MSK Flashcards

(32 cards)

1
Q

OA you are thinking

A

Wear and tear problem

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2
Q

Progressive joint disorder characterized by slow destruction of the normal collagen architecture

A

OA

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3
Q

After what age do we think wear and tear and is it an older or younger issue

A

After the age of 35 we think of OA it’s an older problem

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4
Q

One or more joint involved and asymmetric

A

This is classic of OA you have wear and tear of the joint at multiple injuries at different points and due to overuse of one versus the other

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5
Q

Pain with OA how does that look

A

Typical gets WORSE at the end of the day. If you are on your feet or knee all day

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6
Q

In the morning patients with OA how does they present

A

Stiffness

The stiffness last less than 30 minutes a highlight of this disorder

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7
Q

Common areas we find OA

A

Wrist the knees and the hips

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8
Q

Classic findings on xray of OA

A

Narrowing of the joint and osteophytes like bone spurs

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9
Q

RA patient population

A

Older and younger females

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10
Q

Findings of RA are symmetrical or asymmetrical

A

Symmetrical

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11
Q

Pain___________ with activity for RA

A

Improves

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12
Q

How is the stiffness in the morning for a patient with RA

A

Lasts LONGER than 30 minutes me as they get moving the stiffness gets better

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13
Q
A
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14
Q

Xray findings of RA

A

Osteopenia

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15
Q
A
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16
Q

If you’re pushing on a wound and you feel crepitus on like a area that has tissue not bone what are you thinking?

A

Necrotizing fasciitis 

17
Q

Etiology of osteoarthritis

A

Wear and tear, over the age of 60 equals a 60% chance of developing osteoarthritis, after 55 women are affected more, are genetics, hematological, and endocrine conditions again, mechanism stressors, such as repetitive microtrauma, prior trauma, recent exposure to certain chemicals if a patient in a motor vehicle crash and there’s trauma to the knee any neurological disorders that again have repetitive


18
Q

Clinical manifestations of OA 

A

Pain in one or more joints, stiffness of affected joint after prolonged sitting, grading or crepitus sensation during range of motion, feeling of instability, locking or bucking of the knees 

19
Q

Physical findings of the OA but patient comes in with edema and red hot knee or elbow other differentials

A

Septic joint or foot flare. Admit but in case it is not OA

20
Q

Bony induration or enlargement of affect joints. These nodes** effusion with warm and or redness involved

A

Heberden and bouchards
Heberden is the DIP and Bouchard is the PIP

Angular deformed and limited ROM with palpable or audible creptius can lap have pain on palpation

21
Q

RA deformity classic presentation

A

Swan neck and boutiner

22
Q

RA patients typically develop

A

Ulnar deviation
The ulnar is going to deviate laterally

24
Q

The bouttanaire deformity tell me about the flexion and extension of the joints

A

Flexion PIP and extension DIP

25
Tell me about the flexion and extension of swan neck
PIP hyperextended and DIP flexion
26
Lab and diagnostics for osteoarthritis
Plain x-rays, anterior posterior and lateral knee films as well as bilateral to compare, synovial fluid analysis, CBC, BMP, bone scans, MRIs and CT’s 
27
Management of OA
The goals are to relieve symptoms, maintain or improve function, limit disability, as much as possible, avoid drug toxicity, a multidisciplinary approach is best, rest and joint protection may be warranted
28
Top way to reduce the wear and tear on a patients joints that are obese
Losing the weight
29
Management for RA
DDMARDS
30
Drugs and management for OA
Acute exacerbation ice therapy Moist heat for the day to day relief or to help with the morning stiffness Salisatye acid or aspirin good starting drug 650mg if they can’t take they can take acetaminophen. NSAIDs are going to give you the biggest relief but the biggest draw back block cox1casade reduce mucus production and affect kidney function GI bleeding worsening hypertension and AKI
31
Medication providing the biggest bang for your buck for OA
NSAIDS
32
Two fold problem with NSAIDS in the adult gero population