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Item Question Flashcards

1
Q

What is PUD

A
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2
Q

Sites

A
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3
Q

Kothay besi hoy?

A

1st part of duodenum

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4
Q

Causative organism

A

H.pylori

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5
Q

Virulence kivabe kore?

A
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6
Q

(H.Pylori)Test kivabe korba?

A

Urea breath test

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7
Q

Enzyme produced by H pylori

A

Urease

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8
Q

What are the defensive mechanism of gastroduodenal mucosa

A
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9
Q

What are the damaging force

A
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10
Q

Classify drugs for peptic ulcer disease

A
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11
Q

What are the systemic antacids

A
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12
Q

What are the non systemic antacid

A
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13
Q

Which is better?systemic or non systemic?

A

Non systemic

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14
Q

why non-Systemic is better?

A

Not absorbed
No acid base abnormalities

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15
Q

Criteria of ideal antacid

A
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16
Q

Criteria

A
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17
Q

Bazar a j antacid pawa jay Tate ki thake?

A

Mg(OH)2 and Al(OH)3

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18
Q

Benefit of combination of magnesium hydroxide and aluminium hydroxide

A

MOD-magnesium hydroxide causes osmotic diarrhea
aluminium hydroxide causes constipation
So in combination they minimise GIT adverse effect

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19
Q

What are the ppi’s?

A
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20
Q

mechanism of Action proton pump inhibitor

A
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21
Q

Adverse Effects of omeprazole?

A
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22
Q

Why ppi cause subnormal vit B12 level?

A

Decreased ifc

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23
Q

Why infection increases in ppi?.

A

HCl secretion less,less first line defence

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24
Q

Why ranitidine was banned?

A

Carcinogenesis
presence of low levels of an impurity called N-nitrosodimethylamine (NDMA). NDMA is classified as a probable human carcinogen (a substance that could cause cancer) based on animal studies.

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25
Adverse effect of ranitidine?
26
Why cimetidine banned?
27
Is cimetidine a drug inducer or drug inhibitor?
Inhibitor Increase warfarin toxicity
28
What happens if you give ranitidine injection rapidly?
Bradycardia Hypotension So we will never give rapidly
29
What is prostaglandin analogue?
Misoprostol
30
NSAID induced peptic ulcer mechanism
NSAID is acidic So it is unionised in acidic media(stomach) It is absorbed to parietal cell. Irritation bleeding perforation korbe
31
Which is best drug for NSAID induced PUD
Misoprostol
32
Why misoprostol used in NSAID induced PUD
Prostaglandin improves mucosal blood flow Inhibits the HCl Regenerative capacity increase Secretion barabe
33
What are the cytoprotective agents?
34
Mechanism of sucralfate?
35
Adverse effects of bismuth
36
Triple therapy with dose?
37
PPI continue korba kotodin?(in triple therapy)
4-6 wk
38
Name laxatives with classification
39
A patient has came with constipation which drug is given?
Firstly non pharmacologic approach Then laxative
40
Mechanism of ispaghula husk
41
Adverse effects of ispaghula husk
42
Contraindications of ispaghula husk
43
Mechanism of lactulose?
44
Adverse effect of lactulose?
45
Indication of lactulose?
46
Contraindications of lactulose?
47
Enema kkhn use korbo?
48
What are the anti emetics?
49
A patient with hyperemesis in 1st trimester,what will you give?
Cyclizine,meclizine
50
What will you give in nausea vomiting motion sickness?
Scopolamine Cyclizine Meclizine Promethazine
51
Cancer chemotherapy patient hole ki DBA?
Ondansetron oi group
52
Indication of metoclopramide?
53
Why metoclopramide given in post partum lactation instead of domperidone?
BBB and placenta cross
54
Which antiemetic cause extra pyramidal syndrom?
55
Metoclopramide mechanism
56
Ondansetron,granisetron indication
57
Mechanism of ondansetron
58
Where is chemoreceptor trigger zone?inside BBB or outside?
59
BBB cross kore na amn drug a emesis hoy?
Ha hote pare..Karon chemoreceptor trigger zone BBB er baire thake
60
Name drugs for IBS?
61
What is the role of TCA in IBS ?
62
What are the drugs for IBD ?
63
What are the drugs given in diarrhea?
64
Why loperamide is used instead other opioid?
65
Composition of ors ?
66
Role of glucose in ORS?

Drugs For PUD (2 decks)

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