lecture 3 Flashcards

(39 cards)

1
Q

What are some common symptoms of concussions?

A

Transient cognitive impairment (confusion, poor concentration, amnesia, loss of consciousness), headache, nasuea, sensory difficulty, irritability, emotion dysregulation

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2
Q

What are some risk factors for TBI

A

male, low SES, impulsivity

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3
Q

What is a TBI?

A

change in brain function that manifests as confusion, b/hr change, altered consciousness, coma, neurophsychological deficts or acute sensory or motor neurological defict that results from blunt or penetrating force to the head.

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4
Q

What are the two types of damage?

A

Focal and Diffuse-caused by different types of impacts

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5
Q

What is a focal type of damage

A

caused by movement across a straight line-brain slams into point of contact and rebounds against opposing point of contact.-sig tissue damage without loss of consciousness

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6
Q

What is a diffused TBI?

A

caused by rotational force producing a swirling motion in brain (destroys axonal tissue-happens when striking broad object like dashboard-widespread damage affecting deeper brain areas than focal)

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7
Q

What are the 3 types of TBI?

A

mild, moderate, severe

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8
Q

What is secondary damage in TBI

A

brain swelling at site of trauma restricting blood flow to brain causing cell death which can spread to neighboring cells-a mild TBI can become more severe with time

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9
Q

What is hypoxia

A

less than necessary oxygen supply for normal cellular function and can lead to brain damage

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10
Q

Who has a greater probability of diffuse injury?

A

Children due to bigger more flexible heads and weaker neck muscles.

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11
Q

Why is the impact of TBI on young brains complicated?

A

because young brains are more plastic and can better adapt to brain damage but can cause severe developmental issues

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12
Q

What area of the brain is most susceptible to damage

A

Frontal lobe (Dorsolateral and Orbitofrontal)

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13
Q

What is the dorsolateral prefrontal cotrex involved in?

A

Problem solving, EF

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14
Q

are effects of damage to the dorsolateral PFC always immediate

A

no because of development

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15
Q

What can damage to the Orbiotofrontal Cortex impact?

A

social/emotional development-inhibiting the ability to read social cues and self regulate causing internal and external b/hrs

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16
Q

What is emotion?

A

Two integrated processes the appraisal (early warning system and the action preparation, manning the stations

17
Q

What is emotional regulation

A

Adaptive changes in emotion to specific situation

18
Q

Emotional Dysregulation

A

Maladaptive changes in emotion related to specific situation-salient features in many PSP

19
Q

Inappropriate emotion expression can be

A

Risk factor for later issues and due to poor social awareness, disregard for social rules, inability to regulate expression

20
Q

What is emotional unresponsivieness?

A

Flat affect (associated with negative outcomes)

21
Q

Abnormal changes of emotion are common in?

A

infants and children with ADHD, internalizing symptoms, and bullies

22
Q

Emotional liability (emotional instability) is common in youth with

A

comorbid internalizing and externalizing disorder, depression aggression, anxiety

23
Q

What are some possible contributing factors to Emotional change

A

Normal development, temperament, parent child relationship, parent PSP, abuse, genetics

24
Q

What are two i words associated with a wide range of PSP

A

inattention and impulsivity (substance use, gambling, antisocial behavior and ADHD)

25
What are the other names ADHD has been called?
Minimal brain damage, hyperkinetic reaction of childhood, hyperkinesis, minimal brain dysfunction, ADD, ADHD
26
What are the core symptoms of ADHD
Attention (associated with academic problems and other issues) and Hyperactivity-impulsivity (heighten value for reward) and impulsivity: difficulty inhibiting a primed behavior
27
What are some ways ADHD manifests in schools
excessive activity leve, difficulty staying seated, always touching something, excessive talking, plays noisily, acts without thinking, interrupts others, difficulty following rules
28
What are the developmental milestones of ADHD
impulsivity first at age 3 to 4 and inattention age 5 yo 7
29
What are some things that make ADHD symptomolgy more obvious
later in the day, task complexity, need for restraint, low level of stimulation, intermittent schedule of consequences, longer delay of reinforcement, low adult supervision
30
What are two major theories of ADHD
Top-down (cognitive control theories) and Bottom up (motivational/energetic factors
31
What does Barkley's theory focus on (top down)
role of self-regulation (poor inhibition negatively impacts self regulation, nonverbal working memory, verbal working memory, reconstitution
32
What type of adhd is most likely to other comorbities
Combined type (CD, ODD, SUD, Anxiety and mood)
33
What are some health outcoms ADHD kids suffer from
accident proneness and injury, sleep problems
34
What are some functional problems of ADHD
motor coordination difficulties, underproductivity in academics, reduced intelligence, social problems
35
What area of the brain is impacted by ADHD
prefrontal cortex (underactivation, decreased blood flow in the prefrontal regions, diminished glucose metabolism, abnormal dopamine activity)
36
What neurotransmitter is associated with ADHD
Dopamine-medication increases noradrenergic activity which is associated with increase concentration
37
What is the heritability coefficient for adhd
.70 (likely due to genes for dopamine and serotonin among others)
38
What are some environmental risks and triggers
inadequate schooling, rapid social tempo, family stress, low birth weight, prenatal exposure to teratogens
39