Lesions of VC Flashcards

(56 cards)

1
Q

3 classifications of voice disorders and pathologies

A
  1. functional
  2. neurological
  3. organic
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2
Q

Functional disorders

a. Types of functional disorders (6)

A
  1. Not organic or neurological
  2. Has to do with the way the VF move, but nerve function is intact

a. functional aphonia (loss of voice without organic cause, usually caused by emotional stress)
b. paradoxical vocal fold movement (PVFM)
c. muscle tension dysphonia (MTD)
d. ventricular phonation
e. traumatic laryngitis
f. puberphonia- voice changes in puberty

  • Functional issue- do a videostroboscopy, look at movement of VF in slow motion- can see bowing (not neurological)
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3
Q
  1. Neurological disorders (2)

2. Neuro conditions

A
  1. vf paralysis
  2. spasmodic dysphonia

Neuro Conditions that effect Voice Quality

  1. PD
  2. ALS
  3. MS
  4. HD
  5. MG
  6. EVT
  7. Pseudobulbar palsy
  8. bulbar palsy
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4
Q

Organic disorders (15)

A
  • pathology (change in the structure)
  1. VF nodule
  2. polyp
  3. cyst
  4. granuloma
  5. contact ulcer
  6. infectious laryngitis
  7. reflux laryngitis
  8. presbylarynx
  9. sulcus vocalis
  10. Reinke’s edema/ polypoid degeneration
  11. leukoplakia/erythoplakia
  12. hyperkeratosis
  13. papilloma
  14. webbing
  15. cancer
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5
Q

Lesions of VF

A

Produce pathological changes such as:

  1. Increasing Mass (alter shape)
  2. Altering Shape
  3. Restricting Mobility
  4. Increasing or Decreasing Tension
    a. Can be unilateral or bilateral
    b. Nodules usually bilateral
    c. Polyps are usually unilateral

*** if gap, breathy escape

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6
Q

MASS LARYNGEAL LESIONS IN CHILDHOOD (12)

A
  1. Cri du chat
  2. Bacteria, viral, and fungal infections
  3. Hypertrophic laryngitis
  4. Papilloma
  5. Laryngomalacia
  6. Congenital laryngeal web
  7. Congenital subglottal stenosis
  8. Congenital cysts
  9. Hemangioma
  10. Polyps
  11. Laryngotracheal cleft
  12. Laryngocele
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7
Q

Cri du Chat (11)

A
  1. Neonates and children (cry of the cat)
  2. Genetic defect (Structural defect)
  3. Micrognathia
  4. Abnormal larynx
  5. Beak-like profile
  6. Microcephaly
  7. Hypotonia
  8. Hypertelorism (eyes are set)
  9. Mental retardation
  10. Midline oral clefts
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8
Q

CRI du CHAT VOCAL CHARACTERISTICS

5

A
  1. High pitched, mewing cry
  2. Painful cry
  3. Flat or rising melody patterns
  4. Strained quality (larynx is abnormal)
  5. Crying on inhalation with inhalatory stridor (some type of obstruction or VF more in the midline)
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9
Q

Bacterial, Viral and Fungal Infections
A. Cause: (2)
B. Result from: (5)

A

A. Cause

  1. Respiratory Distress
  2. Airway Obstruction

B. Result from

  1. Result of herpes simplex virus
  2. Croup viral due to influenza virus affects children between 3 months and 3 years of age
  3. Inflammation and edema in the subglottal area
  4. Epiglottis bacterial
  5. Candidiasis fungal
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10
Q

Fungal laryngitis

A

There is a central white coating that can be quite thin with a surrounding erythema. It seems to be most frequently associated with ADVAIR HFA Inhalers (fluticasone propionate and salmeterol)

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11
Q

Candidiasis

A

Candidiasis or thrush is a fungal infection commonly referred to as a yeast infection

  • fungal infection are more likely to occur when the immune system is suppressed like after chemo
  • Coccidomycosis is from Cali’s central valley
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12
Q

Bacterial infection

1. Epiglottitis

A

A bacterial infection of the larynx is very serious as the swelling can nearly close off the airway in an adult and easily closes off the airway in a child. It is typically called epiglottitis or supraglottitis. The danger lies in the softness of the tissue which can easily expand, particularly the loose tissue of the arytenoids can be drawn in during inspiration. Here the arytenoids are very swollen and limiting the opening of the vocal cords. (closing off the airway)

  • typically improves with antibiotics
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13
Q

Inflammation/Laryngitis

A
  1. Inflammation of the larynx
  2. Anterior is the apex
  3. Posterior is where the arytenoids are
  4. Vocal quality:
    a. Drop the pitch
    b. Deep pitch
    c. Soft intensity
    d. Vocal quality- breathy, hoarse, strained (loud, moderate, or severe)
    e. Most lesions will occur on the edge of the VF
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14
Q

Viral Infections (4)

A
  1. common cold- makes VF pink and swollen, voice deep, and secretions thick
  2. Laryngitis sicca- laryngeal dryness, possibly related to autoimmune disease
  3. Herpes Simplex Virus- HSV1 produces most cold sores
    - HSV2 produces most genital herpes
    - Watery blisters in the skin or mucous membrane
  4. Viral laryngitis- unilareral edema, diplophonia, hoaseness (breathy and harsh)
    - plain harshness (no breathiness)
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15
Q

Viral Coup

A
  1. Viral Croup
  2. Most common form of airway obstruction in children 6 months to 6 years old
  3. Respiratory tract infection causes an upper airway obstruction causing:
    a. Barking cough
    b. Horse voice
    c. Inspiratory stridor
    d. Wheezing
    e. Treated with epinephrine
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16
Q

Laryngeal Papilloma

  1. Location
  2. Size
  3. Etiology
  4. Vocal Symp.
  5. Managment
A
  1. on or around VF
  2. wart like
  3. thought to be viral, but uncertain
  4. a. Breathiness
    b. tension
    c. aphonia
    d. hoarseness
  5. Med surg. w/ Voice therapy (keep occuring)
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17
Q

Congenital Laryngomalacia

A
  1. Redundant arytenoid cartilage mucosa (too much)
  2. Epiglottis omega shaped
  3. Aryepiglottic folds sucked into glottis on inhalation
  4. Aryepiglottic folds blown out on exhalation
  5. Symptoms resolve spontaneously within 6 to 18 months
  6. Other Associated Problems
    a. Gastroesophageal Reflux
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18
Q

CONGENITAL LARYNGOMALACIA VOICE CHARACTERISTICS

A
  1. Congenital laryngeal stridor described as high pitched harsh and fluttering
    a. Becomes worse with crying and feeding
  2. Swallow study needed to rule out aspiration pneumonia
    - Can’t see VF, collapsing of epiglottis gives it a strange fluttering sound

***Can occur in peds and adults, can cause an arytenoid to be dislocated

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19
Q

Congenital Laryngeal Web

  1. Location
  2. Etiology
  3. Vocal Symptoms
  4. Managment
A
  1. General includes the anterior commisure and can extend the length of the VFs
    - attachment can be infra and supra glottal as well as cordal
  2. congenital or acquired
  3. a. elevated pitch
    b. tension
    c. diplophonia
    d. hoarseness–> aphonia
  4. stridor
  5. combination of surgery and voice therapy (web can be caused by anything that damages the larynx) (fumes, burn, or injury)
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20
Q

Congenital posterior glottic web

A
  1. can be anterior, middle or posterior
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21
Q

Congenital subglottal stenosis

A
  1. Below the glottis, below VF
  2. Respiratory problems- may need a trach immediately, kids may need to be on permanent trachs
  3. Arytenoids sit on the back of the cricoid cartilage
  4. Arrested embryonic development of conus elasticus
  5. Maldevelopment of the cricoid cartilage
  6. Obstructive narrowing of the airway
  7. Voice normal to impaired
  8. Stridor present from birth
  9. May require tracheostomy
    * ** narrowing
    - can also have congenital tracheal stenosis
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22
Q

Different types of breathiness (2)

A
  1. Hyperfunctional- harshness, straining (pushing too hard)

2. Hypofunctional- weakness, lethargic (try to see if they can get them together)

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23
Q

Congenital Laryngotracheal Cleft

A
  1. Embryonic failure of dorsal cricoid lamina fusion
  2. Results in an interarytenoid cleft and open larynx posteriorly
  3. Cry weak or aphonic due to cleft preventing apposition of the vocal folds
  4. Aspiration pneumonia
  5. Feeding problems
    - VF are formed properly in the front, open in the back
24
Q

Congenital Cysts

A

Cysts- congenital cysts- unilateral, midline of fold, embedded in the fold (within the body of the cord)

  • Congenital saccular and ventricular cyst - make sure you can tell the difference between vibrato and tremor
  • most likely causes hyperfunctional breathiness and deep pitch
25
Congenital Laryngocele
1. Congenitally large laryngeal ventricle 2. Enlarged by activities that increase intralaryngeal air pressure 3. Air filled or fluid filled 4. Herniation of laryngeal ventricle (space between false and true cords) - born with a weakness (large space between true and false VF)
26
CONGENITAL LARYNGOCELE SYMPTOMS
1. Straining 2. Coughing 3. Vocal abuse 4. Playing wind instruments 5. Glassblowing 6. Hoarseness 7. Inspiratory stridor 8. Dysphagia 9. Medical --> incision --> drainage
27
Congenital Laryngocele 1. What are they 2. Location 3. What happens
1. Sessile fluid filled cysts 2. Arise from laryngeal ventricle (deep in the larynx) 3. a. Displace true and false folds b. Glottic and supraglottic obstruction c. Swallowing problems d. Voice impaired to aphonic e. May obstruct airway --> causes stridor
28
3 types of congenital laryngocele
I. Internal Type - within the thyroid cartilage II. External Type - each protrudes above thyroid cartilage through thyrohyoid membrane III. Combination Type - Internal occurs right above the VF - External is above the thyroid cartilage, can be filled with air or fluid- need to get air or fluid out and then it will go away - Lump in the throat- globus symptom – usually due to tension
29
Congenital Hemangioma
- Uncommon vocal cord lesions and if small they may not cause hoarseness or bleeding (if it is not on the edge) - larger ones can be treated with a laser
30
Laryngeal injuries in children and adults
1. Edema 2. Hematoma- blood clot 3. Fractures of larynx and tracheal stenosis 4. Dislocation of arytenoids 5. Inter-arytenoid fixation 6. Lacerations 7. Vocal Fold Paralysis 8. Laryngeal Web 9. Perforation of the pyriform sinus or esophagus 10. Ulcer and Granuloma of the vocal process 11. Hemorrhage - If a child is hoarse, ask why (sick, what's their baseline, screaming or yelling often, could it be congenital?)
31
Hemorrhage
- Most likely be on the superior surface, it will cause swelling, could be due to getting hit in the neck ( a car accident)
32
Chronic Digestive Diseases (Reflux) 1. GERD 2. LPR
1. GERD a. Gastroesophogeal Reflux Disease b. Chronic digestive disease c. Stomach acid backflows in to esophagus 2. LPR a. Laryngopharyngeal Reflux Disease b. Extension of GERD c. Stomach acid backflows in to esophagus, larynx and pharynx - GERD will affect voice - LPR may cause scalloping on the posterior edge of the larynx
33
Reflux Laryngitis
1. Lots of secretions 2. Scalloping 3. Mucus banding- shows pt. of traumatic impact * *** May help to elevate the head of the bed- need to use bricks, not pillows (use wedges)
34
Laryngeal Trauma
1. Automobile accidents- hitting the steering wheel 2. Gunshot wounds 3. Laryngeal Intubation 4. Nasogastric intubation
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Focal Trauma 1. Laryngeal Intubation 2. Nasogastric Intubation
1. a. Endotracheal tube is too large for the patient’s airway b. Mucosa ulceration leads to granuloma c. Dislocation of arytenoid cartilage 2. a. Damage to posterior cricoarytenoid- abduction of the VF (can’t open) b. Mimic recurrent laryngeal nerve palsy muscle has been traumatized *** Posterior issues
36
Traumatic Laryngitis
1. Low pitch 2. Soft intensity 3. Sluggish movement 4. Vocal quality (diplophonia)
37
Intubation Granuloma 1. Location 2. Etiology
1. a. Vocal process b. bilateral or unilateral 2. Damage resulting from prolonged or faulty intubation to maintain airway ***When you hear granuloma, think posterior (posterior issues are painful, not anterior) - Can have an immature granuloma (voice will be okay, but it will be painful)
38
Vocal Abuse (6)
1. Strenuous speaking 2. Singing 3. Yelling/screaming 4. Coughing (always ask why someone may be hoarse, is it coughing) 5. Throat clearing 6. Velopharyngeal insufficiency - Crosses all boundaries (children for screaming and yelling, for an adult singer it could be abuse or misuse)
39
Vocal Misuse
1. Incorrect use of pitch or loudness (leads to these things) a. Elevated loudness levels, high background noise, heavy machinery, speaking over loud music, hearing loss (someone with a hearing loss may be speaking too loudly) b. Elevated pitch levels, increased loudness leads to increased pitch, emotional stress, excessive muscular tension - Straining with no lesion (low pitch) - Straining with a lesion (high pitch)
40
Vocal Abuse in Children | 1. Hyperadduction of VFs
1. Hyperadduction of the vocal folds a. Inflammation b. Vocal nodules (front) c. Contact ulcers (back of the VF- pain is associated with anything posterior due to the arytenoids rubbing together) - Glottal fry causes the arytenoids to bang together. Lowest note we can produce is glottal fry (sing up a scale from lowest to highest)
41
Nodules (symptoms)
1. Client comments- pretty good in the morning, but deteriorates over the course of the day 2. Vocal fatigue 3. Vocal change 4. Chronic throat clearing 5. Intermittent loss of voice 6. Poor pitch control- may have pitch breaks 7. Deterioration of voice during day 8. Tender strap muscles- does your neck feel sore
42
Description of Nodules 1. Location 2. Size 3. Description (young or mature)
1. Bilateral (unilateral is rare) - juncture of anterior and middle third of VF - unilateral lesion (most likely not a nodule) 2. a. young- soft, normal epithelium, pink b. mature- very hard to cure a mature nodule, need to do surgery prior to voice treatment) - firm, organized epithelium, whitish to yellow
43
Prenodules
- mucus banding can indicate the start of a nodule
44
Why is vocal rest recommended following surgery for VF nodules 1. full abduction 2. quiet whisper 3. strong whisper
1. Cords are drawn wide apart in forceful inspiration (need to teach pts. to use a quieter voice) - need to whisper for at least 3 days post surgery 2. folds slightly separated along the anterior two- thirds and a triangular aperture remains posteriorly. (no stress, but can’t project their voice) 3. (just as abusive as talking)- folds are adducted firmly along the anterior two thirds and air is forced through the posterior triangle with considerable friction. *** If pts. speak right away you can have a web form between the VF
45
Contact Ulcers 1. Location 2. Causes 3. Apparence 4. Who do they develop in?
1. Ulceration of the folds in the arytenoid (posterior) region. 2. This causes ulceration of the covering of the arytenoid region. 3. Visual appearance is a raised granuloma on one side and a crater on the other side. - Pachydermia or an abnormal thickening of the mucous membrane forms. 4. Contact ulcers develop in individuals having deep throaty voices. (VERY IMPORTANT, (fry)) - In therapy teach them to use a softer production and lighter pitch
46
Personality characteristics of individuals with contact ulcers
1. Hyperactivity 2. Emotional reactivity 3. Family problems 4. Aggressive/less mature 5. Difficulty managing stressful situations
47
Characteristic of contact ulcer
1. Extreme tension of the speech musculature coupled with generalized bodily tension 2. Forcing the pitch below the optimum 3. Glottal plosive attack 4. Explosive speech patterns a. predominate speech patterns b. rigid melody or confined pitch c. considerable breath pressure d. hoarse quality - Need to treat the whole person - We need to determine what their optimal pitch is compared to their habitual pitch (the pitch they speak at)
48
Treatment of contact ulcers
Altering the fundamental frequency alters the length, thickness, and tilt of the vocal folds so that on adduction shifting parts of stress occur and the glottal impact does not always fall in the same region. (easy onset to avoid hard contact (more frontal focus)
49
Polyps (airborne irritants) 1. Location 2. Size 3. Etiology 4. Description (2 types) 5. Vocal Characteristics 6. Management
1. Occurs in any vascular organ - can occur on nasal or laryngeal mucosa * *** Primarily UNILATERAL and ANTERIOR 2. Varies from small (6mm) to bigger and obstructive 3. Air born irritants, (smoking, inhalation of toxic fumes) or - Idiopathic ( we don’t know what causes them) 4. Soft globular mass exhibiting mucoid degeneration a. Pedunculated- with a pedicle or foot (stem hanging down) b. Sessile- having no peduncle, but attached directly by a broad base. 5. a. Diplophonia (occurs often with pedunculated polyps) b. Breathiness c. Low pitch d. Intermittent aphonia e. Hoarseness (cords don’t close, so harshness with breathiness, no breathiness- just harsh, and if no breathiness it may be a functional problem ) 6. a. Pretreatment recording and counseling b. Pretreatment photography c. Surgical management- Usually say that polyps need surgery d. Post-op voice rest e. Post-op voice therapy Therapy- eliminating the compensatory behaviors that the patient developed due to the polyp
50
Physiologic Voice Disorders
1. May exhibit diplophonia
51
Etiologies of Diplophonia (5)
*** Two distinct pitches during phonation 1. Unilateral paralysis of true vocal fold 2. Vibration of the ventricular folds (typical) 3. Hyperfunctioning of the vocal mechanism 4. Vocal Folds vibrate at different frequencies (one side lags), you can have edema in one cord and not the order 5. Vocal Fold Pathology (possibly VC masses)
52
Ventricular Phonation
1. Produced by vibration of the false vocal folds. 2. May develop as purely functional or as a substitute voice for true vocal fold pathology. - We use the false VF when we cough, may use false after surgery
53
Dysphonia Plicae Ventricularis (DPV)
1. Ventricular phonation 2. Musculoskeletal tension disorder 3. Low v.s. high pitch- if person uses True and false VF= low pitch because of extra mass a. Just false VF= high pitch 4. Hoarse 5. Diplophonia - Behavior modification- eliminate coughing - If just using the false cords, what is going on with the true cords - could be due to prominent hypertrophy of the false VF
54
6 forms of ventricular phonation 1. habitual origin 2. emotional 3. paralytic 4. cerebral type 5. cerebellar type 6. vicarious function
1. the most frequent and represents the extreme and end stage of hyperkinetic dysphonia due to constant vocal abuse. 2. occurs during times of stress or a crucial period of a psychoneurotic person, an emotional crisis may precipitate a psychogenic dysphonia by over adduction of the ventricular folds. 3. due to paralysis of the true cords, the ventricular folds take over the function of phonic glottal closure. (in this case you want them to use the false cords to phonate) 4. may be a sign of dysarthria resulting from brain disease. As a sign of spasticity the voice may change to choked, rough, low, and a squeezed sound. 5. Lesions of the cerebellum may have ataxic, irregular, labored phonation with spasmodic over contraction of the ventricular folds. 6. a desirable compensatory adjustment when the ventricular folds are substituting for defective vocal folds. (was it compensatory)
55
Presbylaryngis
1. A larynx that exhibits significant signs of aging such as: a. Reduced control over phonation b. Changes in speaking and fundamental frequency c. Reduced pitch range and deterioration of vocal quality d. Loudness, resonance, and timing are also affected - Old age and VFs - You need to think about age when you are treating someone - Do you work on pitch stability? - Strengthening?- don’t do it on VFs with a pathology, it will make it worse - Always make sure it is not neuro related
56
Presbylaryngis 1. Presbyphonia (6)
1. Presbyphonia: acoustic properties associated with ageing in the absence of any other pathology a. Altered pitch b. Roughness c. Breathiness d. Weakness e. Hoarseness f. Tremulousness/instability -Work on reducing breathiness and hoarseness Presbylaryngis is a condition that is caused by thinning of the vocal fold muscle and tissues with aging. The vocal folds have less bulk than a normal larynx and therefore do not meet in the midline. As a result, the patient has a hoarse, weak, or breathy voice. This condition can be corrected by injection of fat or other material into both vocal folds to achieve better closure.”...