Intrapartum management Flashcards

1
Q

Contraindications to forceps delivery

A

Face presentation
Vertical transmission risks
Fracture risk
Relative: suspected fetal bleeding disorder

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2
Q

Vacuum vs forceps for preterm delivery

A

Higher risk of subgleal haematoma and scalp trauma with vacuum
- avoid vacuum if <32/40, caution if 32-36/40

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3
Q

Classification of fetal head position wrt OVD

A

Outlet:
-fetal scalp visible witout parting labia
- OA/ OP, ROA/P, LOA/P
- rotation doesn’t exceed 45 degrees
Low:
- vertex +2, not caput
- <45deg or >45 deg from OA/OP
Mid:
- fetal head no more than 1/5 palpable abdominally
- <45 deg or >45 deg from OA/OP
High:
- not recommended if head >/= 2/5

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4
Q

Placement of vacuum

A

3cm from post fontanelle
Halfway point = flexion point

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5
Q

Indications for OVD

A

Fetal compromise
Maternal:
- shorten second stage (eg med condition)
- fatigue/ exhaustion
Inadequate progress:
-P0: no progress 3 hours passive/ active w analgesia; 2 hours w/out analgesia
- P1: No progress w 2 hours passive/ active w analgesia; 1 hour w/out analgesia

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6
Q

Prerequisites for OVD

A

Verbal consent
Abdo/ VE:
- </= 1/5 palpable per abdo
- Vx presentation
- fully dilated and ROM
- need to know position for instrument placement
- assess caput/ moulding
- ? adequate pelvic
-? US

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7
Q

Preparation for OVD

A

Explain and consent (written consent if trial in OT)
Appropriate analgesia
Mat bladder empty
Aseptic technique
Staff - adequate facilities, lighting, staff, knowledge
Anticipate cx: failure, SD, PPH
Epis scissors: MLE to prevent OASIS, 60* angle

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8
Q

Failure risks with OVD

A

Increased failure risk with:
- BMI >30, EFW >4kg, OP, head >1/5
- vacuum > forceps

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9
Q

Risks assoc w OVD

A

OASIS
- 3rd deg w kiwi 8:100 w forceps
Incr risk of bleeding 4:100
If failure - increase risk of fetal head impaction
Vacuum - cepalohaematoma, retinal hge, maternal worries re baby
– dec risk vaginal/ perineal trauma
– no more likely assoc w CS, low Apgar or need for phototx

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10
Q

Aftercare following OVD

A

IV dose co-amox
Reassess VTE
NSAIDS and paracetamol
Document void
? retention - PVR, UC
Offer physio at 3/12 PN to reduce UI

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11
Q

Factors assoc w breech presentation

A

Prev breech birth
Premature labour
High parity
Multiple pregnancy
Polyhydramnios
Oligohydramnios
Uterine anomales
Pelvic tumour or fibroids
Placenta praevia
Hydrocephaly/ anencephaly
Fetal neuromuscular disorders
Fetal head and neck tumours

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12
Q

Complications breech delivery

A

Cord prolapse 1% (0.5% w cephalic)
Fetal head entrapment
PROM
Birth asphyxia
ICH
Intrapartum death
BPI
Rupture of liver/ kidneys/ spleen
Dislocation or fractures

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13
Q

Mx twin delivery after first twin delivered

A

Assistant to stabilise fetus in longitudinal lie
Continuous CTG
US - FH/lie
Oxytocin when lie confirmed
ARM only when fixed, best with contraction
Aim for birth within 30 min

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14
Q

Mx twin delivery if second twin transverse

A

ECV
Internal podalic version
- one or both feet grasped inside uterus –> breech extraction
- don’t ARM too early –> cord prolapse
- ** caution for head entrapment

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15
Q

Length of inter-twin birth interval

A

30 min

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16
Q

Prevention of OASIS

A

MLE considered for OVD
- 60 degrees from midline when perineum distended
Perineal protection at crowning
Warm compression during second stage

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17
Q

Pointers on OASIS repair

A

OT
Regional/ general anaesthesia
Good lighting
Vaginal pack for excessive bleeding
Avoid figure of 8s- good for haemostasis but cause ischaemia
PR before and after

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18
Q

Technique for repairing anal mucosa

A

Continous or interrupted

19
Q

Technique for repairing IAS

A

Interrupted OR
mattress without any attempt to overlap IAS

20
Q

Technique for repairing full thickness EAS

A

either overlapping OR
end to end

21
Q

Technique for repairing partial thickness EAS

A

end to end

22
Q

Suture choice for OASIS

A

Anorectal mucosa:
- 3-0 polyglactin (less irritation and discomfort than PDS)
IAS/ EAS:
- monofilament (3-0 PDS) OR
- modern braided (2-0 polyglactin)
- mattress without any attempt to overlap
Burying of knots beneath superficial perineal muscles recommended to minimise knot and suture migration to skin

23
Q

Post-op mx OASIS

A

Broad spec Ab - reduce infection and dihiscence
Laxatives
Physio
R/V 6-12/52
Incontinence or pain at review –> refer gynae/ colorectal

24
Q

Prognosis of OASIS

A

60-80% asymptomatic 12/12 after repair

25
Q

Normal umbilical vein and artery blood gas values

A

pH: v 7.35/ a 7.28
pCO2: v 38/ a. 49
pO2: v 29/ a 18
BE: v -4/ a -4
HCO3: v 20/ a 22

26
Q

FBS interpretation

A

pH:
>7.25 - normal - no action
7.20 - 7.25 - borderline - repeat in 30-60min if not delivered
<7.20 - abnormal - deliver (OVD/ CS)

27
Q

What is oxytocin and MOA

A

Mammalian neurohypophysial hormone
Produced in supraoptic and paraventricular nuclei of hypothalamus
= neuropeptide
Causes uterotonic stimulation and myoepithelial cell contraction
Uterine sensitivity to oxytocin markedly increases at onset of labour

28
Q

What is syntocinon and formulation

A

Synthetic nonapeptide identical to oxytocin
5u in 1ml and 10u in 1ml

29
Q

Clinical problems assoc w intrapartum oxytocin use

A

Fetal intolerance
Uterine overcontracting- tachysystole
- = 7 contractions in 15min in nullip/ 5 contractions in 15min in multip
- hypertonus - prolonged contraction
- hyperstimulation - tachysystole or hypertonus –> NRCTG

30
Q

Contraindications to oxytocin

A

Non-reassuring fetal trace
Overcontracting
Prev. scar - prev uterine perf, classical CS or myomectomy
Transverse lie/ oblique presentation
Malpresentation

31
Q

Extra caution with oxytocin in which patients

A

Prev CS
Grand multip
PTL
Hx. of MI/ CVS due to effects on BP and HR
Long QT interval

32
Q

Risks assoc w oxytocin use

A

Fetal hypoxia (esp FGR)
Uterine rupture (multips)
Water intoxication assoc w mat/neonatal hypoNa
Interaction with inhaled anaesthetics –> hypoTN, cardiac rhythm disturbances
PGE may potentiate uterotonic effect

33
Q

Infusion rate of oxytocin - augmentation

A

6-30ml/hr
(1-5mU/min)
Increase by 1-5mU/min every 15-30 minutes up to a max of 30mU/min (180ml/hr)

34
Q

Absorbable suture materials in LSCS + rationale for use

A

Gut
Polyglactin (vicryl) - 60 days
- braided polyfilament
- less likely to loosen at knot
- inc tissue rxn/ infection
Polyglecaprone (monocryl) - t1/2 = 7-14/7
- monofilament
- 20-30% breaking strength at. 2/52
- completely absorbed by 100/7
Polydioxane (PSD) - 200 days
- monofilament
- longest lasting

35
Q

Non-absorbable sutures used at LSCS

A

Clips - remove after 5-7/7
Nylon
Polypropylene (prolene)

36
Q

Needles used at LSCS and rationale

A

Round bodied:
- use @ friable tissues
Cutting needle:
- triangular shape w 3 cutting edges
- penetrates tough tissues (eg skin)
Reverse cutting needle:
- cutting surface at convex edge
- good for tough tissue (eg tendon/ subcut)
- decr risk of cutting through tissue
Point:
- blunt: abdo wall closure, friable tissue
- sharp: pierce and spread tissue w minimal cutting

37
Q

Risk of bad outcome with IOL for postdates

A

1 in 200

38
Q

Intrapartum management of VBAC

A

Review chart and confirm safe for VBAC
Obs team notified
16G IVC, FBC and GXH
One-to-one midwife care
CEFM

39
Q

Signs and symptoms of uterine rupture

A

Prolonged, persistent and profound bradycardia
AbN FHR. suggesting fetal compromise
Abdo pain, acute onset scar tenderness
AbN progress in labour, prolonged first or second stage
Vaginal bleeding
Cessation of prev efficient uterine activity
Loss of station of presenting part
Chest pain or shoulder tip pain
Maternal tachy, hypoTN or shock

40
Q

Absolute contraindications to VBAC

A

classical CS
Uterine rupture
Placenta praevia
Declines VBAC

41
Q

Relative contraindications to VBAC

A

breech
macrosomia
twins
2x previous

42
Q

Risks with VBAC

A

Most serious= uterine rupture, 0.2-0.7%, higher if IOL or augmented labour
- Note: CTG, PVB, maternal decline, pain out of proportion, cessation of regular contractions, change in stage
Subsequent pregnancies: r/o PAS, hyst
Inc TTN w ERCS
Success rate: 72-75%, prev SVD 85-91%
EmLSC

43
Q
A