infections Flashcards

1
Q

Most common organism in preseptal cellulitis

A

Staph Aureus

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2
Q

most common cause of proptosis in children

A

orbital cellulitis

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3
Q

orbital cellulitis

A

often secondary to sinusitis
can have associated subperiosteal abscess
Fevers, decreased VA, RAPD, proptosis, pain with eye movements, restricted EOM, optic disc swelling

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4
Q

organisms most commonly causing orbital cellulitis

A

Staph aureus (most common in children)
Strep pneumo
fungi (phycomycetes - most aggressive)

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5
Q

Ophthalmia Neonatorum

Types and time spans

A

Within first month
papillary conj (cant have follicular reaction 2/2 immature immune system)
Chemical - 0-24 hrs - silver nitrate or povodine iodine
Nessaria Gonorrhea - 1-2 days - Hyperacute conjunctivits
Other bacterial 4-5 days - strep, staph, h. flu, enterococci
HSV - 5-14 days (70% type 2)
Chlamydia 5-14 days - most common neonatal infection

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6
Q

Ophthalmia neonatorum

Diagnosis and treatment by type

A

chemical - no tx
gonococcal - Gram neg intracellular diplococci,
IV ceftriaxone for 7 days, bacitracin oint
bacterial - GS/culture erythromycin/bacitracin oint
HSV - scraping with multinucleated giant cells
IV acyclovir, for 10 days + Zirgan/Viroptic
Chlamydia - gram pos intracytoplasmic inclusions on Giemsa stain
Azithromycin PO, topical erythromycin

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7
Q

Prophylaxis against ophthalmia neonatorum at birth

A

topical tetracycline or erythromycin ointment at birth

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8
Q

Pediatric bacterial conjunctivitis - organisms

A

H Flu (50-65%)
Strep Pneumo (20-30%)
Moraxella (10%)
Staph - rare

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9
Q

Vernal Keratoconjunctivitis

A

seasonal allergic conjunctivitis (warm months)
M > F
RF: Atopic dermatitis and atopy
Findings: Horner trantas dots (eosinophils at limbus) cobblestone papilla on tarsus, limbal follicles, pseudomembranes
Shield ulcer

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10
Q

Ligneous conjuncitvitis

A

rare bilateral psudomembranous conjunctivitis in young girls
Exaggerated response after injury surgery or trauma
white thinckened avascular ligneous mass with highly vascularized raised, friable lesion on peripheral conj
affects all mucous membranes

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11
Q

Kawasaki’s Disease

age, race, findings,treatment, associations

A

children under 5
japanese, but epidemics also with high risk in siblings
fever
bilateral conj, bilateral mild iritis (with perilimbal sparing - hallmark)
cervical lymphadenopathy
oral lesions (strawberry tongue)
rash/desquamation of hands and feet
polyarthritis
TX: aspirin
assoc: coronary arteritis - coronary artery aneurysm (incr risk with steroid tx)

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12
Q

Interstitial keratitis

A

congenital syphilis
starts age 5-20, triggered by minor trauma
3 stages:
progressive - pain, photophobia, KP, ciliary flush, ground-glass corneal opacity
florid - cute inflammatory respons - salmon patch of hutchinson (deep vascularization makes cornea look pink)
Retrogression - vessels meet at center of K
ghost vessels (stromal, just ant to Descemet’s) - late finding
can get secondary glaucoma from iris/angle damage
Tx - steroids, penecillin

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13
Q

congenital syphylis

A

maternal transmission after 4th month
Interstitial keratitis
ectopia lentis
argyll robertson pupils
optic atrophy
panuveitis with salt and pepper retinopathy (usually)
Systemic: hutchinson’s teeth, frontal bossing, saddle nose, deafness, tabes dorsalis, saber shins

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14
Q

Hutchinson’s triad

A

interstitial keratitis
hutchinson’s teeth (peg-shaped)
deafness

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15
Q

HSV

A

asymptomatic primary infection

ocular involvement in 10% of disseminated disease

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16
Q

Perinaud’s oculoglandular syndrome

A

unilateral
Conjunctival granuloma
lymphadenopathy

Etiology: Cat Scratch fever
Tularemia
Pasteurella
LGV
Mycobacteria
Sperochetes
17
Q

Congenital Rubella

A

cataracts (bilateral - pearly white nuclear with retention of lens nuclei)
Live virus within lens (extreme inflammation with extraction)
Salt-and-pepper fundus
glaucoma (with not get both cataract and glaucoma)
microphthalmos (15%)

Also: cardiac defects (PDA), deafness, MR