HTN - waldron Flashcards

1
Q

how do you properly measure BP

A

have pt relax, sitting in a chair at least 5 min
patient should avoid caffeine, exercise, smoking 30 min prior to measurement
emptied bladder
neither patient or observer should talk during rest period/during measurement
clothing free area

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2
Q

when does screening begin for HTN

A

adults ages 18+ without known HTN at every visit

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3
Q

what is stage 1 HTN

A

systolic: 130-139
or
diastolic: 80-89

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4
Q

what is stage 2 HTN

A

systolic: greater than 140
or
diastolic: greater than 90

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5
Q

what is HTN crisis

A

systolic: > 180
and/or
diastolic: >120

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6
Q

what is the definition of primary hypertension

A

result of sustained increases in blood pressure secondary to increased peripheral resistance and blood volume

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7
Q

What is Masked Hypertension

A

controlled office BPs but uncontrolled BPs in out-of-office settings
risk of CVD and all-cause mortality is similar to that noted in those with sustained HTN

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8
Q

what is the etiology of HTN

A

multifactoral
genetic
ethnicity
environmental factors (stress)
dietary factors (sodium, ETOH)
obesity
aging

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9
Q

what are risk factors for HTN

A

obesity
alcohol abuse
sedentary lifestyle
tobacco use
excessive emotional stress
obstructive sleep apnea
poor diet

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10
Q

what is secondary HTN

A

elevated BP caused by another medical condition
sometimes more difficulty with control
underlining medical illness needs to be addressed

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11
Q

what are potential causes to secondary hypertension

A

Obstructive Sleep Apnea (OSA)
chronic kidney disease
primary aldosteronism
renovascular disease
cushing syndrome

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12
Q

what are drug related causes of HTN

A

NSAIDS
glucocorticoids
sympathominmetic agents
stimulants
alcohol
OCP

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13
Q

what are the signs and symptoms for secondary HTN

A

often asymptomatic
persistent or severe headache
SOB
fatigue
epitaxies
anxiety
feeling of pulsation in the neck or head

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14
Q

how is HTN diagnosed

A

History
PMHx
FamHx
medications
social hx (diet, habits)
exposures (lead)

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15
Q

what is the goal for diagnosing HTN

A

identify end-organ damage, signs of secondary HTN, reversible exacerbating factors

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16
Q

what are the labs to assist with dx HTN

A

urinalysis
CBC
fasting lipid profile
CMP
Optional: uric acid, 24hour urine creatinine and protein

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17
Q

what is the management for HTN

A

treatment goal: attain and maintain target BP
reduce cardiovascular and renal morbidity and mortality
patients < 60yo: <140/ <90
patients > 60: <150 / <90

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18
Q

what are the non-pharmacologic treatments for HTN

A

weight loss
heart-healthy diet
sodium reduction
potassium supplementation, preferably by diet
increase Physical activity
no more than 2 standard drinks per day for men and 1 for women
smoking cessation

19
Q

what are the pharmaceutical therapies for HTN

A

Diuretics
BB
RAAS
CCB
others

20
Q

what medications are indicated for antiHTN with DM

A

ACEi or ARBs

21
Q

what medications are indicated for antiHTN with HF

A

ACEi, ARBs, BB, Thiazide diuretics, aldosterone antatgonists

22
Q

what is the primary cause of secondary HTN

A

sleep apnea

23
Q

what medications are indicated for antiHTN with MI

A

BB, ACEi (with LV dysfunction)

24
Q

what medications are indicated for antiHTN with Chronic Kidney disease

A

ACEs, ARBs

25
what medications are indicated for antiHTN with isolated systolic HTN
diuretics (preferred), calcium antagonists
26
what type of medication is more effective at reducing strokes in AA patients
CCBs are more effective than ACEi
27
what HTN medications are recommended in CKD, Heart disease
ACEi/ARB
28
what is resistant HTN
persistent HTN despite 3+ medications - one must be a diuretic at adequate dose
29
what is the most common cause of resistant HTN
poor adherence to therapy
30
what is malignant HTN
severe HTN (BP > 180/110)
31
What is a Hypertensive emergency
WITH end-organ damage dissecting aortic aneurysm acute pulmonary edema acute coronary syndromes (STEMI, NSTEMI, unstable angina) renal injury encephalopathy stroke eclampsia *requires hospitalization
32
what is severe asymptomatic HTN
WITHOUT end-organ damage usually chronic outpatient management
33
what is hypertensive urgency
very high BP (diastolic pressure > 120-130) acute complications are unlikely, so immediate BP reduction not required start 2 drug oral antiHTN combination with close eval of treatment efficacy, continued outpatient very high BP without organ damage commonly occurs in highly anxious patients or those who have had very poor sleep quality over a period of weeks
34
what is the treatment for hypertensive emergencies
admit to ICU immediate short -acting BP reduction with IV drugs - nitroglycerin, nitroprusside, labetalol, esmolol, hydralazine
35
how do you work up hypertensive emergencies
ECG, urinalysis, serum BUn and creatinin chest XR
36
what is hypertensive encephalopathy
brain dysfunction secondary to excessively high BP SUDDEN ONSET (most cases) -HA, V, balance difficulties, confusion complications include seizures, retinal hemorrhage
37
what are risk factors for hypertensive encephalopathy
kidney failure, abrupt medication discontinuation, pheochromocytoma, MAOI + tyramine, eclampsia
38
what are the causes of orthostatic hypotension
inadequate autonomic response (neurogenic) OR insufficient intravascular volume or circulation (non-neurogenic)
39
what are the risks for orthostatic hypotension
older adults (>70yo), polypharmacy, peripheral neuropathy, neurodegenerative d/o
40
what is the pathophysiology of orthostatic hypotension
autonomic dysfunction affecting baroreflex - no change in HR or BP severe volume depletion - increase HR meds 40% have no definite cause
41
what are the symptoms with orthostatic hypotension
in response to change in position, usually vs gravity dizziness/lightheadedness visual blurring neck pain, HA 50% have systolic HTN when supine/sitting
42
what is the treatment for orthostatic hypotension
avoid meds related to orthostasis modify diet treat underlying causes modify activity may consider compression stockings consider medication therapy
43
what are the complications of HTN
increases afterload - leads to congestive HF, increases myocardial oxygen demand arterial damage - accelerated atherosclerosis, aneurysm, dissection, kidney disease, stroke