Module 3 Flashcards
(39 cards)
Blood flow to the heart (right side) anatomy
- superior and inferior vena cava streams blood into the right atrium–> through the tricuspid valve—> letting blood into the right ventricles–> the blood shoots up into the pulmonary valve and then to the pulmonary artery–> which sends deoxygenated blood to the LUNGS. “Tri it before you bi it”
Blood flow to the heart (left side) anatomy
- enters back through the pulmonary veins oxygenated–> left atrium—> shot through the bicuspid (mitral or atrioventricular valve) valve—> then into left ventricle–> goes into the aortic valve—> and then shoots up into aorta and into the BODY “A comes before V so Atriam is on top and ventricles are on the bottom”
A&P for the vascular system
Cardiac Conduction System
Perfusion
the passage of blood, blood substitutes, or other fluids through a vessel into the organs and tissues
Cardiac output
amount of blood being pumped by each ventricle in L/min
Stroke volume
amount of blood ejected with each heartbeat (the average is 70mL)
Ejection fraction
the fraction of blood present in the ventricles that is ejected with each heartbeat (normal EF of Left ventricle is 50-70%)
Stroke volume factors
stroke volume x heart rate
1. Preload- the pressure at the end of diastole and the resultant stretching of the muscle fibers (how much blood comes through the atria)
2. Afterload- total resistance to ejection of blood from the ventricle
3. Contractility- the force that causes the myocardium to contract under any given condition(
Heart auscultation
rewatch lecture for slide 14
Diagnostic Evaluation
rewatch lecture for slide 15
Diagnostic testing for the heart
ECG, stress test, echocardiography, doppler, central venous, pulmonary artery, and intra-arterial blood pressure monitoring
Hypertension
BP of <120/80 is considered normal
BP of 120-139/80-89 is considered prehypertension
BP of 140/90 or higher is considered hypertension
(Peripheral vascular resistance related to the diameter of the blood vessel and the viscosity of the blood) (Blood pressure is the product of the cardiac output multiplied by cardiac resistance)
Risk factors for HTN
sleep apnea, renal changes, oral contraceptive use, possibly insulin resistant, diet high in Na and K
Clinical Manifestations of HTN
CAD, left ventricular hypertrophy, changes in the kidneys, retinal changes, cerebrovascular involvement
Dietary modifications
slide 24
Coronary Atherosclerosis
plaque build up in the vessels, a thrombus are possible to form and therefore cut off the blood flow and cause an MI
Modifiable risk factors for atherosclerosis
diabetes HTN, smoking, high cholesterol,
Manifestations of atherosclerosis
- ischemia- inadequate blood flow that deprives the cardiac muscle of O2
- angina pectoris- chest pain brought by myocardial ischemia
- myocardial ischemia- acute onset of chest pain, SOB, diaphoresis, N/V, extreme fatigue
Management of CAD and angina
Nitrates (nitroglycerin)
HMG-CoA reductase inhibitors (statins)
Antiplatelet medications & anticoagulants (aspirin and heparin or warfarin)
Bile acid-sequestrants (cholestyramine)
Oxygen administration
Ca+ channel blockers (diltiazem)
Beta-blockers (metoprolol)
CABG
rewatch lecture for slide 39
Diagnosis of MI
based on ECG changes and analysis of cardiac biomarkers
1. unstable angina
2. ST-segment elevation MI
3. Non-ST-segment elevation MI
Medications for MI
nitroglycerin, morphine, beta-blockers, ACE inhibitors, statins, thrombolytics, analgesics, therapy and rehabilitation, clopidogril-reduces ability of platelets to stick together, therefore preventing clots from forming
Heart failure
the inability of the heart to pump sufficient blood to meet the needs of the tissues for O2 and nutrients (systole- the contraction of the heart, diastole- filling of the heart)