Midterm 2 Flashcards

(174 cards)

0
Q

Overt vs Covert

A

Overt: fighting
Covert: secretive behaviors - you don’t want to get caught - fire setting, torturing animals, etc.

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1
Q

Antisocial Behavior (ASB)

A
Different types of categories:
Overt vs Covert
Destructive vs Non-destructive
Socialized vs Under Socialized
Verbal vs Physical
Instrumental vs Hostile
Proactive vs Reactive
Direct vs Indirect

Different kinds of ASB that all describe ASB
the heritability of these behaviors are different (SES factors, Neurobiological factors, etc.)

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2
Q

Destructive vs Non-destructive

A

destructive: involves digging or the destruction of items, such as furniture, doors, or toys
non-destructive: maladaptive behavior that does not involve destroying property or items

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3
Q

Socialized vs Under Socialized

A

Socialized: gang fighting
Under Socialized: lone wolf

this difference is principally whether you do it alone or in a group

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4
Q

Instrumental vs Hostile

A

Instrumental: behavior done with an expressed purpose - trying to acquire something - mugging someone for their iPhone
Hostile: more emotional - hostility driven

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5
Q

Proactive vs Reactive

A

proactive: protective behavior
reactive: defensive behavior

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6
Q

ODD

A
Oppositional Defiant Disorder
Age inappropriate behavior, including:
-losing temper
-arguing with adults
-active defiance or refusal to comply
-deliberately annoying others
-blaming others for mistakes or misbehavior
-being "touchy" or easily annoyed
-anger and resentfulness
-spitefulness or vindictiveness
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7
Q

Age inappropriate behavior

A

pattern of behavior that exceeds what we would expect from that particular age group

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8
Q

CD

A
Conduct Disorder
a repetitive/persistent pattern violating basic rights of others or age-appropriate societal norms or rules
-aggression to people and animals 
-destruction of property 
-deceitfulness or theft 
-serious violations of rules
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9
Q

aggression to people and animals

A

eg bullying, threatening, fighting, using a weapon

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10
Q

destruction of property

A

deliberate fire setting

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11
Q

deceitfulness or theft

A

“conning” others

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12
Q

serious violations of rules

A

eg running away, truancy, staying out at night without permission

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13
Q

Prevalence Differences in CD and ODD

A

2%-6% for CD
12% for ODD

this WILL be on the test

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14
Q

Gender Differences in CD and ODD

A

in childhood, antisocial behavior 3-4 times more common in boys
differences decrease/disappear by age 15 (more prevalent in boys than in girls but once you transition into adolescence, that decreases/disappears (may still differ a bit)
boys remain more violence-prone throughout lifespan (more likely to show the overt kinds of aggression); girls use more indirect and relational forms of aggression (more likely to show the covert kinds of aggression)

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15
Q

Heterogeneity of ODD and CD

A

this WILL be on the test (slide 5 week 4)

this was a visual representation of many studies that looked at which kinds of ODD and CD behaviors that tend to correlate (meta-analysis)

  • behaviors can vary from non-destructive to highly destructive (on the vertical axis)
  • behaviors can vary from covert to overt
  • you can map behaviors based on how overt they are vs how covert they are and how destructive they are vs non-destructive

2x2 dimensions (nondestructive vs destructive and covert vs overt) and the quadrants that are empirically unique or distinct

four quadrants (A, B, C, D)

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16
Q

Heterogeneity of ODD and CD: quadrant A

A

property violations (Destructive, Covert)

  • Cruel to Animals
  • steals
  • vandalism
  • firesetting
  • lies
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17
Q

Heterogeneity of ODD and CD: quadrant B

A

Aggression (Destructive, Overt)

  • spiteful
  • cruel
  • assault
  • fights
  • bullies
  • blames others
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18
Q

Heterogeneity of ODD and CD: quadrant C

A

Status Violations (Nondestructive, Covert)

  • Runaway
  • truancy
  • substance use
  • breaks rules
  • swears

violations of pre-adult status
considered anti-social behavior because they are too young

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19
Q

Heterogeneity of ODD and CD: quadrant D

A

Oppositional (Nondestructive, Overt)

  • temper
  • defies
  • argues
  • angry
  • annoys
  • stubborn
  • touchy
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20
Q

Context of ASB

A

it is important to appraise whether it’s normal or not

some ASB is normative
Severity
definition of ASB influences sex differences
social ecology

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21
Q

some ASB is normative

A

adult adjustment and substance use
-there’s evidence that young adults have history with/experimentation with substances as adults - don’t want to overpathologize all behaviors because some of it is developmental

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22
Q

severity

A

ranges from minor disobedience to fighting and violence

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23
Q

definition of ASB influences sex differences

A

more common in boys in childhood, but relatively equal by adolescence

  • how you define ASB will influence these differences
  • when you focus on more overt kinds of ASB then the prevalence will be higher in boys but if you define ASB in a more inclusive way, it may include more girls (covert)
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24
social ecology
pattern of association between peer regard and rates of ASB moderated by neighborhood - generally a negative correlation between the amount of ASB you are and the amount of friends/people who like you (inverse correlation) - in low SES that negative slope is switched, the more ASB you are the more you are regarded socially
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Two types of CD
Childhood/early-onset | Adolescent - onset/limited
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Developmental Taxonomy of ASB (Moffitt, 1993)
if you look at the prevalence of ASB as a function of age - in something called the age crime curve - there is something around 13+ or - a year or so there is a huge increase in overall prevalence of ASB and increase in the number of offenders (a new group of kids around 13 or 14 start becoming antisocial that weren't before) explanation for these 2 things by Moffitt - there are 2 distinct groups of kids, around 13 or 14 years old, one (far less prevalent) are chronically antisocial (through each stage of development 6, 9, etc) "life-course persistent", the other "Adolescence Limited" group (more prevalent) aren't showing ASB throughout development (6, 7, 9-12) and around 13/14 are suddenly antisocial when you follow them past 18/19 etc. they show less ASB - this isn't a perfect representation of all kids but as a whole it is a pretty good representation
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Childhood/early-onset
Life course persistent group - few, mostly boys (~25:1) - ADHD comorbidity and ODD - physical aggression - maternal depression/CD; paternal ASPD (much more diverse for family history in psychopathology - long lineage for psychopathical issues) - neuropsychological deficits and low IQ (particularly low verbal IQ) - account for majority of (violent) crime - 2/3 to 3/4 of all crime - poor prognosis (ie persistent) - the most severe/worst subgroups of kids with conduct problems
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Adolescent - onset/limited
- greater gender equity - few child risk factors - social mimicry - Desist by early adulthood for most s
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few child risk factors
don't differ typically with kids with no ASB at all for family history of psychopathology and IQ - listen to recording
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social mimicry
"tension" between biological maturation and limited status/privileges - you feel like an adult biologically but you don't have the status/privilege to do what you want - can't drive, don't have a job, etc. - ASB as effort to obtain status/privileges (these kids tend to hang out in quadrant C)
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Desist by early adulthood for most
not all especially in low SES context maturation of PFC lines up with the time at which ASB tends to take a significant dip - this could be a factor
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ODD vs. CD
Odd has earlier onset (6 years) vs CD (9 years) similar risk factors for ODD and CD (eg family, peer) but magnitude is stronger for CD CD is often preceded by ODD and many children with CD continue to display ODD; most children with ODD do not progress to CD - if you can recognize the signs of CD before it escalates to setting fires or killing animals then (listen to recording) this may be ODD Odd and CD are separable factors
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Controversies: What to include (ODD/CD)?
``` Sexual Promiscuity (other examples?) Classification of disorders ```
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Sexual Promiscuity
other examples: - it isn't age appropriate, we would have to define differently for both boys and girls - quality of sexual promiscuity would need to be defined there are lots of behaviors that may be like CD but it gets tricky to define
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Classification of disorders
more inclusive and sex specific norms vs universal criteria impact on other forms of psychopathology? gender paradox
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more inclusive and sex specific norms vs universal criteria
one of the tensions in classification of CD - do we try to get specific or have inclusive criteria (and ultimately overdiagnosis)
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impact on other forms of psychopathology?
should we calibrate diagnostic criteria so that all disorders are gender equitable?
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gender paradox
lower base rates are associated with greater impairment (MR - mental retardation) refers to the fact that we know there are sex differences in disorders - this tells us that in the gender where that disorder is less common, often times that gender has worse outcomes - we don't know what mediates that - this could reflect that gaps in what we know (how to treat the other gender) - if you are in the gender in which it is less prevalent, you will stand out more than someone in the gender in which it is more prevalent indirect/relational aggression - the mean girls clip - more covert, nondestructive, target of the aggressive act is different (social status, reputation) what kind of damage can you do through social means
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Overt vs Covert ASB: Twin studies
if these are different things should they be treated differently? they looked at twin studies that separately measured overt (aggression) and Covert (rule breaking) and look at relative heritability found that the relative contribution of genetic, shared and nonshared environments are different between overt and covert, doesn't prove anything but is consistent with the idea that aggression and rule breaking behavior are meaningfully separate
40
Hostile Attribution Bias
-there are individual differences in the population who, when exposed to different experiences, interactions with other people that are vague, they have a tendency to attribute intentionality of hostility to the behavior of others (especially when the intentions are vague) Integrates normal theories of attribution Measurement: videotape and vignettes child abuse --> aggression/ASB
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Integrates normal theories of attribution
we rely a lot and borrow from long standing theories in psychology
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measurement (HAB)
videotape and vignettes - show intentionally vague video | -you can measure individual differences - how can we make sense of a situation that is ambiguous (this is a risk factor)
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child abuse --> aggression/ASB
Hypothesized mediator: Hostile attribution bias partially mediated this relationship what is the explanation? what does that tell us about how child abuse impacts socio-cognitive processes? - HAB may underly it - the child will develop this in social cognition or attribution which may facilitate aggression and antisocial
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Causes of Conduct Problems
Gentetic influences - biologically based traits like difficult early temperament or hyperactivity-impulsivity may predispose certain children - adoption and twin studies support genetic contribution, especially for overt behaviors - different pathways reflect the interaction between genetic and environmental risk and protective factors - maternal smoking and conduct problems
45
maternal smoking and conduct problems
this WILL be on the test!! mothers who smoked? Heritability of conduct problems very low mothers who did not smoke? heritability of conduct problems very high looked at relative heritability of conduct problems - all of the kids in the study have conduct problems heritability differed substantially as a function of being born to a mother who smoked vs being born to a mother who didn't smoke - the mother who smoked conduct problems due to the smoking and not as much genetics - those whose mothers didn't smoke behaved similar to what would be expected in the population - so conduct problems were due to genetics - EQUIFINALITY
46
Psychopathy
not a DSM disorder but highly studied likely a "subset" of individuals with antisocial personality disorder (in it's extreme form it is rare in the population) difficult to diagnose because it is LESS about observable behavior and MORE about motivation and internal states
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Psychopathy vs. Criminal Behavior
psychopathy is not the same thing as criminal behavior but they are related Psychopathy: - cleckley's (1941) the Mask of Insanity - specific form of personality disorder Criminal Behavior: - violation of laws, norms, or rights of others - relatively common in the population (even normative) exclusive focus on behavior will lead to the over-diagnosis of psychopathy (eg forensic settings) the bar is lower for criminal behavior than it is for psychopathy - you can't assume that all criminals are going to be psychopathic - you can't assume that it is that common in the population (and you CANT focus on behavior)
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16 original features of psychopathy (cleckley)
- superficial charm and good intelligence - absence of delusions or other irrational thoughts - absence of nervousness/neuroses - unreliability - insincerity - lack of remorse or shame - antisocial behavior - failure to learn from experience - pathological egocentricity - poverty in affective relations - lacking in insight and interpersonal relations - fantastic and uninviting behavior with or without drink - suicide rarely committed - impersonal sexual acts - failure to follow any life plan often it will include a criminal act but there is a ruthlessness, urgency and superficiality associated with psychopathic traits- parasitic lifestyle (go from person to person and use/abuse/manipulate)
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Structure of Psychopathy
Factor 1 | Factor 2
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Factor 1
personality based - charm, grandiosity (well spoken) - lying, manipulation - lack of remorse - lack of empathy - lack of emotional depth not that they don't experience emotion, they just don't experience it to the same depth - especially when it involves other people's distress they show diminished physiological indicators that reflect emotional reactivity
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Factor 2
Behavioral representation - early behavior problems - juvenile delinquency - boredom - irresponsibility - violence
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Callous-unemotional (CU) traits
one of the defining characteristic traits of psychopathy low empathy, lack of guilt, shallow emotions - core feature of psychopathy - associated with earlier onset of ASB, greater variety of offending, and more "official" police contact - more treatment resistant - less affected by negative consequences of their behavior, especially on others, or punishment (failure to learn from experience) - differences in processing negative emotional stimuli (no differences for positive emotional stimuli)
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Fear conditioning and psychopathic traits
deafness to fear in boys with psychopathic tendencies - had them listen to voice recordings of people saying neutral words and the tone conveyed the emotion (happy, disgust, anger, sadness, fear) - more inaccurate in identifying fear compared to comparison group - if fear is knocked down, there are many things that they would be willing to do that others (normal) would avoid because of the fear of consequences
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Correlates of psychopathy
how do we know psychopathy is a real thing and not just antisocial criminal behavior excessive use of instrumental aggression executive function deficits blunted response to fear and sadness in others
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excessive use of instrumental aggression
-reactive aggression is also elevated but instrumental is more specific to psychopathy compared to criminals in general
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executive function deficits
- cognitive/behavioral inflexibility (eg gambling tasks) - sometimes really impulsive but sometimes very good at paying attention to details - inability to alter stimulus-response associations to changing contingencies (have trouble changing strategies on the card task)
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blunted response to fear and sadness in others
- no response to "moral social referencing" | - parenting and callous-unemotional traits
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Physical Symptoms of Anxiety
``` Increased heart rate fatigue increased respiration nausea stomach upset dizziness blurred vision dry mouth muscle tension heart palpitation blushing vomiting numbness sweating ``` these symptoms are not SPECIFIC to anxiety
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Cognitive Symptoms of Anxiety
worry, apprehension, thoughts concerned with emotional or bodily danger, poor concentration ``` thoughts of being scared or hurt thoughts or images of monsters or wild animals self-deprecatory or self-critical thoughts thoughts of incompetence or inadequacy difficulty concentrating blanking out or forgetfulness thoughts of appearing foolish thoughts of bodily injury images of harm to loved ones thoughts of going crazy thoughts of contamination ``` attention problems are not just common in ADHD -burden the responsibility of the psychologist to decide if the attention problems are due to ADHD, anxiety or another disorder
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Behavioral Symptoms of Anxiety
compulsions, escape/avoidance, aggression ``` avoidance crying or screaming nail biting trembling voice stuttering trembling lip swallowing immobility twitching thumb sucking avoidance of eye contact physical proximity clenched jaw fidgeting ```
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Anxiety
a mood state characterized by strong negative emotion and bodily symptoms of tension in anticipation of future danger or misfortune (future-oriented)
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Fear
Present-oriented emotional reaction to current danger, characterized by alarm and strong escape tendencies (often but not always irrational...anxiety disorders)
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Panic
sudden and unexpected fight/flight response in absence of obvious danger/threat, hallmark features: physiology and unexpected
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anxiety vs. fear and panic
these are all expressions of negative mood know what they have in common and what is different
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Anxiety and Fear
both involve physiological arousal both can be adaptive -fear trigger "fight or flight" -anxiety increases preparedness
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fear trigger "fight or flight"
may save life eg unusual strength or endurance body reacts in a way that will utilize the proper/necessary resources for survival
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anxiety increases preparedness
moderate levels improve performance | eg achievement on tests or athletic achievement during games/competition
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Anxiety as adaptive?
yerkes dodson law: arousal and performance optimal level of memory is at the mid level of arousal or stress (of the arc) increasing alertness -->optimal level --->increasing emotional arousal optimal level of performance is somewhere in between no arousal and high arousal
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Normal fears, anxieties, worries, and rituals
moderate fear and anxiety are adaptive, and emotions and rituals that increase feelings of control are common (not pathological or uncommon)
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normal fears
from the developmental perspective: what is normal at one age can be debilitating at an older age whether a fear is normal also depends on its effect and how long it lasts the number and type of fears change/decline over time (ie developmental perspective is necessary!)
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Anxiety Disorders
heterogeneity - spans a range of physical, cognitive and behavioral disorders very diverse symptoms and we need to be mindful that there are commonalities but also significant differences within the types of anxiety disorders ``` 7 Primary DSM 5 anxiety disorders separation anxiety disorder (SAD) selective mutism specific phobia social anxiety disorder (social phobia) panic disorder with versus without specifier agoraphobia generalized anxiety disorder (GAD) ```
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generalized anxiety disorder (GAD)
- excessive (more than expected), uncontrollable anxiety and worry about many events and activities on most days (unfocused or diffuse anxiety - anxiety about a whole range of things - nonspecific) - worry excessively about minor everyday occurrences (what they're going to wear), even when they see they are making themselves and others unhappy (it's not an awareness issue) - accompanied by at least one somatic symptom (eg headaches, stomach aches, muscle tension, trembling)
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prevalence and comorbidity of generalized anxiety disorder (GAD)
3% to 6% of children - equal rate in boys and girls High rates of other anxiety disorders and depression
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onset, course, and outcome of generalized anxiety disorder (GAD)
average age of onset: 10-14 years (rare in young children) older children have more symptoms that may diminish with age symptoms more likely to persist over time when it appears earlier (earlier symptoms emerge, the more severe the course- worse prognosis)
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social phobia
social anxiety/social phobia is a marked, persistent fear of social or performance requirements that expose the child to scrutiny and possible embarrassment - anxiety over mundane activities-concern with being negatively evaluated - negative emotions pertaining to social evaluation of their performance - most common fear is doing something in front of others - eating in front of people, public speaking - more likely than other children to be highly emotional, socially fearful adn inhibited, sad, and lonely - when they experience negative emotions it tends to last longer - generalized social phobia: the most severe form involves fear of most social situations - this is not just about eating 0 this is most social activities - if there are 2 or 3 other ppl in the room
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prevalence, comorbidity, and course of social phobia
- 6-12% of children (more prevalent than GAD) - more common in girls, who are more concerned with social competence and interpersonal relationships than are boys - two-thirds also have another anxiety disorder - most common comorbid disorders: specific phobia or panic disorder; 20% of social phobic adolescents suffer from major depression and may self-medicate with alcohol and other drugs (high comorbidity) - common age of onset: early to mid-adolescence; rare under age 10 (at puberty, both genders are venturing on new social navigation - up until this point, boys played with boys and girls played with girls - prevalence increases with age and may be predicted by early rejection by peers selective mutism - failure to talk in specific social situations - may be a form of social phobia; seen in 0.5% of children
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Panic attack
sudden, overwhelming period of intense fear or discomfort accompanied by characteristics of the fight/flight response (identical to the feeling of almost being hit by a truck) rare in young children, common in adolescents; related to pubertal development, not age (increases in prevalence across development)
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panic disorder
recurrent unexpected panic attacks followed by at least one month of persistent concern about having another attack, constant worry about the consequences, or a significant change in behavior related to the attacks (anticipatory anxiety - they think about the panic attack trying to figure out the rhyme or reason to what may trigger it and what will happen the next time it comes - high anticipatory anxiety and situation avoidance may lead to agoraphobia (negative reinforcement is powerful in our understanding of how anxiety disorders emerge, maintained and how they can be treated - avoidance is a classic example of negative reinforcement because it gets rid of the thing we fear - momentarily)
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agoraphobia
fear of being alone in or avoiding certain places or situations fear of having a panic attack in situations where escape would be difficult or help is unavailable
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prevalence and comorbidity of panic disorder
panic attacks common (3-4% of teens) panic disorder less common (about 1% of teens) panic attacks are more common in females than males and are related to stressful life events (gender is a moderator of the relationship between risk factor and the stressful life events) comorbidity: most commonly other anxiety disorders or depression (may be at risk for suicidal behavior or alcohol/drug abuse)
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onset, course, and outcome for panic disorder
age of onset for first panic attack: 15-19 years; 95% of PD adolescents are postpubertal (largely if not exclusively postpubertal onset) worst prognosis of all anxiety disorders (most difficult to treat and worst outcomes academically, socially, etc.)
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sympathetic nervous system
fight or flight response - requires activation by some perception of harm/danger - physiological correlates: blood pressure increases, heart rate increases, digestion slows down - cognitive system: attentional shift, hyper-vigilance, nervous, difficulty concentrating - behavioral system: aggression and/or avoidance
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cognitive model of panic
trigger stimulus (internal or external) --> perceived threat (key to getting anxiety started) --> apprehension (applies to everyone) --->bodily sensations (applies to everyone) --> catastrophic misinterpretation (key breakdown cognitively - in response to bodily sensations, people with panic disorder or at risk for it are more likely to misinterpret the bodily sensations) --> perceived threat cognition and behavior, cognition and anxiety are interrelated when you have physiological symptoms - interoceptive anxiety - when we misunderstand, misattribute those physiological signs and symptoms - inaccurately interpret what they actually mean
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parasympathetic nervous system
rest and digest response - conservation of energy, digestion occurs, low BP - restorative properties
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associated characteristics of Anxiety disorders
cognitive disturbances (within and across anxiety disorders)
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cognitive disturbances
(within and across anxiety disorders) - disturbance in how info is perceived and processed - threat-related attentional biases - cognitive errors and biases - social and emotional deficits - anxiety and depression
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threat-related attentional biases
selective attention to potentially threatening/dangerous info -anxious vigilance or hypervigilance permits the child to avoid potentially threatening events - if you have some degree of awareness or concern around germs and threat-related attentional biases you will pay extreme attention to the people around you and whether they sneeze and wash their hands -really good at perceiving threat in an environment where there is no threat
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cognitive errors and biases
- perceptions of threats activate danger-confirming thoughts -see themselves as having less control over anxiety-related events than other children - if you ask them to rate themselves they are more inclined to rate themselves as being more in control of their anxiety than others
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social and emotional deficits
- low social performance/high social anxiety - social withdrawal, loneliness, low self-esteem, difficulty initiating and maintaining friendships - deficits in understanding emotion/differentiating between thoughts and feelings (when they become intertwined is when we have problems)
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Theories and causes: family and genetic risk of anxiety disorders
family and twin studies suggest - 33% of the variance in childhood anxiety symptoms is genetic, although identical twins do not have the same types of anxiety disorders (adhd is substantially more heritable) - a general disposition to become anxious is what is inherited; the form of anxiety that takes place is a function of shared/non-shared environmental influences (influence on genetic predisposition to become anxious) - highest genetic influence for obsessive-compulsive behaviors and shyness/inhibition - serotonin and dopamine systems are related to anxiety (Glutamate an GABA as well) - genes are linked to broad anxiety-related traits (eg behavioral inhibition); small contributions from multiple genes, not direct link with specific genes
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anxiety and depression
social phobia, GAD, SAD, and multiple anxiety disorders (not specific phobia) commonly associated with depression
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Gender, Ethnicity, and Culture of anxiety disorders
Higher incidence in girls likely due to genetic vulnerabilities and gender role orientations cultural differences in patterns of referral (which kind are referred or not is not random), help-seeking behaviors, diagnoses, and treatment may be related to parental education the experience of anxiety is pervasive across cultures ethnicity and culture may affect the expression, developmental course, and interpretation of anxiety symptoms cultural differences in traditions, beliefs, and practices affect occurrence and symptoms of anxiety behavior lens principle: child psychopathology reflects a mix of actual child behavior and the lens through which others view it in a child's culture - (is it a character flaw, a behavioral flaw, how does the culture see that disorder)
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Theories and causes: neurobiological factors of anxiety disorders
no single structure/neurotransmitter controls the entire anxiety response system; several interrelated systems work together - hypothalamic-pituitary-adrenal (HPA) axis (regulates stress reactivity), limbic system, prefrontal cortex, other cortical and subcortical structures (emotional reactivity and regulation are related but separable constructs) - overactive behavioral inhibition system (BIS) implicated; BIS may be shaped by early life stressors (part of - listen to recording - that regulates inhibitory control) - brain abnormalities: over-excitable amygdala have been implicated in children who are anxious and/or behaviorally inhibited (shyness or negative emotion - passive, nervous)
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Theories and causes: Family Factors of anxiety disorders
``` parenting practices (eg rejection, over-control, overprotection, modeling anxious behaviors) may be contributors to childhood anxiety disorders parents of anxious children seen as over-involved, intrusive, or limiting child's independence; critical and fewer positive interactions with their children (helicopter parent - hover, overinvolved - self efficacy - listen to recording) lower parental expectations for children's coping abilities (what they think their children are able to cope with - under predict incorrectly that they will struggle with adversity) ```
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Risk factors of anxiety disorders
Temperament (relatively stable and relatively enduring - listen to recording) -reactivity and regulation (Rothbart) - two facets of temperament -kagan proposed temperamental differences related to inborn differences in brain structure and chemistry (focused on behavior inhibition) -inhibited children have: higher resting heart rates greater increase in pupil size in response to unfamiliar (novel things) higher levels of cortisol (released) - measure of stress reactivity listen to recording for slide 22 this is related to ADHD but they are different - this has more of an emotional construct
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Treatment Techniques
Exposure Coping modeling reinforcement the fear thermometer
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Treatment: exposure therapy
central "ingredient" is most, if not all, effective treatments (exposure related therapies) what is exposure? - multiple, controlled, strategic exposure to anxiety producing situations/objects/occasions - imaginal stimuli: images, memories (imagine situations that make them anxious) - interoceptive: sensations associated with anxiety/fear (having the person experience physiology associated with fear/anxiety producing situation - get them habituated - accustomed - to it, process of being exposed to something with greater regularity so you don't have the reactivity you're used to) - in vivo - situations, people, places, objects "in the moment" - real world experience Exposure based therapies are highly effective for many anxiety disorders (ie "trans-diagnostic" - larger spanning multiple diagnoses - multiple types of anxiety disorders) listen to recording
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Coping
(adaptive strategies other than avoidance, escape) avoidance and escape negatively reinforce maladaptive strategies -control your breathing patterns - trying to improve regulation of physiology
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modeling
decatastrophize what the patient thinks is going to happen - do it so they can see that it won't be as bad as they think
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reinforcement
praise when they make progress, etc.
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the fear thermometer
a way to gage if it's too much fear exposure
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Cognitive behavioral therapy
teaches how thinking contributes to anxiety - "maladaptive" thoughts increase anxiety symptoms and ineffective coping strategies (eg avoidance) - goal is to provide skills and combat problematic thinking - example: "I KNOW that if I meet strangers, something TERRIBLE and embarrassing will happen" - cognitive restructuring: gently CHALLENGE the (rational) basis for the negative cognition (reconstruct people's thoughts) - "didn't you go out last week? nothing catastrophic happened from what I remember..."
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PTSD and OCD
DSM-IV listed as anxiety disorders PTSD now included in DSM-5 in section on "Trauma - and stress or -related Disorders" OCD now within "Obsessive Compulsive and Related Disorders" (eg hoarding, body dysmorphic disorder)
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Obsessive-Compulsive Disorder
OCD recurrent, time-consuming, disturbing (to the person) obsessions (persistent and intrusive thoughts, ideas, impulses, or images) and compulsions (repetitive, purposeful, and intentional behaviors) performed to relieve the anxiety -OCD is extremely resistant to "reason" -OCD children often involve family members in rituals (they get so distressed that they are - listen to recording) -rituals fail to provide long-term relief from anxiety, resulting in time-consuming, never-ending cycle of obsessions and compulsions -often leads to severe disruptions in normal activities, health, social and family relations, and school functioning
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obsessions
obsessions increase anxiety intrusive, recurring, and uncontrollable thoughts or urges experienced as irrational (they know they are irrational)
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compulsions
compulsions decrease or reduce anxiety (negatively reinforcing) impulse to repeat certain behaviors or mental acts (eg hand washing, checking, repeating a word, counting) extremely difficult to resist the impulse (this is driven by affect) may involve elaborate behavioral rituals
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PTSD
Post-traumatic Stress Disorder Symptoms grouped into 3 categories: 1. re-experiencing the traumatic event 2. Avoidance of stimuli 3. Increased arousal
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re-experiencing the traumatic event (PTSD)
nightmares, intrusive thoughts, or images (listen to recording)
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Avoidance of stimuli (PTSD)
Refuse to walk on street where rape occurred (behavioral manifestation) Emotional Numbing
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Emotional Numbing (PTSD)
decreased interest in others feels distant or separate from others unable to experience positive emotions
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Increased arousal (PTSD)
insomnia, hypervigilance, exaggerated startle response
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insomnia (PTSD)
difficulty falling asleep and also difficulty staying asleep (due to nightmares, etc.) (listen to recording)
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hypervigilance
difficulty relaxing, hyper aware of one's environment (listen to recording)
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exaggerated startle response
listen to recording
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(PTSD) Prevalence and Comorbidity
although at least 2/3 of children experience at least one potentially traumatic event by age 16, most don't develop PTSD a large national sample: 6-month prevalence for adolescents ages 12-17 was 3.7% for boys, 6.3% for girls -comorbidity: depression and/or substance abuse
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(PTSD) Onset, Course and Outcome
onset and course depend on age of child when trauma occurs and nature of the trauma; onset may be delayed for months or years (typically more recent than years - but weeks or months is a common timeline) in some cases may persist for a lifetime many factors affect recovery, like nature of the traumatic event, child characteristics, social support -cognitive - behavioral therapy may be helpful
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Reactivity
How negative (listen to recording)
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Emotion Regulation
how well can we regulate that negative emotion (reactivity)?
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Feelings into words study
goal: to examine three forms of verbalization during exposure therapy - tension btwn purely behavioral approaches vs cognitive and behavioral approaches (it's thought that maybe cognitive part doesn't add all that much) listen to recording reappraisal distraction affect labeling - how might it work? (people verbalize their emotional states) - neuroimaging and treatment evidence additional exposure on day 2 then 1 week follow-up
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Feelings into words study procedures; affect labeling
create and speak a sentence including a negative word to describe the spider and a negative word or two to describe their emotional response to the spider
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Feelings into words study procedures; reappraisal
create and speak a sentence including a neutral word to describe the spider and a neutral work to describe reaction reconstruct your appraisal of a negative stimulus
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Feelings into words study procedures; distraction
create and speak a sentence involving object or piece of furniture at home and where it is found distract from the negative stimulus
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Feelings into words study outcomes
Skin conductance reactivity - affect labeling subjects show less of a physiological response to exposure to spider behavioral approach - how close did they get to the spider? - affect labeling may better help (not perfectly) the ability to regulate negative affect (not reappraisal or distraction)
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Mood Disorders
run the spectrum from severe depression to extreme mania and involve extreme, persistent, or poorly regulated emotional states
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two general DSM-V categories of mood disorders
depressive disorders | bipolar disorder
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depressive disorders
``` excessive unhappiness (dysphoria) and loss of interest in activities (anhedonia) -irritability is one of the most common symptoms, occurs in 80% of clinic-referred, depressed children ```
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bipolar disorder
mood swings from deep sadness to high elation (euphoria) and expansive mood (mania)
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anhedonia
what used to be pleasurable is no longer pleasurable
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Major Depressive Disorder DSM 5
MDD 5 or more of 9 symptoms (including at least 1 of depressed mood and anhedonia) in the same 2-week period; each symptom must be a change from previous functioning MDD is often said to be episodic recurrent subclinical depression
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MDD is often said to be episodic
symptoms tend to dissipate over time | -they don't last for years - it is a discrete period of time
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recurrent
once depression occurs, future episodes likely (known as kindling) - once you develop one episode, you become more vulnerable to a recurrence of episodes -average number of episodes is 4
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subclinical depression
sadness plus 3 other symptoms for 10 days | significant impairments in functioning even though full diagnostic criteria are not met
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9 symptoms of MDD
``` this WILL be on the test Depressed mood (subjective or observed) loss of interest or pleasure change in weight or pleasure insomnia or hypersomnia psychomotor retardation or agitation (observed) loss of energy or fatigue worthlessness or guilt impaired concentration or indecisiveness thoughts of death or suicidal ideation or suicide attempt ``` depressed mood may present as irritability in children: very relevant in assessment of pediatric bipolar
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Comorbidity (MDD)
as many as 90% of youngsters with depression have one or more other disorders; 50% have two or more - most common comorbid disorders in clinic-referred youngsters are anxiety disorders (especially GAD), specific phobias, separation anxiety disorders - depression and anxiety are more visible as separate, co-occurring disorders as severity of disorder increases and child gets older - other common comorbid disorders are dysthymia, conduct problems, ADHD, substance-use disorder - 60% have comorbid personality disorders, especially borderline personality disorder
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Persistent Depressive Disorder DSM 5
depressed mood for most of the day, for more days than not, for 2 years or longer (difference btwn this and MDD is the timing/chronicity and severity) Presence of 2 or more symptoms
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Persistent Depressive Disorder Symptoms
Poor appetite or over-eating insomnia or hypersomnia low energy or fatigue low self esteem impaired concentration or indecisiveness hopelessness (never without symptoms for more than 2 months) children/adolescents: mood can be irritable and duration must be 1 year or longer
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Who might be dysthymic?
Eyore from winnie-the-pooh
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Child Depression
same criteria for adult and child depression -irritability may be 'substituted' for depressed mood Developmental aspects -less dysphoria and hopelessness and more depressed 'appearance' and somatic complaints (pre-adolescents) -anhedonia & psychomotor retardation increase whereas somatic low self-esteem decrease w/age -comorbidity: separation anxiety disorder among younger children and eating disorders and substance abuse among teens
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Epidemiology of Child Depression: prevalence
6 months of prevalence: 1-3% school age, 5-6% teens lifetime: 15-20% adolescents clinical: 8-15% school-age, adult more stable than child -->adult
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Epidemiology of Child Depression: sex diffs
equal in childhood; 2x girls>boys | -this persists throughout the rest of development
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Epidemiology of Child Depression: outcomes
dropout, suicide, substance abuse, etc.
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Epidemiology of Child Depression: comorbidity (youth)
``` median odds ratio: 8.2 anxiety 6.5 CD 5.5 ADHD one disorder causes another; comorbidity is a distinct disorder listen to recording for more details ```
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Response to Life Events
not all stressful life events affect everyone the same way risk of MDD is much higher for affected MZ and DZ co-twin with the presence of severe life event than unaffected MZ or DZ co-twin Affected MZ co-twin highest because of genetic pairing either variable could be the moderator (listen to recording) G x E (listen to recording) - slide 10
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Affective Dimensions in Depression and Anxiety Disorders
this WILL be on the test Tripartide model you can use these three facets (Negative Affect, Positive Affect, Somatic Arousal) to give additional grasp of what disorder you're dealing with negative affect alone doesn't help us at all because all 3 groups are high in negative affect Positive affect helps with Anxiety disorders (moderate) but not depressive disorders or comorbid anxiety disorders and depressive disorders (low) somatic arousal helps with depressive disorders (moderate) but not anxiety disorders or comorbid anxiety disorders and depressive disorders (high)
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Gender Differences in Depression
MDD twice as common in women than men -similar discrepancy occurs in many countries (but not all) some biological and psychological factors: -girls twice as likely to experience sexual abuse -women more likely to experience chronic stressors (importance of "relationship stress") -girls and women more likely to worry about body image -women may react more intensely to interpersonal loss -women spend more time ruminating; men tend to distract (ruminating may intensify and prolong sad moods) listen to recording
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Etiology of Mood Disorders : Psychological Factors
Cognitive Theories: - Beck's theory of mood disturbance - negative triad - negative schemata - negative schemata cause cognitive biases ``` Overgeneralization Arbitrary Inference Selective Abstraction Learned Helplessness Pessimistic Attributional Style Hopelessness Theory ```
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negative triad
negative view of: self world future
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negative schemata
tendency to see the world negatively
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negative schemata cause cognitive biases
tendency to process info in negative ways
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Cognitive Theory of Depression
Cognitive: negative or maladaptive belief systems - thinking errors - cognitive schemas
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thinking errors
negative automatic thoughts - mind reading: "he thinks I'm a loser" - labeling: "I'm a failure" - catastrophizing: "It's awful if I get rejected. I can't bear it." - All or nothing: "Things never work out for me." - discounting positive: "It's no big deal. Anyone could do that."
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cognitive schemas
internal structures that guide info processing and stimulate self-criticism - "he doesn't like me. I must be unloveable." - "I have no reason to be depressed." - "If I fail this, I must be a failure."
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Etiology of Mood Disorders : Psychological Factors: Overgeneralization
Drawing a sweeping generalization based on a single event | -you walk by a friend who fails to say hello and you think, she hates me
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Etiology of Mood Disorders : Psychological Factors : Arbitrary Inference
``` drawing a conclusion in the absence of sufficient information -you notice a student in your class sleeping and you conclude, I'm a boring instructor ```
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Etiology of Mood Disorders : Psychological Factors : Selective Abstraction
focus on one element while ignoring all others | -you see a small dent on your car and lament, my car is ruined
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Etiology of Mood Disorders : Psychological Factors : Learned Helplessness
Seligman's experiments with dogs uncontrollable negative events --> person learns to be "helpless" (what's the point of going out if everyone hates me) -tendency to attribute negative outcomes to internal, global and stable factors vs. positive outcomes to external, specific and unstable -cycles: expectations often unrealistic which help perpetuate dissatisfaction
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Etiology of Mood Disorders : Psychological Factors : Pessimistic Attributional Style
"attributional biases" internal: "it's my fault" stable: "i'll always fail" global: "i fail at everything"
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Etiology of Mood Disorders : Psychological Factors : Hopelessness Theory
listen to recording (slide 16)
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Psychological Treatment of Depression
Cognitive Behavioral Therapy - monitor and identify automatic thoughts - behavioral activation
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monitor and identify automatic thoughts (CBT)
help them to recognize that what we think affects how we feel and what we feel influences what we do, etc. - dispute automatic negative thoughts - replace negative thoughts with more neutral or positive thoughts
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behavioral activation (CBT)
largely assigning homework to patient - pleasant activity scheduling - dismantling studies show BA by itself to be as efficacious as complete CBT protocol
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Cycle of Depression
negative thoughts (there's no point trying) lead to low mood (feel guilty, discouraged, inadequate and worthless) lead to reduced behavior (become less active, avoid people and situations)
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Bipolar Disorder
BD a striking period of abnormally and persistently elevated, expansive, or irritable mood, alternating with or accompanied by one or more major depressive episodes -elation and euphoria can quickly change to anger and hostility if behavior is impeded; elation and euphoria may be experienced simultaneously with depression -controversy involves difficulty in identifying BP in young people, who show extreme variability and overlap of symptoms with other childhood disorders -significant impairment: previous hospitalization, MDD, medication, co-occurring disruptive behaviors, anxiety disorders, psychotic symptoms, suicidal ideation/attempts -symptoms include restlessness, agitation, sleeplessness, pressured speech, flight of ideas, sexual disinhibitiion, surges of energy, expansive grandiose beliefs
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Classification of mood disorders
unipolar | bipolar
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unipolar
unipolar single episode unipolar recurrent dysthymia
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bipolar
bipolar I bipolar II cyclothymia
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Ranges of Bipolar Mood
``` Mania Hypomania Normal mood Mild Depression Major Depression ```
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Bipolar Disorder Mood Cycling
This WILL be on the test Bipolar I: most common type: have the full range of Mania to Major Depression Bipolar II: in terms of positive mood it tops out at hypomania but goes all the way down to Major Depression Cyclothymia : goes from hypomania to mild depression
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Treatment of Bipolar Disorder
psychological treatments Family focused treatment (FFT) lithium newer mood stablizers
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Treatment of Bipolar Disorder: Psychological
not the first line or sole treatment | psychoeducational approaches: provide info about symptoms, course, triggers and treatments
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Treatment of Bipolar Disorder: Family focused treatment (FFT)
-educate family about disorder, enhance family communication, improve problem solving
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Treatment of Bipolar Disorder: lithium
up to 80% receive some relief with this mood stabilizer potentially serious side effect -lithium toxicity
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Treatment of Bipolar Disorder: newer mood stablizers
anticonvulsants (eg depakote) antipsychotics (eg zyprexa) both also have serious side effects