Knee 2 (test 3) Flashcards
(93 cards)
attachments/path of the ACL
attaches centrally and anteriorly on tibial plateau
runs posteriorly, superior, and lateral
attaches at lateral aspect of intercondylar fossa
ACL restrains what
excessive anterior tibial glide
secondary restraint to tibial IR
prevalence of ACL injury
20% all knee injury
younger females
non modifiable risk factors for ACL injury
female > males
2 weeks following start of period = ligament laxity
bony morphology:
-narrow intercondylar femoral notch
-post tibial slope and hyperextension both correlated with non contact ACL
congenital hypermobility
modifiable risk factors for ACL injury
high shoe-surface interaction/friction
high BMI
possible risk factors we can change for ACL injury
bracing = inconsistent benefit
muscle strength
-lower overall with ACL tears
-ham/quad ratio strength
loading pattern
why would hamstring quad ratio affect ACL injury
predicts LE control
hamstrings prevent anterior translation
lower ratio present in females vs males
what altered loading patterns affect the ACL
impaired LE control
loading patterns happen earlier and nearly 2x faster with impaired control
thus this causes decreased knee flexion with larger ground reaction forces/harder landing (land with stiff knee to offset possible knee valgus that would occur otherwise)
what can you see with impaired LE control
increased dynamic knee valgus and hip add
very easy to see with vertical drop test
poor control is defined by
significant valgus movement
knee is medial to foot
reduced control is defined by
some valgus movement
knee not entirely medial to foot
good control is defined by
no valgus
knee vertical to toes
what might you see with the trunk with ACL injury/modifiable risk factor
greater trunk lean toward support limb
greater trunk RT toward support limb
what does it mean that those at higher risk for ACL injury have greater activation of visual motor strategy vs sensory motor strategy
relying heavily on their eyes for focus
have them close their eyes and movement is poorer/less stable
risk factors for secondary ACL injury
same as primary ACL plus excessive femoral IR moment
% of ACL injuries that are contact vs non contact
non contact = 50-70%
contact = 30%
functional questionaires for ACL
international knee documentation committee (IKDC)
Knee Outcome Survey (KOS)
typical sprain S&S
empty painful endfeels (acute)
limited motion if acute, excess if not
popping
pain with distraction
joint laxity
+ special tests for ligament
symptoms of ACL injury
consistent with any ligament injury
effusion, popping, AND giving away following trauma
WBing activities with likely giving away
signs of ACL injury
consistent for ligament sprain plus
ROM is limited paingul particularly with hyper ext and IR
special tests for ACL
anterior drawer + (possible - if HS tight)
lachmans (possible false neg due to blocked anterior glide via severe swelling that tightens capsule, HS guarding, or meniscal tear)
+ pivot shift
other possibly + for additional tissue damage (i.e. meniscus, MCL, etc)
ACL injuries are often accompanied by arthrogenic muscle inhibition of quads due to what factors
pain
effusion (joint swelling)
-involved knee inhibition
-uninvolved knee inhibition
-amount of swelling not always correlated to amount of inhibition
joint laxity/giving away
muscle weakness/correlation
NOT due to denervation
ACL injuries are often accompanied by arthrogenic muscle inhibition of quads due to what factors
pain
effusion (joint swelling)
-involved knee inhibition
-uninvolved knee inhibition
-amount of swelling not always correlated to amount of inhibition
joint laxity/giving away
muscle weakness/correlation
NOT due to denervation
what does arthrogenic muscle inhibition mean
it starts at the joint/because of a joint issue