Module - 5 - Neurological Emergencies Flashcards
- You are preparing to transport a twenty-year-old man weighing 200 pounds with a history of a self-inflicted gunshot wound to the head. He is intubated with A/C ventilator settings of FIO2 0.5, Vt 600, I/E 1:2, flow 5 L, RR 10, PIP 30. Vital signs are BP 100/60, HR 66, and SaO2 94%. ICP reading of 28. His cerebral perfusion pressure is approximately
A. 100 mmHg
B. 70-90 mmHg
C. 60 mmHg
D.
- D: The intracranial contents have three components: cerebrospinal fluid (CSF), blood volume, and the brain. As mean systemic arterial pressure increases, cerebral arterial blood vessels constrict, preventing the increase in blood volume and flow that would normally occur. If the mean systemic arterial blood pressure decreases, the cerebral arteries dilate, increasing cerebral blood flow. A mean systolic arterial pressure of approximately 60-140 mmHg, cerebral blood flow may be maintained in a constant state.
- What is the initial clinical presentation that may indicate that ICP may be increasing?
A. Hypotension
B. Deteriorating level of consciousness
C. Tachypnea
D. Tachycardia
- B: All neurologic emergencies can lead to coma. During patient assessment, it is useful to use a systematic approach in evaluating the comatose patient and establishing a baseline differential diagnosis. The Glasgow Coma Scale (GCS) is widely used to measure the severity of coma in patients and is therefore and indicator of prognosis.
- You are transporting an eighteen-year-old female patient with a history of being ejected from a motor vehicle accident. She is currently awake and oriented to person, place, and time; however, she is slow to respond. Vital signs are a BP of 70/42, HR 68, RR 26, SaO2 95%, temp. 98.8°F. Hemodynamic readings are CVP 3, CI 2.0, and SVR 600. ICP reading at 6 with a urine output of 100 mL over the last two hours. Your patient is exhibiting signs and symptoms of
A. Herniation
B. Hypovolemic shock
C. Spinal cord injury
D. Diabetes insipidus
- C: The patient is presenting with signs and symptoms of neurogenic shock: tachypnea, normal heart rate, and hypotension. Hemodynamic parameters to indicate the presence of neurogenic shock would include a decreased SVR
- You are transporting a forty-year-old male diagnosed with a subarachnoid hemorrhage. Which of the following assessment findings can be associated with his diagnosis?
A. Presence of doll’s eyes reflex
B. Positive Battle’s sign
C. Positive Brudzinski’s sign
D. Absence of ipsilateral pupillary dilation
- C: Positive Brudzinski’s sign can indicate the presence of a subarachnoid hemorrhage as well as meningitis. Severe neck stiffness causes the patient’s hips and knees to flex when the neck is flexed.
- You arrive on the scene to manage a fall victim. She presents with a BP 70/palp, HR 62, RR 24, Sats 96%. EMS reports brief LOC but now has a GCS of 14. You note a deformity of the right femur, and she is complaining of neck pain. The clinical presentation is most likely a diagnosis of
A. Neurogenic shock
B. Hypovolemic shock
C. Epidural bleed
D. Subdural bleed
- A: Spinal cord injury can lead to neurogenic shock. The patient is presenting with tachypnea, hypotension, and a normal heart rate but can also be present with bradydysrhythmias because of loss of sympathetic tone secondary to the spinal cord injury.
- Pupillary dilation in response to the oculomotor nerve insult that occurs in uncal herniation is a result of
A. Loss of parasympathetic stimulation
B. Loss of sympathetic stimulation
C. Parasympathetic overstimulation
D. Sympathetic overstimulation
- A: The innermost part of the temporal lobe, the uncus, can be compressed so that it goes by the tentorium and places pressure on the brain. The uncus can compress the third cranial nerve, which can affect the parasympathetic input to the eye on the side of the affected nerve, causing the pupil on the affected side to dilate and fail to constrict in response to light as it should.
- Which formula can be used when calculating a cerebral perfusion pressure (CPP)?
A. [(DBP × 2) + SBP] divided by 3
B. MAP − ICP
C. ICP − DBP
D. [(DBP + 2) × SBP] divided by 3
- B: MAP − ICP = CPP.
- An early sign of tentorial herniation would be
A. Doll’s eyes reflex
B. Ataxic breathing
C. Paralysis below the diaphragm
D. Ipsilateral pupillary dilation
- D: Ipsilateral pupil dilation on the affected side.
- You have been requested to transport a thirty-two-year-old male involved in a two-car motor vehicle collision in which the right side of his head struck the “A-post.” Right middle meningeal artery damage has been noted by CT with right-sided “mass effect” resulting. You would expect which of the following?
A. Epidural hematoma
B. Ventricular collapse
C. Cranial midline shift to the left
D. All of the above
- D: All of the above. The middle meningeal artery runs in a groove on the inside of the cranium beneath the pterion, which is vulnerable to injury at this point, where the skull is thin. A blow or fracture of the temporal bone is often the cause of a rupture of the middle meningeal artery, which may cause an epidural hematoma (occurs between the skull and the dura). There is often significant “mass effect” with compression of the ipslateral lateral ventricle and dilatation of the opposite lateral ventricle due to obstruction of the foramen of Monro. Emergency treatment requires decompression of the hematoma, usually by craniotomy.
- The patient presents with a skull fracture that appears to have a central focal point with multiple fractures outward on radiography. This skull fracture would be described as
A. Linear
B. Linear stellate
C. Diastatic
D. Depressed
- B: Linear stellate is a skull fracture with multiple linear fractures radiating from the site of impact. A growing skull fracture (GSF) also known as a craniocerebral erosion or leptomeningeal cyst due to the usual development of a cystic mass filled with cerebral spinal fluid is a rare complication of head injury usually associated with linear skull fractures of the parietal bone in children below three years of age. There are four major types of skull fractures: linear, compressed, distatic, and basilar.
- A head-injured patient would most likely experience an increased ICP as a result of which action?
A. Hip flexion
B. Gagging on the ETT
C. Adduction of the arms
D. Rotation of the head
E. All of the above
- E: All of the above are considered movements/stimulators that can increase ICP. The intubated patient who is restless or who resists ventilatory support is increasing their ICP, which can be extremely critical. Seizures that develop during transport should be promptly treated because they produce hypoxia and cause increased ICP. Intravenous administration of benzodiazepines is indicated for initial seizure management. Hypotension has been found to contribute to the mortality and morbidity of head-injured patients. The patient’s mean arterial pressure (MAP) should be maintained at more than 90 mmHg. Fluids and blood products should be administered to maintain blood pressure.
- You are transporting an awake multisystem trauma patient from a small rural facility with the following vital signs: BP 200/66, HR 56, RR 20-36, SaO2 97%, and temp. 99.9°F. Further assessment reveals a large laceration to the occipital area of the head, with bleeding controlled, and is moving all extremities. Pupils are reactive to light and equal at 4 mm with extraocular movements intact. The patient’s clinical presentation is suggestive of which of the following?
A. Demonstrating signs/symptoms of cushing’s triad
B. Already herniated and will likely deteriorate further
C. Demonstrating signs/symptoms of Brown-Séquard syndrome
D. Demonstrating signs/symptoms of hypovolemic shock
- A: The clinical presentation of Cushing’s triad is the triad of widening pulse pressure (rising systolic, declining diastolic), change in respiratory pattern (irregular respirations), and bradycardia. It is a sign of increased ICP, and it occurs as a result of the Cushing reflex. The normal average range for ICP is 0-10 mmHg.
- You are transporting a thirty-year-old female who was involved in a single vehicle rollover two hours prior to your arrival. She has a swan catheter in place with the following values: CVP 2, CI 2.0, PA S/D 12/6, wedge 7, SVR 400. Vital signs: BP 80/48, HR 46, RR 24, SaO2 90%. The patient’s clinical presentation is suggestive of which diagnosis?
A. Hypovolemic shock
B. Septic shock
C. Left ventricular failure
D. Neurogenic shock
- D: The patient is presenting with neurogenic shock. The SVR
- The expected average normal cerebral perfusion pressure range (CPP) is
A. 80-100 mmHg
B. 50-60 mmHg
C. 70-90 mmHg
D. >100 mmHg
- C: Normal cerebral perfusion pressure range is 70-90 mmHg.
- The average normal ICP range is
A. 0-10 mmHg
B. 10-20 mmHg
C. 20-30 mmHg
D. >30 mmHg
- A: Normal ICP range is 0-10 mmHg, but range can go as high as 15 mmHg.
- The formula to calculate a mean arterial pressure (MAP) is
A. 2/3 DBP × SBP
B. [(DBP × 2) + SBP] divided by 3
C. [(SBP × 2) + DBP] minus 3
D. [(DBP + 2) × SBP] divided by 3
- B: MAP = [(DBP × 2) + SBP] divided by 3.
- The patient presents with the following hemodynamic parameters: CVP 1, CI 1.7, PA S/D 12/6, wedge 6, and SVR 300. Vital signs are 78/40, HR 60, RR 16, SaO2 98%. The most likely cause is
A. RVMI
B. Neurogenic shock
C. Septic shock
D. Hypovolemic shock
- B: SVR
- Classic picture of neurogenic shock presents with
A. Hypertension
B. Absence of tachycardia
C. Cool skin
D. Pallor
- B: Loss of sympathetic tone below the level of the injury results in loss of autoregulation, a decrease in vascular tone, and inability of the heart to increase its intrinisic rate. The classic picture of neurogenic shock presents with the absence of tachycardia.
- You are transporting a patient with a spinal cord injury above T6 level. His baseline vital signs prior to lift off: BP 160/80, HR 62, RR 20. During transport, the patient begins to complain of a throbbing headache with nasal stuffiness. Your assessment reveals that the patient is becoming increasingly agitated. His skin color is flushed and profusely diaphoretic. Repeat vital signs are a BP 206/100, HR 52, RR 26. Your initial management of the patient would be
A. Insert a foley catheter
B. Administer nitroglycerin to help reduce blood pressure
C. Hang a Nipride drip if diastolic is greater than 130 mmHg
D. Do nothing because increased HTN is expected with altitude and spinal cord injuries
- A: Autonomic dysreflexia (AD), also known as “autonomic hyperreflexia or hyperreflexia,” is a potentially life-threatening condition, which can be considered a medical emergency requiring immediate attention. AD occurs most often in spinal cord-injured individuals with spinal lesions above the T6 spinal cord level. Acute AD is a reaction of the autonomic (involuntary) nervous system to overstimulation. This condition is distinct and usually episodic, with the patient experiencing remarkably high blood pressure (often with systolic readings over 200 mmHg), intense headaches, profuse sweating, facial erythema (redness), flushing of the skin above the level of the lesion, goosebumps, nasal stuffiness, bradycardia, apprehension, anxiety, and a “feeling of doom.” An elevation of 40 mmHg over baseline systolic should be suspicious for dysreflexia. Catheterization of the bladder or relief of a blocked urinary catheter tube may resolve the problem. If the noxious precipitating trigger cannot be identified, drug treatment is needed to decrease elevated ICP until further studies can identify the cause.
- You have been requested to transport a forty-year-old male fall victim of approximately 25-30 feet, three hours prior to your arrival. Your assessment reveals a greater motor weakness in upper extremities than in lower extremities, with varying degrees of sensory loss. The clinical presentation may suggest which of the following spinal cord syndrome?
A. Brown-Séquard
B. Central cord
C. Anterior cord syndrome
D. Neurogenic shock
- B: Central cord syndrome is the most common spinal cord injury (SCI) syndrome. This syndrome is unlike a complete lesion and causes loss of all sensation and movement below the level of the injury. Remember “you can dance, but you can’t clap.”
- Hypothermia, low levels of 2,3-DPG, and hypocarbia can cause the oxyhemoglobin dissociation curve shift to go
A. Up
B. Down
C. Right
D. Left
- D: A left shift causes an increase in the affinity, making the oxygen easier for the hemoglobin to pick up but harder to release. Refer to the table for review of causes.
- In addition to glucose, which electrolyte must be maintained within normal limits when managing a head-injured patient?
A. Calcium
B. Magnesium
C. Potassium
D. Sodium
- D: Low serum sodium levels following traumatic brain injury (TBI) can lead to extracellular volume depletion and cerebral edema. These can all result in dangerous increases in ICP. Hypertonic saline can help avoid the negative effects of hyponatremia by increasing serum sodium levels in the acute phase of head trauma care (Johnson and Criddle, 2004; Suarez, 2004). Maintaining serum sodium levels of 145-155 mmol/L is likely to achieve this goal. Serum sodium levels should be maintained no higher than 155 mmol/L. Higher levels are dangerous. Patients with serum sodium levels higher than 160 mmol/L are at increased risk for treatment-related renal failure, pulmonary edema, and heart failure. If serum sodium levels remain above 160 mmol/L for more than 48 hours, the risk of these problems increases even more. Furthermore, if serum sodium levels climb beyond 160 mmol/L, patients are at risk for seizures. The target serum osmolarity is less than 320 mOsmol/L. At higher levels, patients are at increased risk for treatment-related renal failure (Qureshi and Suarez, 2000; Suarez, 2004).
- You are transporting a twenty-year-old male, with penetrating head and facial trauma. During transport, the patient complains of a severe headache, nausea, and vertigo. Your assessment reveals nuchal rigidity, aphasia, dysphasia, along with the patient having episodes of vomiting. What is your diagnosis?
A. Pneumothorax
B. Pneumocephalus
C. Neurogenic shock
D. Hypercapnia
- B: Pneumocephalus is the presence of air or gas within the cranial cavity. It is usually associated with disruption of the skull: after head and facial trauma, tumors of the skull base, after neurosurgery, or with scuba diving (rare). The CT scan of patients with a tension pneumocephalus typically show air that compresses the frontal lobes of the brain, which results in a tented appearance of the brain in the skull known as the Mount Fuji sign. The name is derived from the resemblance of the brain to Mount Fuji in Japan, a volcano known for its symmetrical cone. The presenting symptoms of pneumocephalus vary widely, but headache is almost always present. Experience with diagnostic pneumocephalus has shown that the headache is not induced by the intracranial air alone but that the dura mater must be stretched for pain to occur. Nausea, vomiting, vertigo, nuchal rigidity, aphasia, dysphasia, hemiplegia, and obtundation have all been associated with pneumocephalus, yet all are nonspecific symptoms. Treatment options for pneumocephalus vary. In some cases, the condition resolves on its own with some watchful waiting, application of oxygen, and surgery if not resolving in a timely fashion.
- Calculate the following patient’s cerebral perfusion pressure (CPP): BP 150/75, HR 140, RR 28, SpO2 100%, CVP 2, ICP 25.
A. 98
B. 125
C. 65
D. 75
- D: MAP = [(75 × 2) + 150] divided by 3 = 100. CPP = 100-25 = 75 mmHg























