Midterm Flashcards
(48 cards)
TBI definition
alteration in brain fx or other pathology caused by external force; not congenital or degenerative
leading causes of TBI
falls (35%), MVA (17%), struck by/against (17). Blasts for military. 2.2 million ER visits a year
why is TBI underreported?
have to lose consciousness to count?; coding by dr/ER; don’t go to ER
TBI ages
15-24; under 5 and over 75
primary injuries
localized brain injury (OHI); polar damage (CHI, contracoup); diffuse injury (DAI)
localized head injury/open-head injury
penetrating or missile injuries producing focal damage (less tissue involved); acts of violence; localized can also be CHI; OHI has better outcome due to less intracranial pressure
diffuse/polar damage/closed head injury
acceleration/deceleration (MVA); linear/translation vs. angular/rotational; coup/countrecoup; more brain tissue involved; often front and back
coup/contrecoup
coup= site of impact/front, contrecoup = side opposite injury/back
diffuse axonal injury (DAI)
shearing of nerve tissue b/c of ridges of cranium= white matter injured; key to prognosis; damage to white matter, corpus callosum, internal capsule (everything passes through here!), grey matter, brain stem tracts
midline shift
ventricles moved over after impact
systemic TBI issues: concomitant injuries
hypoxemia (02); hypotension (BP); anemia, hyponatremia (sodium–action potential), infection
hyponatremia and dysphagia =
pontine injury b/c salt converted too quickly
hematoma
bleeding out: epidural (solid, clotted blood separating dura mater and skull), subdural (clotted blood between inner surface and dura), intracerebral (blood thinners first)
intracranial pressure
edema (with blood) and hydrocephalus (CSF–VP shunt); ventricles may be enlarged when losing brain tissue; craniectomy may be done to relieve pressure (cut skull); Richmond bolts can monitor ICR pressure
other intracranial concerns
infections (meningitis, encephalitis, abscess); epilepsy (anti-seizure meds can also be cognitively dulling); vasospasm (constriction in arteries)
coma
after CNS damage or depression; state without eye opening, obeying commands, uttering words
Glasgow Coma Scale
subjective assessment of consciousness related to E (eye opening) + M (motor response) + V (verbal response); severe = 8 or below; mod = 9-12; mild =13-15; also used for predictions of outcome; eyes out of 4, motor out of 6, verbal out of 5
likelihood for a second or more TBI?
3-4 x more likely
coma scales
Disability Rating Scale; Glasgow Outcome Scale; Sickness Impact Profile; Katz Adjustment Scale; Galveston Orientation and Amnesia Test
decorticate vs. decerebrate
flexion (body inward) vs. extension (body outward)
neuro eval early stages
consciousness, motor response (decorticate vs. decerebrate), muscle tone (spastic/flaccid), orbicularis reflex (tap on head and blink), pupil size, resting eye position, other ocular
neuro eval later stages
smell and taste, visual acuity and fields, facial symmetry and mvm’t, motor function (body), hearing, phonation and swallow, gait and balance, cognition
psychosocial considerations
premorbid factors (personality, hx, substance), impaired cog. fx. (memory and attention), emotional status (depression), environmental responses
Reitan and Wolfson Model of Cog Fx
sensory input–attention, concentration, memory–lang. and visuospatial skills–concept formation, reasoning, logic