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N202 Exam 3 Unit 2 > MI 18 Questions > Flashcards

MI 18 Questions Flashcards

(61 cards)

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1
Q

HR*SV

A

CO

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2
Q

CO*BMI

A

CI

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3
Q

Length of Cardiac Fiber & amy of bld in ventricle during diastole –> Force of contraction

A

Starlings Law

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4
Q

Normal Ejection Fracture

A

60-70%

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5
Q

Leading cause of morbidity and mortality

A

MI

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6
Q

Transmural Necrosis

A

4-6h

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7
Q

Subendocardial Necrosis

A

w/in minutes

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8
Q

Q wave

A

Zone of infarction

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9
Q

ST Elevation

A

Injury

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10
Q

T wave inversion

A

Ischemia

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11
Q

Location of Infarct determined by

A

EKG

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12
Q

Leads V1-V4

A

Anterior (LAD)

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13
Q

Leads II, III, aVf

A

Inferior (RCA)

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14
Q

Leads V5-V6, I, aVl

A

Lateral, Circumflex

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15
Q

Healing process post MI begins

A

in 24h

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16
Q

Post MI tissue is vulnerable

A

10-14d

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17
Q

Most Indicative Lab

Dx Acute MI

A

Troponin (protein)

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18
Q

Determine extent and tx of MI

A

Serial EKG
ST-elevation (1mm+/-)–> (Fireman’s hat)
Appear w/in few hrs

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19
Q

ST elevation returns to normal post MI

A

~2weeks

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20
Q

Normal Q

A

<1mm

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21
Q

Q wave changes (post MI) appear..

A

hours to days after MI..

Does not return to norm.

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22
Q

Primary goal of Tx (MI) acute phase

A

RELIEVE PAIN***

then control arrhythmias & stop progression

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23
Q

Greatest S/S of Ischemia

A

pain

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24
Q

Most common SE of MI

A

arrhythmias–> constant monitoring

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25
First line anti arrhythmic
Amiodarone
26
Decrease REMODELING of ventricle, ventricular dilation, CHF & mortality
ACE Inhibs
27
Increase in Temp post MI
Indicates myocardial necrosis process
28
PTCA w/in
90mins of MI
29
ONLY FOR WITNESSED V-FIB ARREST W/ ROSC
Therapeutic Hypothermia | -->improved neurological o/c
30
Hypothermia Protocol Temp
32-34C for 18-24h Monitored w/ bladder probe Ice packs and cooling blankets
31
Cause sudden death 2h post AMI
V. Fib
32
Sinus Brady post AMI
40% inferior wall | No Tx unless S/S
33
Most common reperfusion rhythm tolerated well no Tx
Accelerated Idioventricular Rhythm
34
hypersensitivity to necrotic products Pericarditis w/ pleural effusions 10d-3mo Post MI
Dressler's Syndrome
35
Pleuritic pain w/ breathing Friction Rub Left Pleural Effusion Fever
Dressler's Syndrome
36
Ventricular Septal Rupture
Medical Emergency | Blood shunted from LV-->RV
37
New, Loud systolic murmur Progressive dyspnea Tachycardia Pulmonary Congestion
Ventricular Septal Rupture Cardiac cath/ Sx in OR-->patch Nipride (afterload reducers) Lasix (decrease preload)
38
5-6d post MI Sudden neck vein distention Decreased BP (hypotention) PEA
Cardiac Rupture | Rapid, Typically death
39
1st line antiarrhythmic
Amiodarone
40
For PVC's & Vtach give
Lidocaine
41
decrease infarct sz, pre/after load, HR/ contractility
BB
42
decrease remodeling of ventricle
ACE | SE: Cough
43
Therapeutic Hypothermia only for
witnessed V-fob arrest with ROSC
44
Tx PVC
IV Lido, Pronestyl, Cordarone
45
Most common re-perfusion rhythm
Accelerated Idioventricular Rhythm (No Tx)
46
Common cause of Acute Pulmonary Edema
Left Sided HF
47
Paroxysmal Nocturnal Dyspnea | Coughing w/ frothy blood tinged sputum
Acute Pulmonary Edema
48
Digoxin
Positive Inotropic Agent | Cardiac glycoside
49
Dobutamine (Dobutrex)
B-adrenergc agonists | + Inotropic Agent
50
SE of Dobutamine
Increased Ventricular Irritability | Increased O2 demand by myocardium
51
Dobutamine -->
increased contractility and renal, mesenteric, coronary, & cerebral blood flow
52
Venous Arterial Smooth Muscle Vasodilator
Nipride
53
``` Cyanide Toxicity N/V Confusion Tinnitus Hypotension ```
Nipride
54
Biventricular Pacing | Resynchronization
only when asynchronous btwn ventricles
55
loss of 40% fund myocardium -->
Cardiogenic Shock
56
Dopamine
Vasopressor Increases Contractility, CO, BP Use central line-vessicant
57
SE Dopamine
HA, Palpitations, Tachycardia | Tissue Sloughing
58
Dobutamine Decreases
PAP & PVR
59
Dobutamine Increases
CO, SV (minimal HR & BP) Preferred when no hypotension
60
Inocor decreases
preload and afterload | relaxes vascular smooth muscle
61
3 purposes of Intra-Aortic Ballon Pump
Relieves the LV workload Forces blood into Coronary arteries Decreases Afterload

N202 Exam 3 Unit 2 (3 decks)

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