MCBG S16 Nutrition, Diet And Body Weigth. Flashcards

(56 cards)

1
Q

Name some types of metabolism.

A
Oxidative pathways 
Fuel storage and mobilisation
Biosynthesis
Detoxification
Catabolic processes
Anabolic processes
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2
Q

What work is energy needed for?

A
Biosynthetic work
Electrical work
Mechanical work
Osmotic work
Transport work 

BEMOT

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3
Q

Where do we get energy from to drive these processes?

A

Chemical bonds

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4
Q

What is the SI unit of food energy?

A

kJ

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5
Q

Define 1kcal.

A

The amount of energy needed to raise the temperature of 1kg of water by one degree Celsius.

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6
Q

How many kJ is 1 kcal equal to?

A

4.2kJ

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7
Q

What are the major dietary carbs?

A
Starch 
Sucrose 
Lactose 
Fructose
Glucose
Maltose
Glycogen
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8
Q

Where is sucrose found commonly and what is it made of?

A

Table sugar

Glucose and fructose disaccharide.

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9
Q

Where is lactose found and what is it made of?

A

Milk

Galactose glucose disaccharide.

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10
Q

What is maltose made of?

A

2 glucose - disaccharide.

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11
Q

What are the 9 essential amino acids that cannot be synthesised?

A

If Learned This Huge List May Prove Truly Valuable

Isoleucine, lysine, threonine, histidine, leucine, methionine, phenylalanine, tryptophan and valine.

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12
Q

Children and pregnant women have high rates of protein synthesis and therefore may also require what amino acids?

A

Arginine, Tyrosine and cysteine,.

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13
Q

What is another name for lipid?

A

triacylglycerols TAGS

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14
Q

Why do fats yield more energy upon oxidation than carbs or proteins?

A

Contain less O2 and therefore are more reduced.

Means have potential to be more oxidised and therefore release more energy.

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15
Q

Name 2 essential fatty acids that cant be synthesised in the body?

A

Linoleic and linolenic

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16
Q

What minerals are important for bones and teeth structure?

A

Calcium and phosphorus

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17
Q

How much Na+ K+ and Cl- should be given in IV fluids?

A

1mmol/kg/day.

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18
Q

What volume of water should be given via IV per day?

A

30ml/kg/day

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19
Q

Name the 4 fat soluble vitamins and a disease that can arise from a deficiency in them.

A

A - Xerophthalmia - dryness of the cornea and conjuctiva
D - rickets
E- neurological abnormalities
K - defective blood clotting

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20
Q

What are some examples of fibre?

A

Cellulose
Pectin
Lignin
Gums

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21
Q

What is high fibre shown to reduce?

A

Cholesterol and risk of diabetes.

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22
Q

What is low fibre lined to?

A

Bowel cancer and constipation.

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23
Q

Why can humans not break down cellulose?

A

No enzyme to break beta 1,4 glycosidic bond.

24
Q

What is the recommended daily intake of fibre per day?

25
What is a DRV?
Dietary reference value - series of estimates of the amount of energy and nutrient needed by different groups of health UK population.
26
What is RNI?
Recommended nutrient intake. | Requirement for 97.5% of the population.
27
What is EAR?
Estimated average requirement | 50% of population requirements are met.
28
What is LRNI?
Lower recommended nutrient intake. | Requirements of 2.5% of the population are met.
29
Energy requirements vary depending on?
Age, sex, body composition and physical activity.
30
What is daily energy expenditure the sum of?
BMR, DIT and PAL.
31
What is BMR?
Basal metabolic rate - Maintenance of cells - Functioning of organs - maintaining body temperature
32
What is DIT?
Diet-induced thermogenesis
33
What is PAL?
Physical activity level.
34
What factors affect BMR?
``` Body size - SA Gender Temperature of environment Body temperature Endocrine status - hyperthyroidism increases BMR. ```
35
What does voluntary physical activity represent?
The energy demands of skeletal muscle, heart muscle and respiratory muscles.
36
Hoe long can energy be stored in adipose?
40 days
37
What is obesity?
Excessive fat accumulation in adipose tissue which impairs health
38
What does BMI have to be to be considered obese?
>30
39
What is a weakness with BMR?
Muscular people - classified as obese incorrectly
40
What is an alternative measure instead of BMR?
Waist to hip ratio
41
A greater proportion of fat in upper body compared to jumps is associated with increased risk of what diseases?
``` TII diabetes Insulin resistance hyperinsulism Hypertension Hyper lipidemia Stroke Premature death ```
42
What is marasmus?
Form of severe malnutrition characterised by protein deficiency.
43
By what percentage does body weight have to be decreased by to be classed as marasmus?
62%
44
What nutritional deficiencies cause marasmus?
Energy Protein Vitamins and minerals Dehydration
45
Explain the biochemistry behind the emaciated appearance of people with marasmus?
Fat stores mobilised due to negative energy balance FAs release and loss of body fat occurs Converted to ketone bodies as a source of energy for the CNS CNS and RBCs need cant use FAs and need glucose. Glucose released from glycogen stores in the liver. Once exhausted muscle protein is broken for to amino acids for gluconeogenesis resulting in muscle mass loss.
46
What changes can be observed in someone with marasmus?
Very little body fat and muscle mass
47
What can the loss of ability to replace and repair tissue is marasmus lead to?
Thin mucosal surfaces and impaired secretory functions in the GI tract Normochromic anaemia - RBC count decreased but haemoglobin conc in RBC is normal Pituitary hormones are affected
48
What are some cardiovascular implications of marasmus?
Heart muscle thins leading to impairment of heart function: Bradycardia and hypotension. Brain can be affected in severe forms.
49
What are symptoms of Kwashiorkor?
``` Oedematous legs Sparse hair Flaky skin Swollen abdomen Thin muscles but fat Present. Fatigue Poor immune fucntion Poor healing of damaged tissues ```
50
What causes kwashiorkor?
Normal calorific intake but very low protein intake
51
Why does hepatic dysfunction result from kwashiorkor?
Liver unable to make lipoproteins due to dietary deficiency of essential amino acids. Accumulation of lipid in liver.
52
How does insufficient protein intake result in oedema?
Insufficient protein means not enough AAs to synthesis albumin and other plasma proteins., Reduction in oncotic pressure of plasma, Reducing amount of fluid that moves back into circulation at venous end.
53
Why can the limbs of someone with Kwashiorkor look deceptively healthy?
Oedema hides the underlying muscle wasting and poor growth.
54
Why can kwashiorkor sufferers not be given large amounts of proteins straight away?
Down regualtion of enzymes in urea cycle | Leads to ammonia build up
55
How should food be reintroduced?
Slowly with small amounts, increased at intervals. | Monitor.
56
What can re-feeding syndrome result inm?
Confusion, coma, convulsion and death.