - Short attention span
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ENTEROBIASIS (Pinworms) Nursing Care Management
- identifying the parasite, eradicating the organism, and preventing reinfection. - Parents need clear, detailed instructions for the tape test - Pinworm specimens are collected in the morning as soon as the child awakens and before the child has a bowel movement or bathes.
64
INTESTINAL PARASITIC DISEASE GENERAL NURSING CARE MANAGEMENT
- Assistance with identification of the parasite - Administration of treatment of the infection as prescribed (Antiparasitic medications) - Prevention of initial infection or reinfection
65
INGESTION OF INJURIOUS AGENTS
1. Heavy Metal Poisoning 2. Lead Poisoning
66
HEAVY METAL POISONING
1. occur from the ingestion of a variety of substances - Lead (most common) - Iron - Mercury 2. Treatment involves chelation - use of a chemical compound that com bines with the metal for rapid and safe excretion.
67
LEAD POISONING
1. Facts - Ingestion leads to serious damage to the brain and nervous system, kidneys, and red blood cells. - In most instances of acute childhood lead poisoning, the source is non intact lead based paint in an older home or lead contaminated bare soil in the yard. 2. Signs and symptoms at the beginning - irritability - headache - fatigue and abdominal pain 3. S/sx for Low levels at 5 µg/dL - learning and behavioral problems 4. S/Sx for High levels - seizures - cognitive challenges - coma - death
68
SOURCES OF LEAD POISONING
- Toys manufactured in countries where restrictions on lead are lax - Soil around the exterior of the house - Dust or fumes created by home renovations - Pottery made with lead glazes, jewelry made from lead or lead alloys - Older lead based water pipes - Lead dust brought home clothing of the caregivers who work with lead products.
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LEAD POISONING Therapeutic Management
Chelation is the term used for removing lead from circulating blood and, theoretically, some lead from organs and tissues.
70
Lead Poisoning Nursing Care Management
prevent the child’s initial or further exposure to lead.
71
CHILD MALTREATMENT
1. Facts - refers to any form of abuse or neglect that harms a child’s well being. - can have severe, long term effects on a child's physical, emotional, and psychological development 2. S/Sx: - Physical signs: Bruises, burns, fractures, malnutrition - Emotional signs: Depression, anxiety, withdrawal, aggression - Behavioral signs: Fear of certain adults, self harm, sudden changes in school performance - Sexual abuse indicators: Inappropriate sexual behavior or knowledge, difficulty walking/sitting
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Types of Child Maltreatment
1. Physical abuse - intentional use of force that causes harm, injury, or physical suffering 2. Emotional (Psychological) Abuse - repeated behaviors that harm a child’s self worth or emotional development. 3. Sexual Abuse - Involves any sexual act forced upon a child. 4. Neglect - most common form of maltreatment. - failure of a parent or other person legally responsible for the child’s welfare to provide for the child’s basic needs
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Types of Neglect
1. Physical neglect -deprivation of necessities, such as food, clothing, shelter, supervision, medical care, and education. 2. Emotional neglect - failure to meet the child’s needs for affection, attention, and emotional nurturance.
74
CHILD MALTREATMENT Nursing Care Management
1. Each child may manifest different responses to neglect, depending on the situation and developmental age of the child. 2. goal of the interview is to determine whether the child is in a safe environment and whether the caregiver has the skills and resources to care for the child. 3. Report it to... - to Department of Social Welfare and Development (DSWD) - Council for the Welfare of Children (CWC) - Local Barangay and Social Welfare Offices or law enforcement - Philippine National Police Women and Children Protection Center (PNP WCPC) 4. Provide support to the child by listening and reassuring them 5. Encourage professional help , such as counseling or medical care 6. Intervene safely if possible (if you are in a position to help)