Limp in adults & children profoma Flashcards

(39 cards)

1
Q

What should you ask in a history where a child presents with a limp?

A

Age- this affects what they could have

Gender

Onset- gradual or sudden

previous episodes

Pain levels

Systemic symptoms- suggests infection or JIA

Joint swelling

Hx of trauma

Birth history -breach or oligohydramnios (lack of amniotic fluid)

Developmental histroy - when did they start walking?

Family history of DDH, SUFE, Perthes, inflammatory diseases or autoimmune conditions.

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2
Q

What are some differentials for limping in children?

A

Septic arthritis

Irritable hip - transient synovitis - common in boys aged 4-8 & symptoms usually settle w/in 2 weeks.

Reactive arthritis

Trauma & fracture e.g. Toddlers fracture (subtle un-displaced spiral fracture of the tibia, usually in preschool child).

Slipped upper femoral epiphysis (SUFE)

Developmental Dysplasia of the hip (DDH)

Perthes disease

Osteomyelitis

Occult trauma - trauma not apparent on initial presentation. Including NAI

Juvenile idiopathic arthritis (JIA)

Malignancy e.g. osteosarcoma, Ewing sarcoma.

Osgood-Schlatter disease - pain & swelling of tibial tuberosity & patella tendon.

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3
Q

Differentials for limping categorised by age?

A

All ages:
- infection
- JIA
- NAI

Infants (1-3y)
- Late presenting DDH
- Irritable hip
- NAI or occult trauma

Childhood (3-11y)
- Perthes disease (p= primary so 3-10)
- irritable hip
- SUFE (s= secondary so 11-14 y)
- NAI

Adolescence
- NAI
- SUFE
- Infection

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4
Q

Presentation of limp in children

A

Rapid onset of pain:
- trauma or infection

Gradual onset of pain:
- Perthes

Weeks of knee/hip pain then sudden increase in pain
- SUFE

Pain in groin mimicking septic arthritis
- Transient synovitis of the hip

Night pain gradually getting worse with analgesics not helping
- Malignancy

Limp & leg length difference with no pain
- DDH

Muscle imbalance or wasting with painless limp
- Neuromuscular disorder e.g. cerebral palsy

Systemically unwell e.g. fever, drowsy, irritibility, not eating…
- JIA or infection

Recent infection e.g. UTI, otitis media…
- Transient syntovitis

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5
Q

Investigations for limping in children

A

Always exclude septic arthritis first

Blood tests:
- Septic arthritis- high WCC, ESR & CRP
– JIA or transient synovitis - mildly increased WCC, ESR & CRP.
- Muscular dystrophy - high creatinine kinase.

Kocher criteria- differentiates between transient synovitis from septic arthritis, since both cause hip pain.
- 1-4 score. 5= higher change of septic arthritis
1. WCC > 12,000 cells/mm⁻³
2. Inability to bear weight
3. Fever > 38.5°C
4. ESR > 40 mm/h

X-rays:
- Subluxation in DDH
- Damaged femoral head & acetabulum in Perthes’ disease.
- SUFE
- Evidence of infection - they are initially normal.
- Fracture
- Hip = AP & frog leg lateral views; Knee = AP & lateral views.

Ultrasound:
- can identify swelling from inflammatory diseases, septic arthritis, transient synovitis & early Perthes disease. -

MRI
- can identify inflammation, osteomyelitis & tumours.
- Rarely performed.

Urinalysis - looks for infection.

Blood culture - excludes septic arthritis.

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6
Q

Presentation of limping in adults

A

NOTE: we don’t get a normal gait until age 3-4 yrs.

Look for STRAWS
- S-hort leg gait
- T-rendelenberg gait
- R-igid: do the limbs move flexibly?
- A-ntalgic Gait
- W-eakness
- S-upratentorial gait - split into 2 types (hemiplegic & diplegic)

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7
Q

Describe a Hemiplegic gait

A
  • Unilateral weakness & spasticity
  • Arm - flexed, adducted & internally rotated.
  • Leg - extended, foot plantar flexed
  • Circumduction - circular motion.
  • e.g. Spinal cord lesion or post stroke.

NOTE: view image on notes!

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8
Q

Describe a diplegic gait

A
  • Bilateral weakness & spasticity.
  • Hips& knees-flexed&abducted.
  • Ankles-extended &internally rotated
  • The knees are forced together due to spasticity in the adductor muscles resulting inleg overlapwhen walking (a.k.a. scissoring gait).
  • In an attempt to overcome this adduction, the patientcircumducts both legduring the swing phase.
  • Narrow base, drags legs w/ toe walking
  • e.g. Cerebral palsy

NOTE: view image on notes

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9
Q

Describe a trendelenburg gait (waddling/ myopathic)

A
  • Caused by unilateral weakness of hip abductor muscle.
  • Patient is unable to weight bear on that side.
  • Pelvic drop when walking.
  • Waddling

Possible causes:
- Short femoral neck
- Gluteal nerve damage
- Hip replacement surgery
- Hip fracture

NOTE: view image on notes

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10
Q

Describe an antalgic gait

A
  • Short stance phase
  • Long swing phase on the painful leg/foot
  • Avoids heel strike
  • Avoids weight-bearing on the painful side.
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11
Q

Describe a Parkinsonian gait

A
  • Small steps, shuffling
  • Bradykinesia- slowness of movement
  • Rigidity
  • Tremor
  • Stooped head
  • Freezing & difficulty initiating steps

NOTE: view image on notes

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12
Q

Describe an Ataxic gait

A
  • Clumsy, staggering movements
  • Alcohol intoxication mimics this.
  • Wide base
  • Body may sway when standing still
  • Cannot heal-toe walk
  • e.g. Cerebellar issues

NOTE: view image on notes

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13
Q

Describe a Neuropathic gait

A
  • Foot drop
  • High stepping gait - lift knee high enough not to drag foot on floor.
  • Unilateral causes - L5 radiculopathy or Peroneal nerve palsy.
  • Bilateral causes - peripheral neuropathies e.g. diabetic neuropathy, Charcot-Marie-Tooth disease & Amyotrophic Lateral Sclerosis (ALS).

NOTE: view image on notes

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14
Q

Differential diagnosis for limping in adults

A

Ligament tear:
- PCL
- ACL

Tendinopathy:
- Patella tendon
- achilles tending
- hamstring tendon

Nerve entrapment:
- Sicatica
- L5-S1 entrapment

Malignancy

Infection
- septic arthritis
- osteomyelitis

Backpain:
- spinal stenosis
- Herniated disc
- disciitis

Muscle weakness
- trendelenburg

Inflammatory:
- RA
- Reactive arthritis
- psoriatic arthritis
- ankylosing spondylitis

OA

Parkinsons

Fractures & dislocations

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15
Q

Management of limping in adults

A
  1. Pain relief e.g. NSAIDs
  2. Weight reduction
  3. Physiotherapy
  4. Orthotics & aids- walking stick in contralateral hand.
  5. Surgical - Joint replacement or osteotomy.
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16
Q

Risk factors for Developmental Dysplasia of Hip (DDH)

A
  • Breech delivery
  • First degree family history
  • Oligohydramnios - too little amniotic fluid.
  • Packaging disorders in the womb
  • First born

More common in girls

17
Q

Presentation of DDH

A

Late presentation if not found on screening:
- Painless limp & abnormal gait
- Leg length discrepancy
- Restricted range of movement- loss of abduction.
- Asymmetric skin creases
- Subluxation or dislocation of hip

18
Q

Examination & Investigation for DDH

A

Barlow test- test of instability. Involves dislocating the babies hip. The test is positive if you are able to dislocate the hip.

Ortolani test- reduction of the dislocated hip. “Clunk, click”.

Common in new borns but negative after 3 months in many cases.

Ultrasound better on babies btw 0-6 months because the femoral head is cartilage so won’t show on x-rays

NOTE: view image on notes

19
Q

Management for DDH

A

Pavlik harness- if severe- prevents hip abduction & extension)

Closed reduction - involves being in a cast for 6 weeks

Open reduction of the hip - at 18 months.

Open reduction with femoral or pelvic osteotamies

20
Q

What is SUFE?

A

the growth plate at the top of humerus is fractured, & femoral head slips out of position.

21
Q

Risk factors for Slipped Upper femoral epiphysis (SUFE)

A
  • Male
  • Obesity
  • Endocrine association - Hypothyroidism, Hypopituitarism…

Aged 8-15 most common

22
Q

Presentation of SUFE

A

Previous history of gradual thigh, knee or hip pain w/ sudden worsening of pain

Pain worse on activity

Subtle painful limp - antalgic gait.

Externally rotated - femoral head is displaced posteriorly & inferiorly.

Loss of internal rotation of leg in flexion

Note: view notes for diagram

23
Q

Investigation from SUFE

A

X-ray - need AP & frog leg views. Frog leg views are more helpful.
- dislocation of femoral head is postero-inferiorly

X-ray:
- melting ice cream cone appearance visible through line of Klein)

Frog view:
- straight line through femoral neck anterior to epiphysis
NOTE: view x-ray of notes!-very important

24
Q

Management for SUFE

A

Surgical fixation w/ screws- mild to moderate.

Open reduction w/ osteotomy - for severe.

This is an emergency because avascular necrosis of the femoral head can occur.

Under 10, endocrine problems or if you don’t think they’ll come back - then prophylactically pin the other side, too.

25
What is Perthes disease?
Degenerative condition affect hip joints of children Due to avascular necrosis of femoral head
26
Risk factors for Perthes disease
- Male - Skinny hyperactive children (but obese children do badly) - Possible causes: clotting disorders, passive smoking, genetic, environmental. - 4-10y (older children do badly)
27
Presentation of Perthes Disease
- Gradual onset of hip pain - Reduced range of movement - particularly abduction. - Painful limp - Leg length discrepancy - Stiffness
28
Investigations for Perthes
x-ray early changes: - widening of joint space Later changes: - decrease femoral head size (flattening)
29
Management for Perthes disease
- Usually no treatment required as disease self-limiting. - Physio for ROM - Hip containment - try to keep the ball & socket congruent so there is less chance of OA in later life.
30
Presentation of cerebral palsy
Painless limp
31
Presentation of JIA
- Last more than 3 weeks - Patient younger than 16 - 4 or less joints affected- usually medium sized joints e.g.g knees, ankle, elbows- pain + swelling - Signs of joint inflammation - limp - Pain or irritable & reluctant to mobilise - Fatigue, malaise & other systemic symptoms - ANA may be positive- associated w/ uveitis
32
Epidemiology of transient synovitis
- 70% boys - Age 3-10 yrs MOST common cause of hip pain in children Associated w/ viral infection
33
Management of transient synovitis
- Resolves on own. - Give NSAIDs - Can mimic septic arthritis so send to hospital to exclude this diagnosis.
34
Presentation of transient synovitis
- Reactive inflammation of synovium - Often after viral illness- 1-2 weeks - often unilateral in hip, thigh, groin or knee - may be asymptomatic - tenderness/pain during passive movement - limited abduction + internal rotation - antalgic gait (avoids weight bearing) - may have low grade fever
35
Epidemiology of Osgood-Schlatter
- occurs due to overuse (particularly in adolescents) - seen in active kids between 10-15 years old - more common in men
36
Pathophysiology of Osgood-Schlatter
Inflammation of the insertion point of the patella tendon at the tibial tuberosity - overuse injury, repetitive quadriceps contraction creates traction on tibial tuberosity leading to micro avulsion fractures creating tendinous inflammation - severe cases may suffer a complete tibial tubercle avulsion fracture
37
Presentation of Osgood-Schlatter
- anterior knee pain exacerbated by movement, relieved by rest - pain, tenderness + visible swelling over tibial tubercle - limping - tends to be asymmetrical but can be bilateral - initially bump is tender due to inflammation, however as bone heals the lump will become hard + non-tender
38
Management for Osgood-Schlatter
- short term analgesia + NSAID use - rest - modification of activities/physio - If all else fails surgical ossicle resection, excision of tibial tuberosity
39
Investigations of Osgood-Schlatter
mainly clinical- examination On X-ray may see: - elevation of tibial tubercle - fragmentation of tibial tubercle - soft tissue swelling - calcification/thickening of patellar tendon