LA ENT Flashcards

ears (192 cards)

1
Q

MC cause of conductive and SN hearing loss

A

conductive: ceruman impaction
SN: prebycusis

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1
Q
A
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2
Q

damaged ossicle, mastoiditis, OM, FB, ETD, secondary to URI, Perf TM, fluid, otosclerosis, cholesteatoma, tumors

A

causes of conductive HL

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3
Q

CNS lesion, neuro d/o, aminoglycosides, loops, meniere, neuroma, labyrinthitis, infection

A

causes of SN HL

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4
Q
A

vertigo

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5
Q

SE of reglan, promethazine

A

extrapyramidal, tardive dyskinesis, dystonia, parkinsonism

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6
Q

dangers of dopamine blockers

reglan, promethazine

A

neuroleptic malignant syndrome

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7
Q
A

Neuro leptic malignancy syndrome
stop dopamine blocker

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8
Q
A

anticholinergics: meclizine, scopolamine,

only 2 meds

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9
Q

avoid anticholinergics in what pts

A

acute angle glaucoma, BPH with urinary retention

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10
Q

can follow a URI. episodic vertigo. 1 minute duration.

No hearing loss, ataxia, or tinnitus.

A

BPPV

do hallpike manuver

dec salt, meclizine

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11
Q

Has hearing loss. Episodic vertigo, No relation to movement, last hours

A

menieres

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12
Q
A
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13
Q

treatment?

A

vestibular on left

laby on right. (unilateral HL)

1st line glucortcoids

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14
Q
A

peripheral on left

central on right

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15
Q

After ETD or URI. PAINLESS otorrhea (brown and odor),
Conductive HL

A

Cholesteatoma

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16
Q

After URI. Fullness, popping, underwater feeling, fluctuating conductive HL, tinnitus

A

ETD

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17
Q

Percussion TENDER posteriorly, FEVER, deep ear pain(worse at night)

next step?

A

Mastoiditis
CT with contrast!! IV antibx (IV vanco plus (piper/taz, ceftazadine, cefepime)

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18
Q

Otalgia, sudden pain relief with bloody otorrhea.
Can lead to cholesteatoma.

avoid what

A

TM perf

aminoglycosides

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19
Q

Autosomal dominant. Slowly progressive hearing loss especially at low frequencies.

next step?

A

Otosclerosis

Tone audiometry and hearing aids.

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20
Q

Abnormal bony overgrowth of foot plate of stapes, conductive, hearing loss, family history, Autosomal dominant

A

Otosclerosis

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21
Q
A

Cholesteatoma

conductive

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22
Q
A

acoustic vestibular neuroma

MRI and audiometry

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23
Q
A

trigeminal CN 5

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24
abducens CN 6
25
facial CN 7
26
how is the eustachian tube different in kids
shorter, narrower, and more horizontal
27
MC organism in acute OM
strep pneumo
28
serous otitis media on left acute otitis medial on right
29
most sensitive test for acute otitis media tx
pneumatic otoscope, TM will not mobilize amox 1st choice
30
MC organism in chronic OM
pseudomonas
31
MC organism in otitis externa
pseudomonas
32
pain with tragus pulling, purulent discharge tx?
otitis externa topical antibx
33
painless, purulent otorrhea, ear fullness, conductive hearing loss
chronic OM
34
ear disorder that peaks at 6-18 months
acute OM
35
malignant necrotizing otitis externa CT/MRI next. Bx to confirm.T ADMIT and IV antipsedomonal antibx Cipro 1st line.
36
granulomatous tissue at cartilaginous part of ear canal
malignant necrotizing otitis externa
37
otoscope: effusion with TM retracted/flat. hypomobility with air. No signs of inflammation
serous OM with effusion. resolves on its own
38
what antibiotic is ototoxic?
aminoglycosides avoid in TM perf
39
hordeolum on right chalazion on the left
40
entropian on left ectropian on right
41
Blockage of nasolacrimal duct
dacrocystitis
42
dacrocystitis
43
blepharitis
44
blow out fx
45
dry on left wet on left Macular degeneration?
46
cotton wool spots, DM retinopathy
47
DM retinopathy
48
DM retinopathy
49
DM retinopathy
50
circinate ring in dm retinopathy
51
blot and dot hem in dm retinopathy
52
papilladema in htn retinopathy
53
retinal detachment
54
next step
get opthalm, emergency! lay supine towards side of retinal detachment
55
ice rink; corneal abrasion
56
limbal flush: keratitis
57
hazy cornea
, keratitis
58
hypopyon, bacterial keratitis. And 1 more
59
dendritic corneal ulceration: herpes keratitis
60
amblyopia. think strabismus
61
clouding of the Lens (versus clouding of cornea)
clouding of the Lens: cataract (clouding of cornea = glaucoma)
62
globe rupture
63
globe rupture
64
leukocoria, retinoblastoma
65
retinal blastoma
66
hyphema
67
central artery retinal occlusion
68
boxcar
central artery retinal occlusion
69
blood and thunder
central vein retinal occlusion
70
marcus gunn pupil . optic neuritis and retinal detachment
71
moa of acetazolamide
decreases production of aqueous humor and CSF production
72
dangerous glaucoma
acute angle closure
73
glaucoma common in blacks and asians
open angle with blacks. closed angle with asians.
74
slow, progressive painless bil. peripheral vision loss, halos(worse at night) sometimes cupping of optic disc tx
chronic open angle glaucoma 1st line is prostaglandin analogs(latanoprost to decrease IOP)
75
precipitants: mydriasis, anticholinergics, adrenergic meds sudden severe unilateral ocular pain, halos, tunnel vision, blurred vision appears unwell
acute angle closure glaucoma
76
exam: conjunctiva red, cloudy cornea, pupil fixed and dilated optic disc cupping/blurring next step
acute angle closure lie on back, pilocarpine drops need iridotomy
77
pathogen of hordeolum
mc st aureus
78
upper eyelid common finding
chalazion
79
hordeolum gland
meibomian found under eyelid
80
entropion vs ectropion
entropion is inverted.
81
FB sensation, tearing ,red eye condition due to spasm of orbiculis oculi muscle
entropion
82
tearing, dry eye symptoms, red conjunctiva condition due to relaxation of orbiculis oculi muscle
ectropion
83
hyphema, teardrop shaped pupil, obscured red reflex enophthalmos more than exopthalmos exam for what
afferent pupillary defect put on rigid eye shield
84
unilateral, severe pain, swelling, redness, tearing, drainage to medial side of lower lid area treatment
anterior dacrocystitis warm compresses and antibiotic (clindamycin, vanco + ceftriaxone)
85
mucopurulent drainage from punta. No signs of infection.
posterior dacrocystitis dacrocystorhinostomy
86
meimobian gland dysfunction is most common cause of what
posterior blepharitis
87
crusting, scaling, red rimming, flaking
blepharitis
88
sunken eye what bones are involved
orbital blowout zygomatic, palantine, maxillary bone
89
most common kind of orbital blowout
inferior
90
diplopia(upward gaze), dec acuity, epistaxis, orbital emphysema, parathesias to gums/lips next step/ avoid what
orbital blowout order CT avoid sneezing
91
CT: teardrop line
orbital blowout
92
most common cause of permanent blindness and vision loss in older adults
macular degeneration
93
most common type of macular degeneration
dry/atrophic
94
bilateral gradual central vision field loss(detailed/colored), microopsia, metamorphopsia(straight lines bent)
macular degeneration
95
ocular pain with eye movement, opthaloplegia with diplopia(EOM weakness), proptosis, visual changes, eyelid edema and erythema
septal orbital cellulitis
96
order what for a septal orbital cellulitis next step
high resolution CT admit and IV vanco plus (ceftriaxone or cefotaxime)
97
unilateral ocular pain, eyelid erythema/edema. next step
preseptal orbital cellulitis
98
MRSA coverage for preseptal orbital cellulitis
clindamycin
99
drusen bodies (white-yellow spots on outer retina.
dry macular degeneration
100
most common kind of diabetic retinopathy
nonproliferative
101
AV nicking, abnormal light reflexes on dilated tortuous arteriole
hypertensive retinopathy
102
hypertensive retinopathy
103
AV nicking hypertensive retinopathy
104
flame & dot hemorrhages, cotton wool spots, soft/hard exudates, microaneurysms
hypertensive retinopathy
105
most common primary intraocular malignancy in childhood
retinoblastoma
106
ocular ultrasound shows an intraocular calcified mass
retinoblastoma
107
leukocoria(presence of abnormal white reflex instead of the nml red reflex; may develop strabimus or nystagmus next step
retinoblastoma order a ocular u/s after dilated opthalmic exam
108
small moving flashing lights, floaters, progressive unilateral vision loss, decreased peripheral and central + shafers sign clumping of brown color pigment vitreous cells in anterior vitreous humor resembling tobacco dust
retinal detachment
109
avoid what in retinal detachment and what to do when diagnosed
miotic drops lay supine with head turned towards affected side
110
ocular pain, tearing, red eye, photophobia ice rink/linear abrasion
ocular FB and/or corneal abrasion
111
size of corneal abrasion to treat and tx antibx
over 5 mm, patch for no longer than 24 hours e mycin ointment contact lens wearers-pseudomonas- topical cipro or oxofloxacin
112
1st symptom with ocular fb/corneal abrasion
decreased visual acuity
113
MC pathogen with bacterial and viral conjunctivitis
bacterial: staph aureus viral: adenovirus
114
why get a fluorscein stain with bacterial conjunctivitis?
to rule out keratitis or abrasions
115
red, itchy, nml vision, ipsilateral preauricular lymphadenopathy, copius watery tearing. poss mucoid discharge. may be bilateral symptoms in 2-3 days
viral conjunctivitis
116
marked pruritis to eyes, usually bilateral
allergic conjunctivitis
117
opthalmia neonatorium conjunctivitis pathogens day 2-5 and 5-7
gonococcal chylamydia trachomatis
118
pathogens for bacterial keratitis
s aureus, strep, pseudomonas-cls wearers
118
opthalmia neonatorium conjunctivitis conjunctivitis occurs due to what chemical given prophylaxis?
silver nitrate e mycin ointment given immediately after birth
119
ocular pain, photophobia, eye redness, vision changes, discharge, tearing, FB sensation, hazy cornea, limbic injection, hypopyon if severe "difficulty keeping eye open"
bacterial keratitis
120
what disease may rapidly progress and be sight threatening
bacterial keratitis
121
findings with fluorescein uptake with bacterial keratitis
increased uptake, more than an abrasion
122
do not patch eye in what disorder
bacterial keratitis
123
herpes keratitis is a reactivation of virus in what ganglion
trigeminal
124
acute onset unilateral ocular pain, photophobia, eye redness, blurred vision, watery discharge hazy cornea, limbic injection, conjunctiva erythema, preauricular lymphadenopathy major cause of blindness in US *what will you find on fluorescein stain?
dendritc corneal abrasion
125
phenytoin, alcholism, sedating medications that can affect labyrinthitis, inner ear disorder, menieres all can cause what symptom
nystagmus
126
occurs between sclera and conjunctiva, bleeding could be due to blepharitis, corneal abrasion/ulcers, FB, increased BP, trauma
subconjunctival hemorrhage
127
drifting eye name 2 types
strabismus esotropia, deviated inward (nasally) exotropia, deviated outward (temporally)
128
diplopia, scotomas or amblyopia asymmetric corneal reflex
strabismus
129
hirschberg test for what
strabismus
130
cover/uncover test for what
looks for latent strabismus
131
1st line treatment for strabismus
eye patch to nml eye
132
what if strabismus persists over 4-6 months of age
referral for intermittent manifest strap to decrease incidence of amblyopia
133
transient monocular vision loss over visual field, temporary curtain/shade comes down then resolves within an hour
amaurosis fugax
134
etio: migraine aura, lupus, arteritis, retinal embolie
amaurosis fugax
135
most common cause of blindness in the world
cataracts
136
TORCH syndrome
toxoplasmosis, rubella, CMV, HBV neonatal cataracts
137
painless, slow progressive blurred vision loss over mnths to years. absent red reflex poss diplopia, halos around lights, avoid driving at night and reading
cataracts
138
emergency central retinal artery or vein occlusion
artery
139
acute sudden painless monocular vision loss, may be preceded by amaurosis fugax
central retinal artery occlusion
140
fundoscope: pale retina with cherry red macula. Boxcar appearance of vessels.
central retinal artery occlusion
141
sudden onset, monocular vision loss, may be painless or not etio: HTN, DM, glaucoma, smoking, hypercoagulable state, multiple myeloma
central retinal vein occlusion
142
blood and thunder hemorrhages, optic disc swelling, retinal vein dilation
central retinal vein occlusion
143
marcus gunn pupil possible relative afferent pupillary defect
central retinal vein occlusion
144
which is worse, alkali or acidic burns to eye
alkali, causes liquidification necrosis
145
what happens with acidic eye burns
coagulative necrosis
146
ocular pain!, decreased vision, blepharospam(cant open eyelid), photophobia next step
Chemical burn immediate irrigation until pH is 7-7.4 with lactated ringers
147
unilateral severe ocular pain and photophobia, eye redness, tearing, blurred or dec vision constricted pupil, "cell and flare"
anterior uveitis
148
blurred or decreased vision, floaters, may not be painful. constricted pupil, "cell and flare"
posterior uveitis
149
cell and flare
uveitis
150
treatments for anterior and posterior uveitis
anterior: topical glucocorticoids post: systemic glucocorticoids
151
can occur with ethambutol, chloramphenicol. MS or autoimmune. most common in woman and young pts 20-40yo acute mono-ocular vision loss and decrease in color vision
optic neuritis
152
marcus gunn pupil fundoscopy: 2/3 of nml disc cup retrobulbar or 1/3 of disc swelling and blurring(papillitic)
optic neuritis
153
headache, n/v, vision may be disturbed for a few seconds; usually bilateral fundoscopy: swollen optic disc and blurred margins. due to increased intraocular pressure
papilledema
154
what tests to order with papilledema
MRI/CT to rule out mass effect lumbar puncture if increase in CSF pressure
155
treatment for papilledema
acetazilomide
156
strept pharyngitis. rapid antigen test, if neg, throat culture
157
peritonsillar abscess
158
retropharyngeal abscess
159
leukoplakia left candiaiasis right
160
erythroplakia
161
most common type
lichen planus reticular local glucocorticoids
162
HA, sore throat, fever HOARSENESS
acute pharyngitis
163
FEVER, no viral sx, anterior cervical lymphadenopathy pharyngeal edema/exudate tx
strep! PCN!
164
muffled hot potato voice, drooling, trismus, severe unilateral pharyngitis, high fever
peritonsillar abscess
165
uvula deviation to contralateral side, buldging of soft palate, ant cervical lymphadema, poss referred ear pain
peritonsillar abscess
166
test for peritonsillar abscess
CT
167
torticollis, fever, drooling, anterior cervical lymphadenopathy, lateral neck mass
retropharyngeal abscess
168
cotton like feels
candidiasis
169
yellow gray center
apthous ulcer
170
white patchy lesion cannot be scaped off
oral leukoplakia
171
painless, erythematous, soft, velvety patch, on mouty, floor, soft palate, ventral tongue
erythroplakia
172
hoarseness, aphonia, dry scratchy throat
laryngitis
173
rapidly spreading cellulitis of floor of mouth fever, chills, malaise, stiff neck, dysphagia, drooling, muffled void, stridor if severe. no lymphadenopathy
ludwig's angina
174
ludwig angina test of choice
CT
175
sudden onset of very firm and tender gland swelling with purulent discharge.
acute bacterial sialadenitis, suppurative order CT
176
sudden onset fever, ulcerative lesions of gingiva friablilty with vesicles on mucous membrae of mouth. dew drops on a rose petal
acute herpetic gingiovo stomatitis
177
vesicles that rupture
acute herpetic pharyngotonsillitis
178
painless, white smooth, corrugated hairy plaque along lateral border to mucosa cannot be scraped off
oral hairy leukoplakia
179
painful swelling in her right cheek, exacerbated during meals. She reports a recent episode of flu-like symptoms, including fever and malaise, erythema and tenderness over the area of the right parotid gland. Palpation of the gland expresses purulent material from the Stensen’s duct, leukocytosis
. A diagnosis of acute bacterial sialadenitis is made. She is started on IV antibiotics targeting Staphylococcus aureus, advised on adequate hydration, and sialogogues to stimulate saliva flow. Warm compresses to the affected area are also recommended. She is scheduled for a follow-up in one week to assess response to treatment.
180
malaise and swelling of his face. He has no significant past medical history, but it is documented in his chart that his mother declined the recommended standard immunizations for children because of personal beliefs. Vital signs are stable, with the exception of a mild fever. In addition to the facial swelling, physical exam is also notable for swelling around the testes. There are no rashes.
This classic presentation points toward mumps parotitis – fever, bilateral parotid gland swelling, and occurring within a timeframe post-MMR vaccination (while vaccine effectiveness is high, it isn't 100%).
181
______disorder that is not premalignant. It is an Epstein-Barr virus-induced lesion that occurs almost entirely in HIV-infected patients Generally affects the lateral portions of the tongue, although the floor of the mouth, the palate, or the buccal mucosa may also be involved TX: Unlikely to progress to squamous cell carcinoma
Oral hairy leukoplakia is a separate Treatment with zidovudine, acyclovir, ganciclovir, foscarnet, and topical podophyllin or isotretinoin Therapy is usually not indicated
182
_____is an oral potentially malignant disorder that presents as white patches of the oral mucosa that cannot be wiped off with gauze. (compare this to oral candidiasis) Tobacco use (smoked and especially smokeless), alcohol abuse, HPV infections Leukoplakia is in itself a benign and asymptomatic condition. However, some patients will eventually develop squamous cell carcinoma (SCC)
Oral leukoplakia DX: The diagnosis of leukoplakia is suspected in patients presenting with a white lesion of the oral mucosa that cannot be wiped off with gauze and that persists after eliminating potential etiologic factors, such as mechanical friction, for a six-week period Biopsy is indicated for any undiagnosed leukoplakia TX: For 2–3 circumscribed lesions, surgical excision Destructive therapies (e.g., laser ablation, cryosurgery), medical therapies (e.g., retinoids, vitamin A, carotenoids, NSAIDs), and watchful waiting with close clinical and histologic follow-up
183
The child has had a high fever, sore throat, and stridor. She has a muffled voice and is sitting up on the stretcher drooling while leaning forward with her neck extended. The patient's parents are adamantly against vaccinations, claiming that they are a "government conspiracy." You order a lateral neck x-ray, which shows
a swollen epiglottis. The patient recovered following treatment with prednisone and ceftriaxone.
184
a 17-year-old female complaining of a painful rash on her cheek. She says that it has come and gone a few times before and that she usually can feel itching and a tingling discomfort before a break out of the lesions. On physical exam, you observe clusters of small, tense vesicles on an erythematous base.
Orolabial herpes (gingivostomatitis) is the most prevalent form of mucocutaneous herpes infection. Overall, the highest rate of infection occurs during the preschool years. Female gender, history of sexually transmitted diseases, and multiple sexual partners have also been identified as risk factors for HSV-1 infection. Primary herpetic gingivostomatitis usually affects children below the age of 5 years. It typically takes the form of painful vesicles and ulcerative erosions on the tongue, palate, gingiva, buccal mucosa, and lips. Edema, halitosis, and drooling may be present, and tender submandibular or cervical lymphadenopathy is not uncommon. Hospitalization may be necessary when pain prevents eating or fluid intake. Systemic symptoms are often present, including fever (38.4 ° to 40 ° C [101 ° to 104 ° F]), malaise, and myalgia. The pharyngitis and flulike symptoms are difficult to distinguish from mononucleosis in older patients. The duration of the illness is 2 to 3 weeks, and oral shedding of the virus may continue for as long as 23 days. Recurrences typically occur two or three times per year. The duration is shorter and the discomfort less severe than in primary infections; the lesions are often single and more localized, and the vesicles heal completely by 8 to 10 days. Pain diminishes quickly in 4 to 5 days. UV radiation predictably triggers recurrence of orolabial HSV-1, an effect that, for unknown reasons, is not fully suppressed by acyclovir. Pharmacologic intervention is therefore more difficult in patients with orolabial infection.
185
a 33-year-old male presents to the emergency department with a 3-day history of severe throat pain, fever, and difficulty swallowing. He mentions that the pain has progressively worsened, now radiating to his ear, and describes a feeling of tightness and swelling in his neck. He also reports a muffled voice and difficulty breathing, particularly when lying down. His medical history is notable for untreated dental caries and a recent upper respiratory tract infection. On examination, he is febrile with a temperature of 39.2°C (102.6°F). Inspection of the oropharynx is limited due to trismus. There is noticeable swelling and tenderness to palpation on the left side of his neck, with overlying erythema and warmth. His voice is hoarse, and he appears to be in respiratory distress with stridor noted on auscultation. Laboratory tests reveal leukocytosis. A CT scan of the neck with contrast shows a large abscess in the left parapharyngeal space with surrounding cellulitis, consistent with a
deep neck infection. The patient is admitted to the intensive care unit for airway monitoring given his respiratory distress. Immediate intravenous broad-spectrum antibiotics are initiated, and an urgent otolaryngology consultation is obtained for possible surgical drainage. He is also evaluated by a dentist for management of his dental caries, which is suspected to be the source of the infection
186
a 14-year-old who is brought to your Emergency Department (ED) with an intractable nosebleed. Pinching of the nose has failed to stop the bleeding. In the ED, a topical vasoconstrictor is tried but also fails to stop the bleeding.
Persistent bleeding despite anterior packing, especially with visualization in the posterior oropharynx, indicates a posterior source. A posterior balloon pack is designed to tamponade a posterior bleed and is the most appropriate next step. These patients must be admitted to the hospital and prompt consultation with an otolaryngologist is indicated.
187
a 6-year old female who is being seen for a routine well-child exam is noted to have multiple teardrop-shaped growths partially obstructing the nasal passages. The child has a history of chronic sinusitis and recurring ear infections. As an astute PA, you order a sweat chloride test.
Nasal polyps, while relatively common in adults, are much less frequent in young children. Their presence in a child should prompt consideration of cystic fibrosis (CF), an inherited condition affecting the function of exocrine glands. Early diagnosis and treatment of CF are crucial.
188
a 13-year-old boy with clear fluid discharge from his nose for 2 days duration. This has also been associated with sneezing. On nasal exam, the mucosa and turbinates appear edematous and slightly bluish. He has swollen dark circles under his eyes and a transverse nasal crease.
Along with a minimally sedating oral antihistamine intranasal corticosteroids are considered first-line treatment for moderate to severe allergic rhinitis. They reduce inflammation and symptoms such as sneezing, itching, and nasal congestion. The can be administered regularly or as needed. For predictable exposures it's best to initiate therapy two days before, continuing through, and for two days after the end of exposure.
189
a 34-year-old previously healthy male with complaints of facial pressure and rhinorrhea for the past 3 weeks. The patient reports that several weeks prior, he had a “common cold,” which resolved. However, he has since developed worsening facial pressure, especially over his cheeks and forehead. He reports over 1 week of green-tinged rhinorrhea. His temperature is 100.1 F (37.8 C), blood pressure is 120/70 mmHg, pulse is 85/min, and respirations are 15/min. The nasal exam reveals edematous turbinates and purulent discharge. The patient has facial tenderness with palpation over the involved sinus.
ABRS should be diagnosed when signs and symptoms of acute rhinosinusitis (ARS) (purulent nasal drainage plus nasal obstruction, facial pain-pressure or both) persist without improvement for at least 10 days or if signs and symptoms worsen within 10 days after initial improvement.
190
MC pathogen chronic sinusitis
staph aureus bacterial aspergillus fungal