mmusculoskeletal Flashcards

1
Q

osteoarthritis

A

breakdown of articular cartilage leading to damage of bone; osteophytes form in joint space causing narrowing and decreased movement causing progressive degen.

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2
Q

risk factors of osteoarthritis

A

old age, female, obesity, labor-intensive occupations, sports activities

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3
Q

manifestations of osteoarthritis

A

pain when moving under stress (relieved by rest), hard & boney, stiff joints, morning stiffness (~30 min), usually impacts weight bearing joints (hips, knees, cervical spine, lumbar spine)

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4
Q

osteoarthritis bony growths

A

heberden- distal and bouchard- proximal

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5
Q

osteoarthritis assessment

A

crepitus over joint, mild effusion from inflammation, non-systemic, xray shows decreased joint space and osteophyte formation, subchondral bones may appear thick

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6
Q

osteoarthritis does not appear

A

bilaterally, it can but it does not have to

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7
Q

osteoarthritis management

A

decrease pain/stiffness, improve/maintain mobility, exercise t preserve joint, weight loss, OT/PT, orthotics and walking devices, NSAIDs/steroids, severe cases may require arthroplasty

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8
Q

osteoporosis

A

bone resorption (osteoclast) > bone formation (osteoblast) causing thinning of the bone

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9
Q

osteoporosis can lead to…

A

compression FX in T and L spine, FX in hips and wrists

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10
Q

osteoporosis risk factors

A

small frame, female gender, ethnicity, aromatase inhibitor use, nutritional factors, autoimmune diseases, steroid use, immobility, diabetes

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11
Q

onset of osteoporosis age

A

men around 60-70 and women around 50-60

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12
Q

manifestations of osteoporosis

A

low bone mineral density on DEXA scan, rounding of upper back (dowagers hump), osteoporotic FX, otherwise asymptomatic

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13
Q

osteoporosis assessment

A

Xray show radiolucency if significant demineralization (undetectable otherwise), dual energy xray (DEXA) provides bone mineral density of spine and hips

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14
Q

prevention of osteoporosis

A

balanced diet high in calcium and vit. D, regular weight bearing exercises (20-30 min/day)

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15
Q

weight training stimulates…

A

bone mineral density

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16
Q

management of osteoporosis

A

pharmacologic therapy to improve bone density (bisphosphenates or alendronate), hip FX managed with joint replacement, compression FX managed conservatively

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17
Q

osteoporosis nursing interventions

A

teach pt how to prevent worsening, manage pain, improve bowel elim. to avoid FX, injury prevention

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18
Q

osteomalacia

A

inadequate mineralization of bone caused by Vit. D deficiency causing soft/weakened bones

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19
Q

causes of osteomalacia

A

failure to absorb calcium, excessive calcium loss, GI disorders, liver disease, kidney disease, renal insufficiency, hyperparathyroidism, malnutrition

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20
Q

osteomalacia assessment

A

xray shows general demineralization and can show compression FX; labs show low Ca, low Phos, elevated alkaline phosphatase; bone biopsy show increased osteoid

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21
Q

osteoid

A

demineralized cartilaginous bone matrix aka pre-bone

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22
Q

why low calcium and low phosphorus in osteomalacia?

A

d/t bone unable to mineralize

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23
Q

why elevated alkaline phosphatase in osteomalacia?

A

indicative of increased bone turnover

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24
Q

management of osteomalacia

A

treat underlying cause if possible, Vit. D and Ca supps, sun exposure, if kidney disease is problem then activated form of vit D prescribed, if dietary is cause then recommend diet change, deformities may require braces

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25
Q

pagets disease

A

idiopathic; hyperactive osteoclast cause increased compensation of osteoblast creating larger but weaker bones; causing of pathologic fractures, bowing of legs, commonly affects skull, femur, tibia, pelvis, vertebrae

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26
Q

paget manifestation

A

usually asymptomatic, bowing of femur and tibia, spine is bent forward, bone may be warm and tender, pain is moderate aching and increases with weight bearing

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27
Q

paget disease assessment

A

elevated alkaline phosphatase, normal Ca level, demineralized and overgrowth mosaic pattern on Xray, diagnose with xray or bone biopsy

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28
Q

management of paget

A

NSAIDs, Ca with Vit. D, bisphosphonates to slow excessive bone remodeling

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29
Q

osteomyelitis

A

infection of bone resulting in inflammation, necrosis, and new bone formation; >50% d/t S. aureus, can extend to soft tissues and joints adjacent; if not properly treated then bone abscess can form

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30
Q

3 types of osteomyelitis

A

hematogenous: blood borne, contiguous-focus: surgery directly induced infection to bone, vascular insufficiency: diabetes oof PVD

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31
Q

manifestations of osteomyelitis- hematogenous

A

sudden onset, septic, systemic, fever and tachycardi

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32
Q

manifestations of osteomyelitis- contiguous-focus

A

no septic like, surface overlying bone is swollen, warm, painful

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33
Q

manifestations of chronic otesomyelitis

A

nonhealing ulcer overlying infected bone

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34
Q

manifestations of diabetic osteomyelitis

A

nonhealing FX, impaired immune response, foot ulcer >2cm is suspicious

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35
Q

osteomyelitis iassessment

A

acute: Xray, bone scan, MRI, leukocytosis, elevates ESR (inflammation marker), ~50% of cultured wounds are positive
chronic: xray, bone scan, ESR and WBC are normal, anemia is possible

36
Q

management of osteomyelitis

A

delay elective orthopedic surgery if infection present, sterile technique for surgery, prophylactic Antibx, prompt drain removal, aseptic post-op care, debridement, antibiotic (long term)

37
Q

osteomyelitis nursing interventions

A

pain management, improve mobility, control infection, promote home based/transitional care

38
Q

septic arthritis

A

infection of joint; mortality for single infected joint is 11%

39
Q

septic arthritis risk factors (who is more at risk of developing this)

A

older age, diabetes, RA, skin infection, alcoholism, Hx of joint surgery, IV drug use

40
Q

manifestations of septic arthritis

A

warm/painful/swollen joint, decreased ROM, chills/fever/leukocytosis, half of all cases are the knee

41
Q

septic arthritis assessment

A

infectious work up, aspiration/examine/culture (will be purulent pus filled) synovial fluid, CT/MRI, bone scan

42
Q

septic arthritis management

A

prompt treatment is primary, broad spect. antibx, aspirate synovial fluid periodically/therapeutic aspiration, splinting, pain relief, progressive ROM, potential for joint fibrosis, watch for recurring

43
Q

nursing interventions for septic arthritis

A

pain relief, improve physical mobility, control infection, promote home/community/transitional care

44
Q

arthroplasty

A

replacing joint with artificial joint (commonly knee and hip); used for extensive damage, pain, or limited function; result of RA, osteoarthritis, osteonecrosis, or congenital malformation

45
Q

nursing interventions for arthroplasty

A

positioning for comfort, care of incision, pain management, early ambul. educate on home care, monitor for PE/DVT/shock/infect./dislocation/Pneum

46
Q

types of soft tissue injuries

A

contusion: soft tissue bruise, strain: pulled muscle (overstretched), sprain: ligaments and supporting muscle fiber around joint (d/t twisting), dislocation: articular surfaces of the joint, subluxation: partial or incomplete dislocation

47
Q

managing soft tissue injury

A

Rest, ice, compression, elevation to reduce edema, then immobilization

48
Q

types of FX

A

closed or simple, open or compound/complex, intra-articular

49
Q

avulsion FX

A

Fx where fragment of bone has been pulled away by tendon and its attachement

50
Q

comminuted FX

A

Fx in which bone has splintered into several fragments

51
Q

compression FX

A

Fx in which bone has been compressed (common in vertebral Fx)

52
Q

depressed FX

A

Fx in which fragments are driven inward (commonly seen in skull)

53
Q

epiphyseal Fx

A

Fx through epiphysis

54
Q

greenstick FX

A

Fx in which one side of bone is broken and other side is bent (common in children)

55
Q

impacted FX

A

Fx in which a bone fragment is driven into another bone fragment

56
Q

oblique Fx

A

a Fx occurring at an angle across the bone (less stable than transverse Fx)

57
Q

open Fx

A

Fx in which damage also involves skin or mucous membrane (aka compound Fx); increased risk of infection

58
Q

Pathologic Fx

A

Fx that occurs through an area of diseased bone (osteoporosis, bone cyst, etc.); can occur with or without trauma

59
Q

simple Fx

A

Fx that remains contained with no disruption of the skin integrity

60
Q

spiral Fx

A

Fx that twists around the shaft of the bone

61
Q

stress Fx

A

Fx resulting from repeated loading of bone and muscle

62
Q

Transverse Fx

A

Fx that is straight across the bone shaft

63
Q

manifestation of Fx

A

loss of function, shortening, edema, deformity, crepitus, ecchymosis

64
Q

FX assessment

A

Health HX, comorbidities, Pain, VS, respiratory status, LOC, signs/symptoms of shock, neurovascular assessment of extremity, bowel/bladder elim, BS, I&O (hip Fx), skin condition, anxiety/coping

65
Q

managment of Fx

A

immediate immobilizatin, covering open wounds, Fx reduction, fracture reduction then immobilize

66
Q

3 types of reduction

A

closed reduction: manual traction; open reduction: surgical; delayed reduction: waiting for pt to stabilize prior to intervention

67
Q

2 types of immobilizations

A

external fixation: casts, bandages; internal fixation: plates and screws

68
Q

FX nursing interventions

A

elevate, monitor for neurovascular compromise (5 Ps), monitor for normal elimination (hip Fx), isometric muscle exercises, encourage ADLs, pain management, pt education, wound care

69
Q

Risk factors for FX healing

A

> 40, corticosteroids, NSAIDs, avascular necrosis (AVN), bone loss, tobacco, comorbidities, local trauma, inadequate immobilization, malalignment, premature weight bearing, infection, local malignancy

70
Q

acute FX complications

A

shock, fat embolism, compartment syndrome, DVT, PE, DIC, infection, loss of bladder control (hip FX), hemorrhage

71
Q

chronic FX complications

A

delayed union, malunion, nonunion, AVN of bone, complex regional pain syndrome (CRPS), heterotrophic ossification (benign bone growth in atypical location)

72
Q

Casts/splints/braces assessment

A

assess injury/treat lacerations/wounds; assess neurovascular status: 5 Ps

73
Q

5 Ps of neurovascular assessment

A

Pain, pulse, pallor, parasthesia, paralysis

74
Q

casts/splints/braces complications

A

pressure injuries, disuse syndrome (muscle weakness/wasting or joint stiffness), acute compartment syndrome

75
Q

compartment syndrome

A

increased pressure within compartment that impairs blood flow ad compromises tissue viability

76
Q

compartment syndrom assessment

A

Pain- intense disproportionate to injury, pallor, puselessness, parasthesia- numbness/tingling, paralysis, compartment swelling- taut shiny skin with potential blistering

77
Q

management of severe compartment syndrome

A

fasciotomy

78
Q

external fixator devices assessment and management

A

used to manage open FX with tissue damage; monitor for infect., elevate, educate pt, never adjust device

79
Q

traction

A

immobilizes by applying pulling fors to body; can be done to skeletal or skin (ex: halo vest for cervical spine)

80
Q

nursing intervention for skin traction

A

maintain alignment, report pain, assess pressure points, DVT prophylaxis, foot exercise q1Hour, position changes

81
Q

amputation

A

can be congenital or traumatic or caused by condition like PVD, infection, malignancy; done to improve pain, disease process, or improve function

82
Q

amputation assessment

A

neurovascular assessment and functional status, signs/symptoms of infect, nutritional status, health problems, psychological status/grief/coping

83
Q

amputation nursing interventions

A

pain relief, wound care, resolving grief/body image issues, promote independence, assist pt to achieve physical mobility

84
Q

acute amputation compications

A

post-op hemorrhage, infection, poor wound healing/skin breakdown, dehiscense

85
Q

delayed amputation complications

A

phantom limb pain, joint contracture (prolong immobilization reduces ROM)