Midterm Flashcards
(74 cards)
Phases of Swallowing
Oral preparation
Oral phase (oral transit)
Pharyngeal
Esophageal
Oral Prep Phase
1 swallowing -place food in mouth, manipulate/chew food Sensory recognition of food Labial closure, seal and stripping Buccal tension Soft palate is pulled down and forward Bolus manipulation Chewing Bolus formation
Oral Phase
(1-1.5 seconds) step 2 Propel bolus posteriorly Posterior movement of bolus Tongue tip and lateral margins of tongue held against alveolar ridge Central groove at midline of tongue Lingual stripping
Pharyngeal Phase
(1second) step 3 Transit bolus vertically through pharynx Velopharyngeal closure Hyolaryngeal elevation Laryngeal closure Base of tongue retraction Cricopharyngeal opening
Esophageal Phase
(8-20 seconds) step 4
Transit bolus vertically through esophagus
UES closes as soon as bolus passes into the esophagus
Bolus transits through esophagus via peristalsis
Swallowing vs. feeding disorder
Swallowing disorder as problems with one of four stages of swallowing.
Feeding disorder-may have difficulty bringing bolus to oral cavity
Definition of Dysphagia
Impairment in any of four phases of swallow may be symptom of underlying disease patient has.
Depending upon where damage was if had stroke.
Patient may lose weight, may have regurgitation.
May have reduced sensation.
Need saliva to help break down bolus in smaller pieces to help trigger swallow.
Consequences of Dysphagia
Could be result of stroke, head and neck cancer, certain medications.
-Medical consequences- depends on disease. Could develop aspiration pnemonia, poor nutrition and hydration.
-Psychosocial consequences- patient part of senior group see show and lunch once a week. Now life has changed impacts functioning because of emotions now. -Celebrations and occasions are involved around eating impacts psychosocial
-Clinical management- clinical swallow exam. Get case history, chart review, make determination of patient after swallow exam to see if requires instrumental exam to further assess, then determine treatment options and strategies (diff. postures? Or diet changes?)
Clinical Examination
Instrumental Examination
Treatment Options
Signs and Symptoms of Dysphagia
Difficulty recognizing food- cognitive function impacted
Difficulty controlling food/ liquid – do they regurgitate
Difficulty controlling saliva- constantly drooling? Open mouth no tight lip seal
Coughing
Recurrent pneumonia
Unexplained weight loss- important symptoms as well as tiredness
Gurgly voice – sound like gargling all the time. Wet vocal quality maybe food or liquid is going into airway. Not coughing to protect airway.
Any perceptual changes that they notice in their bodily functions. Leads patient to seek medical help, sometimes patients ignore symptoms until it gets too bad they seek help. Common complaint-food getting stuck.
-signs are objective measures of behavior that people elicit during examination.
Ex: lab findings to find out patient has pneumonia, or white blood cell count is really high. Sometimes symptoms overlap.
Diagnosis and Treatment of Dysphagia
Define feeding and swallowing problem Identify etiology Determine appropriate diagnostic tests Plan approach Implement treatment Monitory progress Evaluate progress Slp responsible for assessment and intervention of swallowing disorders. -chart review, patient interview to possibly identify etiology. Determine tests..
Who Manages Dysphagia
Speech-language pathologist- primary person
Otolaryngologist- important for upper digestive tract
Gastroenterologist- lower digestive issues. If give Modified barium swallow. Give patients different food consistencies. Notice reflux but don’t diagnose-ask radiologist. Refer to gastro.
Radiologist
Neurologist- neurological based disorder, stroke, tbi
Dentist- if require any prosthetic device for oral cavity
Nurse- important because they are with the patients all the time. Help feed patients
Dietition- responsible for establishing type of diet
Occupational Therapist- may need different feeding utensils
Pulmonologist-physician/respiratory therapist- if patient is on trach or ventilator dependent. Not regular members of the team. But could have a role depending on primary diagnosis
Settings for Dysphagia Patients
Acute Care Setting- hospital
Sub-acute Care Setting- more medical monitoring. Could be rehab within hospital or skilled nursing facility. Intensive speech but medically clear
Skilled Nursing Facility- more long-term care. Patient may have alzheimer’s . Care for patient and keep patient safe
Home Health Care- slp receives eval and conducts eval and treatment. May do more cognitive rehabilitation
Aging is determined by
Passage of years
Genetic make-up
Environmental factors
Swallowing Changes in normal aging
Changes in oral cavity
Loss of dentition and loss of bone mass
Reduced taste sensation
Changes in the pharynx/ larynx
Reduced maximal hyolaryngeal movement (reserve)
Ossification of thyroid and cricoid cartilages
Lowering of larynx to C7 (>70 y.o.)
Presbyphagia
Normal age related changes in swallow function in healthy individuals
Increased duration of oral transit
Slight increase in frequency and extent of oral residue reduced sensation
Uncoupling of oral and pharyngeal stages
Delay in triggering of pharyngeal swallow
Delayed hypolaryngeal elevation
Increase in frequency and amount of pharyngeal residue
Common complaints with COPD
Xerostomia- dry mouth Heartburn Chest pain Mouth breathing lightheadedness Restless leg syndrome Sleep apnea
Scleroderma
causes skin to thicken and harden. Lose energy and strength. Throw up food, lose weight. Difficult breathing, lose sense of balance. Respiration and swallowing needs to be coordinated.
What is affected if there is cortical damage?
Anterior L or R hemisphere:
Oral phase impairment
Apraxia: no tongue response or searching tongue movement
Delayed oral transit
Pharyngeal phase impairment
? Delayed pharyngeal swallow
Note: Bilateral damage ↑ prevalence and severity
What is affected if there is brainstem damage?
Lower brainstem (medulla)
Oral phase may or may not be intact (depending on CNXII involvement)
Pharyngeal phase impairment
Swallow reflex may be absent initially
Delayed pharyngeal swallow
Unilateral pharyngeal weakness
Reduced hyolaryngeal elevation → reduced UES opening
Complicating Factors
Medical history (previous strokes)
Concomitant diseases (comorbidities)- combo of COPD or respiratory diseases, heart disease, TBI in addition? Impacts prognosis
Post-stroke complications-
Cognitive impairments- able to follow directions, alert, oriented, aware and able to perceive their problems?
Age
What is affected with TBI
More diffuse damage
Coup, contrecoup
Shear
Swelling
Oral preparation, transit and/or pharyngeal phases may be imparied
Complications:
Physical damage to larynx, other structures
Cognitive deficits-follow directions, express verbally, answer questions, be oriented to person, place, time
Impulsiveness-severe risk for aspiration depending on what region is affected
? Compliance
depends on area affected
Alzheimer’s complications
Characteristics
Agnosia for food – lack of awareness. Don’t open mouth in response to food
slow acceptance
slow or absent initiation of oral phase (abnormal bolus hold)
Apraxia for feeding- difficulty motor initiation and initiating oral stage. Knowing to form bolus.
Apraxia for swallowing
difficulty initiating oral stage (abnormal bolus hold)
-progressive in nature. Lose memory skills, not aware of surroundings.
Delayed pharyngeal swallow
Reduced strength of pharyngeal wall contraction- moving bolus down the pharynx into the esophagus
Reduced hyolaryngeal elevation
Parkinson’s complications
Oral phase
Slow initiation of movement
Reduced jaw opening
Reduced lingual range of motion and strength
Slow oral transit due to ‘tongue pumping’ (i.e. repetitive anterior to posterior lingual movement with reduced lowering of the back of tongue)
Reduced base of tongue retraction
Reduced pharyngeal wall contraction
Reduced hyolaryngeal elevation (reduced airway protection and UES opening)
Amyotrophic Lateral Sclerosis
UMN and LMN damage
Oral phase:
Reduced lip closure
Reduced tongue mobility
Reduced bolus control
Reduced bolus propulsion (reduced tongue pressure)
Pharyngeal phase
Reduced base of tongue retraction
Reduced pharyngeal wall contraction
Reduced hyolaryngeal elevation (reduced airway protection and UES opening)
Earlier onset of dysphagia if UMN damage predominates