Knee 2 (test 3) Flashcards

(93 cards)

1
Q

attachments/path of the ACL

A

attaches centrally and anteriorly on tibial plateau

runs posteriorly, superior, and lateral

attaches at lateral aspect of intercondylar fossa

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2
Q

ACL restrains what

A

excessive anterior tibial glide

secondary restraint to tibial IR

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3
Q

prevalence of ACL injury

A

20% all knee injury

younger females

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4
Q

non modifiable risk factors for ACL injury

A

female > males

2 weeks following start of period = ligament laxity

bony morphology:
-narrow intercondylar femoral notch
-post tibial slope and hyperextension both correlated with non contact ACL

congenital hypermobility

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5
Q

modifiable risk factors for ACL injury

A

high shoe-surface interaction/friction

high BMI

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6
Q

possible risk factors we can change for ACL injury

A

bracing = inconsistent benefit

muscle strength
-lower overall with ACL tears
-ham/quad ratio strength

loading pattern

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7
Q

why would hamstring quad ratio affect ACL injury

A

predicts LE control

hamstrings prevent anterior translation

lower ratio present in females vs males

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8
Q

what altered loading patterns affect the ACL

A

impaired LE control

loading patterns happen earlier and nearly 2x faster with impaired control

thus this causes decreased knee flexion with larger ground reaction forces/harder landing (land with stiff knee to offset possible knee valgus that would occur otherwise)

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9
Q

what can you see with impaired LE control

A

increased dynamic knee valgus and hip add

very easy to see with vertical drop test

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10
Q

poor control is defined by

A

significant valgus movement

knee is medial to foot

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11
Q

reduced control is defined by

A

some valgus movement

knee not entirely medial to foot

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12
Q

good control is defined by

A

no valgus

knee vertical to toes

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13
Q

what might you see with the trunk with ACL injury/modifiable risk factor

A

greater trunk lean toward support limb

greater trunk RT toward support limb

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14
Q

what does it mean that those at higher risk for ACL injury have greater activation of visual motor strategy vs sensory motor strategy

A

relying heavily on their eyes for focus

have them close their eyes and movement is poorer/less stable

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15
Q

risk factors for secondary ACL injury

A

same as primary ACL plus excessive femoral IR moment

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16
Q

% of ACL injuries that are contact vs non contact

A

non contact = 50-70%

contact = 30%

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17
Q

functional questionaires for ACL

A

international knee documentation committee (IKDC)

Knee Outcome Survey (KOS)

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18
Q

typical sprain S&S

A

empty painful endfeels (acute)
limited motion if acute, excess if not
popping
pain with distraction
joint laxity
+ special tests for ligament

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19
Q

symptoms of ACL injury

A

consistent with any ligament injury

effusion, popping, AND giving away following trauma

WBing activities with likely giving away

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20
Q

signs of ACL injury

A

consistent for ligament sprain plus

ROM is limited paingul particularly with hyper ext and IR

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21
Q

special tests for ACL

A

anterior drawer + (possible - if HS tight)

lachmans (possible false neg due to blocked anterior glide via severe swelling that tightens capsule, HS guarding, or meniscal tear)

+ pivot shift

other possibly + for additional tissue damage (i.e. meniscus, MCL, etc)

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22
Q

ACL injuries are often accompanied by arthrogenic muscle inhibition of quads due to what factors

A

pain

effusion (joint swelling)
-involved knee inhibition
-uninvolved knee inhibition
-amount of swelling not always correlated to amount of inhibition

joint laxity/giving away

muscle weakness/correlation

NOT due to denervation

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22
Q

ACL injuries are often accompanied by arthrogenic muscle inhibition of quads due to what factors

A

pain

effusion (joint swelling)
-involved knee inhibition
-uninvolved knee inhibition
-amount of swelling not always correlated to amount of inhibition

joint laxity/giving away

muscle weakness/correlation

NOT due to denervation

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23
Q

what does arthrogenic muscle inhibition mean

A

it starts at the joint/because of a joint issue

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24
muscle inhibition of quads with ACL injury leads to
atrophy/more inhibition/weakness; deficits common 2-4 years after injury determined by observation, palpation, and muscle testing need to take seriously and communicate importance of staying on top of exercises to pt can also affect other side/knee as well
25
PT Rx/prognosis for ACL injury if they are not returning to high risk activities
most can return to low risk activity without sx with good outcomes
26
3 primary/early goals for ACL injury
full/near full ROM (especially ext) minimal to no swelling quad activation/endurance/coordination
27
how to ensure ACL pts obtain full ROM
immediate mobilization in PT for ROM, pain, and minimizing immobilization effects full ext should be obtained no later than 4 weeks (wont happen with everyone, especially if other structures like meniscus are involved) -predicts ext at 12 weeks and lower risk of OA
28
what may indicate quad activation/endurance with ACL injury
SLR without extension lag (bend in leg) quad set > 90% of uninvolved side (could be misleading if uninvolved side is also experiencing inhibition)
29
PT Rx for ACL strain
early WBing as symmetrical as possible w/o detrimental effects POLICED functional bracing (comes later); more beneficial with ACL deficiency, conflicting support with ACL reconstruction weak support for continuous passive motion devices
30
why is bracing inconsistently supported for injury
little accessory movements/translations can still occur within brace for ACL, outside of hyperext, brace wont prevent any other motions that could also cause pain
31
MT for ACL
initiate post op
32
describe purpose of neuromuscular electrical stimulaiton for MET for ACL
neuromusclular electrical stimulation (NMES) for activation/coordination/strength: significant increase in quad strength 'no significant changes with function isometrics at varying angles based upon symptoms discontinue once quad index > 80% uninvolved side
33
general MET parameters for ACL
assumptions must be made about arthrogenic muscle inhibition intense resistive training without inducing pain must be performed eventually emphasize both concentric and eccentric training
34
non WBing vs WBing activities guidelines for ACL
load is generally greater with non WBing activities than WBing because open chain/non WBIng only activates quads but WBing will generally activate HS and other muscle groups as well to counteract the force of the quads and provide more symmetrical muscle activation across the joint non WBing or OKC activities are less of a concern than in the past greatest loads are found within 50 degrees of full ext with both WBing and non WBing (so i.e. you can do a knee ext from 90 to 45 only or with closed chain you dont want the pt to squat with knees over toes)
35
how does the stress on the ACL change with squatting, lunging, and leg press
increases with knees beyond toes decreased with fwd trunk lean
36
walking guidelines/general exercise guidelines with ACL injury
with walking there is as much load as non WBing knee ext due to repetitive terminal knee ext this is several times greater than other WBing activities this means that both OC and CC knee activities early and often is OKAY...especially if they are walking and using correct trunk and LE control
37
goals for hamstring strength and coordination emphasis of ACL Rx
males: hams > 66% of quad activity females: jams > 75% of quad activity in females this value could be skewed if there is quad inhibition present
38
neuromusclular training that should be present in an ACL Rx
normal strength does not equal proper neuromusclular or LE control trunk proprioception and kinesthesia to minimize lean and twist
39
components of LE proprioception and kinesthesia that should be incoorporated with neuromuscular training for ACL Rx
minimize frontal and transverse plane motion promote sagittal plane knee and trunk flexion decrease GRF with softer landing progressive speed and difficulty emphasis on jump landing and balance
40
timing for MET Rx for ACL
needs to be at least 2-3x/week for 6-10 months bilateral, for cross edu = less deficit compared to only exercising involved knee
41
effectiveness of blood flow restriction
no better than intensive exercise can increase growth hormone but can also decrease myostatin which would limit cell growth (cancels out)
42
importance of motor learning for ACL MET
important to improve movement patterns use language they can understand and help cue proper movement
43
examples of motor learning with a external focus
improved balance/coordination higher vertical jump more force production greater knee flexion softer landing
44
how could you improve motor learning by adding observation to practice
competition with others (motivation/responsibility) real time feedback/post exercise feedback
45
plyometrics for ACL
increased loading with rate of deceleration (need to control descent with good movement patterns) vertical jump similar loading to NWB ext on ACL (with proper movement patterns)
46
what % of ACL tears include meniscal tears
22-86%
47
how can meniscal involvement with ACL tear affect recovery
if a partial meniscectomy there is no change if there is a meniscal repair performed = slower progressions due to greater protection required for meniscus healing may slow achievement of full ROM due to decreased WBing required for said protection
48
how many ACL injuries have bone bruises involved
80% have bone bruise
49
what is the general timeline for recovery with ACL tears involving a bone bruise
if skeletally immature (young) healing = 2 weeks to 3 months skeletally mature healing = 1 month to 1 year average is 3.2 months overall bone bruise is a delaying factor that leads to more difficulty reaching full ext and proper quad function (inhibition)
50
what happens if there is a MCL tear with ACL
MCL tears generally are not surgically repaired if ACL is torn then often surgeons will wait 10 weeks for MCL to heal before repairing ACL (better outcomes vs earlier sx)
51
precuations if there is MCL injury/involvement
only sagittal plane activity for 4-6 weeks limit tibial ER and valgus stresses
52
what % of ACL injuries involve articular cartilage defect
36%
53
what are the treatment options if there is articular cartilage involvemetn
debridement (abrasion) = WBAT for 3-5 days and no delays to ACL rehab osteoarticular transport system (OATS) and autologous chondrocyte implantation (ACI) (cultured) = most conservative guidelines and greatest delays microfx of bone underneath to stimulate bleeding = NWB for 2-8 weeks; delay to ACL rehab ** dont need to know specifics; just know if articular cartilage is repaired then it will delay ACL timeline
54
order of effectiveness of 3 articular cartilage repair options
OATS > ACI > Microfx
55
why is ACL hard to heal/often surgically fixed
clotting repair is inhibited by synovial fluid few return to high risk activity without sx due to continual instability
56
what % ACLs are surgically repaired
65%
57
what are the 3 arthroscopic techniques for ACL reconstruction
bone patellar tendon bone (BPTB) grafts- have both autograft and allogract semitendinosus/gracilis (SGT) graft
58
what is allograft vs autograft
allograft = cadaver autograft = from pt
59
how does pre op weakness affect outcomes of ACL sx
the stronger you start the better you are likely to come out
60
describe a BPTB autograft
incision over opposite patellar tendon remove middle 1/3 of: bone of patella, patellar tendon, and bone of tibial tuberosity
61
what is a complication that may occur with BPTB autograft
up to 1/3 develop anterior knee pain
62
graft strength timeline
initial weakining within 1st 4 weeks incorporation of graft into bone at 6-8 weeks dense fibrous tissue at 8-12 weeks
63
symptom difference with BPTB allograft
symptoms improve faster than the graft incoorporates into the body pt may feel like they can due more since they didn't have the extra trauma of an autograft but it actually takes longer for the donor graft to incorporate into the body than if it was their own tissue
64
graft strength for allograft
incorporation if graft into bone at 8-12 weeks dense fibrous tissue at 8-12 weeks delayed timeline and longer rehab
65
why might one choose an allograft vs auto
if pediatric pt = dont want to interrupt growth plate if pt has already had multiple ACL repairs and no longer has viable tissue to donate can avoid anterior knee pain from graft being taken
66
advantages of STG graft
prepubescent youth to avoid growth plate complications avoids anterior knee pain
67
Rx if STG graft is performed
may start pure strengthening of hamstrings at 6-8 weeks delay heavy strengthening with hams for 12 weeks
68
prognosis for ACL recovery
18-24 months post op muscle weakness and impaired neuromuscular control remain all grafts and bone show continued healing on imaging inhibition, atrophy, and weakness are common out to 2 years and 4 years post op (even in both LEs)
69
prognosis for BPTB grafts at 40 months
45% resumed pre injury level 29% returned to competitive sport
70
failure rate for ACL
up to 30% 75% of 2nd tears occur between 18 and 24 months reduced injury rate by waiting at least 9 months to return to play *worse if meniscal or articular cartilage involvement or ext lag
71
describe PCL
thicker/stronger attaches central/posterior on tibial plateau runs superior/anterior attaches anteriorly on medial aspect of intercondylar fossa
72
PCL primarily restrains what
excessive posterior tibial glide and IR
73
least injured knee ligament
PCL
74
etiology of PCL sprain
hyperflexion primarily but also some with hyperext
75
S&S of PCL injury
consistent with any ligament injury limited and painful ROM (least pain in ER) + PCL special tests
76
special tests for PCL
quads active post drawer post sag reverse pivot shift
77
PT RX for PCL
ligament Rx plus emphasis on limiting posterior tibial gliding
78
describe MCL
flat broad ligament with 2 bands runs from medial condyles of femur and tibial
79
restraint of MCL
anterior band = limits flexion posterior band = limits hyperext
80
MCL attaches to
medial meniscus posterior capsule adj muscle and tendon units SO if you have a pt with MCL sprain you need to check other surrounding structures
81
most injured knee lig
MCL
82
etiology of MCL sprain
excessive valgus and or ER stress and or hyper ext
83
S&S of MCL sprain
general ligament S&S plus impaired ROM least limits with IR + MCL special tests adn possibly medial meniscus special tests TTP
84
special tests for MCL
valgus stress at 0 and 30 degrees more extended position tests other structures like cruciates and capsules
85
Rx for MCL
ligament RX plus early protection with valgus and ER stress and end ranges of flx/ext most wont need sx bc ligament is extraacrticualr and can scar/heal on blended capsule
86
describe the LCL
round cordlike ligemetn attaches to lateral condyle of femur and fibular head no attachment to menisci primary restraint to excessive varus and ER stresses
87
prevalence of LCL injury
string ligament so rarely injured
88
etiology of LCL injury
excessive varus and or ER stress and hyperext
89
S&S of LCL injury
consistent woth ligament injury plus ROM limited and painful especially in ext and ER + LCL special tests TTP
90
special tests for LCL
varus in 0 and 30
91
PT Rx for LCL injury
like ligament Rx plus early protection with varus and ER stress may need sx because ligament remains from the capsule even though its extraarticular
92
MET for all sprains
combo of supervised and HEP better than either alone combo of open and closed chain exercises coordination training