Integumentary disorders Flashcards

(25 cards)

1
Q

5 layers of epidermis

A

Stratum corneum - Luciderm - granulosum - spinosum - basale

- Vit d produced in last 2, nutrients and fluid diffuse to epidermis via blood vessels in the dermis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Dermis

A
  • connective tissue (elastin and collagen)
  • nerve endings, lymph and blood vessels
  • receptors for pain, pressure, heat, & cold
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Functions of skin

A

First line of defense! innate immunity

  • prevents excessive fluid loss
  • controls body temp
  • sensory info against environmental hazards
  • synthesizes ad activates Vit d
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Older adults

A
  • flattening of the basement membrane
  • loss of elastic fibers
  • weakened collagen
  • decreased subcutaneous fate
  • thinner stratum corneum
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Skin lesion treatment

A
  • antihistamine (itching)
  • local wound care
  • systemic meds if severe
  • surgical removal
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Descriptive information

A

Location of skin lesion

  • length of time lesion has been present
  • change that have occurred over time
  • physical appearance: color, elevation, texture, type of exudate, presence of pain or pruritus
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Pressure injury

A

Prolonged pressure on bony prominence, friction, shearing, maceration, dehydration, malnutrition, poor circulation
- 2 hrs in bed, 15 min in chair, 35 degree S-L, head of bed < 30 degree

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Pressure injury stages

A
  1. Alteration in intact skin temp, consistency, sensation, persistent redness
  2. Loss of epidermis/dermis, blister abrasion, partial thickness
  3. Full thickness / subcutaneous, deep crater
  4. Full thickness, necrosis, muscle, bone, tendon, capsule
    - unstageable: Escher / necrotic tissue requires debridement
    - deep tissue injury
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Treatment of pressure injury

A
  • pressure relief
  • wound care: clean, debride, infection control, moisture balance, nutrition, address circulation
  • exercise
  • hi volt e stim
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Diabetic (neuropathic) wounds

A
  • typically on the plantar surface
  • callus common on wound edge
  • tunneling common
  • foot deformities contribute to excess pressure
  • decreased “protective sensation” leads to lack of feeling irritants
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Diabetic wounds

A
  1. Risk factors
    - poorly controlled blood sugars
    - poor nutrition
    - obesity
    - peripheral neuropathy
  2. Testing: monofilament for protective sensation
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Treatment of diabetic wounds

A
  1. Prevention of infection
  2. Off loading
  3. Surgical debridement
  4. Growth factors
  5. Manage blood glucose levels
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Venous wounds

A
  • venous insufficiency
  • gaiter area of leg
  • edema
  • superficial wound (no tunneling)
  • lots of drainage
  • common with severe lymphedema
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Treatment of venous wounds

A
  • wound care
  • compression
  • treat underlying medical conditions (fluid control and infections)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Arterial wounds

A
  • caused by poor blood perfusion
  • dry wound bed
  • gangrene
  • punched out wound edge
  • DO NOT COMPRESS
  • many require vascular surgery to restore blood flow
  • high amputation risk
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Inflammatory skin disorders

A

A broad category that includes many conditions, ranging in severity from mild to itching to serious medical health complications

17
Q

Treatment of inflammatory skin disorders

A
  • refer to physician (meds and phototherapy)
  • identify and remove irritant
  • apply emollients
18
Q

Auto-immune: psoriasis

A
  • chronic inflammatory skin disorder of unknown origin
  • onset usually occurs in the teen years
  • has remissions and exacerbations
  • lesions are commonly found on the face, scalp, elbows, and knees
19
Q

Auto-immune: scleroderma

A
  • a group of rare, progressive diseases that involve the hardening and fighting of the skin and connective tissues
  • may occur as a skin disorder, or it may be systemic, affecting the viscera
  • excess collagen
  • unknown cause
20
Q

Eschar

A
  • a slough or dead tissue that sheds from healthy skin

- eschar may be shed naturally or with debridement to prevent infection

21
Q

PT interventions

A
  1. Debridement / wound care
    • remove eschar and non-viable tissue
    • sharp debridement, autolytic, enzymatic, mechanical, maggots
  2. Techniques to increase ROM/function
    • stretching, splinting, positioning, compression
  3. Techniques to increase muscle strength/function
    • exercise
    • assess for appropriate assistive devices
22
Q

Pressure garments

A
  • control edem and the formation of hypertrophic scars by applying pressure to the wounded area
  • aid in reducing the effects hypertrophic scarring there by reducing scarring and deformities
23
Q

Have you made these connections?

A
  1. S&S = impairments
  2. Healing is predictable (time and sequence)
  3. Diagnosis does not equal acuity
24
Q

Cellulitis

A
  • common, potentially serious bacterial skin infection
  • swollen, red area of skin, feels hot and tender, may spread rapidly
  • occurs when one or more types of bacteria enter through a break in the skin
  • typically caused by staph or strep
  • can occur at any location on the body, but is most common in the lower legs
25
Impetigo
Highly contagious infection, spread by direct contact with hands, eating utensils, and towels - usually caused by staph or strep - commonly on the face - presents as small vesicles that develop into yellowish-brown crusts that is red beneath they crust and exudes a honey colored liquid - treatment includes topical antibiotics in the early stages