lungs Flashcards

(43 cards)

1
Q

nebuliser vs inhaler

A

inhaler - hand held usually with a spcer

nebs- a machine that attached to a mask that turns liquid into a mist , usually in more sever condtions and in hospitals but some people can have nebs at home

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2
Q

COPD exacerbation what to give

A
  1. oxygen (need to check if they are a retainer or not because not all copd patients are retainers)
  2. bronchodilators (salbutomol/ipatrorpium)
  3. steroids
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3
Q

why is PE so hard to diagnose

A

can give a normal chest x ray

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4
Q

PERC SCORE

A

used to rule out PE definitvely

basically its if you really doubt a PE but you really want to make sure. as long as its zero, it means very low PE chance no need to do anythign. but if even one postive do a dimer

age >50
sats on room air <95
tachy >100
unilateral leg swelling
hempomptysos
hormone oestrogen use (not progesterone)
prior dvt or pe

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5
Q

what do you ALWAY ASK IN PATIENT IF SUSPECT PE

A

S.O.B
CHEST PAIN
INSPIRATORY PAIN
HEMOMPTYSIS!!!!

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6
Q

wells score interpatation

A

> 4 PE is likely order imaging
<4 PE is unlikely but do a d dimer to rule out

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7
Q

CI to ct Pa

A

contrast allergy

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8
Q

PERC VS WELLS

A

WELLS very high suspiciosn
PERC - very low

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9
Q

antibiotics given in pneumonia (just goofgle|)

A

usually would be amox but if allergic then move to macrolides like clarithromycin OR

doxycycline

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10
Q

Peak flow what is it and why

Vs spirometer

A

A device used to see how much air you can forcibly exhale from your lungs in 2 seconds

Take a deep breath in and then forcibly exhale for 2 seconds

We use it to diagnose asthma? but also to monitor as you do it regularly to see if getting better or worse

Spirometer is more reliable
More expensive

You generally do a peak flow first then spirometery confirms

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11
Q

What does an ABG show you and why would you perform it

A

Shows you if patient is hypoxic
Gives you lactation
Gives you bircarb and co2 levels

Perform it if a person looks clinically unwell or is respiratory conditions and also kidney conditions but also others like heart failure etc

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12
Q

ways to diagnose asthma

A

FeNo
eosiniphilia
spiromeery

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13
Q

meds you should be wary of in astham

A

BB
NSAIDS

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14
Q

Definition astha

A

acute inflammation of airways, that is reversible cause bronchial hyperreactivity with mucouse secretion

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15
Q

will you airways be the same after years of astham

A

no due to the chronic inflammation there will be remodeling

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16
Q

RF ASTHAM

A

ATOPY-exzema, rhnitis
FH
premature
Respiratory infections in infancy
Prematurity and low birth weight
Obesity

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17
Q

symptoms asthma

A

chets pain
dyspnea
wheezing
cough

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18
Q

why is asthma worse at night

A

beacsue you are sleeping on you rback so more tagantion but we dont know really the reason why

19
Q

if you suspect asthma what would you start of with

A

FEno or eosiniphils level

then spriomteryr with revrsibility test

direct bronhcical challenge ~ not first line usually done when those above have not given a conclusive answer

note that all these tests can be falsley negative in paitents treated with inhaled CS

20
Q

INVESTIGATIONS FOR ASTHMA ATTACK

A

ABG - to see hypoxia and co2 levels
x ray - want to see if any trigger for the attack or any complication such as pneumothorax

21
Q

what should the co2 level be in asthma

A

low due to hyperventilation but if its high it means near fatal

22
Q

management of asthma attack

A

salbuotmol nebulized if that does not work or its a sever attack you can addyo on to ipratropium (anitcholinergic)

Give prednisolone 40-50mg orally, or IV hydrocortisone if the patient is unable to swallow

Can consider IV magnesium sulphate and/or aminophylline if the patient is not responding to nebulisers

If the patient continues to deteriorate despite maximal therapy, they may require intubation and ventilation in an intensive care setting (for example in cases of severe hypoxia or exhaustion)

23
Q

effect of adrenaline on airways

24
Q

nomral technique for inhaler at home

A

take 1 puff every 30/60 secnds upo to 10 puffs if not better ambulance

25
Bad signs of asthma
bradycardia bradypnea hypotensive hypercapnea silent chest
26
normal sp02
94-98%
27
Remember, a ‘normal’ PaCO2 in an asthma exacerbation is not reassuring. A normal or raised PaCO2 is significantly concerning as this indicates that the patient is becoming tired and is failing to ventilate effectively. These patients need urgent discussion with a senior clinician and critical care.
28
doses of cs to give in astham
40/50 mg pred iv hydrocortison - 100mg Continue prednisolone 40-50mg daily for at least five days after the exacerbation or until recovery.
29
when would we consider giving mg so4
Consider giving a single dose of IV magnesium sulphate (1.2-2g infusion) to patients with: Acute severe asthma who have not had a good initial response to inhaled bronchodilator therapy Life-threatening or near-fatal asthma Magnesium sulphate should only be used following consultation with a senior clinician.
30
when to give aminothyilline
should only be given by a senior clinician
31
in terms of giving fluids whats the limit
can give up to 4 times boluse (which adds up to 2000 in nomral patients of 1000 in heart failure/kidney failure
32
what are rhonchi examples
mucous in the airways, heard best centrally rather than peripherally as its usually the larger airways affected. any condition with secretions in the lungs copd bronchiactasis pneumonia CF
33
rhocnhi and coughing
can be cleared if cough as its mucous
34
monophonci vs polyphonic wheeze
one airway vs multiple airways (asthma /COPD)
35
veiscular breathing
normal no pause between inspiration longer than expiration and louder expiration quieter (because its passive)
36
what transmits sounds better
solids and fluids increases transmission
37
are crackles affected by coughin
depends on the cause e.g if due to secretions then if you cough it can clear the crackles or they improve! as youve mobilised the mucous but if its edema - the fluid has no where to go
38
kussmaul breathing
associated with metabolic acidosis hence DKA, BODY IS TRYING TO COMPENSATE BY REPSIRATORY ALKOSLOSIS TO BLOW OF THAT EXCESS CO2 BY A DEEP LABOURED BREATHING
39
PHYSIOLOGY OF BREATHING IN MEATBOLIC ACIDOSS
initially the body hyperventilates shallow and quick to get rid of the co2 but this cant be maintained and eventually breathing becomes slower and deeper. this is what we call kussmaul.
40
type 1 and type 2 resp failure
type 1 - just hypoxemia pulmonary edema, pneumonia , ards type 2 - hypoxia and hypercapnea copd, asthma, neurological disorderees
41
ARDS everything about it
its due to a primary cause leads to non cardigenic pulmonary edema pt looks clinically unwell causes lungs , pneumonia, systemic, sepsis, pancreatitis, DIC usually managed in ICU diagnosis - cxr,(edema) Abg (type 1), amylase - common cause
42
berlin criteriai
used to determine if ARDS
43