Intro to visual perception Flashcards

1
Q

History - Extramission

A

-reached out of your eyes + you touch things through your eyes

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2
Q

History-Democritus

A

-realised vision was occurring in the brain
-subtle atoms come from object into eyes
-see by eyes alone
-visual carter sees, eyes are just the way in
-visual images are burned
-vision is a relative not absolute sense-depends on context

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3
Q

History-Plato

A

-Actively opposed of experiment
-vision is very complicated
- can’t predict now things will look
- no one will ever be able to explain it
-resulted in no experiments done for hundreds of years

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4
Q

History -Alhazen

A

-Did experiments
-drew accurate anatomical eyes
-light rays coming off an object carry info to you
-light travels in a straight line
- while light composed of many colours

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5
Q

History-Leonardo da Vinci

A
  • To make his paintings good had to understand how light works
  • As things go far away become less saturated
    -first view of real eyes
    -very accurate drawing
    -understood anatomy not physics of eye
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6
Q

History-Hermann von Helmholtz

A

-invented numerous things for looking into eye
- can’t look at someones eye because you need light to see and you block it when looking at someone
-look through telescopes with 2 lights placed nett to head that bane oft mirror + into eye
-Able to see balk of eye
-invented ophthalmoscope

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7
Q

since Helmholtz

A

-visual system is most studied understood part of brain
-provides quantitative, experimentally -verified link between changes in cells of the eye how we see

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8
Q

Iris

A

-muscle to expand + contract to control amount of light entering

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9
Q

Anatomy

A
  • correa = main lens
    -Aquas humour: fluid in anterior chamber
    -iris = muscle
    -pupil = hole in iris
  • ciliary body = makes aqueous humor
    -ciliary muscle = controls lens shape
    -virous humour = jelly between lens retina
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10
Q

Travelling light

A

-light comes from bottom
-cones + rods send signals to lots of cells
-cascade of info travelling to ganglion cells
-very complicated
-wiring under photorecepters act as little computer
-light enters retina is processed + computed rich signal is sent to brain
- L, M + s rods
-rods operate at night
- cones operate when there is more light

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11
Q

The fovea

A

-foveal pit formed by ganglion cells pulled back to help us see better
-gives photorecepters unimpeded view of world so central vision isn’t impacted by cell presence
- This is where all high resolution vision is so very little short wavelength because it is densely packed
- No rods in central - used to see in daytime
-no blue cones (short wavelength)
- more red than green cones
-red- long wavelengths

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12
Q

Rods

A
  • more sensitive to light than cones
    -provide low-resolution blurry black + White night vision
    -cores give you sharp colour vision in light
  • Because there are no rods in the ford you can’t see things you directly look at in the dark
    -Rods peak as you move away from centre
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13
Q

Adaptation- photoreceptors adapt to the average light

A

-if you KeeP something in the same place on your retina, photoreceptors adapt to it + it becomes harder to see
- prolonged fixation causes disappearance because photoreceptors constantly ask What is the normal thing + need to adapt to local env
-photoreceptors first see difference but don’t like it so change sensitivity so cant see difference

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14
Q

Adapt- photonreceptors respond to lights of different intensities

A

-schnapf et al 1990
- Although light pulses increase linear, gap between lines on graph are not constant
-once a photoreceptor is adapted, you flash light at different intensities
-response depends on adaptation state

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15
Q

where is the blind spot?

A
  • 15 degrees from centre (fovea)
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16
Q

what do you see in your blind spot?

A

-Blind spot doesn’t look like a hole
- it is hard to see even with 1 eye closed
-Brain makes stiff to fill it in
-Brain makes up most of your visual field

18
Q

Recording from cells in the retina

A

-mountcastle, Kuffler, Barlow
- Bright bar moved ground on screen
-electrodes placed in an anathetised cat brain
-electrons are in contact with 1 + neurons in the visual cortex
-experimenter waves stimulus around until finch place that stimulates neuron
- Amplifier takes signal from cortex + increases them to display on screen
-Neuron only responds when centre is illuminated indicates visual field it detects

19
Q

receptive Fields

A

-on-centre RF may pool from signals on the retina
-Array f core cells contribute to whole RF
-Different sub-regions of RF
-signals from centre core are pod
-signals surrounding are neg
-Ada them = output that’s sent from retina to brain by ganglion cells

20
Q

receptive fields overlap

A

-photoreceptors can provide excitatory input to 1 RF+ an inhibitory to another
-indiv cones contribute to different RF
-RFs can have excitatory and inhibitory regions
-Rs can be on or off centre
-on centre like while spots on dark background
-Bottom lines dark on white

21
Q

Lateral inhibiton

A

-in the Rf the surround inhibits the centre
-Antagonism between neighbouring region is known as lateral inhibition
- Neg surrounds interact with pos centre

22
Q

can we see the effects of RF’s Hermann

A

-Hermann + scintillating grid
-one Rf is positioned over a junction
-receives bright input to centre + surrounding
-Rf can be between lines receiving input to centre less on sides

23
Q

why do we have RF?

A
  • respond to structured inputs
    -Pattern matching
    -ignores changes in average brightness
    -Turns visual world into an invariant code: contrast
  • combo of light adaption + RF (ganglion cells) lets us extract a constant representationof the world
24
Q

Myobia -short sightedness

A

-exposure to outdoor light can welp prevent short sightedness
-eyeball is too long

25
Hyperopia presbyopia-long sightedness
- Hyperopia = genetic origin -presbyobia = lens hardens with age you can get both presbyobia + myopic -Age-related macular deg on = 50% of eye problems -glaucoma = 18% -diabetic retinopathy=17%
26
Glaucoma-crushed wires
-damage to nerve cells (wiring) because high intraocular Pressure -Aqueous humour cant drain from eye - Because angle between corned iris is closed OR -because trabecular meshwork tissue inside that drainage system is blocked = pressure build up -central visual field is fine but surrounding is dark + minimal
27
Macular Degeneration-peeling walls
-degen of retina + death of photoreceptors often age related -centre of visual field is invisible -50% of cases -cells in centre Start breaking down then expands out -wet form = blood vessels start leaking -drugs can stop this to slow it down -drastic occurance -out of no where
28
Diabetic retinopathy-leaking pipes
-vision is lost in patches across visual field -Blood vessels leak -Treatment = laver beam to shut down blood vessels