Jakirlic Flashcards

(34 cards)

1
Q

Increased diameter of ONH

A

Stage 3 moderate

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2
Q

Normal temporal disk,

No elevation

A

Stage 1 very early

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3
Q

Complex metamorphopsia

A

Parietal tumor/stroke, parietal seizures and migraine. Persist covering both eyes

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4
Q

Brain tumor can cause

A

Complex hallucinations

Simple and complex illusions

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5
Q

Migraines

A

Simple hallucinations

Complex metamorphopsia

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6
Q

Pareto-occipital tumor/stroke causes

A

Complex Illusion:
Diplopia not changing covering both eyes
Polyopia
Palinopsia

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7
Q

Drug abuse

A

Can cause complex illusion called Palinopsia

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8
Q

Bilateral Occipito-Temporal lobe tumor/stroke

A

Complex Illusion-akinetopsia “motion blindness “

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9
Q

Hallucinations cause : simple

A

Retinal disease (PVD, tear, RD)
Optic nerve disease
Migraine: visual aura

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10
Q

Hallucinations cause : complex

A
Dementia
Metabolic disease
Illegal drug use
Occipital tumor/stroke
Occipital epilepsy
Charles Bonnet Syndrome
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11
Q

Transient Visual Loss-Monocular

A

Incipient retinal vascular occlusion (CRAO, CRVO)
Retinal vasospasm (ocular migraine)
GCA
Papilledema (IIH) (usually bilateral but can be unilateral due posture change)
Amaurosis fugax (hypoperfusion)
Cardiac valvular disease

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12
Q

Most common cause of monocular vision loss

A

Amaurosis fugax (hypoperfusion) d/t carotid artery stenosis

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13
Q

Most common cause of binocular visual disturbance:

A

migraine

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14
Q

Non-migraine TVL

A

sign of serious vascular disease

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15
Q

Transient Visual Loss-Binocular

A

Migraine
Papilledema (IIH)
Vertebrobasilar insufficiency
Postural hypotension

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16
Q

Amaurosis Fugax

A

Sudden onset of severe visual loss in one eye
All or part of monocular visual field
Lasts seconds to minutes
Completely resolves within 10 minutes (can last up to 1 hour)
Fellow eye unaffected
No other neurologic symptoms
No symptoms of GCA
50+ year olds with cardiovascular risk factors

17
Q

Amaurosis Fugax: examination

A

Normal ocular examination in both eyes

May visualize Hollenhorst plaque in retinal arteriole

18
Q

Amaurosis Fugax Workup

A
Laboratory;
CBC with differential
ESR & CRP
Hypercoagulable state in young patients
Carotid artery investigation
19
Q

Ocular Ischemic Syndrome

A

Entire eyeball suffers from hypoperfusion
Severe ICA stenosis or occlusion
GCA

20
Q

Ocular Ischemic Syndrome Symptoms

A

Transient monocular visual loss or blur improved with laying down
Orbital ache improved on laying down
Dull ocular pain all around the eye that improves laying down.
Transient neurologic symptoms: contralateral weakness or numbness

21
Q

Ocular Ischemic Syndrome signs

A

Decreased BCVA
Decreased IOP
Mild anterior uveitis
Retinal venous engorgement and tortuosity
Mid-peripheral retinal hemorrhages (classic sign)
Iris neovascularization
Cataract (forms quicker)

22
Q

Ocular Ischemic Syndrome workup and treatment

A

Investigations: carotid doppler, ESR, CRP
Treatment
Carotid endarterectomy: symptomatic patients with severe 70-99% stenosis

23
Q

Vasospastic monocular TVL

A

retinal migraine
Typically young adult
Same eye affected every time

24
Q

Vertebrobasilar Insufficiency

A

Age 50+
Sudden onset visual loss in both eyes
Vision recovers completely within minutes (can take up to 1 hour)

25
Chiasmal Disease
``` Vision loss; Typically painless Progressive Bilateral asymmetric Headache possible ```
26
Anything to Anterior to chiasm and LGN
optic atrophy and contralateral APD possible
27
Posterior the LGN,
no optic atrophy and RAPD.
28
Post-Chiasmal Disease
Homonymous: nasal VF of one eye and temporal VF of fellow eye Congruous: nasal and temporal defects closely resemble each other Decreased VA not common unless lesion also involves optic nerve or occipital lobes Usually due to cardiovascular disease (stroke)
29
Optic Tract Lesions
Tumors Ischemic stroke Aneurysms of superior cerebellar or PCA
30
Lesions of optic radiations
``` Vascular occlusions (common causes) Primary & secondary tumors Trauma Incomplete incongruous quandranopsias Neurologic deficits predominate ```
31
Neurologic symptoms predominate | Temporal Lobe Lesions
Seizures Hemiparesis (loss of motor control ) Hemisensory loss (loss of sensation) Aphasia
32
Neurologic symptoms predominate | Parietal Lobe Lesion
Hemiplegia (paralysis of one side of the body.) Hemisensory loss Visual neglect Aphasia
33
Lesions of the visual cortex
will Not have any neurologic defect .
34
Calcarine cortex lesions;
always due to stroke, completely Congruous no neurological deficits +/- macular sparing