MSK Flashcards
(248 cards)
What is knee OA including etiology (modifiable and non-modifiable), S +S and imaging findings
Degeneration of articular cartilage and subchondral bone leading to joint space narrowing, rubbing of bone on bone leading to osteophyte formation
Etiology
Non-modifiable
-Age
-Gender (F>M) - hormonal, weight distribution, body shape (q-angle)
-Genetics
-Congenital malformation
Modifiable
-Obesity
-High impact activities
-Inactivity
-Muscle weakness - rely on passive structures
-Trauma
-Decreased proprioception - not optimal loading
-Joint mechanics (may or may not be able to modify)
S + S
Insidious onset
Morning stiffness (<30 mins) - if >1 hour it is more likely inflammatory (RA)
Pain with activity - worse with weight bearing, squatting, stairs, static postures, rissing after sitting, excessive activity (e.g., walking), fall in barometric pressure
Joint line tenderness
Decreased ROM / strength / function
Bony enlargement
Crepitus
Muscle atrophy, swelling, warm knee, feelings instability (decrease joint space = ligaments lax or quad inhibition), genu varum or valgum
Imaging
Decreased joint space
Osteophyte formation
What is ulnar tunnel syndrome? Including etiology, S + S , special tests
Compression of ulnar nerve as it passes through Guyon’s canal - between pisiform and hook and hamate
Etiology
Trauma - FOOSH with or without hamate fracture
Chronic pressure
Space occupying lesion
Extended use of crutches
Higher risk of cyclist, baseball catches, karate players and use of jack hammers
S +S
Pain and paresthesia in ulnar nerve distribution
Motor weakness of muscles innervated by ulnar nerve
Decreased grip strength
Fatigue with repetitive or sustained activities
Severe - claw hand and atrophy of hypothenar eminence
Special tests
Froment’s sign - pinch paper and pull away - need to use adductor pollicus and if not then go in to thumb flexion instead
Guyon canal compression test
Tinel’s test over guyon’s canal
ULTT ulnar nerve
Nerve conduction velocity test
Interventions
Activity modification
Cock up splint - into extension
Ergonomic and padded equipment
Frequent changes of hand positions
Nerve mobilization
Medical - NSAIDs, corticosteroid injections, guyon’s canal release
What is an ACL? What is an ACL tear? Including etiology and S+S
Runs back, up and lateral (BUL) - from medial tibial condyle to lateral femoral condyle
Two bands
Anterior medial - taut in flexion - more tested with anterior drawer
Posterolateral - taut in extension - more tested with lachman’s
Resists anterior drawer, medial rotation of tibia and valgus/varus on tibia
Epidemiology - F>M - hormones and q-angle
Etiology
Excessive anterior translation of tibia
Contact
-A valgus force from lateral side of knee
-Terrible triad - ACL,MCL,medial meniscus
Non-contact
-Pivoting or cutting - ER of tibia, or internal rotation of tibia or femur with planter foot
-Rapid deceleration - because quads working eccentric - pull tibia forward
-Forceful hyperextension - cause of contraction of quads
S + S
Hear audible pop or snap, tearing sensation
Pain
Contrast, throbbing, aching
Increased with movement or weight bearing
Hemarthrosis
Joint effusion
Knee “giving out” or feelings of instability - because of swelling inhibiting quads or because of actual instability
Limited ROM
What is the slipped capital femoral epiphysis? Including etiology, S +S, interventions
Fracture through growth plate (physis) causing slippage of end of femur (metaphysis)
Shaft moves anterior (and external rotated), head moves posterior
Seen in adolescent M>F
Etiology
Obesity
Family history
Endocrine disorders
S + S
Pain in hip or anterior thigh
Pain with activity
ROM limitations - flexion, abduction, IR
Limp present
Interventions - surgery - same rehab as ORIF
Get screw through but don’t change the femoral head cause more risk for avascular necrosis
What is the S + S, special tests and interventions of CTS?
S +S
Paresthesia and pain in median nerve distribution
Worse with sustained or repetitive wrist movements
Nocturnal numbness and pain
Relieved by “flicking”
Weakness and clumsiness in hand - decrease grip strength and frequently dropping objects
Severe - atrophy and thenar eminence and lumbricals 1 and 2
Special tests
Tinel’s test
Phalen’s test - reverse prayer
Reverse Phalen’s - Prayer
Carpal compression test
Resisted APB - only one exclusively innervation by median nerve
ULTT median nerve
Nerve conduction velocity test
Electromyography (EMG)
Interventions
Activity modification
Splinting in neutral
Mobility techniques
Nerve mobilization
Tendon gliding exercises
Joint mobilization
Improve muscle performance (no symptoms)
Gentle multi-angle muscle setting
Progress to resistance and endurance
Fine-finger dexterity
Medial - NSAIDs, corticosteroids, carpal tunnel release surgery
What is herpes zoster?
AKA shingles
Viral infection of nerve causing skin rash along the typical dermatomal pattern (stripe like in Tx, just on one side)
May be with fever
Describe the fitting of forearm crutches
Handgrips
-2 inch lateral, 6 inch anterior
-Hand grips at wrist crease, ulnar styloid, greater trochanter (elbows 20-30 degrees)
Cuff
-Proximal 1/3rd forearm, 1-⅕ below olecranon or elbow cuff
-Squeeze together or pull apart for proper fit (partial contact but not binding
What is a PCL? What is a PCL tear? Including etiology and S + S?
Goes from tibial lateral plateau to the medial femoral condyle
Resists posterior translation, medial rotation of tibia and valgus/varus force on tibia
Stronger and thicker than ACL - less likely to tear
Etiology
Posterior translation of tibia on femur (typically knee flexion)
Dashboard injury
Fall on knees in hyperflexion
Sudden forceful hyperflexion or hyperextension
S + S
Pain
Contrast, throbbing, aching
Increased with movement, especially kneeling or stairs
Hemarthrosis
Joint effusion
Limited ROM in acute stage
Increased passive extension ROM
Genu recurvatum (knees hyperextended on observation)
Have functional instability - so less likely to come to clinic cause don’t notice it as much
What are the different weightbearing restrictions?
NWB - affected limb bearing no weight in any circumstance
-ONLY CRUTCHES AND STANDARD WALKER
TTWB - toe touch weight bearing - foot may touch for balance only (<20%)
PWB - prescribed based on percentage of weight that can be applied (25%, 50%) -
-Use two bathroom scale method (or limb load monitor, heel sensors)
WBAT - as much weight as comfortable
FWB - no restrictions, no gait aid necessary
*increase in weight bearing - progression, bone adapts to stress - Wolfe’s law
What are the components for neuro scan for physical exam of cervical spine? What is it testing?
-Myotome - group of muscles or muscle supplied by spinal nerve
-Dermatomes - area of skin supplied by spinal nerve
-Reflexes - involuntary and instant response to a stimuli
-Special tests
What is Sever’s disease? Including etiology, S + S, interventions
Traction apophysitis in calcaneal insertion of achilles tendon
Epidemiology - overuse injury in children, ages 7-10
Etiology
Repeated tension on growth plate of calcaneus
Growth spurt - bone growing faster than muscle
Over-pronation - increase stretch of tendon
Increase incidence in sports that involve running / jumping (especially hard surfaces)
S + S
Heel pain (posterior plantar side)
Worse with calf use - walking, running, jumping
May present with antalgic gait
Pain with pressure over medial and lateral calcaneus in area of growth plate
May present with decreased passive DF
ALREADY on stretch and inhibited by pain
Interventions
Manage pain
Decrease parameters of aggravating activity
Foot orthotics
Stretching
Heel lifts - decrease stress of tendon
Explain the common foot deformities
Club foot
Congenital deformity, etiology unknown - genetics or environment or intrauterine malpositioning
Most common is congenital talipes equinovarus (CTEV)
Congenital - from birth, talipes - foot and ankle, equinovarus - horse like and in varus
Ankle - PF, rearfoot - varus, midfoot - adduction and supination
foot in in PF and turned inwards
Can be rigid (can’t move, complicated surgery) and flexible (can move a lot - serial casting gradually)
During gait walking on edge laterally of foot and the heels are off the ground
Pes cavus
Longitudinal arches are accentuated with abnormally short muscles on the sole of the foot
Leads to rigid foot with poor ability to adapt to stress and absorb shock (similar to stress #/ shin splints, causes plantar fasciitis, heel stress #)
Pes planus
Medial longitudinal arch is reduced
Can cause no other problems
Can lift arch and see if it changes the pain
Hallux rigidus
Extension of great toe is limited due to OA of MTP
Hallux valgus
Great toe deviations medially (along with MT head)
Causes - tight pointed shoes, hereditary factors
Bunion = medial side callus, thickened bursa and exostosis (bone)
Can use toe wedge, change footwear, intrinsic foot exercises, mobilization or get fusion, osteotomy
Claw toe
Hyperextension of MTP and flexion of PIP and DIP
PIP and MTP get callus formation
Hammer toe
Extension of MTP and flexion of PIP (DIP may be flexed, neutral or extended)
Callus at PIP
Mallet toe
Flexion of DIP
Callus at DIP or tip of toe
all three hereditary, poor shoes, muscle imbalance - common in RA
Describe single point canes
Advantages - high mobility, unload limb, stairs, lower energy expenditure
WBAT or FWB
Disadvantages - minimal balance deficits, low stability
Explain disc displacement in TMD?
Disc displacement with reduction
-Click 1 : Reduction of disc
-Click 2 : Dislocation of Disc
*typically anterior disc displacement
Disc displacement without reduction
-Closed locked - anterior disc displacement, can’t open
-Open locked - posterior disc displacement, can’t close
What is the applied anatomy for the shoulder joint?
Sternoclavicular joint - attaches shoulder to axial skeleton, ligaments very strong, more likely to fracture clavicle
Acromioclavicular joint - attached scapula to axial skeleton
Glenohumeral joint
-ER>abduction>IR - closed packed
-30 degrees flexion, 60 degrees abduction, internal rotation - loose packed
Scapulothoracic joint - false joint since no capsule and no bony articulation
What is double crush syndrome?
Nerve compression in more than one place for same nerve
Proximal compression increases vulnerability at distal point
Name and explain the types of kyphosis deformities
Round back
-Entire spine is kyphosis - decreased pelvic inclination (20 degrees) with thoracolumbar and thoracic kyphosis
-Associated with FHP and rounded shoulders
Scheuermann’s disease
-Uneven growth = excessive wedge shape increasing kyphosis
-Typically T10-12
-Rare congenital and/or degenerative weakening of vertebral end plate
-Most common deformity in adults, 2nd decade, “growing pains”
Hump back
-Gibbus (localized, sharp, posterior angulation) in Tx caused by structural deformity (e.g., anterior wedging) due to fracture, tumour or bone disease
-May or may not have pelvic inclination
Flat back
-Decrease pelvic inclination and decrease curve in Tx - mobile though
Dowager’s hump
-Increased kyphosis, typically postmenopausal OP with older women creating anterior wedge fracture across several vertebrae (result from trunk flexion)
-Happens in upper and middle Tx, decreases height
What is an achilles rupture? Including Etiology, S + S, special tests and interventions
Full grade III tear (~2-6cm proximal to calcaneal insertion)
Epidemiology: M>F, athletes in 30s-40s
Etiology
Direct trauma (direct blow / cut)
Rapid and forceful concentric contraction
Eccentric overload
Corticosteroid use - weakens connective tissue - more likely to rupture
S + S
Audible snap / tear
Swelling
Warmth, bruising
Obvious limp - because no PF
Palpable gap
Gross decrease in strength in PF
Present with pain
Special tests
Thompson’s test - squeeze calf, ankle in PF
Interventions
PT (conservative)
-Cast immobilization in max PF (4 weeks) then gradually progress for 4 weeks reduced DF
-Crutches NWB
-Progressive ROM and strengthening exercise following
-Proprioception exercises
-higher risk of re-rupturing
Surgical management
-Rupture repair (suturing)
-Immbolization post-op same as conservative approach - strength and ROM may be difficult to regain
-higher risk of complications but less likely to re-rupture
What is plica syndrome? Including S + S, special tests and interventions
Irritation to the plica (commonly medial plica) that causes inflammation of synovial sack where the area of the plica becomes thicker - this may catch between patella and femur causing further irritation and inflammation
Plica - fold of synovial membrane - embryological remnant
S + S
Intermittent anteromedial knee pain
Pain with prolonged standing, squatting, sitting, stairs
Tenderness on plica - medial side
Audible clicking or snapping
Knee may give way, present with pseudo-locking (not real, just catching), catching
Special tests
Hughston’s plica test
Mediopatellar plica test
Patellar bowstring test
Interventions
Decrease inflammation
Decrease aggravating activities
Taping to offload plica
Address muscle imbalance and possible patellar tracking
Surgical - partial or full removal of plica
What are the borders and contents of the thoracic outlet?
Borders
-Anterior - pect minor, coracoid process, clavicle
-Posterior - scapula, UFT
-Medial - 1st rib, scalenes
-Lateral - axilla
Content
-Brachial plexus
-Subclavian artery
-Subclavian vein
What is the etiology of PFPS?
Extrinsic - sudden or drastic changes in training regime
Intrinsic - abnormal patellar tracking (normally 60-90 degrees most contact, at 20- contact at apex, 90- contact at base, odd facet - side of patella - likely irritated and painful)
Increased Q angle
Measured from ASIS to mid patella, and mid patella to tibial tubercle
Angle of quad muscles and patellar tendon - represents angle of force of the quads
If increase in q-angle = larger bias to lateral quads = patella tracks more lateral
Q- angle in females 15-18, males 12-15
Q-angle <12: patella alta (high sitting patella)
Q-angle >18: patella baja (low sitting patella), genu valgum, subluxing patella - since pull patella laterally out of trochlea, PFPS
Muscle and fascial tightness
ITB tightness - lateral tracking
Patella retinaculum tightness (especially laterally)
Ankle PF tight (tight calves) → decrease ankle DF, subtalar pronation (not proper push offs), increased tibial IR (tibial torsion) - changes q-angle since tibial tubercle has changes, changes pull of quads
Hip muscle weakness
Weakness in hip abductors and ERs may results in adduction of the femur and valgus at the knee and possibly IR of femur
Glute med weakness = TFL compensation and overuse = increase femoral internal rotation
Glute med - abduction, external rotation and extension
TFL - abduction, internal rotation and flexion
*changes q-angle, TFL pulls it in to internal rotation or makes ITB more taut - patella track in different way
VMO insufficiency
Disuse or inhibition (because of joint swelling and pain)
Weakness or poor timing of VMO = lateral drifting of patella
Lax medial retinaculum
Describe arthokinematics
Accessory movements that occur between joint surfaces
Types of movement
-Slide - one point of one meets new point on other - e.g., tires sliding to a stop
-Roll - new point on one meet new point on another - tires moving normally
-Spin - doesn’t move, same surfaces but spins
-Traction
-Distraction - pulling at a right angle
-Compression
*in periphery - name based on the distal segment, in spine - named based on the vertebrae above compared to the one below
Direction of movement
What is spinal stenosis? How does it present in Lx?
Narrowing of the central canal (central stenosis) or lateral canal (lateral stenosis) which may compress the nerve roots or spinal cord
Onset is insidious and usually >60
Due to osteophytes, spondylosis, ligament thickening
May result in neurogenic claudication - neural leg pain
Better with flexion, worse with extension
What is a Colles’ fracture? Including etiology, complications, S + S and interventions
Distal radial fracture where the fragment moves more dorsally
-SMITH’S fracture - the distal segments moves more anterior
Complications
-Compression neuropathy (commonly median nerve)
-CRPS
-Arthritis
From FOOSH - with wrist extended
-SMITH’s - with wrist flexed
-More common in osteoporotic women - because they have a weak radius therefore it is the first thing to give)
S +S
-“Dinner fork” deformity
-Dorsal wrist pain and TOP
-Swelling - which may cause the compression of the median nerve
-May have bruising or paraesthesia
-Difficulty lifting or grasping
Interventions
-Medical - immobilization (either stable in cast (thumb spica), or unstable/displaced in ORIF)
-PT - mobilization, strengthening
-In cast - move everything above and below - NO PRO/SUP - makes sense cause radius moves around ulna = pain and delayed healing