Lesson 6 Flashcards

1
Q

neoplasm

A

brain/spinal cord cancer

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2
Q

cerebral palsy

A

damage to brain neurons/malformation of brain tissues

hypoxic brain injuries

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3
Q

what causes CP

A
injury involving labour/delivery 
infection
Rh factor incompatibility 
high bilirubin levels
can occur in labour
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4
Q

T/F CP is classed by a degree of altered mobility/brain necrosis

A

T!

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5
Q

CP clinical features

A
lack of coordination 
tremors 
speech problems 
seizures 
lots of deficits
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6
Q

T/F CP progresses

A

F!

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7
Q

CP prehospital care

A

keep calm/comfortable
learn baseline
transport their movement aids

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8
Q

MS… who’s it more common in?

A

develops at ages 20-40

women
europeans
temperate zones (MB)

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9
Q

what is MS

A

body attacks itself (attacks myelin)

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10
Q

loss of myelin…

A

interferes with signal conduction

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11
Q

T/F only certain types of nerve fibres are affected with MS?

A

F! all nerve fibres are affected

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12
Q

lower body MS symptoms

A

heaviness, weak, stiff and numb

neck flexion can cause pain

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13
Q

MS attacks

A

progress over several days
peek at about a week
recover in several weeks/months

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14
Q

T/F MS symptoms worsen with increased body temp

A

T!

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15
Q

T/F complete recovery is impossible in the early stages of the disease

A

F!

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16
Q

MS prehospital care

A

keep calm/comfortable

transport their movement aids

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17
Q

ALS forms

A

sporadic sALS

familial fALS

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18
Q

sporadic ALS

A

most common form
onset is age 56
if you are diagnosed younger progression is slower

19
Q

ALS causes

A

possibly neuroglia secreting a neurotoxin?

20
Q

T/F only sensory neurons are affected by ALS

A

F! only motor

21
Q

ALS loss of neurons leads to…

A

muscle atrophy

22
Q

ALS clinical features

A

upper extremity weakness/hand dexterity
cramps/twitching
speech impairment

23
Q

ALS prehospital treatment

A

comfort care

understand patients wishes

24
Q

what’s more severe upper or lower body symptoms MS

25
MS clinical features
vision loss blurred vision emotional changes
26
familial ALS
genetic mutation | less common
27
ALS
degeneration of the upper motor neutrons in the cerebral cortex and the lower motor neutrons of the brainstem/spinal cord
28
muscular dystrophy
skeletal muscle degeneration genetic - gene defect can cause muscles to weake n not just a childhood disorder
29
what replaces skeletal muscle in MD
fat and fibrous connective tissue causes a loss in motor function
30
MD clinical features
spinal cord deformity pneumonia cardiac abnormalities difficulty moving/breathing/speaking/swallowing
31
MD prehospital care
comfort care transport movement aids ventilator support?
32
polio transmission
oral to oral | fecal to oral
33
how odes polio enter the body
through the bloodstream in the GI tract
34
what does the polio virus invade?
motor neurons of the spinal cord/medulla destroys nissl bodies
35
__% of infected neurones die pithing the first week polio
50
36
t/f polio symptoms can return kin 15-40 years
T!
37
polio clinical features
``` fever GI issues headache sore throat muscle pain weakness ```
38
t/f most people suffering paralysis during polio remain that way
F! usually recover in a year
39
polio prehospital care
comfort care manage airway
40
parkinsons
most common chronic neurogenerative disease more in men onset of 55-60 cause is unknown
41
what causes Parkinson's
dopamine production is permanently reduced
42
what does dopamine do
smooth muscle contraction
43
parkinsons clinical features
``` slow movement tremors muscle rigidity aura dementia loss of balance ```
44
parkinsons prehospital care
comfort care | emotional support