Hemiplegia Flashcards

(32 cards)

1
Q

What histories are important in hemiplegia?

A

Headache, seizures, LOC

Speech deficits, sensory loss and weakness of face/limbs

Risk factors for stroke (HTN, smoking, DM)

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2
Q

What classic upper limb posture will be adopted in hemiplegia?

A

Arm held to the side, elbow flexed and fingers and wrist flexed on to the chest

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3
Q

What classic lower limb posture will be adopted in hemiplegia?

A

Limb extended at both hip and knee

Foot plantar flexed and inverted

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4
Q

What will be the muscular symptoms in hemiplegia?

A

Unilateral weakness

Increased tone, hyper-reflexia and upgoing plantar response (UMN deficit)

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5
Q

Which upper limb muscle groups are most affected by hemiplegia?

A

Shoulder abducters

Elbow extensors

Wrist and finger extensors

Small hand muscles

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6
Q

Which lower limb muscle groups are most affected by hemiplegia?

A

Hip flexors

Knee flexors

Dorsiflexors and evertors of the foot

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7
Q

What are some special things that you should look for on examination in hemiplegia?

A

Homonymous hemianopia and sensory inattention

Horners syndrome

Carotid bruits

Speech defects

AF/murmurs

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8
Q

If the patient had horner’s syndrome contralateral to the hemiplegia what does that suggest?

A

Carotid dissection

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9
Q

What are some geriatric causes of hemiplegia?

A

Vascular event (thrombosis, embolism, haemorrhage)

Tumour

Subdural haematoma

Syphilis

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10
Q

What are some paediatric causes of hemiplegia?

A

MS

Sickle cell disease (most common?)

Neoplasm

Trauma

Neurosyphilis

Intrarcanial infection

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11
Q

If you have a patient that has hemiplegia secondary to an intracranial infection, what else should you look for?

A

Underlying AIDS

Otitis media

Cyanotic heart disease

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12
Q

What is first line therapy for a suspected stroke?

A

Aspirin as soon as possible

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13
Q

What is a possible complication of aspirin therapy for stroke?

A

Increased risk of haemorrhagic stroke

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14
Q

For ischaemic stroke, what early changes can you sometimes see on CTB?

A

Loss of white/grey matter differentiation

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15
Q

What investigations should you perform in a patient with hemiplegia?

A

CTB/MRI

FBE, BSL, INR/aPTT

ECG, pO2

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16
Q

What are some adult causes of hemiplegia?

A

Seizures and postictal deficits

Migraine

Systemic infection

Neoplasm

17
Q

Why is it important to measure BSL in acute stroke patients?

A

Hyperglycaemia associated with stress response of the stroke associated with poor prognosis

Hyperglycaemia worsens ischaemic damage

Hyperglycaemia worsens penumbra salvage

Hyperglycaemia worsens results of re-canalisation and increases chance of intracerebral haemorrhage

18
Q

T/F BSL monitoring is important in acute stroke management, but intensive serum glucose control is not

A

T

No difference found in morbidity or mortality with intense glucose control vs normal therapy

Intense control only associated with higher risk of iatrogenic hypoglycaemia

19
Q

Is it important to assess if the person can swallow correctly?

A

Yes

Dysphagia is a common complication but important for preventing aspiration pneumonia

20
Q

Should tPA (alteplase) be given in the setting of acute ischaemic stroke, and if so, how much?

A

It should, and within 3 hours (but asap after CT/MRI)

0.9mg/kg, 10% of which should be given as a bolus and 90% of which should be infused over 1/24

Maximum dose of 90mg, thus maximum bolus of 9mg

21
Q

How should a TIA patient be managed?

A

Aspirin

Advise re risk factors

Ultrasound/digital subtraction angiography of carotid arteries

MRI

22
Q

Why Is it important to differentiate a carotid TIA from a vertebrobasilar TIA?

A

Carotid TIA more amenable to surgery

TIA in anterior circulation is usually more serious prognostically than one in posterior circulation

23
Q

What features are classically present in an carotid TIA?

A

hemiparesis

Aphasia

Amaurosis fugax

24
Q

What features are classically present in a vertebrobasilar TIA?

A

Two of vertigo, dysphagia, ataxia and drop attacks

Bilateral or alternating weakness or sensory symptoms

Sudden bilateral blindness in patients aged >40

25
When is carotid endarterectomy indicated in the case of TIAs?
When carotid stenosis is close to 99% (not 100%, and not much below 80%)
26
Carotid angioplasty (stenting) vs endarterectomy for carotid TIAs?
Possibly better results with angioplasty
27
What is RIND?
Reversible ischaemic neurological disease Like a TIA, but resolves within 1/52 rather than 1/7
28
What is the major risk factor for the development of lacunar infarcts?
HTN
29
Where can lacunar infarcts occur in the brain?
Internal capsule Pons Basal ganglia Thalamus
30
What deficit would a lacunar infarct in the internal capsule produce on examination?
Partial hemiparesis or hemisensory impairment
31
What deficit would a lacunar infarct in the pons produce on examination?
Ataxia of cerebellar type Partial hemiparesis
32
What is the pathogenesis of lacunar infarcts?
Hyaline arteriolosclerosis causing occlusion of small arteries in the brain Or Rupture of Charcot-Bouchard microaneurysms that produce a haematoma, which resolves leaving an area of infarct