III. Maternal & Fetal Monitoring Flashcards

(126 cards)

1
Q

How many stages of labor?

A

3

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What stage is the longest stage?

A

Stage one

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Begins with uterine contractions and continues until cervix fully dilated to 10cm.

A

Stage 1

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Begins with cervical dilated to 10 cm until delivery of Fetus

A

Stage 2

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

stage: Until delivery of placenta

A

stage 3

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

The ideal labor epidural should cover sensory loss from ____ to ____.

A

T10 to S5

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

First Stage of labor:

Afferent/Efferent nerve impulses from the lower uterine segment and cervix cause visceral pain

A

Afferent

Afferent = sensory
Efferent = motor

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

First Stage of Labor:

Nerve cell bodies are located in the dorsal root ganglia of ____ to ____.

A

T10 to L1

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What dermatome level is T10

A

umbilicus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

what stage has pain that is poorly localized?

A

Stage 1

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

what stage of labor includes somatic pain that is well localized?

A

Stage 2

Afferent nerves innervating the vagina and perineum causes somatic pain which is better localized.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Stage 2 Labor:

somatic pain impulses (from the vagina and perineum) travel primarily via the ____ to dorsal root ganglia of S1-S5

A

pudendal nerve

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

How to assess a sensory nerve block:

A
  1. Explain the procedure and purpose to the patient
  2. Choose tool (ice cube, cold alcohol 4x4, broken tongue depressor)
  3. Establish Control (Ice): place the ice on an area well away from the possible dermatome cover such as the neck or face and ask if they feel cold.
  4. Apply the ice to an area likely blocked on the same side of the body and ask the patient, does this feel the same cold or different?
  5. Apply ice to areas above and below this point until it is clear at which level the top and bottom of the block is covered.
  6. Repeat the procedure on the opposite side of the body, as blocks may be uneven or unilateral.
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Dermatome level:

C4

A

Clavicles (C is 4 clavicles)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Dermatome Level:

T4

A

Nipples (T is 4 tips of the nips)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Dermatome Level:

T6

A

Xiphoid

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Dermatome Level:

T10

A

Umbilicus (0 looks like belly button)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Dermatome Level:

S1

A

Pinky Toe

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Dermatome Level:

L1

A

Inguinal Line (top of bathing suit tan line - L1 is Lifeguard 1)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

name of syndrome that affects pregnant women when laying supine = ↓ venous return and CO.

A

Aortocaval Compression Syndrome

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

When does aortocaval compression syndrome begin during pregnancy?

A

16-20 weeks gestation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Signs and symptoms of Aortocaval Compression Syndrome?

A
  • HoTN
  • Pallor
  • Sweating
  • Nausea & Vomiting (2/2 to HoTN)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

Treatment for Aortocaval Compression Syndrome?

A

Left Uterine Displacement (LUD)

  • wedge under right hip
  • 15º tilt
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

LIst HTN disorders experienced during pregnancy:

A
  1. Gestational HTN/Pregnancy Induced HTN (PIH)
  2. Pre-Eclampsia
  3. Eclampsia
  4. Sever Pre-Eclampsia
  5. HELLP Syndrome
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
What is considered Gestational HTN/PIH?
139/89 after 20 weeks
26
Etiology of Gestational HTN/PIH
**1. Abnormal sensitivity to catecholamines & hormones 2. Fetal maternal antigen antibody reaction's** 3. Production of vasoactive prostaglandins (Thromboxane A & Prostacycline)
27
What is a major symptom of preeclampsia that is missing in Gestational HTN/PIH?
proteinurea
28
Gestational HTN/PIH usually resolves by ____ postpartum and treatment is NOT needed.
12 weeks
29
Preeclampsia: BP: ____ Proteinurea: ____ Sx: ____
>140/90 >300mg/24 hr *Edema, Headaches, Visual disturbances, Hyperreflexia*
30
Severe Preeclampsia: BP: ____ Proteinurea: ____ Sx: ____
160/110 >5g/24 hr *HELLP Syndrome (*8Hemolysis, Thrombocytopenia*, ↑Liver Enzymes, ↓PLT count)*
31
The presence of what hallmark symptom graduates preeclampsia to eclampsia?
Seizures
32
Because these HTN pregnancy disorders can affect ____, we should monitor closely and give careful consideration to regional anesthetics.
blood coagulation
33
What coag studies are we interested in for pregnant HTN patients?
Full coag studies: - PT - PTT - Fibrinogen
34
Most providers require what range of platelets IOT consider regional anesthesia?
70-100k *consider trend in PLT count*
35
Is it safe to remove epidural catheter if PLT low?
Catheter should be left in Get another set of labs and wait until PLT is safe range (100+ ideally), maybe for couple days
36
HELLP syndrome is a life threatening condition and is considered a sub-variant of ____.
Severe Pre-Eclampsia
37
HELLP stands for:
H: Hemolysis (RBC breakdown = Hgb breakdown = ↑Bilirubin) EL: Elevated liver enzymes (ALT, AST) LP: Low PLT count/thrombocytopenia (<100k)
38
What major vital may be normal with HELLP syndrome, delaying diagnosis?
BP
39
HELLP Tx:
- Transfusion - Bedrest - Continuous monitoring of mom & baby - Mg - manage HTN if present
40
At what point do we treat HTN in pregnant patients?
BP >159/109
41
What are common drugs used to treat PIH?
- Oral Hydralazine (most common) - Labetalol - Clonidine - Nifedipine - NTG/SNP
41
What are specific treatments for Preeclampsia?
1. Manage HTN (same as PIH) 2. Seizure Prophylaxis: Mg Sulfate (2 g/15min ≤ 4-6 g loading dose followed by 1-2 g/hr) 3. Definitive treatment is delivery of baby **[BQ] (Board Question)**
42
Specific treatments for Eclampsia
1. Prevent aspiration 2. Manage airway 3. Control seizures - Midazolam 1-2 mg - Ativan 2-4 mg - Diazepam 5-10 mg *(Textbook answer)* - THEN *Mg Sulfate 1-2 g/hr*
43
Mg Sulfate helps with both seizure prophylaxis and ____.
BP management
44
If mother starts seizing while pregnant, what procedure will take place immediately.
STAT c-section
45
uses for Mg Sulfate:
1. Prevention of Eclampsia & Seizures 2. Tocolytic (inhibits uterine contraction, slow/stops premature labor) 3. Cerebral Protectant for premature babies
46
Therapeutic level of Mg Sulfate
4-8 mEq/L (4.8-9.6 mg/dL)
47
Patients receiving Mg Sulfate should be closely monitored for ____.
Mg Toxicity
48
T/F: Mg Toxicity symptoms are easily discernable in pregnant patients.
False *are the first symptoms of magnesium toxicity are fatigue, nausea and vomiting, blurred vision, EKG changes. These symptoms are not uncommon in otherwise healthy pregnant patients*
49
What EKG changes are a result of magnesium toxicity?
1. Prolonged PRI 2. Widened QRS
50
Mg Sulfate dose dependent side effects table picture
51
Treatment for Mg toxicity?
1 g Calcium Gluconate IV over 10 min
52
What consideration should anesthetist make if a patient is on magnesium at therapeutic levels?
1. Mg potentiates NMB : *Reduce doses of Rocuronium* 2. Tocolytic ∵ can cause post-partum hemorrhage: Hemabate, Methergine, *Pitocin*
53
3 types of fetal monitoring
1. Auscultation 2. Electronic Fetal Monitoring 3. Internal fetal monitoring
54
2 Auscultation methods:
1. Fetoscope (low-tech) 2. Portable Doppler
55
Benefits of Auscultation methods:
1. Quick & portable 2. Detects Baseline 3. FHR rhythm and dysrhythmias 4. Helps detect changes in heart rate 5. Differentiates maternal from fetal HR
56
Limitations of Auscultation methods:
1. No printout: *cannot assess changes over time* 2. Not continuous: *acute changes can be missed* 3. Cannot assess variability 4. Maternal position limiting (supine difficult) 5. Difficult to use on obese patients
57
Electronic Fetal Monitoring (EFM) uses what two external belts?
1. Doppler (FHR monitor) 2. Tocodynamometer (contraction monitor)
58
The tocodynamometer uses a ____ that records uterine contraction duration & intervals.
pressure transducer
59
Electronic fetal monitoring picture
60
Electronic fetal monitoring picture 2
61
EFM Benefits:
1. Noninvasive 2. Continuous documentation 3. Not labor intensive 4. Shows variability (over time) 5. *Works well independent of pt position*
62
EFM Limitations:
1. Restricts patient movement 2. Not ECG but measures cardiac motion 3. Double counting or picks up maternal heart tones *(especially if mom HR is tachycardic)* *newer technology is wireless and does NOT restrict movement*
63
Internal Fetal Monitoring uses a ____ ____ electrode.
fetal spiral
64
what must occur before the fetal spiral electrode is used for fetal monitoring?
The **amniotic sac must be broken** in order to allow obstetrician to place electrode vaginally through uterus onto **baby's forehead**.
65
What method is most accurate assessment of fetal HR?
Internal Fetal Monitoring via Fetal Spinal Electrode (FSE)
66
Benefits of FSE:
1. Detects FHR variability 2. Detects dysrhythmias (R-R interval monitoring on EKG) 3. Mother may have mobility *new tech has wireless models to allow for ambulation*
67
Limitations of FSE:
1. **Can be used only after the membranes of the amniotic have ruptured (after "water breaks”)**: ↑risk of infection (invasive) 2. Discomfort during placement 3. Contraindicated in patient’s with herpes outbreak ↑risk of transferring virus to baby 4. Contraindicated in HIV+ women due to infection risk. *likely not an option during early labor*
68
2 methods of monitoring uterine activity:
1. Tocodynamometer: external 2. Intrauterine pressure catheter (IUPC): Internal
69
Main limitation of external tocodynamometer
hard to use in obese patients and dependent on position
70
Main benefit of external tocodynamometer:
noninvasive
71
Main benefits of IUPC:
1. More accurate uterine pressure (mmHg) monitoring 2. Accurate timing of heart rate changes with contraction
72
Main limitation of IUPC:
Invasive
73
Where is tocodynamometer placed?
over the fundus of uterus *superior of the two belts on the abdomen*
74
Internal Fetal Monitoring Fetal Spinal Electrode Picture
75
How do we determine the baseline of the FHR?
the average FHR over 10 min, rounded to the nearest 5 bpm
76
Normal fetal heart rate (FHR)
110-160
77
what does fetal heart rate best indicate clinically (i.e., what other vital is it closely correlated with)?
how well oxygenated the baby is (oxygenation status)
78
Two classifications of clinical changes in fetal heart rate:
1. Accelerations 2. Decelerations (Early, Late, Variable)
79
FHR: The Graph Display the intervals between the vertical red lines represent ____ minute/s.
1 minute
80
FHR: The Graph Display Fetal heart tracing is displayed in the **lower/upper** pane?
upper
81
FHR: The Graph Display what is displayed in the lower pane of the FHR graph?
uterine activity
82
FHR Display Graph Picture
83
what is the normal beat-to-beat variability in a healthy fetus?
5-25 bpm *measured from peak HR to lowest HR*
84
beat to beat variability: Minimal: _____ Moderate: _____ Marked: _____
Minimal: >5 Moderate: 5-25 Marked: >25
85
The following situations can decrease B-B variability:
1. **Hypoxia/Acidosis** 2. Congenital anomalies ø Anencephalic fetus: born w/o part of brain/skull 3. Medication Administration (↓CNS + Cross Placenta) ø Narcotics ø Sedatives/Anesthetics ø High dose anticholinergics
86
If signs of fetal distress are present what should be done first?
alert obstetrician immediately
87
Determine variability:
Normal "Moderate" Variability
88
Determine variability:
Minimal *BUT this is sign of distress: there should be some degree of variability coinciding with contractions*
89
Fetal Tachycardia range:
>160 bpm
90
causes of fetal tachycardia
1. Recovery following asphyxia 2. Maternal or fetal infection 3. Catecholamine administration 4. Tachydysrhythmias 5. Thyrotoxicosis
91
Fetal Bradycardia range:
<110 *for >2 MINUTES*
92
Causes of fetal bradycardia
1. Acute hypoxia ø Idiopathic and benign if short lived 2. Drugs: ø Beta Blocker ø LA administration
93
Most dangerous fetal rhythm
FHR < 60 bpm *will result in cardiac decompensation*
94
what are some things nurses may do as first line if baby is experiencing bradycardia?
- supplemental O2 - change mom position (all fours)
95
If baby's heart rate does not return to normal (after bradycardia) after a couple minutes, what will the next course of action?
STAT C-section
96
Temporary increase in FHR
Acceleration
97
requirements to considered "acceleration"
<32 weeks: 10 bpm **>15 sec, but <2 min** *>32 weeks: 15 bpm **>15 sec, but <2 min***
98
Causes of fetal accelerations
Fetal movement Fetal stimulation Maternal contractions
99
Accelerations are said to correlate with ____.
fetal well-being *reassuring sign; if they occur often mother may get supplemental O2*
100
Gradual decrease and return to baseline
Deceleration
101
the lowest point of decel (slowest FHR):
Nadir
102
Early or late decels are termed in relation to ____.
uterine contraction
103
EARLY decels are said to occur when the FHR decreases coincide with ____.
onset of uterine contractions
104
Early Decels: Time from onset to the lowest point of deceleration is ____.
≥30s
105
Early decels: ____ occurs with the peak of contraction.
Nadir
106
Common etiology of early decels:
**Fetal head compression** (2/2 uterine squeeze) Mild hypoxia (well-tolerated)
107
Early decels are caused by ____ produced when the head is compressed by uterine contraction.
vagal response
108
Onset and depth of early decelerations mirror ____.
the shape of the contractions
109
Early decelerations tend to be proportional to ____.
the strength of the contraction.
110
Early decels graph picture
111
T/F: Early decels are typically benign, common, and not an emergent situation.
True
112
Graphically speaking, what is the difference in early and late decels?
- Late decels do NOT initiate with the onset of contraction; rather, the **onset is at or after peak contraction.** - **The Nadir occurs AFTER peak contraction (shifted right).**
113
Late decelerations are characterized by decreasing FHR waveform and a return to baseline. The onset to Nadir is ____ seconds.
>30 sec
114
Common etiology of late decelerations:
**Uteroplacental insufficiency** (prolonged asphyxia & fetal hypoxia) *Hypoxia* causes bradycardia, not contraction like in early decels
115
What type of decel?
Late decels
116
An abrupt, visually apparent decrease in the FHR below the baseline & recovery
variable deceleration
117
Variable decelerations have what onset to nadir?
<30 seconds
118
Common etiology of variable decelerations:
1. Cord Compression ø Due to oligohydramnios *(↓ Amniotic Fluid)* 2. Sustained head compression (vagal response)
119
Variable decelerations have a ____ relationship to contraction.
variable (duh)
120
if decel has a Nadir that occurs after peak contraction, it is either a "LATE" decel or "VARIABLE" decel; how can we decide?
LATE: onset to Nair > 30sec VARIABLE: onset to Nadir < 30 sec ("abrupt"
121
What type of decel?
Variable decel
122
What type of decel?
Variable Decel
123
The more severe & sustained the bradycardia = the more severe the hypoxia = the higher the ____ of the baby
stress
124
Besides sustained bradycardia, what are the two worst rhythms?
- late decels - variable decels
125
VEAL CHOP mnemonic picture