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Blood Vessels > Immune System > Flashcards

Immune System Flashcards

(71 cards)

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1
Q

Already ready to fight

A

Innate

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2
Q

Gets better at fighting over time

A

Adaptive

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3
Q

Skin better at keeping pathogens out

A

Innate defenses

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4
Q

Macrorgansims takes up space on skin by pathogenic flora

A

Innate defenses

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5
Q

Getting rid of it Hurts us

A

Microbiome

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6
Q

Present in wet entries to our body

A

Mucosa

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7
Q

Still have a layer of epidermis that acts as a physical wall

A

Additional specialized defenses

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8
Q

Digest pathogen on food

A

Avoid of stomach acid

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9
Q

Degrade pathogenic cells

A

Digestive enzymes

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10
Q

Forms a sticky trap for microorganisms in our lungs and digestive tract

A

Mucin

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11
Q

Second line of innate defenses guards against pathogens that enter deeper tissue

A

Primary static defenses

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12
Q

Phagocytosis

A

Macrophages
Neutrophil
Dendritic cell

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13
Q

After ingesting the offending pathogen

A

Phagosome

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14
Q

Containing cuasic enzymes bind with the vesicle containing the pathogen forming a phagonlusome

A

Lysosome

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15
Q

Blocking the lysosome

A

Mycobacterium tuberculosis

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16
Q

Waiting in tissue like skin and the digestive tract to ingest and destroy invaders

A

Macrophages

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17
Q

Call for backup from neutrophils in the blood

A

Cytokines

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18
Q

Degranulate release toxic granules on and around the pathogen

A

Neutrophils phagocytosis

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19
Q

Non specific methods

A

Damage other tissue

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20
Q

Present in surface tissue and can phagocytose

A

Dendritic cells

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21
Q

Best at antigen presentation through their

A

MHC-2

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22
Q

Involved in inflammatory response

A

Innate defense

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23
Q

Mast cells have bags of

A

Histamines

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24
Q

Very similar to mast cells

A

Basophils

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25
Injured cells release chemical signal
Pro inflammatory cytokines
26
Release histamine
Mast cells
27
Histamine encourages
Vasodilation
28
Increase vascular permeability
Histamine
29
Encourages recruitment of more leukocytes
Histamine
30
Eat pathogens and tease cytokines calling for assistance
Macrophages
31
Show how sn aid a fight
Neutrophils
32
Made up of cell debris and neutrophils that have litterbugs eaten themselves to death
Plus
33
Pathogens can’t handle wide swings In temp and Turk tip tempt o kill them enough
Fever
34
Cel easily survive this range
Fever
35
Part of innate defenses
Nk
36
Macrophages, dendritic cell, B cells take chunks of the pathogen and attach it to there cell membrane
Major histocompatibility complex-2
37
Also called paPc
B cells
38
Protein takes up exogenous antigens
Major histocompatibilty complex
39
Proteins that are captured floating around and presents them on surfaces
Exogenous antigens
40
Destroy pathogens as well as inflammation and fever
Neutrophils and macrophages
41
Immune can fight pathogens they have seen before
Adaptive response
42
Portions of a pathogen that were destroyed after phagocytosis are attached to the Mhc-2 on surface of -
Professional antigen presenting cells
43
B cells produce
Unique antibodies
44
Naive B cells produce a few antibodies and attach them to their cell surface
Membrane bound antibodies
45
Rights know you ha e the means to protect your body against a new artificial Virus
From the antibodies your body already has
46
B cells in lymph nodes bump | Into pathogen they have a. Small chance there _ can bind with pathogens surface antigens
B cell receptor
47
Once a B cell is activated
No longer naive
48
It phagocytosis the pathogen and
Attaches the antigen to its own mhc-2
49
Long term immunological memory of previyos pathogens
Memory B cell
50
Antibody factories for this current round of infection
Plasma cell
51
Antibodies produced by plasma cells travel through ecf and blood looking for
More of that pathogen antigen to bind
52
In thymus T cells differentiate into 2 types
Helper T cells | Cytotoxic T cells(killer)
53
T cells are lymphocytes and have a protein on their surface called
T cell receptor
54
Huge amount of variety in there shape and can only bind to a small percentage of antigens
TCR
55
When a Particular TCR is able to bind to an paPc, Mhc-2
Helper T cell is activated
56
Go out and turn into an infection
Effector helper T cell
57
Stored and enable a fast response during next infection
Memory helper T cells
58
Bind to an antigen is activated | Ensures resources are directed to the most efficient cells
Only TCR
59
Perform two major actions
Helper effector T cells
60
Constantly being created on every cell of the body
Proteins
61
DNA or rna and the cells own genetic machinery starts to express the viral information Creates more virus protein Peices
Viral infections
62
Broken genes create odd new proteins
Cancerous cells
63
Constantly presenting any protein in any cell
Mhc-1
64
The immune system checks these proteins aa weld or not slef When a protein is aberrant the immune system targets for destruction
Mhc-1
65
Produce normal proteins and frequently scooped up by Mhc-1
Should not be destroyed
66
Non self Protoens
Mhc-1 alerts that something is wrong
67
Cyctoxic T cells and nk cells don’t bind with mhc-2
Attach to Mhc-1
68
NKattach to 2 thongs glycoproteins the stimulate the NK cell to kill suspect cell
Activating ligands
69
NK cell attach to 2 things doesn't care if it presenting a normal virus or not
MHC-1 complexes
70
they vote to kill you or not predisposed to kill
natural killer
71
A blend of adaptive an d innate, anotbodies bout to a pathogen increase the rate of destruction by NK cells
Antibody Dependant Cell-mediated Cytotoxicity

Blood Vessels (4 decks)

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